Search results for "Glucuronides"

showing 10 items of 13 documents

Metabolism of propafenone and verapamil by cryopreserved human, rat, mouse and dog hepatocytes: comparison with metabolism in vivo

2003

In the present study we examined the metabolism of [(14)C]propafenone (P) and [(14)C]verapamil (V) using cryopreserved human, dog (Beagle), rat (Sprague-Dawley) and mouse (NMRI) hepatocytes. The percentage ratios of the metabolites were identified after extraction by HPLC with UV and radioactivity detection. Phase-II metabolites were cleaved using beta-glucuronidase. Metabolism of the drugs by cryopreserved hepatocytes was compared with that in the respective species in vivo. All phase-I and -II metabolites known from in vivo experiments: 5-hydroxy-P (5-OH-P); 4'-hydroxy-P (4'-OH-P); N-despropyl-P (NdesP) and the respective glucuronides, were identified after incubation with cryopreserved h…

Time FactorsPropafenoneIn Vitro TechniquesPharmacologyCryopreservationRats Sprague-DawleyHydroxylationMicechemistry.chemical_compoundDogsGlucuronidesPropafenoneSpecies SpecificityIn vivomedicineAnimalsHumansIncubationAgedCryopreservationPharmacologyChemistryGeneral MedicineMetabolismMiddle AgedIn vitroRatsVerapamilBiochemistryHepatocytesVerapamilAnti-Arrhythmia Agentsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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An investigation of the stability of free and glucuronidated 11-nor-delta9-tetrahydrocannabinol-9-carboxylic acid in authentic urine samples.

2004

Preanalytical stability of a drug and its major metabolites is an important consideration in pharmacokinetic studies or whenever the analyte pattern is used to estimate drug habits. Firstly, the stability of free and glucuronidated 11-nor-delta9-tetrahydrocannabinol-9-carboxylic acid (THCCOOH, THCCOOglu) in authentic urine samples was investigated. Random urine samples of cannabis users (n = 38) were stored at -20, 4, and 20 degrees C up to 15 days and up to 5 days at 40 degrees C, and alterations of the analyte pattern during storage were followed by liquid chromatography-tandem mass spectrometry. Secondly, the influence of pH (range 5.0-8.0) on the stability of the analytes was studied us…

AnalyteSubstance-Related DisordersHealth Toxicology and MutagenesisCarboxylic acidMetaboliteUrineToxicologyHigh-performance liquid chromatographyMass SpectrometryAnalytical Chemistrychemistry.chemical_compoundGlucuronidesPharmacokineticsDrug StabilityEnvironmental ChemistryHumansDronabinolDiagnostic Errorschemistry.chemical_classificationChemical Health and SafetyChromatographyForensic MedicineHydrogen-Ion ConcentrationSubstance Abuse DetectionchemistryΔ9-tetrahydrocannabinolGlucuronideArtifactsJournal of analytical toxicology
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Resveratrol metabolites inhibit human metastatic colon cancer cells progression and synergize with chemotherapeutic drugs to induce cell death.

2012

Scope Resveratrol (RSV) has been proposed to prevent tumor growth; nevertheless, these preventive effects are controversial since RSV pharmacokinetics studies show a low bioavailability. Recent clinical trials show that patients with colorectal cancer and receiving oral RSV have high levels of RSV conjugates in the colorectum, mainly RSV-3-O-sulfate (R3S), RSV-3-O-glucuronide, and RSV-4′-O-glucuronide. However, their potential biological activity has not yet been established. This study thus investigated in human colorectal cancer cell lines whether RSV main metabolites retain anticarcinogenic properties as their parental molecule. Methods and results Proliferation, apoptosis assays and cel…

Programmed cell deathColorectal cancerCell SurvivalvirusesApoptosisBiologyResveratrolPharmacologychemistry.chemical_compoundGlucuronidesIn vivoCell Line TumorStilbenesmedicineHumansCell ProliferationCell Cyclevirus diseasesBiological activityDrug Synergismrespiratory systemCell cyclemedicine.diseaseAntineoplastic Agents PhytogenicchemistryCell cultureApoptosisResveratrolColonic NeoplasmsFood ScienceBiotechnologyMolecular nutritionfood research
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In silico methods for metabolomic and toxicity prediction of zearalenone, α-zearalenone and β-zearalenone.

