Search results for "Goats"

showing 9 items of 69 documents

Impairment of the modulatory role of nitric oxide on the endothelin-1-elicited contraction of cerebral arteries: a pathogenetic factor in cerebral va…

1997

OBJECTIVE: Nitric oxide (NO) and endothelin-1 (ET-1) are two endothelium-derived factors probably involved in the pathogenesis of cerebral vasospasm after subarachnoid hemorrhage (SAH). Our aim was twofold, i.e., to ascertain whether endothelial and nonendothelial NO modulates the contractile response of cerebral arteries to ET-1 and to analyze whether this relationship might be impaired after experimental SAH. METHODS: Rings of middle cerebral artery from goats in the control group and from goats with SAH were set up for isometric tension recordings. SAH was induced 3 days before the experiments by infusion of 10 ml of autologous arterial blood through a catheter previously inserted into t…

medicine.hormonemedicine.medical_specialtySubarachnoid hemorrhageEndotheliumCerebral arteriesNitric OxideEndothelinsCerebral vasospasmInternal medicinemedicine.arterymedicineAnimalscardiovascular diseasesEndothelin-1business.industryVascular diseaseGoatsVasospasmCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesMicroscopy Electronmedicine.anatomical_structureIschemic Attack TransientVasoconstrictionAnesthesiaMiddle cerebral arterycardiovascular systemCardiologySurgeryNeurology (clinical)Endothelium VascularbusinessNeurosurgery
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Direct and neuromodulatory effects of histamine on isolated goat cerebral arteries.

1992

1. The effects of histamine on isolated goat middle cerebral artery were examined using two experimental approaches: recording of isometric tension and measurement of [3H]-noradrenaline efflux. 2. Cumulative addition of histamine (10(-7)-3 x 10(-2)M) and 2-pyridylethylamine (2-PEA, 10(-6)-3 x 10(-2)M) produced concentration-dependent contractile responses. Preincubation with diphenhydramine (10(-7), 10(-6)M) or cimetidine (10(-7), 10(-6)M) competitively inhibited the histamine-induced contractile response. 3. Endothelium denudation enhanced the contractile effects of histamine. 4. Transmural electrical stimulation elicited contractions which were enhanced by histamine (10(-7)M), 2-PEA (10(-…

medicine.medical_specialtyCerebral arteriesAdrenergicStimulationHistamine H1 receptorIn Vitro TechniquesMuscle Smooth Vascularchemistry.chemical_compoundNorepinephrineInternal medicineMedicineAnimalsReceptors Histamine H1CimetidinePharmacologybusiness.industryGeneral NeuroscienceGoatsCerebral ArteriesDimapritElectric StimulationEndocrinologychemistryMechanism of actionFemalemedicine.symptombusinessHistaminemedicine.drugHistamineMuscle ContractionJournal of autonomic pharmacology
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Effects of endothelin-1 on the cerebrovascular bed of the goat.

1991

Abstract The aim of the present study was to analyze the effects of endothelin-1 (ET-1) in the cerebrovascular bed of the goat, the importance of endothelial integrity and the contribution of extracellular Ca2+ to these responses. We measured isometric tension and 45Ca2+ uptake in isolated middle cerebral arteries, and cerebral blood flow (CBF) in unanesthetized animals. ET-1 elicited concentration-dependent contractions which were potentiated in arteries without endothelium. Ca2+-free medium and nicardipine inhibited, and Bay K 8644 potentiated the ET-1-induced contractions. ET-1 enhanced 45Ca2+ uptake in isolated arteries. Injections of ET-1 directly into the cerebral circulation decrease…

medicine.medical_specialtyEndotheliumCerebral arteriesNicardipineIn Vitro TechniquesMuscle Smooth VascularCerebral circulationNicardipineInternal medicinemedicine.arteryIsometric ContractionmedicineAnimalsPharmacologyChemistryCalcium RadioisotopesEndothelinsGoatsAnatomy3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterEndothelin 1Acetylcholinemedicine.anatomical_structureEndocrinologyCerebral blood flowCerebrovascular CirculationMiddle cerebral arteryPotassiumCalciumFemaleBlood vesselmedicine.drugEuropean journal of pharmacology
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Ca2+ entry blockers inhibit prostaglandin F2 alpha-induced cerebrovascular contractile responses in goats.

