Search results for "Histocompatibility Antigens Class I"

showing 8 items of 208 documents

Stimulation with carbachol alters endomembrane distribution and plasma membrane expression of intracellular proteins in lacrimal acinar cells.

2000

The events that lead to Sjogren's autoimmune processes in the lacrimal gland remain poorly understood. The acinar cell's responses to acute cholinergic stimulation include release of secretory products across the apical plasma membrane (apm) and a number of processes related to traffic between endomembrane compartments and the basal-lateral plasma membranes (blm), such as recruitment of Na, K-ATPase, accelerated recycling, and accelerated transcytosis of secretory IgA. We tested the hypothesis that stimulation-induced acceleration of endomembrane traffic is accompanied by changes in compartmentation and increased blm expression of proteins that are normally sequestered in endomembrane compa…

medicine.medical_specialtyAcid PhosphataseImmunoblottingGolgi ApparatusStimulationBiologyCholinergic AgonistsCathepsin BCathepsin BCellular and Molecular Neurosciencesymbols.namesakeInternal medicinemedicineAcinar cellAnimalsEndomembrane systemCells Culturedrab5 GTP-Binding ProteinsDifferential centrifugationEnzyme PrecursorsCell MembraneHistocompatibility Antigens Class IIMembrane Proteinsalpha-GlucosidasesGolgi apparatusGalactosyltransferasesCathepsinsSensory SystemsStimulation Chemicalbeta-N-AcetylhexosaminidasesCell biologyOphthalmologyEndocrinologySjogren's SyndromeTranscytosisrab GTP-Binding ProteinssymbolsCarbacholElectrophoresis Polyacrylamide GelFemaleRabbitsSodium-Potassium-Exchanging ATPaseIntracellularExperimental eye research
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Preeclampsia, a pregnancy-specific disease, is associated with fetal monocyte activation.

2001

The maternal syndrome of preeclampsia is an exclusively pregnancy-related illness involving multiple organs and severe forms may be complicated by HELLP (hemolysis, elevated liver enzymes, low platelets) syndrome. Recently, it has been proposed that both normal pregnancy and preeclampsia are associated with a systemic activation of the nonspecific maternal immune system and that, in particular, monocytes have a central role in the adjustment of maternal immune functions in pregnancy. Here we have investigated the role of the fetal nonadaptive immune system in normal term delivery, uncontrollable preterm labor, and preeclampsia. We demonstrate that spontaneous delivery at term as well as pre…

medicine.medical_specialtyHELLP SyndromeHELLP syndromeImmunologyLipopolysaccharide ReceptorsInflammationBiologyMonocytesPreeclampsiaImmune systemFetusPre-EclampsiaPregnancyInternal medicinemedicineImmunology and AllergyHumansreproductive and urinary physiologyFetusPregnancyInnate immune systemInterleukin-6MonocyteHistocompatibility Antigens Class IHistocompatibility Antigens Class IImedicine.diseaseFetal BloodEndocrinologymedicine.anatomical_structureembryonic structuresImmunologyFemalemedicine.symptomClinical immunology (Orlando, Fla.)
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The Presence of Gestational Diabetes is Associated with Increased Detection of Anti-HLA-class II Antibodies in the Maternal Circulation

2006

Problem Gestational diabetes (GD) may be associated with temporarily reduced immune tolerance toward alloantigens for the time of pregnancy. The aim of this study was to assess anti-HLA-class I and -II antibodies as markers for an aberrant immunostimulation in women with GD. Method of study The percentage of anti-HLA-class I and -II antibodies was estimated in women with GD, normal term delivery and fetal distress, which was confirmed by demonstrating low cord blood pH for this patient group. These antibodies may cross the placental barrier and cause interleukin-6 (IL-6) release from fetal monocytes by cross-linking monocytes with antibody-loaded cells. Therefore we estimated the percentage…

medicine.medical_specialtyImmunologyModels BiologicalFetal DistressAntigenIsoantibodiesPregnancyInternal medicinemedicineFetal distressHumansImmunology and AllergyFetusPregnancybusiness.industryHistocompatibility Antigens Class IIObstetrics and GynecologyHydrogen-Ion ConcentrationFetal Bloodmedicine.diseaseGestational diabetesDiabetes GestationalTolerance inductionFetal circulationEndocrinologyReproductive MedicineCord bloodLeukocytes MononuclearFemalebusinessAmerican Journal of Reproductive Immunology
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Granulocyte-macrophage colony-stimulating factor-cultured bone marrow-derived macrophages reveal accessory cell function and synthesis of MHC class I…

