Search results for "Herpes"

showing 10 items of 307 documents

H-2-linked murine cytotoxic T cell responses specific for sendai virus-infected cells

1978

CBA (H-2k) mouse-derived lymphochoriomeningitis virus and herpes simplex virus-specific cytotoxic T lymphocytes lyse virus-infected target cells compatible on either the H-2k or H-2D region. In contrast, CBA, C3H and AKR (H-2k) mouse-derived sendai virus-specific cytotoxic T lymphocytes (CTL) fail to lyse H-2D-compatible virus-infected cells. A similar lack of H-2D region-associated lytic activity was found with C57BL/6 and C57BL/10 (H-2b) mice as well as with the recombinants B10.A (2R) [Kb-Db] and B10.A (4R) [Kk-Db]. On the other hand, BALB/c (H-2d) mice and A/J (H-2a) mice do generate H-2Dd-associated sendai virus-specific CTL. These results are in contrast to those obtained with (CBA X …

Cytotoxicity ImmunologicGenotypeT-LymphocytesvirusesImmunologychemical and pharmacologic phenomenaVirusMajor Histocompatibility ComplexEpitopesMiceGenotypeAnimalsLymphocytic choriomeningitis virusSimplexvirusImmunology and AllergyCytotoxic T cellGeneMice Inbred BALB CParamyxoviridae InfectionsbiologyHerpes Simplexbiology.organism_classificationVirologySendai virusParainfluenza Virus 1 HumanMice Inbred C57BLCTL*RickettsiaLytic cycleMice Inbred CBAEuropean Journal of Immunology
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Cloning and expression of the complement receptor glycoprotein C from Herpesvirus simiae (herpes B virus): protection from complement-mediated cell l…

2003

Simian herpes B virus (SHBV) is the herpes simplex virus (HSV) homologue for the species MACACA: Unlike in its natural host, and unlike other animal herpesviruses, SHBV causes high mortality in accidentally infected humans. SHBV-infected cells, like those infected with HSV-1 and equine herpesvirus types 1 and 4, express complement C3 receptor activity. To study immunoregulatory functions involved in susceptibility/resistance against interspecies transmission, the SHBV glycoprotein C (gC(SHBV)) gene (encoding 467 aa) was isolated. Sequence analysis revealed amino acid identity with gC proteins from HSV-2 (46.9 %), HSV-1 (44.5 %) and pseudorabies virus (21.2 %). Highly conserved cysteine resi…

Cytotoxicity ImmunologicHerpes B virusvirusesComplement Pathway AlternativeMolecular Sequence DataHerpesvirus 1 CercopithecineComplement receptorBiologyTransfectionmedicine.disease_causeVirusCell LineViral Envelope ProteinsVirologymedicineAnimalsHumansAmino Acid SequenceCloning MolecularPeptide sequenceSequence Analysis DNAbiology.organism_classificationVirologyComplement systemHerpes simplex virusCell cultureComplement C3bReceptors Complement 3bAlternative complement pathwayJournal of General Virology
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Combining dasatinib with dexamethasone long-term leads to maintenance of antiviral and antileukemia specific cytotoxic T cell responses in vitro

2012

Maintaining graft versus leukemia (GvL) and antivirus responses of cytotoxic T cells (CTLs) while suppressing graft-versus-host disease (GvHD) remains a challenge after allogeneic bone marrow transplantation. Clinical observations indicate that combining glucocorticoids with multi-tyrosine-kinase inhibitors could be a successful therapeutic approach. We and others have shown that the BCR-ABL/SRC kinase inhibitor dasatinib may enhance or suppress T cells in vitro. In this report, we evaluated combination effects of dasatinib and dexamethasone on CD3 + and virus-specific CD8 + T cells directly ex vivo and on antigen-specific leukemia-reactive and alloreactive CD8 + T cell clones. Functional o…

Cytotoxicity ImmunologicHerpesvirus 4 HumanCancer ResearchNaive T cellT cellDasatinibDrug Evaluation PreclinicalReceptors Antigen T-CellCytomegalovirusApoptosisT-Cell Antigen Receptor SpecificityBiologyLymphocyte ActivationCell DegranulationDexamethasoneAntigenHLA AntigensT-Lymphocyte SubsetsGeneticsmedicineHumansCytotoxic T cellAntigens ViralProtein Kinase InhibitorsMolecular BiologyCells CulturedDegranulationDrug SynergismT-Lymphocytes Helper-InducerCell BiologyHematologyDasatinibThiazolesPyrimidinesmedicine.anatomical_structureImmunologyCancer researchCytokinesK562 CellsMemory T cellCell DivisionCD8Signal TransductionT-Lymphocytes Cytotoxicmedicine.drugExperimental Hematology
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A tyrosinase peptide presented by HLA-B35 is recognized on a human melanoma by autologous cytotoxic T lymphocytes

1999

We previously described different cytotoxic T lymphocyte (CTL) clones isolated from the blood lymphocytes of a melanoma patient after in vitro stimulation with autologous tumor cells. These CTL clones recognized at least 2 distinct antigens on the melanoma cells. Here, we show that one of them consists of a peptide derived from tyrosinase and presented by HLA-B35. The peptide is 9 amino acids long and has the sequence LPSSADVEF. It can be presented by the 2 major B35 allelic subtypes, B*3501 and B*3503. As HLA-B35 is one of the most frequent HLA-B specificities, being present in about 20% of Caucasian individuals, it may be a useful target for peptide-based immunotherapy of melanoma.

