Search results for "Guano"

showing 10 items of 193 documents

Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohnʼs disease

2009

Background: Oxidative stress is considered a potential etiological factor for Crohn's disease (CD). We characterized the reactive oxygen species (ROS) generated in immune peripheral cells of CD patients, as well as their antioxidant enzyme status and the presence of oxidative damage. In addition, mitochondrial function (ΔΨm) was analyzed to detect the possible origin of ROS. Methods: Cells were obtained from patients at the onset of disease, prior to any treatment. Experiments were repeated when patients were in clinical remission. A set of experiments was carried out in a group of CD patients in persistent morphological remission. Controls were healthy volunteers who were not receiving any…

AdultMaleMitochondrial DiseasesMitochondrionBiologyPharmacologymedicine.disease_causeInflammatory bowel diseaseAntioxidantsMonocytesNitric oxideSuperoxide dismutasechemistry.chemical_compoundCrohn DiseaseMalondialdehydemedicineHumansImmunology and AllergyLymphocytesMembrane Potential Mitochondrialchemistry.chemical_classificationReactive oxygen speciesSuperoxide DismutaseSuperoxideGastroenterologyDeoxyguanosineHydrogen PeroxideCatalaseOxidantsmedicine.diseaseOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineCatalaseCase-Control StudiesImmunologybiology.proteinFemaleReactive Oxygen SpeciesOxidative stressGranulocytesInflammatory Bowel Diseases
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Oxidative stress biomarkers in four Bloom syndrome (BS) patients and in their parents suggest in vivo redox abnormalities in BS phenotype.

2007

Objective: To evaluate an association of Bloom syndrome (BS) phenotype with an in vivo prooxidant state. Methods: The following endpoints were measured in 4 BS patients, their 6 parents, and 78 controls: a) leukocyte and urinary 8-hydroxy-2′-deoxyguanosine (8-OHdG); b) blood glutathione (GSSG and GSH), c) plasma levels of some plasma antioxidants (uric acid, UA, ascorbic acid, AA, α- and γ-tocopherol), and of glyoxal (Glx) and methylglyoxal (MGlx). Results: Leukocyte 8-OHdG levels were significantly increased in the 4 BS patients vs. 40 controls (p = 0.04), while the urinary 8-OHdG levels were non-significantly increased in BS patients. Glutathione disulfide levels and GSSG/GSH ratio were s…

AdultMaleParentsmedicine.medical_specialtyglyoxalAdolescentClinical Biochemistrymedicine.disease_causechemistry.chemical_compoundIn vivoInternal medicinemedicinemethylglyoxalLeukocytesHumansBloom syndromeChildoxidative streGlutathione DisulfideMethylglyoxalDeoxyguanosineGeneral MedicineGlutathioneMiddle AgedAscorbic acidmedicine.diseaseGlutathioneOxidative StressEndocrinologyPhenotypechemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidGlutathione disulfideBloom syndromeFemaleOxidation-ReductionOxidative stressBiomarkersBloom SyndromeClinical biochemistry
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Olive oils high in phenolic compounds modulate oxidative/antioxidative status in men

2004

The aim of the present study was to evaluate whether olive oils high in phenolic compounds influence the oxidative/antioxidative status in humans. Healthy men (n = 12) participated in a double-blind, randomized, crossover study in which 3 olive oils with low (LPC), moderate (MPC), and high (HPC) phenolic content were given as raw doses (25 mL/d) for 4 consecutive days preceded by 10-d washout periods. Volunteers followed a strict very low-antioxidant diet the 3 d before and during the intervention periods. Short-term consumption of olive oils decreased plasma oxidized LDL (oxLDL), 8-oxo-dG in mitochondrial DNA and urine, malondialdehyde in urine (P < 0.05 for linear trend), and increased HD…

AdultMaleTime FactorsMedicine (miscellaneous)Urinemedicine.disease_causeDNA MitochondrialAntioxidantschemistry.chemical_compoundDouble-Blind MethodPhenolsMalondialdehydemedicineHumansPlant OilsPhenolsFood scienceOlive OilGlutathione PeroxidaseNutrition and DieteticsCross-Over StudiesDose-Response Relationship DrugCholesterolCholesterol HDLDeoxyguanosineMalondialdehydePostprandial PeriodDietLipoproteins LDLDose–response relationshipVegetable oilPostprandialchemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineOxidation-ReductionOxidative stress
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Urinary metals and metal mixtures and oxidative stress biomarkers in an adult population from Spain: The Hortega Study

