Search results for "Heart defects"

showing 10 items of 57 documents

Polyamines and microbiota in bicuspid and tricuspid aortic valve aortopathy

2019

Polyamines are small aliphatic cationic molecules synthesized via a highly regulated pathway and involved in general molecular and cellular phenomena. Both mammalian cells and microorganisms synthesize polyamines, and both sources may contribute to the presence of polyamines in the circulation. The dominant location for microorganisms within the body is the gut. Accordingly, the gut microbiota probably synthesizes most of the polyamines in the circulation in addition to those produced by the mammalian host cells. Polyamines are mandatory for cellular growth and proliferation. Established evidence suggests that the polyamine spermidine prolongs lifespan and improves cardiovascular health in …

Heart Defects Congenital0301 basic medicineAortic valveVascular smooth muscleHeart Valve Diseases030204 cardiovascular system & hematologyGut floraSystemic inflammation03 medical and health sciences0302 clinical medicineBicuspid aortic valveBicuspid Aortic Valve Diseasemedicine.arteryPolyamines and microbiotaAscending aortaPolyaminesAnimalsHumansMedicineSettore MED/05 - Patologia ClinicaBicuspidMolecular BiologyAortabiologybusiness.industrymedicine.diseasebiology.organism_classificationGastrointestinal MicrobiomeCell biologyEndothelial stem cell030104 developmental biologymedicine.anatomical_structureAortic ValveDisease Progressioncardiovascular systemTricuspid Valvemedicine.symptombicuspid and tricuspid aortic valve aortopathybusinessCardiology and Cardiovascular Medicine
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Exome sequencing in a child with neurodevelopmental disorder and epilepsy: Variant analysis of the AHNAK2 gene

2022

Background The AHNAK2 gene encodes a large nucleoprotein expressed in several tissues, including brain, squamous epithelia, smooth muscle, and neuropil. Its role in calcium signaling has been suggested and to date, clear evidence about its involvement in the pathogenesis of clinical disorders is still lacking. Methods Here, we report a female 24-year-old patient diagnosed with a cardio-facio-cutaneous-like phenotype (CFC-like), characterized by epilepsy, psychomotor development delay, atopic dermatitis, congenital heart disease, hypotonia, and facial dysmorphism, who is compound heterozygote for two missense mutations in the AHNAK2 gene detected by exome sequencing. Results This patient had…

Heart Defects CongenitalAHNAK2 borderline intellectual functioning epilepsy facio-cardio-cutaneous-like phenotype NGS exomefacio-cardio-cutaneous-like phenotypeFaciesNGS exomeSettore MED/39 - Neuropsichiatria InfantileFailure to ThriveNucleoproteinsEctodermal DysplasiaNeurodevelopmental DisordersAHNAK2borderline intellectual functioningGeneticsHumansepilepsyExomeFemaleMolecular BiologyGenetics (clinical)Molecular Genetics & Genomic Medicine
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Cardio-facio-cutaneous syndrome and gastrointestinal defects: report on a newborn with 19p13.3 deletion including the MAP 2 K2 gene

2022

Abstract Background Cardio-facio-cutaneous syndrome (CFCS) belongs to RASopathies, a group of conditions caused by mutations in genes encoding proteins of the rat sarcoma/mitogen-activated protein kinase (RAS/MAPK) pathway. It is a rare syndrome, with about 300 patients reported. Main clinical manifestations include facial dysmorphisms, growth failure, heart defects, developmental delay, and ectodermal abnormalities. Mutations (mainly missense) of four genes (BRAF, MAP 2 K1, MAP 2 K2, and KRAS) have been associated to CFCS. However, whole gene deletions/duplications and chromosomal microdeletions have been also reported. Specifically, 19p13.3 deletion including MAP 2 K2 gene are responsible…

Heart Defects CongenitalComparative Genomic HybridizationEctodermal DysplasiaPregnancyCFCS RASopathies Contiguous gene syndrome Array-CGH Genotype-phenotype correlations HPS Case reportFaciesHumansFemaleSyndromeHernia UmbilicalFailure to ThriveItalian Journal of Pediatrics
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Studies of malformation syndromes of man XIB: the cerebro-hepato-renal syndrome of zellweger: Comparative pathology

1976

A study of 11 autopsied cases of the cerebro-hepato-renal syndrome of Zellweger (ZS) is reported. All cases had severe, persistent congenital hypotonia, hepatic lobular disarray, renal cortical cysts and pulmonary hypoplasia. Many had cardiovascular malformations, hepatomegaly, cerebral cortical gyral maldevelopment and pancreatic islet hyperplasia. Additional, less frequent findings are delineated. Results of iron content studies of hepatic and renal tissues are related to age of survival and possible development of fibrosis.