1991

We examined the effects of extracellular Ca2+ withdrawal and of Ca2+ entry blockers on goat cerebrovascular responses to prostaglandin F2 alpha (PGF2 alpha). We measured isometric tension in isolated middle cerebral arteries, and cerebral blood flow (CBF) in unanesthetized animals. PGF2 alpha produced concentration-dependent contractions of isolated arteries. The contractions were partially inhibited by incubation in Ca(2+)-free medium (by 63.1 +/- 1.8% without ethyleneglycol-bis-(beta-amino-ethylether)-N,N,N',N'-tetra-a cetate (EGTA), and by 82.4 +/- 3.7% with EGTA). The Ca2+ entry blockers inhibited PGF2 alpha-elicited contraction and relaxed PGF2 alpha-precontracted arteries (nicardipine…

medicine.medical_specialtyMuscle RelaxationNicardipineCerebral arteriesProstaglandinIn Vitro TechniquesDinoprostCerebral circulationchemistry.chemical_compoundInternal medicinemedicine.arteryIsometric ContractionmedicineAnimalsNimodipineEgtazic AcidPharmacologyGoatsCerebral ArteriesCalcium Channel BlockersEndocrinologychemistryVasoconstrictionCerebrovascular CirculationCirculatory systemMiddle cerebral arteryVerapamilCalciumFemalemedicine.drugEuropean journal of pharmacology
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Endothelial modulation of 5-hydroxytryptamine-induced contraction in goat cerebral arteries.

1993

Abstract 1. 1. In isolated goat middle cerebral artery segments, 5-hydroxytryptamine (5-HT, 10 −8 −3 × 10 −5 M) caused concentration-dependent contractions, with EC 50 = 2.1 (1.9−2.5) × 10 −7 M and E max = 60 ± 2% of 50 mM KCl-induced contraction. 2. 2. Mechanical removal of endothelium significantly increased the E max (91 ± 8%) and did not change the EC 50 value of 5-HT-elicited contractions. 3. 3. Incubation of unrubbed arteries with the irreversible inhibitor of EDRF, gossypol (10 −5 M), significantly increased the contractile response to 5-HT ( E max = 77 ± 4%). 4. 4. Incubation of unrubbed arteries with the competitive inhibitor of the NO synthesis, N G -nitro - l -arginine (L-NOARG) …

medicine.medical_specialtySerotoninContraction (grammar)ArginineEndotheliumCerebral arteriesIndomethacinProstacyclinIn Vitro TechniquesArginineNitric OxideNitroarginineMuscle Smooth Vascularchemistry.chemical_compoundInternal medicinemedicine.arteryIsometric ContractionmedicineAnimalsPharmacologyChemistryGoatsEndothelium-derived relaxing factorGossypolAnatomyCerebral ArteriesEpoprostenolEndocrinologymedicine.anatomical_structureMiddle cerebral arteryFemaleEndothelium Vascularmedicine.symptomVasoconstrictionmedicine.drugMuscle ContractionGeneral pharmacology
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Changes in the adrenergic mechanisms of cerebral arteries after subarachnoid hemorrhage in goats.

1994

We have examined the effects of experimental subarachnoid hemorrhage (SAH), induced by delivering autologous blood into the subarachnoid space, on the adrenergic mechanisms of the goat cerebrovascular bed. To achieve this, the response to noradrenaline was recorded both in vivo, by measuring cerebral blood flow in unanesthetized animals, and in vitro, by recording isometric tension in isolated cerebral arteries. In addition, we checked the function of adrenergic innervation by measuring the tritium efflux evoked by electrical stimulation in cerebral arteries preloaded with [3H]-noradrenaline, and we examined this innervation by using both fluorescent and electron transmission microscopy. Al…

medicine.medical_specialtySubarachnoid hemorrhageCerebral arteriesAdrenergicNorepinephrine (medication)NorepinephrineCerebral vasospasmInternal medicinemedicineAnimalscardiovascular diseasesbusiness.industryVascular diseaseGoatsBrainCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesMicroscopy ElectronEndocrinologymedicine.anatomical_structureCerebral blood flowIschemic Attack TransientNerve DegenerationSurgeryFemaleVascular ResistanceNeurology (clinical)Subarachnoid spacebusinessAdrenergic FibersBlood Flow Velocitymedicine.drugNeurosurgery
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Modulatory action of acetylcholine on cerebrovascular sympathetic neurotransmission