2020

Zearalenone (ZEA), α-zearalenol (α-ZEL) and β-zearalenol (β-ZEL) (ZEA's metabolites) are co/present in cereals, fruits or their products. All three with other compounds, constitute a cocktail-mixture that consumers (and also animals) are exposed and never entirely evaluated, nor in vitro nor in vivo. Effect of ZEA has been correlated to endocrine disruptor alterations as well as its metabolites (α-ZEL and β-ZEL); however, toxic effects associated to metabolites generated once ingested are unknown and difficult to study. The present study defines the metabolomics profile of all three mycotoxins (ZEA, α-ZEL and β-ZEL) and explores the prediction of their toxic effects proposing an in silico w…

In silicoMetaboliteToxicologyArticleAmes test03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyMetabolomicsGlucuronidesCytochrome P-450 Enzyme SystemIn vivoAnimalsMetabolomicsComputer SimulationMycotoxinZearalenoneZebrafish030304 developmental biology0303 health sciencesChemistryIn silicofood and beverages04 agricultural and veterinary sciencesGeneral Medicine040401 food sciencePASS onlineEndocrine disruptorBiochemistryBlood-Brain BarrierMetaToxZearalenoneSwissADMEReactive Oxygen SpeciesPredictionFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Molecular docking-based design and development of a highly selective probe substrate for UDP-glucuronosyltransferase 1A10

2018

Intestinal and hepatic glucuronidation by the UDP-glucuronosyltransferases (UGTs) greatly affect the bioavailability of phenolic compounds. UGT1A10 catalyzes glucuronidation reactions in the intestine, but not in the liver. Here, our aim was to develop selective, fluorescent substrates to easily elucidate UGT1A10 function. To this end, homology models were constructed and used to design new substrates, and subsequently, six novel C3-substituted (4-fluorophenyl, 4-hydroxyphenyl, 4-methoxyphenyl, 4-(dimethylamino)phenyl, 4-methylphenyl, or triazole) 7-hydroxycoumarin derivatives were synthesized from inexpensive starting materials. All tested compounds could be glucuronidated to nonfluorescen…

0301 basic medicineMutantGlucuronidationPharmaceutical ScienceUGT1A10030226 pharmacology & pharmacySubstrate Specificity7-hydroxycoumarin derivativechemistry.chemical_compound0302 clinical medicineDrug DiscoveryCRYSTAL-STRUCTUREGlucuronosyltransferaseta116ta317AFFINITYchemistry.chemical_classificationChemistry3. Good healthMolecular ImagingMolecular Docking Simulation7-hydroxycoumarin317 Pharmacyin silicoMolecular MedicinefluorescenceUDP-glucuronosyltransferaseEXPRESSIONENZYMEStereochemistryIn silicoKineticsFLUORESCENT-PROBETriazoleta311103 medical and health sciencesGlucuronidesMicrosomesXENOBIOTICSHumansUmbelliferonesFluorescent DyesGLUCURONIDATIONta1182glucuronidationfluoresenssiSubstrate (chemistry)drug metabolism030104 developmental biologyEnzymeDRUG-METABOLISMDrug DesignMolecular ProbesMutationMutagenesis Site-DirectedORAL BIOAVAILABILITYDrug metabolism
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Metabolism of apigenin by rat liver phase I and phase II enzymes and by isolated perfused rat liver

2004

The metabolism of apigenin, a low estrogenic flavonoid phytochemical, was investigated in rat using liver models both in vitro (subcellular fractions) and ex vivo (isolated perfused liver). In vitro, phase I metabolism led to the formation of three monohydroxylated derivatives: luteolin which was the major metabolite (K(m) = 22.5 +/- 1.5 microM; V(max) = 5.605 +/- 0.090 nmol/min/mg protein, means +/- S.E.M.), scutellarein, and iso-scutellarein. These oxidative pathways were mediated by cytochrome P450 monooxygenases (P450s). The use of P450 inhibitors and inducers showed that CYP1A1, CYP2B, and CYP2E1 are involved. In vitro studies of phase II metabolism indicated that apigenin underwent co…

MaleFMN ReductaseMetabolite[SDV]Life Sciences [q-bio]Pharmaceutical ScienceIn Vitro TechniquesMethylation030226 pharmacology & pharmacyMass Spectrometry03 medical and health scienceschemistry.chemical_compoundGlucuronides0302 clinical medicineCytochrome P-450 Enzyme SystemAnimalsApigeninEnzyme InhibitorsRats WistarLuteolinBiotransformationChromatography High Pressure LiquidComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidsPharmacologySex Characteristics0303 health sciencesbiologySulfatesScutellareinCytochrome P450MonooxygenaseDiosmetinRats3. Good health[SDV] Life Sciences [q-bio]KineticsLiverBiochemistrychemistryApigeninbiology.proteinRATFemaleSpectrophotometry UltravioletLuteolinNADPDrug metabolismSubcellular Fractions
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Carboxyl nonsteroidal anti-inflammatory drugs are efficiently glucuronidated by microsomes of the human gastrointestinal tract.

