Search results for "Granulomatosis"

showing 10 items of 34 documents

Cytotoxic effects of antibodies to proteinase 3 (C-ANCA) on human endothelial cells.

1994

SUMMARY Autoantibodies directed against cytoplasmic antigens of neutrophils (ANCA), especially those with specificity for proteinase 3 (PR-3) and myeloperoxidase, are valuable markers for differential diagnosis and monitoring of disease activity in Wegener's granulomatosis (WG) and other vasculitides. Till now, several concepts concerning a direct role of antibodies against PR-3 in the pathogenesis of WG have been discussed. Recently we were able to show that these antibodies recognize PR-3 translocated into the membrane of human endothelial cells. The aim of this study was to investigate putative cytotoxic effects of antibodies to PR-3 on human endothelial cells. Antibodies were obtained b…

Cytotoxicity ImmunologicC-ANCAEndotheliumMyeloblastinImmunologyAutoantigensChromatography AffinityAntibodies Antineutrophil CytoplasmicAntigenProteinase 3medicineImmunology and AllergyCytotoxic T cellHumansLupus Erythematosus SystemicCells CulturedAutoantibodiesMixed Connective Tissue DiseasebiologySerine EndopeptidasesAntibody-Dependent Cell CytotoxicityGranulomatosis with PolyangiitisEndothelial stem cellmedicine.anatomical_structureSjogren's SyndromeMyeloperoxidaseImmunologybiology.proteinEndothelium VascularAntibodyResearch ArticleClinical and experimental immunology
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Antibodies to proteinase 3 increase adhesion of neutrophils to human endothelial cells

1993

SUMMARY The detection of anti-neutrophil cytoplasmic antibodies (ANCA), especially those with specificity for proteinase 3, is important in the diagnosis and in monitoring disease activity of Wegener's granulomatosis and related vasculitides. An ubiquitous feature of all ANCA-associated acute vascular injury is lytic necrosis. Adhesion of neutrophils to endothelium is a fundamental early step of the inflammatory response. Recently we were able to show that ANCA recognize their target antigen (proteinase 3) translocated into the membrane of human endothelial cells. The aim of this study was to investigate the effect of ANCA on the adhesion of neutrophils to human endothelial cells. Incubatio…

EndotheliumNeutrophilsMyeloblastinImmunologyFluorescent Antibody TechniqueEnzyme-Linked Immunosorbent AssayAntibodies Antineutrophil CytoplasmicAutoimmune DiseasesFlow cytometryProteinase 3E-selectinCell AdhesionmedicineHumansImmunology and AllergyCell adhesionCells CulturedAutoantibodiesMixed Connective Tissue Diseasebiologymedicine.diagnostic_testSerine EndopeptidasesGranulomatosis with PolyangiitisAdhesionFlow CytometryEndothelial stem cellmedicine.anatomical_structureImmunoglobulin GImmunologybiology.proteinEndothelium VascularAntibodyE-SelectinCell Adhesion MoleculesResearch ArticleClinical and Experimental Immunology
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EULAR/PRINTO/PRES criteria for Henoch-Schonlein purpura, childhood polyarteritis nodosa, childhood Wegener granulomatosis and childhood Takayasu arte…

2010

EULAR/PRINTO/PRES Objectives To validate the previously proposed classification criteria for Henoch-Schonlein purpura (HSP), childhood polyarteritis nodosa (c-PAN), c-Wegener granulomatosis (c-WG) and c-Takayasu arteritis (c-TA).Methods Step 1: retrospective/prospective webdata collection for children with HSP, c-PAN, c-WG and c-TA with age at diagnosis <= 18 years. Step 2: blinded classification by consensus panel of a representative sample of 280 cases. Step 3: statistical (sensitivity, specificity, area under the curve and.-agreement) and nominal group technique consensus evaluations.Results 827 patients with HSP, 150 with c-PAN, 60 with c-WG, 87 with c-TA and 52 with c-other were compar…

Genetics and Molecular Biology (all)myalgiaVasculitismedicine.medical_specialtyHenoch-Schonlein purpuraAdolescentIgA VasculitisClassification criteriaInternational CooperationImmunologychildhood polyarteritis nodosaBiochemistryGeneral Biochemistry Genetics and Molecular BiologyRheumatologyhemic and lymphatic diseasesTerminology as TopicSchoenlein-HenochmedicineHumansImmunology and Allergycardiovascular diseasesArteritisChildPurpurac-Wegener granulomatosisAdolescent; Child; Epidemiologic Methods; Granulomatosis with Polyangiitis; Humans; International Cooperation; Polyarteritis Nodosa; Purpura Schoenlein-Henoch; Takayasu Arteritis; Terminology as Topic; Rheumatology; Immunology; Biochemistry Genetics and Molecular Biology (all); Immunology and Allergycriteria; children; Henoch-Schönlein purpura; childhood polyarteritis nodosa; Wegener granulomatosis; Takayasu arteritis; EULAR; PRINTO; PRESPolyarteritis nodosabusiness.industryGranulomatosis with Polyangiitismedicine.diseaseTakayasu ArteritisDermatologyPolyarteritis NodosaSurgeryHenoch-Schönlein purpuraIgA vasculitisSettore MED/38 - PEDIATRIA GENERALE E SPECIALISTICAmedicine.symptomEpidemiologic MethodsGranulomatosis with polyangiitisVasculitisbusinessRheumatismc-Takayasu arteriti
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In vitro production of anti-neutrophilocyte-cytoplasm-antibodies (ANCA) by Epstein-Barr virus-transformed B-cell lines in Wegener's granulomatosis.

