Search results for "Histocompatibility"

showing 10 items of 473 documents

Human CD8+ memory and EBV-specific T cells show low alloreactivity in vitro and in CD34+ stem cell–engrafted NOD/SCID/IL-2Rγcnull mice

2013

Current strategies in cellular immunotherapy of cancer and viral infections include the adoptive transfer of T cell receptor (TCR) and chimeric antigen receptor engineered T cells. When using transient RNA expression systems in clinical studies, multiple infusions with receptor-redirected T cells appear necessary. However, in allogeneic hematopoietic stem-cell transplantation, repeated transfer of donor-derived T cells increases the risk of alloreactive graft-versus-host disease. We investigated naive-derived (T N ), memory-derived (T M ), and Epstein Barr virus-specific (T EBV ) CD8 + T cell subsets for alloreactivity upon redirection with RNA encoding a cytomegalovirus-specific model TCR.…

Herpesvirus 4 HumanCancer ResearchT-LymphocytesT cellAntigens CD34Mice SCIDStreptamerCD8-Positive T-LymphocytesBiologyImmunotherapy AdoptiveMiceInterleukin 21GeneticsmedicineAnimalsHumansTransplantation HomologousCytotoxic T cellIL-2 receptorAntigen-presenting cellMolecular BiologyInterleukin 3Histocompatibility TestingHematopoietic Stem Cell TransplantationCell BiologyHematologyNatural killer T cellmedicine.anatomical_structureImmunologyCancer researchImmunologic MemoryInterleukin Receptor Common gamma SubunitExperimental Hematology
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Analysis of the (H-2b x H-2k)F1Restricted Response to Insulin.

1984

The aim of these studies was to characterize the (H-2b X H-2k)F1-unique restriction element(s) responsible for presentation of bovine insulin (BI) to a long-term cultured T-cell line (BK-BI-1.2). (B10.BR X bm12)F1 spleen cells, which express a normal Ab alpha Ak beta molecule but a mutated Ak alpha Abm12 beta product on their cell surface, were perfectly able to act as BI-presenting cells. Antibody inhibition experiments with antibodies directed at I-Ak products revealed that monoclonal antibody 10-2.16, which reacts with the Ak beta polypeptide chain, abrogated BI-directed T-cell proliferation, whereas antibody H116-32.R5 with specificity for the Ak alpha chain was not inhibitory. These re…

Heterozygotemedicine.drug_classT-LymphocytesImmunologyGlutamic AcidAlpha (ethology)Context (language use)BiologyLymphocyte ActivationMonoclonal antibodyEpitopeEpitopesMiceGlutamatesmedicineAnimalsInsulinBeta (finance)Histocompatibility Antigens Class IIAntibodies MonoclonalGeneral MedicineGlutamic acidBiochemistrybiology.proteinAntibodyAlpha chainScandinavian Journal of Immunology
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Isolation and characterization of Oct-4+/HLA-G+ mesenchymal stem cells from human umbilical cord matrix: differentiation potential and detection of n…

2008

The presence of multipotent cells in several adult and embryo-related tissues opened new paths for their use in regenerative medicine. Extraembryonic tissues such as umbilical cord are considered a promising source of stem cells, potentially useful in therapy. The characterization of cells from the umbilical cord matrix (Wharton''s Jelly) and amniotic membrane revealed the presence of a population of mesenchymal-like cells, sharing a set of core-markers expressed by "mesenchymal stem cells". Several reports enlightened the differentiation capabilities of these cells, even if at times the lack of an extensive characterization of surface markers and immune co-stimulators expression revealed h…

HistologyCell Culture TechniquesClinical uses of mesenchymal stem cellsCell SeparationBiologyUmbilical CordHLA AntigensHumansAmnionMolecular BiologyCell ProliferationStem cell transplantation for articular cartilage repairHLA-G AntigensSettore BIO/16 - Anatomia UmanaMultipotent Stem CellsHistocompatibility Antigens Class IMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAmniotic stem cellsCell BiologyTelomereCord liningCell biologyMedical Laboratory TechnologyMesenchymal stem cells Umbilical cord matrix Differentiation protocols Tolerogenic properties Self-renewal markersAmniotic epithelial cellsImmunologyStem cellOctamer Transcription Factor-3BiomarkersAdult stem cellHistochemistry and Cell Biology
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The role of the human cytomegalovirus UL111A gene in down-regulating CD4+ T-cell recognition of latently infected cells: implications for virus elimi…