Heart Defects CongenitalLiver CirrhosisMalePathologymedicine.medical_specialtySiderosisCirrhosisRenal cortical cystsPancreatic islet hyperplasiaIronKidneyNeurologic ManifestationsPulmonary hypoplasiaMaldevelopmentFibrosisHumansMedicineAbnormalities MultipleRadiology Nuclear Medicine and imagingbusiness.industryInfant NewbornBrainInfantGeneral MedicineKidney Diseases Cysticmedicine.diseaseBile Ducts IntrahepaticNeonatal hypotoniaLiverConnective TissuePediatrics Perinatology and Child HealthFemaleHepatic fibrosisbusinessHepatomegalyEuropean Journal of Pediatrics
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Clinical Delineation Of A Subtype Of Frontonasal Dysplasia With Creased Nasal Ridge And Upper Limb Anomalies: Report Of Six Unrelated Patients

2017

IF 2.259; International audience; Frontonasal dysplasias are rare congenital malformations of frontonasal process-derived structures, characterized by median cleft, nasal anomalies, widely spaced eyes, and cranium bifidum occultum. Several entities of syndromic frontonasal dysplasia have been described, among which, to date, only a few have identified molecular bases. We clinically ascertained a cohort of 124 individuals referred for frontonasal dysplasia. We identified six individuals with a similar phenotype, including one discordant monozygous twin. Facial features were remarkable by nasal deformity with creased ridge and depressed or absent tip, widely spaced eyes, almond-shaped palpebr…

Heart Defects CongenitalMale0301 basic medicineChoanal atresiaNoseBiologyfrontonasal dysplasiaChoanal AtresiaFacial BonesEncephaloceleCohort StudiesCraniofacial Abnormalities03 medical and health sciences0302 clinical medicineExome SequencingGeneticsmedicineHumansAbnormalities MultipleFrontonasal dysplasia[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsGenetics (clinical)Exome sequencingEncephalocelenasofrontal encephaloceleCorpus Callosum AgenesisInfantAnatomymedicine.diseasePhenotype030104 developmental biologyPalpebral fissuremedicine.anatomical_structure[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsnasal malformationFaceEtiologyUpper limbFemaleAgenesis of Corpus Callosum030217 neurology & neurosurgery
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Array-CGH and clinical characterization in a patient with subtelomeric 6p deletion without ocular dysgenesis

2011

Subtelomeric terminal 6p deletion has been recognized as a clinically identifiable syndrome including facial dysmorphism, malformation of the anterior eye chamber, hearing loss, heart defect and developmental delay. Genotype –phenotype correlations of previously published patients have been strongly suggested anterior eye segment anomalies as one of major malformation of the syndrome if the critical 6p25 region containing the FOXC 1 gene. In addition it has been hypothesized the presence in this region of one or more genes involved in hearing loss. We report on a case of terminal 6p deletion in a 47, XYY karyotype. Further characterization of the deletion with array comparative genome hybri…

Heart Defects CongenitalMaleHearing lossDevelopmental DisabilitiesKaryotypeBiologyEyeDysgenesisSettore MED/38 - Pediatria Generale E SpecialisticaChromosome 19GeneticsmedicineHumansarray-CGH.Eye AbnormalitiesGeneGenetics (clinical)Genetic Association StudiesIn Situ Hybridization FluorescenceGeneticsComparative Genomic Hybridizationeye abnormalitieInfantKaryotypeForkhead Transcription Factorshearing loSubtelomereAnterior Eye SegmentSettore MED/03 - Genetica MedicaChromosomes Human Pair 6FOXC1medicine.symptomChromosome Deletionchromosome 6p deletionComparative genomic hybridization
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Motor development of infants with univentricular heart at the ages of 16 and 52 weeks

2013

Purpose To compare the motor development of patients with hypoplastic left heart syndrome (HLHS) and other types of univentricular heart (UVH) with peers who are healthy at the ages of 16 and 52 weeks. Methods Motor development was assessed with the Alberta Infant Motor Scale (AIMS). Results Both the 23 patients with HLHS and the 13 patients with UVH had lower total AIMS scores in both observations than the controls. At the age of 52 weeks, patients with HLHS had significantly lower scores in all 4 AIMS subscales, whereas patients with UVH had lower scores only in the prone and standing subscales. Conclusion Motor development of patients with HLHS or UVH is delayed during the first year of …