1991

1. Acetylcholine (10 micrograms/min) diminished the electrically-induced cerebral blood flow reductions. Atropine (1-2 mg) partially blocked this inhibitory effect. 2. Exogenously administered noradrenaline (1-10 micrograms) and tyramine (50-500 micrograms) reduced cerebral blood flow but this effect was unchanged by acetylcholine infusion. 3. Acetylcholine inhibited the nonadrenergic component of the electrically-induced contraction at a concentration greater than or equal to 10(-6) M and potentiated the adrenergic component at a concentration greater than or equal to 10(5) M. Atropine 10(-7) M) inhibited both of these effects. In addition, acetylcholine (10(-4) M) enhanced the electricall…

medicine.medical_specialtySympathetic Nervous SystemContraction (grammar)Cerebral arteriesTyramineAdrenergicTetrodotoxinIn Vitro TechniquesSynaptic TransmissionMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundIsometric ContractionInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPharmacologyChemistryGoatsMuscarinic acetylcholine receptor M3Cerebral ArteriesTyramineAcetylcholineElectric StimulationAtropineEndocrinologyCerebrovascular CirculationFemaleAcetylcholinemedicine.drugGeneral Pharmacology: The Vascular System
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Endothelin action on goat cerebral arteries.

1990

Abstract Cumulative application of endothelin-1 (human) markedly constricted goat isolated cerebral arteries in a concentration-dependent manner. Contractile responses were not affected by removal of endothelial cells. Removal of extracellular calcium or addition of the calcium channel blocker nicardipine (10−7 M) failed to abolish responses to endothelin. The results suggest that the endothelium-independent constriction of cerebral arteries produced by endothelin cannot be explained solely by voltage-dependent calcium channels. The contractile responses are likely to be mediated by stimulation of specific receptors for this peptide.

medicine.medical_specialtymedicine.drug_classCerebral arteriesNicardipinePharmaceutical Sciencechemistry.chemical_elementStimulationCalcium channel blockerBiologyCalciumIn Vitro TechniquesMuscle Smooth VascularNicardipineInternal medicinemedicineAnimalsPharmacologyVoltage-dependent calcium channelEndothelinsGoatsCerebral ArteriesEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemCalciumFemaleEndothelium VascularEndothelin receptormedicine.drugBlood vesselMuscle ContractionThe Journal of pharmacy and pharmacology
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Effect of seasonality on physico-chemical and microbiological properties of nicastrese milk and artisanal cheese

2021

ABSTRACT Nicastrese is an indigenous Italian goat breed reared in the Calabria region under semi-extensive practices. From January to June, the milk yield of 400 multiparous Nicastrese goats was evaluated. In addition, tank milk and ripened cheese samples were subjected to physico-chemical and microbiological analyses with the aim to assess the effect of the seasonality on quality parameters. The volatile organic compounds profile of the ripened Nicastrese cheese samples was evaluated. Results showed that the seasonality had a significant effect on milk fat, lactose, urea, citric acid contents and acidity. Microbiological analysis of tank milk samples revealed variability among the differen…

microbial countsFood HandlingNicastrese goat raw goat's milk and cheese microbial counts VOCsraw goat's milk and cheeseMicrobiology03 medical and health scienceschemistry.chemical_compoundFecesraw goat’s milk and cheesefluids and secretionsCheesemicrobial countGeneticsmedicineAnimalsNicastrese goatFood scienceNicastreseLactoseMolecular Biology030304 developmental biology0303 health sciencesVolatile Organic CompoundsbiologyBacteriaGoatsMicrobiota0402 animal and dairy sciencefood and beveragesVOCs04 agricultural and veterinary sciencesSeasonalitybiology.organism_classificationmedicine.disease040201 dairy & animal scienceBreedFecal coliformMilkchemistryFermentationUreaListeriaFemaleSeasonsCitric acid
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