2009

AbstractThe capacity of human cytomegalovirus (HCMV) to establish and maintain a latent infection from which it can later reactivate ensures its widespread distribution in the population, but the mechanisms enabling maintenance of latency in the face of a robust immune system are poorly understood. We examined the role of the HCMV UL111A gene, which encodes homologs of the immunosuppressive cytokine interleukin-10 in the context of latent infection of myeloid progenitor cells. A UL111A deletion virus was able to establish, maintain, and reactivate from experimental latency in a manner comparable with parental virus, but major histocompatibility complex class II levels increased significantl…

Human cytomegalovirusCD4-Positive T-LymphocytesIsoantigensMyeloidGenes Viralmedicine.medical_treatmentImmunologyPopulationCytomegalovirusDown-RegulationBiologyIn Vitro Techniquesmedicine.disease_causeBiochemistryAutoantigensHerpesviridaeVirusImmune systemmedicineHumansProgenitor celleducationMyeloid Progenitor Cellseducation.field_of_studyHistocompatibility Antigens Class IICell BiologyHematologymedicine.diseaseVirologyVirus LatencyCytokinemedicine.anatomical_structureImmunologyCytomegalovirus InfectionsHost-Pathogen InteractionsGene DeletionBlood
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Human cytomegalovirus pp71 stimulates major histocompatibility complex class i presentation of IE1-derived peptides at immediate early times of infec…

2013

ABSTRACT Suppression of major histocompatibility complex (MHC) class I-mediated presentation of human cytomegalovirus (HCMV) peptides is an important mechanism to avoid CD8 T lymphocyte recognition and killing of infected cells. Of particular interest is how MHC class I presentation of essential regulatory immediate early (IE) proteins of HCMV can be effectively compromised at times when known viral immunoevasins are not abundantly expressed. The tegument protein pp71 had been suggested to be involved in MHC class I downregulation. Intriguingly, this polypeptide is also critically engaged in the initial derepression of the major IE gene locus, leading to enhanced expression of IE proteins I…

Human cytomegalovirusCD74virusesImmunologyCytomegalovirusBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsViral ProteinsDownregulation and upregulationVirologyMHC class ImedicineHumansDerepressionAntigen PresentationAntigen processingMHC class I antigenHistocompatibility Antigens Class Ivirus diseasesbiochemical phenomena metabolism and nutritionmedicine.diseaseUp-RegulationInsect ScienceImmunologyCytomegalovirus Infectionsbiology.proteinPathogenesis and ImmunityPeptidesJournal of virology
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The immunogenicity of human and murine cytomegaloviruses.

2000

Cytomegaloviruses are strictly host-species-specific. During an aeon of co-evolution, virus and host have found an arrangement: the productive and cytopathogenic cycle of viral gene expression is held in check by the host's immune response. As a consequence, cytomegalovirus disease is restricted to the immunocompromised host. The virus has evolved strategies to avoid its elimination and eventually hides itself in a silent state, referred to as 'viral latency'. Redundant molecular mechanisms have been identified by which cytomegaloviruses interfere with antigen presentation pathways to 'evade' immune control. In the annual period covered by this review, the IE1 protein was revisited as an im…

Human cytomegalovirusMuromegalovirusvirusesImmunologyAntigen presentationCongenital cytomegalovirus infectionCytomegalovirusImmunodominanceBiologyVirusImmediate early proteinImmediate-Early ProteinsViral Matrix ProteinsMiceViral ProteinsAntigenmedicineImmunology and AllergyAnimalsHumansAntigen PresentationImmunogenicityHistocompatibility Antigens Class IIvirus diseasesReceptors Antigen T-Cell gamma-deltamedicine.diseasePhosphoproteinsVirologyKiller Cells NaturalImmunologyCurrent opinion in immunology
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Inhibition of CD1 antigen presentation by human cytomegalovirus.

2008

ABSTRACTThe betaherpesvirus human cytomegalovirus (HCMV) encodes several molecules that block antigen presentation by the major histocompatibility complex (MHC) proteins. Humans also possess one other family of antigen-presenting molecules, the CD1 family; however, the effect of HCMV on CD1 expression is unknown. The majority of CD1 molecules are classified on the basis of homology as group 1 CD1 and are present almost exclusively on professional antigen-presenting cells such as dendritic cells, which are a major target for HCMV infection and latency. We have determined that HCMV encodes multiple blocking strategies targeting group 1 CD1 molecules. CD1 transcription is strongly inhibited by…

Human cytomegalovirusTranscription GeneticvirusesImmunologyAntigen presentationCD1Cytomegaloviruschemical and pharmacologic phenomenaMajor histocompatibility complexmedicine.disease_causeMicrobiologycomplex mixturesCell LineAntigens CD1Immune systemAntigenVirologyMHC class ImedicineHumansCells CulturedAntigen PresentationbiologyImmunityhemic and immune systemsmedicine.diseaseVirologyProtein TransportHerpes simplex virusGene Expression RegulationInsect Sciencebiology.proteinPathogenesis and Immunitylipids (amino acids peptides and proteins)Journal of virology
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Suppression of CD8+ T cell recognition in the immediate-early phase of human cytomegalovirus infection.