2004

Limited studies have been carried out on the biotransformation of carboxyl nonsteroidal anti-inflammatory drugs (NSAIDs) in the liver. However, the role of the intestine in NSAID metabolism has not been investigated. In this report, the contribution of UDP-glucuronosyltransferases (UGTs) in the human gastrointestinal (GI) tract from five donors to the glucuronidation of the NSAIDs, RS-ketoprofen, S-naproxen, RS- and S-etodolac, was investigated. UGT activity and, for some donors, mRNA levels were evaluated. All NSAIDs were glucuronidated throughout the GI tract; however, glucuronidation was low in stomach and duodenum as compared to the remainder of the intestine. RT-PCR analysis demonstrat…

medicine.medical_specialtyBiophysicsGlucuronidationAdministration OralPharmacologydigestive systemBiochemistryGene Expression Regulation EnzymologicFirst pass effectGlucuronidesNaproxenInternal medicineMicrosomesmedicineHumansRNA MessengerGlucuronosyltransferaseMolecular BiologyChromatography High Pressure LiquidGastrointestinal tractChemistryReverse Transcriptase Polymerase Chain ReactionStomachHuman gastrointestinal tractAnti-Inflammatory Agents Non-SteroidalUGT2B7Gastrointestinal Tractmedicine.anatomical_structureEndocrinologyKetoprofenDuodenumMicrosomeEtodolacBiochimica et biophysica acta
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Partition coefficient, blood to plasma ratio, protein binding and short-term stability of 11-nor-Delta(9)-carboxy tetrahydrocannabinol glucuronide.

2002

11-Nor-Delta(9)-carboxy tetrahydrocannabinol glucuronide (THCCOOglu) is a major metabolite of tetrahydrocannabinol in blood. Despite its mass spectrometric identification already in 1980, further physicochemical data of THCCOOglu have not been established. Therefore, the octanol/buffer partition coefficient P and the blood to plasma ratio b/p for THCCOOglu concentrations of 100 and 500ng/ml were investigated. Protein binding of the glucuronide was established from spiked albumin solutions at a level of 250ng/ml as well as from authentic samples. The data were compared to those of 11-nor-Delta(9)-carboxy tetrahydrocannabinol (THCCOOH). In addition, the short-term stability of THCCOOglu in pl…

OctanolBlood Specimen CollectionChromatographyMetaboliteAlbuminPlasma protein bindingForensic MedicineMass spectrometryMass SpectrometryPathology and Forensic MedicinePartition coefficientchemistry.chemical_compoundGlucuronideschemistryDrug StabilityBlood plasmaHumansDronabinolGlucuronideLawProtein BindingForensic science international
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Urinary tetrahydroaldosterone as a screening method for primary aldosteronism: a comparative study

2003

Abstract Background The major aldosterone metabolite 3α,5β tetrahydroaldosterone reflects up to 45% of the aldosterone secretion. Its 24-h urinary excretion is likely to provide an accurate index of the daily aldosterone production and to be an indicator for primary aldosteronism (PA). Methods In a prospective study, the validity of tetrahydroaldosterone as a screening test for PA was evaluated in comparison to serum potassium, plasma aldosterone, plasma renin activity, plasma aldosterone/renin activity ratio (PARR), as well as 24-h urinary aldosterone-18-glucuronide and free aldosterone. A total of 111 normotensive individuals, 412 PA patients and 1453 essential hypertensive patients, were…

AdultMalemedicine.medical_specialtyAdolescentmedicine.drug_classEssential hypertensionSensitivity and SpecificityPlasma renin activitychemistry.chemical_compoundGlucuronidesPrimary aldosteronismInternal medicineHyperaldosteronismReninInternal MedicinemedicineHumansMass ScreeningProspective StudiesAldosteroneMass screeningScreening proceduresAldosteronebusiness.industryMiddle Agedmedicine.diseaseHyperaldosteronismEndocrinologychemistryMineralocorticoidPotassiumFemalebusinessAmerican Journal of Hypertension
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Impact of glucuronide interferences on therapeutic drug monitoring of posaconazole by tandem mass spectrometry.

2010

Abstract Background: Posaconazole is a novel antifungal drug for oral application intended especially for therapy of invasive mycoses. Due to variable gastrointestinal absorption, adverse side effects, and suspected drug-drug interactions, therapeutic drug monitoring (TDM) of posaconazole is recommended. Method: A fast ultra performance liquid chromatography-tandem mass spectrometry (UPLC-MS/MS) method for quantification of posaconazole with a run-time <3 min was developed and compared to a LC-MS/MS method and HPLC method with fluorescence detection. Results: During evaluation of UPLC-MS/MS, two earlier eluting peaks were observed in the MRM trace of posaconazole. This was only seen in p…

Observer VariationPosaconazoleElectrosprayChromatographyAntifungal Agentsmedicine.diagnostic_testChemistryBiochemistry (medical)Clinical BiochemistryAntifungal drugGeneral MedicineTriazolesMass spectrometryTandem mass spectrometryHigh-performance liquid chromatographyGlucuronidesTherapeutic drug monitoringTandem Mass SpectrometrymedicineHumansDrug MonitoringGlucuronidemedicine.drugClinical chemistry and laboratory medicine
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