1991

The frequent detection of anti-neutrophilocyte-cytoplasm-antibodies (ANCA) in patients with Wegener's granulomatosis (WG) led to the supposition that this disease might be of autoimmune nature. For some authors assume that Epstein-Barr virus (EBV) infection of human B-lymphocytes besides polyclonal activation could reveal the cryptic immune status against different autoantigens in patients with autoimmune diseases we investigated EBV-transformed B-lymphocytes from patients with Sjögren's syndrome, mixed connective tissue disease, WG and healthy blood donors. Two stable B-cell lines (Ho3, We1) could be established. Inhibition experiments showed that antibodies produced by transformed B-lymph…

Herpesvirus 4 HumanImmunologyBlotting WesternKidney GlomerulusFluorescent Antibody TechniqueCross ReactionsIn Vitro Techniquesmedicine.disease_causeVirusAntibodies Antineutrophil CytoplasmicMixed connective tissue diseaseAntigenmedicineImmunology and AllergyHumansB cellAgedAutoantibodiesB-LymphocytesbiologyInterleukin-6Granulomatosis with PolyangiitisMiddle Agedmedicine.diseaseCell Transformation ViralEpstein–Barr virusVirologymedicine.anatomical_structureImmunoglobulin MPolyclonal antibodiesImmunoglobulin GImmunologybiology.proteinKeratinsAntibodyClone (B-cell biology)Autoimmunity
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The Role of Inflammation in Lymphoma

2014

Human lymphomas usually develop in specialized tissue microenvironments characterized by different populations of accessory stromal and lymphoid cells that interact with malignant cells. A clinical role of the tumor microenvironment has recently emerged, bringing new knowledge and suggesting new ideas and targets for treatment. This chapter analyzes the microenvironment in human lymphomas highlighting the role of inflammation in their pathogenesis. Microenvironmental specificity is detailed according to different models including classic Hodgkin lymphoma (HL), follicular lymphoma (FL), diffuse large B-cell lymphoma (DLBCL), peripheral T-cell lymphoma, unspecified and angioimmunoblastic T-ce…

InflammationPathologymedicine.medical_specialtyTumor microenvironmentStromal cellLymphomatoid granulomatosisFollicular lymphomalymphomaInflammationBiologymedicine.diseaseBCL10Lymphomaimmune system diseaseshemic and lymphatic diseasesmedicinemedicine.symptomBrentuximab vedotinmedicine.drug
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Splenic Graulomatosis

2015

Learning objectives Background Findings and procedure details Conclusion Personal information References

Inflammationgenetic structureseducationUltrasoundAbdomenSplenic GranulomatosisTissue characterisationMRInfectionContrast agent-intravenousSettore MED/36 - Diagnostica Per Immagini E RadioterapiaSpleenCT
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A human renal cancer line as a new antigen source for the detection of antibodies to cytoplasmic and nuclear antigens in sera of patients with Wegene…

1991

Autoantibodies directed against cytoplasmic antigens of neutrophils (ANCA), especially proteinase 3 (C-ANCA), have proved to be a useful clinical tool to support the diagnosis or to monitor disease activity in Wegener's granulomatosis (WG). Till now, human neutrophil granulocytes have represented the major antigen source used to detect antibodies in WG by the immunofluorescence technique (IFT). We have tested serum samples of 164 patients with different connective tissue diseases (50 suffering from clinically active WG) performing IFT on a human renal cancer line (SK-RC11) and have found antibodies against the nuclear and cytoplasmic antigens in 39 patients. C-ANCA+ sera displayed a charact…

Liver CirrhosisTime Factorsmedicine.drug_classNeutrophilsImmunologyBlotting WesternFluorescent Antibody TechniqueImmunofluorescenceMonoclonal antibodyAutoantigensMonocytesSerologyCell LineArthritis RheumatoidScleroderma LocalizedAntigenProteinase 3medicineImmunology and AllergyHumansLupus Erythematosus SystemicAnti-neutrophil cytoplasmic antibodyAutoantibodiesMixed Connective Tissue Diseasemedicine.diagnostic_testbiologyTumor Necrosis Factor-alphaGranulomatosis with PolyangiitisBiological Transportmedicine.diseaseVirologyMolecular biologyKidney NeoplasmsSjogren's SyndromeAntibodies Antinuclearbiology.proteinInterferonsAntibodyGranulomatosis with polyangiitisGranulocytesJournal of immunological methods
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Long-term efficacy of remission-maintenance regimens for ANCA-associated vasculitides.

