Search results for "HORMONES"

showing 10 items of 1169 documents

Mechanism of leptin expression in breast cancer cells: role of hypoxia-inducible factor-1α

2007

We reported previously that the obesity hormone leptin is overexpressed in breast cancer biopsies. Here, we investigated molecular mechanisms involved in this process, focusing on conditions that are associated with obesity, that is, hyperinsulinemia and induction of hypoxia. By using quantitative real-time PCR, immunofluorescent detection of proteins and enzyme-linked immunosorbent assays, we found that treatment of MCF-7 breast cancer cells with high doses of insulin or the hypoxia-mimetic agent CoCl2, or culturing the cells under hypoxic conditions significantly increased the expression of leptin mRNA and protein. Notably, the greatest leptin mRNA and protein expression were observed und…

Transcriptional ActivationCancer Researchmedicine.medical_specialtyActive Transport Cell NucleusBreast NeoplasmsBiologymedicine.disease_causeleptinbreast cancerInternal medicineCoactivatorGene expressionTumor Cells CulturedGeneticsmedicineHumansInsulinHIFp300-CBP Transcription FactorsPromoter Regions GeneticMolecular BiologyCell NucleusRegulation of gene expressionBinding SitesLeptin receptorLeptinPromoterCobaltHypoxia-Inducible Factor 1 alpha SubunitCell HypoxiaGene Expression Regulation NeoplasticEndocrinologyhyperinsulinemiaCarcinogenesisChromatin immunoprecipitationhormones hormone substitutes and hormone antagonistsProtein BindingOncogene
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Estrogen receptor (ER)-mediated transcriptional regulation of the human corticotropin-releasing hormone-binding protein promoter: differential effect…

2004

CRH-binding protein (CRH-BP) regulates activation of the hypothalamic-pituitary-adrenal (HPA) axis by binding and inhibiting CRH. We investigated for the first time transcriptional regulation of the human CRH-BP promoter using transient transfections. Estrogen receptors (ERs) contributed to ligand-independent constitutive activation of the promoter, whereas in the presence of estradiol ERalpha induced and ERbeta repressed promoter activity in a dose-dependent manner. TNFalpha inhibited promoter induction by ERalpha in the absence and presence of estradiol. Three ERE half-sites in the CRH-BP promoter bound ERalpha and ERbeta in an EMSA, and disruption of ERE half-sites by site-directed mutag…

Transcriptional Activationendocrine systemTranscription Geneticmedicine.drug_classResponse elementEstrogen receptorBiologyResponse ElementsEndocrinologymedicineTranscriptional regulationTumor Cells CulturedAnimalsEstrogen Receptor betaHumansPromoter Regions GeneticMolecular BiologyPsychological repressionConserved SequenceEstradiolNeurosecretionTumor Necrosis Factor-alphaEstrogen AntagonistsEstrogen Receptor alphaGeneral MedicineTransfectionMolecular biologyTamoxifenEstrogenPituitary GlandMutationTumor necrosis factor alphaCarrier Proteinshormones hormone substitutes and hormone antagonistsTamoxifenmedicine.drugMolecular endocrinology (Baltimore, Md.)
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Dissecting signaling and functions of adhesion G protein-coupled receptors

2012

G protein-coupled receptors (GPCRs) comprise an expanded superfamily of receptors in the human genome. Adhesion class G protein-coupled receptors (adhesion-GPCRs) form the second largest class of GPCRs. Despite the abundance, size, molecular structure, and functions in facilitating cell and matrix contacts in a variety of organ systems, adhesion-GPCRs are by far the most poorly understood GPCR class. Adhesion-GPCRs possess a unique molecular structure, with extended N-termini containing various adhesion domains. In addition, many adhesion-GPCRs are autoproteolytically cleaved into an N-terminal fragment (NTF, NT, α-subunit) and C-terminal fragment (CTF, CT, β-subunit) at a conserved GPCR au…