1988

The antigen-mediated activation of a number of T cell clones by bone marrow (BM) cells cultivated in the presence of various colony-stimulating factor (CSF) preparations was investigated. BM macrophages (BMM phi) grown in L929 cell supernatant as a crude source of macrophage colony-stimulating factor (M-CSF) as well as BM cells propagated in the presence of recombinant M-CSF exhibited transient antigen presentation potential to some T cell clones, being maximal on day 7 and having declined to a low level by day 19 of in vitro culture. Treatment of these long-term-cultivated BMM phi populations with recombinant interferon-gamma (IFN-gamma) resulted in predominant antigen presentation capacit…

medicine.medical_specialtyT cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsBone Marrow CellsMajor histocompatibility complexLymphocyte ActivationCell LineInterferon-gammaMiceAntigenColony-Stimulating FactorsInternal medicinemedicineImmunology and AllergyCytotoxic T cellAnimalsAntigensAntigen-presenting cellGrowth SubstancesMHC class IIHybridomasbiologyMonocyteMacrophagesHistocompatibility Antigens Class IIGranulocyte-Macrophage Colony-Stimulating FactorMolecular biologyCulture Mediamedicine.anatomical_structureEndocrinologybiology.proteinEuropean journal of immunology
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Role of Human Leukocyte Antigens (HLA) in Autoimmune Diseases

2018

Since the discovery of HLA 60 years ago, it has contributed to the understanding of the immune system as well as of the pathogenesis of several diseases. Aside from its essential role in determining donor-recipient immune compatibility in organ transplantation, HLA genotyping is meanwhile performed routinely as part of the diagnostic work-up of certain autoimmune diseases. Considering the ability of HLA to influence thymic selection as well as peripheral anergy of T cells, its role in the pathogenesis of autoimmunity is understandable. The aim of this paper is to provide a brief overview of the role and current clinical relevance of HLA-B27 in spondyloarthritis and HLA-B51 in Behçet's disea…

musculoskeletal diseases0301 basic medicinemedicine.medical_specialtyReviewBehcet's diseaseDiseaseHuman leukocyte antigenmedicine.disease_causeOrgan transplantationAutoimmune DiseasesAutoimmunityPathogenesisEpitopes03 medical and health sciencesImmune system0302 clinical medicineRheumatologyHLA AntigensInternal medicineHumansImmunology and AllergyMedicineClinical significanceskin and connective tissue diseases030203 arthritis & rheumatologyHLA-B27business.industryHistocompatibility Antigens Class IInutritional and metabolic diseasesmedicine.diseaseRheumatologyeye diseasesstomatognathic diseases030104 developmental biologyRheumatoid arthritisImmunologybusiness
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HLA Class II Differentiates Between Thyroid and Polyglandular Autoimmunity.

2015

The HLA class II genes are susceptibility genes for autoimmune endocrine diseases; however, scarce data are available pertaining to the determinants of genetic susceptibility to polyglandular autoimmunity (PGA). A total of 300 consecutive and unselected patients with either PGA or monoglandular autoimmune thyroid disease (AITD) and 100 healthy control subjects were genotyped for the HLA class II DRB1, -DQA1, and -DQB1 alleles. Compared to patients with AITD and controls, the HLA-DRB1*03 (pc =0.001), *04 (pc<0.001), -DQA1*03 (pc<0.001), and -DQB1*02 (pc =0.001) alleles were increased in patients with PGA. When dividing patients with Hashimoto's thyroiditis (HT) into those with PGA (PGA-HT) v…