Heart Defects CongenitalMalePediatricsmedicine.medical_specialtyHeart VentriclesPhysical Therapy Sports Therapy and RehabilitationFirst year of life030204 cardiovascular system & hematologyMotor ActivityNeuropsychological TestsHypoplastic left heart syndrome03 medical and health sciences0302 clinical medicineChild Development030225 pediatricsmedicineHumansProspective StudiesProspective cohort studyMotor skillRetrospective Studiesbusiness.industryInfantRetrospective cohort studyta3141medicine.diseasePrognosisChild developmentUniventricular heartExercise TherapyMotor SkillsChild PreschoolPediatrics Perinatology and Child HealthFemalebusinessPediatric Physical Therapy
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A population-based study relevant to seasonal variations in causes of death in children undergoing surgery for congenital cardiac malformations

2007

AbstractAimsOur objectives were, first, to study seasonal distribution of perioperative deaths within 30 days after surgery, and late death, in children undergoing surgery for congenitally malformed hearts, and second, to study the causes of late death.MethodsWe analysed a retrospective cohort of 1,753 children with congenital cardiac malformations born and undergoing surgery in the period from 1990 through 2002 with a special focus on the causes of late death. The data was obtained from the registry of congenital cardiac malformations at Rikshospitalet, Oslo, and the Norwegian Medical Birth Registry. The mean follow-up from birth was 8.1 years, with a range from zero to 15.2 years.ResultsD…

Heart Defects CongenitalMalePediatricsmedicine.medical_specialtyTime FactorsAdolescentrespiratory syncytial virussudden deathSudden deathCohort StudiesPostoperative Complicationsrespiratory infectionCause of DeathVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Pediatrics: 760medicineHumansChildDown's syndromeRetrospective StudiesVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Cardiology: 771Norwaybusiness.industryInfant NewbornInfantRespiratory infectionGeneral MedicinemortalityPeer reviewSurvival RatePopulation based studyChild PreschoolPediatrics Perinatology and Child HealthFemaleSeasonsCardiology and Cardiovascular MedicineCongenital cardiac malformationsbusinessCardiology in the Young
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Reduced inotropic support after aprotinin therapy during pediatric cardiac operations

1999

Several reports indicate that aprotinin treatment before and during cardiopulmonary bypass (CPB) might have a protective effect on the myocardium. We evaluated the hemodynamic effects of perioperative aprotinin treatment.We conducted a randomized, double-blind, placebo-controlled trial in 34 infants (mean age, 2.5 years) who had cardiac operations. Half of the patients received high-dose aprotinin therapy. There were no significant differences between the aprotinin and placebo groups with respect to age, weight, sex, aortic cross-clamp time, and CPB time. The following data were recorded at arrival in the intensive care unit 6, 12, 24, and 48 hours after termination of CPB: heart rate, bloo…

Heart Defects CongenitalMalePulmonary and Respiratory MedicineAdolescentPhosphodiesterase InhibitorsHemodynamicsPlaceboHemostaticslaw.inventionAprotininDouble-Blind MethodlawHeart rateCardiopulmonary bypassHumansMedicineEnoximoneAprotininChildEnoximoneCardiopulmonary Bypassbusiness.industryHemodynamicsInfant NewbornInfantFurosemideBlood pressureChild PreschoolAnesthesiaFemaleSurgeryCardiology and Cardiovascular Medicinebusinesshormones hormone substitutes and hormone antagonistsmedicine.drugThe Annals of Thoracic Surgery
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Adjustable tourniquet to manipulate pulmonary blood flow after Norwood operations

2000

Abstract Background . Survival after first-stage palliative Norwood operations for single ventricle with systemic outflow obstruction is mainly dependent on a balanced ratio of pulmonary blood flow to systemic blood flow. Here we report the clinical results using a modified technique that allows a controlled systemic-to-pulmonary shunt flow to prevent pulmonary overcirculation. Methods . From 1995 to 1998, of 26 infants undergoing first-stage palliative Norwood operations, 7 had placement of an adjustable tourniquet around a modified right Blalock-Taussig shunt. Results . Hospital survival was 20 of 26 patients (77%). All 7 patients in whom snaring of the shunt was indicated survived. Two p…

Heart Defects CongenitalMalePulmonary and Respiratory MedicinePulmonary Circulationmedicine.medical_specialtyReconstructive surgeryHeart diseaseHemodynamicsPulmonary Arterylaw.inventionHypoplastic left heart syndromelawHypoplastic Left Heart SyndromeCardiopulmonary bypassHumansMedicineCardiac Surgical ProceduresTourniquetbusiness.industryPalliative CareInfant NewbornInfantTourniquetsmedicine.diseaseSurgeryShunt (medical)Survival Ratemedicine.anatomical_structureVentricleAnesthesiaFemaleSurgeryCardiology and Cardiovascular MedicinebusinessThe Annals of Thoracic Surgery
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