Tumor biologyGeneral NeuroscienceAdhesionComputational biologyBiologyGeneral Biochemistry Genetics and Molecular BiologyCell biologyGPR56History and Philosophy of ScienceHuman genomeSignal transductionCell adhesionReceptorhormones hormone substitutes and hormone antagonistsG protein-coupled receptorAnnals of the New York Academy of Sciences
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Oxytocin prevents the increase of cocaine-related responses produced by social defeat

2018

The neuropeptide oxytocin (OXT) plays a critical role in the regulation of social and emotional behaviors. OXT plays a role in the stress response and in drug reward, but to date no studies have evaluated its implication in the long-lasting increase of the motivational effects of cocaine induced by repeated social defeat (SD). During the social defeat procedure, 1 mg/kg of OXT was administered 30 minutes before each episode of SD. Three weeks after the last defeat, the effects of cocaine on conditioned place preference (CPP), locomotor sensitization and the self-administration (SA) paradigm were evaluated. Our results confirm that raising the levels of OXT during social defeat stress can bl…

UNESCO::PSICOLOGÍAhormones hormone substitutes and hormone antagonists
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Diferències entre sexes en la resposta cardiovascular, hormonal i psicològica a una competició: Variables moduladores i facilitadors de la victòria

2018

Competition is a social interaction where there is a confrontation between individuals or groups to achieve an objective. In our society competition is present transversally, with consequences for individuals. Therefore, competition is considered a social stressor able to elicit important psychobiological responses. From an evolutionary perspective, it has been suggested that, against stress situations, men would be more likely to fight-and-flight, while women would be more likely to tend-and-befriend strategies. Consequently, different psychobiological response to competition can be described depending on sex. A large proportion of studies on competition were based on the biosocial status …

UNESCO::PSICOLOGÍA::Psicología experimentalhormonesheart rate variability:PSICOLOGÍA [UNESCO]emotional statesituational appraisalUNESCO::PSICOLOGÍA:PSICOLOGÍA::Psicología experimental [UNESCO]competitionsex-differences
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High level of plasma estradiol as a new predictor of ischemic arterial disease in older postmenopausal women: the three-city cohort study.

2012

Background Despite evidence that estrogens may be involved in atherothrombosis, the role of endogenous sex steroid hormones in ischemic arterial disease among postmenopausal women remains uncertain. Methods and Results In the Three‐City prospective cohort study of subjects (n=9294) >65 years of age, we investigated the association of total 17β‐estradiol, bioavailable 17β‐estradiol, and total testosterone with the 4‐year incidence of ischemic arterial disease among postmenopausal women who did not use any hormone therapy. We designed a case–cohort study including a random sample of 537 subjects and 106 incident cases of first cardiovascular events. Weighted Cox proportional‐hazards model…

Univariate analysismedicine.medical_specialtyhormonesbusiness.industryEpidemiologyHazard ratiomedicine.diseaseConfidence intervalcardiovascular diseasesEndocrinologyDiabetes mellitusInternal medicinemedicineCardiologyrisk factorswomenCardiology and Cardiovascular MedicineProspective cohort studybusinessBody mass indexTestosteroneCohort studyOriginal ResearchJournal of the American Heart Association
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The pro-opiomelanocortin gene of the zebrafish (Danio rerio)

2003

The cDNA and the gene for pro-opiomelanocortin (POMC) in the zebrafish (Danio rerio) were isolated and analyzed. The gene consists of three exons and two short introns and has a similar overall structural organization as in Homo sapiens. Intron 1 (339 bp) divides the 5(') untranslated region from the coding region while intron 2 (1522 bp) is located between the signal peptide and the sequence encoding ACTH. Transcription starts 26 bp downstream of a TATA box and there is one polyadenylation signal in the 3(') untranslated region. The cDNA comprises of 964 bp with an open reading frame encoding a 222 amino acid hormone prepropeptide that is split into six putative hormones. Sequence comparis…

Untranslated regionendocrine systemPro-OpiomelanocortinMolecular Sequence DataBiophysicsDanioBiochemistryExonAnimalsHumansCoding regionAmino Acid SequenceRNA MessengerCloning MolecularMolecular BiologyPeptide sequenceGeneZebrafishPhylogenyZebrafishGeneticsBase SequencebiologyIntronChromosome MappingCell BiologyZebrafish Proteinsbiology.organism_classificationSequence Alignmenthormones hormone substitutes and hormone antagonistsBiochemical and Biophysical Research Communications
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Er supermosjonisme sunt? : kartlegging av helsevariabler blant mannlige langdistanseløpere og syklister.