Cytotoxicity ImmunologicHerpesvirus 4 HumanCancer Researchmedicine.medical_treatmentAntigen presentationTyrosinase PeptideBiologyTransfectionAntigenTumor Cells CulturedmedicineAnimalsHumansCytotoxic T cellAmino Acid SequenceMelanomaPeptide sequenceAllelesCell Line TransformedB-LymphocytesMonophenol MonooxygenaseMelanomaImmunotherapymedicine.diseasePeptide FragmentsRecombinant ProteinsCTL*OncologyCOS CellsImmunologyCancer researchHLA-B35 AntigenT-Lymphocytes CytotoxicInternational Journal of Cancer
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Low frequency of cytotoxic liver-infiltrating T lymphocytes specific for endogenous processed surface and core proteins in chronic hepatitis B.

1993

To investigate the role of hepatitis B virus (HBV)-specific CD8+ T cells in chronic hepatitis B, the lytic activity of peripheral blood mononuclear cells (PBMC) and liver-infiltrating T cell clones and cytotoxic T cell (CTL) lines stimulated by recombinant vaccinia virus-infected cells were analyzed. Autologous and allogeneic Epstein-Barr virus-transformed B cells infected with vaccinia vectors (VAC) that contain sequences of the surface (S), secretory core (E), cytoplasmatic core (C) VAC antigen of HBV, or the wild-type (WT) VAC served as target cells. ELISA and immunoblotting showed HBV antigen expression in infected cells. Neither PBMC nor C- or E-VAC-stimulated CTL lines showed specific…

Cytotoxicity ImmunologicHerpesvirus 4 HumanT cellGenes MHC Class IVaccinia virusBiologymedicine.disease_causeHepatitis B AntigensAntigenCell MovementmedicineImmunology and AllergyCytotoxic T cellHumansHepatitis B e AntigensHepatitis ChronicHepatitis B virusHepatitisB-LymphocytesHepatitis B Surface AntigensHepatitis Bmedicine.diseasebiology.organism_classificationCell Transformation ViralHepatitis BVirologyHepatitis B Core AntigensRecombinant ProteinsCTL*Infectious Diseasesmedicine.anatomical_structureHepadnaviridaeLiverProtein Processing Post-TranslationalT-Lymphocytes CytotoxicThe Journal of infectious diseases
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Frequency of cytotoxic T lymphocyte precursors to herpes simplex virus type 1 as determined by limiting dilution analysis.

1983

The conditions for establishing a limiting dilution assay to measure cytotoxic T lymphocyte precursors (CTL-P) against herpes simplex virus type 1 (HSV-1) were determined. Analysis by Poisson statistics demonstrated that the estimated frequency of HSV-1-reactive cells in the spleens of normal mice was less than 1/250,000. In contrast, mice immunized previously with infectious HSV-1 demonstrated a CTL-P frequency between 1/3,500 and 1/15,670. The generation of a maximum cytotoxic T lymphocyte response required that mice be primed in vivo with infectious virus. Immunization with inactivated virus either failed to elicit detectable CTL-P frequencies or gave frequencies markedly less than thos…

Cytotoxicity ImmunologicT cellImmunologyPopulationchemical and pharmacologic phenomenaBiologymedicine.disease_causeMicrobiologyVirusMiceImmune systemAntigenmedicineCytotoxic T cellAnimalsAntigens LySimplexviruseducationCytotoxicityCells Culturededucation.field_of_studyVirologyInfectious Diseasesmedicine.anatomical_structureHerpes simplex virusImmunologic TechniquesMice Inbred CBAInterleukin-2ParasitologyImmunizationSpleenT-Lymphocytes CytotoxicViral Infections and ImmunityInfection and immunity
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Herpes-Simplex-virus-specific, H-2Dk-restricted T lymphocytes bear receptors for H-2Dd alloantigen.