2019

Introduction: Few studies have investigated the role of exposure to metals and metal mixtures on oxidative stress in the general population. Objectives: We evaluated the cross-sectional association of urinary metal and metal mixtures with urinary oxidative stress biomarkers, including oxidized to reduced glutathione ratio (GSSG/GSH), malondialdehyde (MDA), and 8‑oxo‑7,8‑dihydroguanine (8-oxo-dG), in a representative sample of a general population from Spain (Hortega Study). Methods: Urine antimony (Sb), barium (Ba), cadmium (Cd), chromium (Cr), cobalt (Co), copper (Cu), molybdenum (Mo), vanadium (V) and zinc (Zn) were measured by ICPMS in 1440 Hortega Study participants. Results: The geomet…

AdultMaleUrine metals010504 meteorology & atmospheric sciencesPopulationchemistry.chemical_elementUrineZinc010501 environmental sciencesPopulation-basedmedicine.disease_cause01 natural scienceschemistry.chemical_compoundChromiumMetal mixturesMalondialdehydeMetals HeavymedicineHumanseducationlcsh:Environmental sciences0105 earth and related environmental sciencesGeneral Environmental ScienceAgedlcsh:GE1-350Cadmiumeducation.field_of_studyDeoxyguanosineGlutathioneMiddle AgedMalondialdehydeGlutathioneCross-Sectional Studieschemistry8-Hydroxy-2'-DeoxyguanosineSpainOxidative stressEnvironmental PollutantsFemaleOxidative stressBiomarkersNuclear chemistryEnvironment International
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Insulin resistance and oxidative stress in familial combined hyperlipidemia.

2007

Oxidative stress is associated with atherosclerosis. Familial combined hyperlipidemia (FCH) is considered as a human model of primary dyslipidemia and atherosclerosis frequently associated with insulin resistance (IR), but there are few data on its possible relation to oxidative stress. The objective of this study was to evaluate oxidative stress status using different markers in subjects with FCH assessing its possible correlation with anthropometric parameters and IR. This was a cross-sectional study. A cohort of 40 FCH patients (20 with IR (HOMAor=3.2) and 20 without IR (HOMA3.2)), and 20 healthy volunteers were included, all of them non-diabetic, normotensive and non-smokers. We measure…

AdultMalemedicine.medical_specialtyAdolescentHyperlipidemia Familial Combinedmedicine.disease_causeAnthropometric parametersInsulin resistanceInternal medicinemedicineHumansAgedGlutathione Disulfidebusiness.industryVascular diseasenutritional and metabolic diseasesDeoxyguanosineMiddle Agedmedicine.diseaseAtherosclerosisGlutathioneLipidsFamilial combined hyperlipidemiaOxidative StressEndocrinology8-Hydroxy-2'-DeoxyguanosineCohortFemaleInsulin ResistanceCardiology and Cardiovascular MedicinebusinessDyslipidemiaOxidative stressAtherosclerosis
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Glutathione levels in blood from ataxia telangiectasia patients suggest in vivo adaptive mechanisms to oxidative stress

2007

Objective: To evaluate an in vivo pro-oxidant state in patients with ataxia telangiectasia (AT). Methods: A set of oxidative stress endpoints were measured in 9 AT homozygotes, 16 AT heterozygotes (parents) and 83 controls (grouped in age ranges as for patients and parents, respectively). The following analytes were measured: (a) leukocyte 8-hydroxy-2-deoxyguanosine (8-OHdG); (b) blood glutathione (GSSG and GSH); and (c) plasma levels of glyoxal (Glx) and methylglyoxal (MGlx). Results: AT patients displayed a significant decrease in blood GSSG (p=0.012) and in MGlx plasma concentrations (P=0.012). A nonsignificant decrease in the GSSG:GSH ratio (p = 0.1) and a non-significant increase in 8-…

AdultMalemedicine.medical_specialtyAdolescentglyoxalClinical Biochemistryataxia telengiectasiamedicine.disease_causeAtaxia Telangiectasiachemistry.chemical_compoundIn vivoInternal medicinemethylglyoxalmedicineHumansataxia telangectasiaoxidative stressglutathioneChildoxidative streMethylglyoxalDeoxyguanosine8-Hydroxy-2'-deoxyguanosineHeterozygote advantageGeneral MedicineGlutathionePyruvaldehydemedicine.diseaseAdaptation Physiological8-Hydroxy-2'-deoxyguanosineEndocrinologychemistryBiochemistryChild PreschoolAtaxia-telangiectasiaFemaleataxia telangectasia; oxidative stress; glutathione; glyoxal; methylglyoxal; 8-Hydroxy-2'-deoxyguanosineOxidative stressTarget organDNA DamageClinical Biochemistry
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AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
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Urinary 8-oxo-7,8-dihydro-2′-deoxyguanosine (8-oxo-dG), a reliable oxidative stress marker in hypertension