2016

Masteroppgave folkehelsevitenskap - Universitetet i Agder 2016 Introduction: Physical activity is well documented as an important factor for good health. There are however some people who train far beyond the national recommendations for 6 physical activity combined with work and family life. The research done on «high volume exercisers» is limited, specially the health variables. Objective: The purpose with this master paper is to investigate if high volume exercise is healthy. By identifying different health variable it may point to how the body responds to high training volume. Method: 41 male high volume exercises (18 to 50 years) were included in this cross-sectional study. The compila…

VDP::Medical disciplines: 700::Health sciences: 800::Other health science disciplines: 829hormonesutholdenhetsutøverepublic healthendurance athletesfolkehelsehormonerdiseasesME516treningsavhengighetsupermosjonismesykdommerbeinmineraltetthetexercise addictionbone mineral density
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Dexamethasone Inhibits the Pro-Angiogenic Potential of Primary Human Myoblasts

2021

Tissue regeneration depends on the complex processes of angiogenesis, inflammation and wound healing. Regarding muscle tissue, glucocorticoids (GCs) inhibit pro-inflammatory signalling and angiogenesis and lead to muscle atrophy. Our hypothesis is that the synthetic GC dexamethasone (dex) impairs angiogenesis leading to muscle atrophy or inhibited muscle regeneration. Therefore, this study aims to elucidate the effect of dexamethasone on HUVECs under different conditions in mono- and co-culture with myoblasts to evaluate growth behavior and dex impact with regard to muscle atrophy and muscle regeneration. Viability assays, qPCR, immunofluorescence as well as ELISAs were performed on HUVECs,…

Vascular Endothelial Growth Factor A0301 basic medicineMuscle tissueCD31endocrine systemQH301-705.5AngiogenesisMyoblasts SkeletalNeovascularization PhysiologicInflammationdexamethasonehuman primary myoblastsArticleCatalysisInorganic Chemistry03 medical and health sciences0302 clinical medicineHuman Umbilical Vein Endothelial Cellsmedicinepolycyclic compoundsHumansMyocyteBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopyHUVECsTube formationChemistryOrganic ChemistryGeneral Medicineco-cultureVEGFCoculture TechniquesMuscle atrophyComputer Science ApplicationsCell biologyChemistry030104 developmental biologymedicine.anatomical_structureGene Expression RegulationCD31medicine.symptomWound healing030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsInternational Journal of Molecular Sciences
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Vasopressor-Sparing Strategies in Patients with Shock: A Scoping-Review and an Evidence-Based Strategy Proposition

2021

Despite the abundant literature on vasopressor therapy, few studies have focused on vasopressor-sparing strategies in patients with shock. We performed a scoping-review of the published studies evaluating vasopressor-sparing strategies by analyzing the results from randomized controlled trials conducted in patients with shock, with a focus on vasopressor doses and/or duration reduction. We analyzed 143 studies, mainly performed in septic shock. Our analysis demonstrated that several pharmacological and non-pharmacological strategies are associated with a decrease in the duration of vasopressor therapy. These strategies are as follows: implementing a weaning strategy, vasopressin use, system…

Vasopressinmedicine.medical_specialtyendocrine systemEvidence-based practicemedicine.medical_treatmentReviewvasopressorshocklaw.inventionnorepinephrineNorepinephrine (medication)Sepsissepsis03 medical and health sciences0302 clinical medicineRandomized controlled triallawmedicineRenal replacement therapyIntensive care medicinebusiness.industrySeptic shockweaningR030208 emergency & critical care medicineGeneral Medicinemedicine.disease030228 respiratory systemShock (circulatory)Medicinemedicine.symptombusinesshormones hormone substitutes and hormone antagonistsmedicine.drugJournal of Clinical Medicine
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