1980

Cytotoxic T lymphocytes generated in the course of an HSV-infection of CBA (H-2k) mice not only lyse syngeneic, virus-infected target cells but also cross-react with noninfected taraget cells expressing the Dd alloantigen. On the effector cell level, this alloreactivity is mediated by virus-specific CTL's that are restricted to H-2Dk determinants. On the prekiller cell level, the anti-HSV-reactive T cells exhibiting cross-reactivity for Dd alloantigen could be positively selected on H-2d spleen-cell monolayers. After differentiation into cytolytic effector cells, target cells expressing Dd alloantigens and syngeneic HSV-infected target were lysed with equal efficiency. The results imply tha…

Cytotoxicity ImmunologicT-LymphocytesImmunologyReceptors Antigen T-Cellchemical and pharmacologic phenomenaMice Inbred StrainsBiologymedicine.disease_causeVirusEpitopesMiceGeneticsmedicineCytotoxic T cellAnimalsSimplexvirusReceptorEffectorH-2 Antigenshemic and immune systemsbiology.organism_classificationMolecular biologyCytolysisCTL*RickettsiaHerpes simplex virusImmunologyImmunogenetics
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In Vivo Protective Effect of Tumour Necrosis Factor   Against Experimental Infection with Herpes Simplex Virus Type 1

1991

C57BL/6 mice, which differ genetically from other strains by their resistance to herpes simplex virus type 1 (HSV-1) infection, were inoculated intraperitoneally with different doses of tumour necrosis factor alpha (TNF-alpha). Mice pretreated with 100 ng, or even 10 ng, of TNF-alpha showed prolonged survival compared to control mice that were infected with 10(7) p.f.u. of HSV-1. Significant protection was observed in mice injected 4 or 8 h prior to or after HSV-1 inoculation, respectively. Protection was also observed when mice which differed at their H-2 locus were treated with TNF-alpha after infection with HSV-1. Interferon could not be detected in the sera of mice at different time poi…

Cytotoxicity ImmunologicTime FactorsNecrosismedicine.medical_treatmentBiologymedicine.disease_causeVirusCell LineNatural killer cellMiceInterferonIn vivoVirologymedicineAnimalsSimplexvirusMice Inbred BALB CTumor Necrosis Factor-alphaHerpes SimplexVirologyRecombinant ProteinsMice Inbred C57BLKineticsHerpes simplex virusCytokinemedicine.anatomical_structureMice Inbred DBAImmunologyTumor necrosis factor alphaInterferonsmedicine.symptommedicine.drugJournal of General Virology
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Anti H-2Dd alloreactivity mediated by herpes-simplex-virus specific cytotoxic H-2k T lymphocytes is associated with H-2Dk.

1980

Herpes-simplex-virus (HSV) specific, H-2k-restricted, immune cytotoxic T lymphocytes also lyse noninfected H-2d target cells. Genetic mapping studies revealed that HSV-specific Dk-restricted CTL cross-react with allogeneic targets expressing Dd alloantigens. Cold target inhibition experiments indicate that only a minority of HSV-specific CTL mediate cross-reactive cytolysis. The data give an example of where the phenomenon of H-2-restricted versus nonrestricted responsiveness is not due to distinct subsets of T cells but solely depends on the antigenic determinants recognized.

Cytotoxicity ImmunologicvirusesImmunologychemical and pharmacologic phenomenaBiologyCross Reactionsmedicine.disease_causeEpitopeEpitopesMiceImmune systemAntigenIsoantibodiesGeneticsmedicineCytotoxic T cellAnimalsSimplexvirusCytotoxicityHistocompatibility Antigen H-2DMice Inbred BALB CH-2 AntigensVirologyCytolysisCTL*Herpes simplex virusImmunologyMice Inbred CBAT-Lymphocytes CytotoxicImmunogenetics
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Influence of dibutyryl cyclic AMP on thymidine uptake by herpes simplex virus infected cells and the intracellular level of cyclic AMP.

1977

Abstract Dibutyryl cyclic AMP inhibits the increase of dThd and BrdUrd transport normally observed after infection with Herpesvirus hominis, type I and II. Incorporation is also reduced. Inhibition of uptake is non-competitive as analysed by the Lineweaver-Burk plot. Addition of this drug to infected cells also reduces the activity of the thymidine kinase (EC 2.7.1.75). Transport of dUrd, dCyd and dAdo is not reduced. 4–8 h after infection with thymidine kinase (+) herpes strains the level of cAMP increases. On infection with a thymidine kinase (−) virus, only a small elevation of cAMP can be shown. It was also found that early addition of actinomycin D or of cycloheximide prevents the incr…

DNA ReplicationUltraviolet RaysDeoxyribonucleosidesBiologyCycloheximidemedicine.disease_causeVirus ReplicationBiochemistry Genetics and Molecular Biology (miscellaneous)Thymidine KinaseVirusCell Linechemistry.chemical_compoundSpecies SpecificitymedicineCyclic AMPSimplexvirusThymine NucleotidesCycloheximideDadoBiological TransportDibutyryl Cyclic AMPMolecular biologyKineticsHerpes simplex viruschemistryBromodeoxyuridineBucladesineThymidine kinaseDNA ViralDactinomycinThymidineIntracellularThymidineBiochimica et biophysica acta
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