2007

The potential use of oxidative stress products as disease markers and progression is an important aspect of biomedical research. In the present study, the quantification of urine 8-oxo-7,8-dihydro-2'-deoxyguanosine (8-oxo-dG) concentration has been used to express the oxidation status of hypertensive subjects. 8-oxo-dG has been simultaneously isolated and assayed in nuclear (nDNA) and mitochondrial DNA (mtDNA). In addition, oxidative stress of mononuclear cells has been estimated by means of GSH and GSSG levels and GSSG/GSH ratio in hypertensive subjects before and after antihypertensive treatment. It is shown that oxidative stress decreases significantly in hypertensive patients after trea…

AdultMalemedicine.medical_specialtyDNA damageUrinary systemUrinemedicine.disease_causeDNA MitochondrialBiochemistryPeripheral blood mononuclear cellchemistry.chemical_compoundInternal medicinemedicineHumansDeoxyguanosineChromatography High Pressure LiquidCell NucleusGlutathione DisulfideDeoxyguanosineGeneral MedicineGlutathioneGlutathioneOxidative StressEndocrinologychemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineHypertensionGlutathione disulfideFemaleBiomarkersOxidative stressFree Radical Research
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Different patterns of in vivo pro-oxidant states in a set of cancer- or aging-related genetic diseases

2008

A comparative evaluation is reported of pro-oxidant states in 82 patients with ataxia telangectasia (AT), Bloom syndrome (BS), Down syndrome (DS), Fanconi anemia (FA), Werner syndrome (WS), and xeroderma pigmentosum (XP) vs 98 control donors. These disorders display cancer proneness, and/or early aging, and/or other clinical features. The measured analytes were: (a) leukocyte and urinary 8-hydroxy-2'-deoxyguanosine (8-OHdG), (b) blood glutathione (GSSG and GSH), (c) plasma glyoxal (Glx) and methylglyoxal (MGlx), and (d) some plasma antioxidants [uric acid (UA) and ascorbic acid (AA)]. Leukocyte 8-OHdG levels ranked as follows: WS>BS approximately FA approximately XP>DS approximately AT appr…

AdultMalemedicine.medical_specialtyDown syndromeXeroderma pigmentosumAdolescentmedicine.disease_causeBiochemistrychemistry.chemical_compoundAtaxia TelangiectasiaPhysiology (medical)Internal medicinemedicineHumansBloom syndromeChildAgedXeroderma PigmentosumMethylglyoxalDeoxyguanosineGlutathioneGlyoxalMiddle AgedAscorbic acidmedicine.diseasePyruvaldehydeGlutathioneEndocrinologyFanconi AnemiaantioxidantschemistryBiochemistry8-Hydroxy-2'-DeoxyguanosineUric acidOxidative streFemaleWerner SyndromeDown SyndromeReactive Oxygen SpeciesOxidative stressBloom SyndromeDNA Damage
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Blood ethanol levels and adenylyl cyclase activity in lymphocytes of alcoholic patients

1999

Abstract Background: The adenylyl cyclase (AC) signal transduction pathway is a target of acute and chronic ethanol actions. This study examined whether AC activity in lymphocyte membranes of male alcoholic patients correlated with blood concentrations of ethanol. Methods: Patients ( n = 13; mean age: 40 ± 8 years) were studied on the day of admission (day 0) and 2 days later under detoxification. Moreover, 13 age-matched male healthy controls (mean age 40 ± 9 years) were included. Lymphocyte membranes were prepared by differential centrifugation whereby blood ethanol was washed out. As a measure of AC activity the formation of cyclic adenosine monophosphate (cAMP) from adenosine triphospha…

AdultMalemedicine.medical_specialtyLymphocyteStimulationGuanosine triphosphateAdenylyl cyclasechemistry.chemical_compoundAdenosine TriphosphateInternal medicineCyclic AMPmedicineHumansCyclic adenosine monophosphateLongitudinal StudiesLymphocytesBiological PsychiatryEthanolForskolinDose-Response Relationship DrugEthanolColforsinMiddle AgedAlcoholismmedicine.anatomical_structureEndocrinologychemistryCase-Control StudiesGuanosine TriphosphatePhosphorus RadioisotopesAdenosine triphosphateAdenylyl CyclasesSignal TransductionBiological Psychiatry
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