2018

International audience; Objective - To compare long-term efficacy of remission-maintenance regimens in patients with newly diagnosed or relapsing antineutrophil cytoplasmic antibodies (ANCA)-associated vasculitides. Methods - The 28-month Maintenance of Remission using Rituximab in Systemic ANCA-associated Vasculitis trial compared rituximab with azathioprine to maintain remission in patients with newly diagnosed or relapsing granulomatosis with polyangiitis, microscopic polyangiitis or renal-limited ANCA-associated vasculitis. Thereafter, prospective patient follow-up lasted until month 60. The primary endpoint was the major-relapse rate at month 60. Relapse and serious adverse event-free …

MaleAzathioprineKaplan-Meier Estimateurologic and male genital diseasesGastroenterologySeverity of Illness Index0302 clinical medicineimmune system diseasesRecurrenceRisk FactorsAzathioprineClinical endpointImmunology and Allergy030212 general & internal medicineskin and connective tissue diseasestreatmentRemission InductionMiddle Aged3. Good healthTreatment OutcomeAntirheumatic AgentsRituximabFemalesystemic vasculitisGranulomatosis with polyangiitisMicroscopic polyangiitisVasculitisRituximabImmunosuppressive Agentsmedicine.drugSystemic vasculitisAdultmedicine.medical_specialtyImmunologyAnti-Neutrophil Cytoplasmic Antibody-Associated VasculitisGeneral Biochemistry Genetics and Molecular BiologyDrug Administration ScheduleAntibodies Antineutrophil Cytoplasmic03 medical and health sciencesRheumatologyInternal medicinemedicineHumanscardiovascular diseasesLymphocyte CountGlucocorticoidsAnti-neutrophil cytoplasmic antibodyAged030203 arthritis & rheumatology[SDV.GEN]Life Sciences [q-bio]/Geneticsgranulomatosis with polyangiitisDose-Response Relationship Drugbusiness.industrymedicine.diseaserespiratory tract diseases[ SDV.GEN ] Life Sciences [q-bio]/GeneticsbusinessAnnals of the rheumatic diseases
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An uncommon presentation of eosinophilic granulomatosis with polyangiitis: a case report

2013

Introduction Eosinophilic granulomatosis with polyangiitis is a rare and potentially fatal disease if not readily diagnosed. Cerebral involvement is extremely rare and clinical presentation as hemorrhagic stroke is even rarer. Case presentation A 58-year-old Caucasian man was admitted to our medical unit because of a computed tomography-diagnosed hemorrhagic stroke with right-sided hemiparesis and fever. A chest computed tomography scan also revealed multiple bilateral pulmonary infiltrates; coronary artery, and carotid and left vertebral artery calcifications were also observed. Empiric antimicrobial therapy with cephalosporins was promptly undertaken; low-molecular-weight heparin was intr…

MalePathologymedicine.medical_specialtyChurg-Strauss polyangiitisSettore MED/09 - Medicina Internaeosinophilic granulomatosiCase ReportSelf MedicationChurg-Strauss SyndromeCoronary AngiographyAdrenal Cortex HormonesEosinophilicMedicineHumansChurg–Strauss syndromeCorticosteroidsLeukocytosisSelf-medicationStrokeLungAnti-neutrophil cytoplasmic antibodyMedicine(all)medicine.diagnostic_testbusiness.industryEosinophilic granulomatosis with polyangiitisGeneral MedicineCerebral InfarctionMiddle AgedSubarachnoid Hemorrhagemedicine.diseaseCoronary arteriesStrokemedicine.anatomical_structureAngiographymedicine.symptombusinessGranulomatosis with polyangiitisImmunosuppressive AgentsSystemic vasculitis
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Vasculitic wallenberg syndrome with detection of anti-proteinase 3 antibodies in the cerebrospinal fluid of a patient with severe Wegener's granuloma…

2000

MalePathologymedicine.medical_specialtyMyeloblastinAutoantigensAntibodies Antineutrophil CytoplasmicCerebrospinal fluidProteinase 3MyeloblastinMedicineHumansLungLateral Medullary SyndromeAutoantibodiesTransplantationKidneyLateral medullary syndromeLungmedicine.diagnostic_testbusiness.industrySerine EndopeptidasesAutoantibodyGranulomatosis with PolyangiitisMagnetic resonance imagingMiddle Agedmedicine.diseaseMagnetic Resonance Imagingmedicine.anatomical_structureNephrologybusinessTomography X-Ray ComputedNephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
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