2012

Human cytomegalovirus (HCMV) interferes with MHC class I-restricted antigen presentation and thereby reduces recognition by CD8+ T-cells. This interference is mediated primarily by endoplasmic reticulum-resident glycoproteins that are encoded in the US2–11 region of the viral genome. Such a suppression of recognition would be of particular importance immediately after infection, because several immunodominant viral antigens are already present in the cell in this phase. However, which of the evasion proteins gpUS2–11 interfere(s) with antigen presentation to CD8+ T-cells at this time of infection is not known. Here we address this question, using recombinant viruses (RV) that express only o…

Human cytomegalovirusVirulence FactorsvirusesAntigen presentationCytomegalovirusCD8-Positive T-LymphocytesCell LineImmune toleranceViral ProteinsViral Envelope ProteinsAntigenVirologyMHC class IImmune TolerancemedicineHumansCytotoxic T cellImmune EvasionbiologyHistocompatibility Antigens Class IRNA-Binding Proteinsvirus diseasesmedicine.diseaseVirologyCell cultureCytomegalovirus InfectionsImmunologybiology.proteinCD8
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Immune evasion proteins gpUS2 and gpUS11 of human cytomegalovirus incompletely protect infected cells from CD8 T cell recognition

2009

AbstractHuman cytomegalovirus (HCMV) encodes four glycoproteins, termed gpUS2, gpUS3, gpUS6 and gpUS11 that interfere with MHC class I biosynthesis and antigen presentation. Despite gpUS2–11 expression, however, HCMV infection is efficiently controlled by cytolytic CD8 T lymphocytes (CTL). To address the role of gpUS2 and gpUS11 in antigen presentation during viral infection, HCMV mutants were generated that expressed either gpUS2 or gpUS11 alone without coexpression of the three other proteins. Fibroblasts infected with these viruses showed reduced HLA-A2 and HLA-B7 surface expression. Surprisingly, however, CTL directed against the tegument protein pp65 and the regulatory IE1 protein stil…

Human cytomegalovirusvirusesAntigen presentationIE1CytomegalovirusCD8-Positive T-LymphocytesVirus ReplicationMajor histocompatibility complexpp65US2Immediate-Early ProteinsViral Matrix ProteinsHLA-B7 AntigenInterferon-gammaViral ProteinsImmune systemViral Envelope ProteinsVirologyHLA-A2 AntigenMHC class ImedicineHumansCytotoxic T cellCells CulturedAntigen PresentationbiologyImmune evasionRNA-Binding Proteinsvirus diseasesbiochemical phenomena metabolism and nutritionPhosphoproteinsmedicine.diseaseVirologyCTL*MutagenesisCTLCytomegalovirus InfectionsMHC class Ibiology.proteinUS11CD8Virology
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Identification of transcripts of the T cell antigen receptor beta chain gene and major histocompatibility complex class II genes in antigen-presentin…

1988

The cloned murine cell line BK-BI-2.6.C6 has previously been shown to exhibit T cell characteristics, to synthesize and express MHC class II molecules, and to present protein antigens to antigen-dependent T cell clones. As a more definitive proof of the T-cell nature of these cells, transcripts of the rearranged T cell antigen receptor (TcR) beta gene were assessed by Northern blot analysis. BK-BI-2.6.C6 cells constitutively transcribe mRNA for the light chain of TcR and express the disulphide-linked alpha, beta TcR heterodimer at the cell surface. In addition mRNA for the polymorphic MHC class II subunits A alpha and A beta as well as for the invariant gamma chain were detected. BK-BI-2.6.…

HybridomasT cellReceptors Antigen T-Cell alpha-betaT-LymphocytesImmunologyT-cell receptorAntigen presentationHistocompatibility Antigens Class IIReceptors Antigen T-CellAntigen-Presenting CellsGeneral MedicineMHC restrictionBiologyMajor histocompatibility complexMolecular biologyCell LineMicemedicine.anatomical_structurebiology.proteinmedicineCytotoxic T cellAnimalsRNA MessengerAntigen-presenting cellCD8Scandinavian journal of immunology
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