musculoskeletal diseasesAdultMalemedicine.medical_specialtyendocrine system diseasesAdolescentEndocrinology Diabetes and MetabolismGraves' diseaseClinical BiochemistryThyroid GlandAutoimmunityImmunogeneticsHashimoto Diseasemedicine.disease_causeBiochemistryThyroiditisAutoimmunityYoung AdultEndocrinologyInternal medicineGenetic predispositionMedicineHumansHashimoto DiseaseGenetic Predisposition to DiseaseAlleleskin and connective tissue diseasesChildbusiness.industryBiochemistry (medical)ThyroidHistocompatibility Antigens Class IIInfantGeneral MedicineMiddle Agedmedicine.diseaseGraves DiseaseEndocrinologymedicine.anatomical_structureChild PreschoolImmunologyFemalebusinessHormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme
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H2-M polymorphism in mice susceptible to collagen-induced arthritis involves the peptide binding groove.

1996

The ability to develop type II collagen (CII)-induced arthritis (CIA) in mice is associated with the major histocompatibilityI-A gene and with as yet poorly defined regulatory molecules of the major histocompatibility complex (MHC) class II antigen processing and presentation pathway. H2-M molecules are thought to be involved in the loading of antigenic peptides into the MHC class II binding cleft. We sequencedH2-Ma, H2-Mb1, andH2-Mb2 genes from CIA-susceptible and-resistant mouse strains and identified four differentMa andMb2 alleles and three differentMb1 alleles defined by polymorphic residues within the predicted peptide binding groove. Most CIA-resistant mouse strains share commonMa, M…

musculoskeletal diseasesImmunologyGenes MHC Class IIMolecular Sequence DataGenes MHC Class IPeptide bindingMice Inbred StrainsMajor histocompatibility complexEpitopeMiceAntigenMHC class IGeneticsAnimalsAmino Acid SequencePhylogenyDNA PrimersMHC class IIPolymorphism GeneticbiologyBase SequenceSequence Homology Amino AcidAntigen processingH-2 AntigensHistocompatibility Antigens Class IIMolecular biologyArthritis ExperimentalHistocompatibilityHaplotypesbiology.proteinCollagenSequence AlignmentImmunogenetics
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Non-conventional forms of HLA-B27 are expressed in spondyloarthritis joints and gut tissue

2016

Objectives Human leukocyte antigen (HLA)-B27 (B27) is the strongest genetic factor associated with development of Ankylosing Spondylitis and other spondyloarthropathies (SpA), yet the role it plays in disease pathogenesis remains unclear. We investigated the expression of potentially pathogenic non-conventional heavy chain forms (NC) of B27 in synovial and intestinal tissues obtained from SpA patients. We also determined the presence of NC-B27 in joints, lymphoid and gastrointestinal tissue from B27 transgenic (TG1) rats with M.tuberculosis-induced SpA. Methods Expression of NC-B27 in human SpA joints and gut and in (21-3 × 283-2)F1 HLA-B27/Huβ2m rat tissue was determined by immunohistochem…

musculoskeletal diseasesalpha-DefensinsHLA-B27 transgenic rat modelGastrointestinal DiseasesCD8 AntigensImmunologyGene ExpressionArticleSpondyloarthropathieAnimalsHumansHLA class I free-heavy chainImmunology and AllergySpondylitis AnkylosingSpondyloarthropathiesskin and connective tissue diseasesHLA-B27 AntigenHLA-B27CD11 AntigensHistocompatibility Antigens Class ISynovial MembraneReceptors KIR3DL2Arthritis ExperimentalR1HLA class I free-heavy chainsRatsDisease Models AnimalSettore MED/16 - ReumatologiaHLA class I free-heavy chains; HLA-B27; HLA-B27 transgenic rat model; Spondyloarthropathies; Immunology and Allergy; ImmunologyBone RemodelingRats Transgenic
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