Search results for "Histones"

showing 10 items of 200 documents

Differential functions of calpain 1 during epithelial cell death and adipocyte differentiation in mammary gland involution

2014

Calpains become activated in the mammary gland early during weaning, cleaving several proteins located mainly in the cell membrane, but also in other organelles such as lysosomes, mitochondria and nuclei. By immunofluorescence and Western blot analysis, we have demonstrated the nuclear translocation of calpain-1 and calpain-2, together with the cleavage of several cytoplasmic nucleoporins in epithelial cells of the lobulo-alveolar compartment. In vivo and in vitro calpain inhibition prevented this nucleoporin degradation. In addition, calpain-1 was also present in the nucleus of non-epithelial mammary tissue cells, concomitant with adipocyte re-differentiation. Calpain-1 was internalized wi…

MaleCellular differentiationBiochemistryHistonesMicechemistry.chemical_compoundHistone H3Mammary Glands AnimalAdipocyteAdipocytesAnimalsLactationMolecular BiologyMammary gland involutionbiologyCalpainCell DifferentiationEpithelial CellsCalpainCell BiologyMolecular biologyNuclear Pore Complex ProteinsProtein TransportHistoneGene Expression Regulationchemistrybiology.proteinH3K4me3FemaleNucleoporinBiochemical Journal
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H3 and H3.3 histone mRNA amounts and ratio in oral squamous cell carcinoma and leukoplakia.

2006

Histone variants (e.g. H3) play an important role in chromatin structure and gene expression regulation of normal cells. Aims of this study were to: (1) estimate H3 and H3.3 histone mRNA expressions and their ratio in oral squamous cell carcinoma (OSCC) and oral leukoplakia (OL); (2) investigate whether H3 and H3.3 variants could play a role in the pathogenesis of OSCC and OL, also conditionally to HPV infection, age, gender, and main habits (tobacco smoking and alcohol drinking) in human beings studied. Twenty-three cases of OSCC and 20 cases of OL were examined in lesion site (LS) and juxtaposed clinically undamaged site (JUS) by RT-PCR for H3 and H3.3 histone mRNA; 13 healthy oral mucosa…

MaleHPVH3.3 histone mRNAAlcohol Drinking"carcinoma"BiologyH3 histone mRNAoral leukoplakiaPathogenesisHistonesleucoplakia"Sex FactorsRisk FactorsmedicineCarcinomaHumansRNA MessengerOral mucosaGeneral DentistryPapillomaviridaeLeukoplakiaAgedRegulation of gene expressionMessenger RNAPapillomavirus InfectionsSmoking"H3 histones"HPV infectionAge FactorsMouth MucosaMiddle Agedmedicine.diseaseMolecular biologyoral squamous cell carcinomastomatognathic diseasesmedicine.anatomical_structureHistoneOtorhinolaryngologyDNA Viralbiology.proteinCarcinoma Squamous CellFemaleMouth NeoplasmsLeukoplakia OralOral diseases
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Global and gene-specific histone modification profiles of mouse multipotent adult germline stem cells

2010

We previously reported the generation of multipotent adult germline stem cells (maGSCs) from spermatogonial stem cells (SSCs) isolated from adult mouse testis. In a later study, we substantiated the pluripotency of maGSCs by demonstrating their close similarity to pluripotent male embryonic stem cells (ESCs) at the epigenetic level of global and gene-specific DNA methylation. Here, we extended the comparative epigenetic analysis of maGSCs and male ESCs by investigating the second main epigenetic modification in mammals, i.e. global and gene-specific modifications of histones (H3K4 trimethylation, H3K9 acetylation, H3K9 trimethylation and H3K27 trimethylation). Using immunofluorescence stain…

MaleHomeobox protein NANOGChromatin ImmunoprecipitationEmbryologyAdult Germline Stem CellsBlotting WesternFluorescent Antibody TechniqueBiologyMethylationPolymerase Chain ReactionCell LineEpigenesis GeneticHistonesMice03 medical and health sciences0302 clinical medicineSOX2GeneticsAnimalsEpigenetics10. No inequalityMolecular Biology030304 developmental biologyHomeodomain Proteins0303 health sciencesGenomeMultipotent Stem CellsSOXB1 Transcription FactorsObstetrics and GynecologyAcetylationNanog Homeobox ProteinCell BiologyFlow CytometryMolecular biologySpermatogoniaChromatinReproductive Medicineembryonic structuresH3K4me3Octamer Transcription Factor-3Chromatin immunoprecipitation030217 neurology & neurosurgeryDevelopmental BiologyBivalent chromatinMolecular Human Reproduction
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FM19G11, a New Hypoxia-inducible Factor (HIF) Modulator, Affects Stem Cell Differentiation Status

2009

The biology of the alpha subunits of hypoxia-inducible factors (HIF alpha) has expanded from their role in angiogenesis to their current position in the self-renewal and differentiation of stem cells. The results reported in this article show the discovery of FM19G11, a novel chemical entity that inhibits HIF alpha proteins that repress target genes of the two alpha subunits, in various tumor cell lines as well as in adult and embryonic stem cell models from rodents and humans, respectively. FM19G11 inhibits at nanomolar range the transcriptional and protein expression of Oct4, Sox2, Nanog, and Tgf-alpha undifferentiating factors, in adult rat and human embryonic stem cells, FM19G11 activit…

MaleHomeobox protein NANOGTranscription GeneticCellular differentiationBiologyResponse ElementsBenzoatesBiochemistryHistonesRats Sprague-DawleyMolecular Basis of Cell and Developmental BiologySOX2EpendymaBasic Helix-Loop-Helix Transcription FactorsAnimalsHumansp300-CBP Transcription FactorsMolecular BiologyEmbryonic Stem CellsHomeodomain ProteinsRegulation of gene expressionSOXB1 Transcription FactorsAcetylationCell DifferentiationNanog Homeobox ProteinCell BiologyTransforming Growth Factor alphaHypoxia-Inducible Factor 1 alpha SubunitMolecular biologyEmbryonic stem cellCell HypoxiaRatsCell biologyAdult Stem CellsGene Expression RegulationPharmaceutical PreparationsBenzamidesStem cellOctamer Transcription Factor-3Chromatin immunoprecipitationHeLa CellsAdult stem cellJournal of Biological Chemistry
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Androgen receptor activation by polychlorinated biphenyls

2013

The exposure to environmental endocrine disrupting compounds (EDC), as polychlorinated biphenyls (PCBs), widely diffused in the environment may produce epigenetic changes that affect the endocrine system. We found that PCBs activate AR transcriptional activity and that this effect is potentiated by the demethylase Jarid1b, a histone demethylase that catalyzes the removal of trimethylation of lysine 4 on histone H3 (H3K4me3), induced by PCB. The aim of the present study was to investigate the effect of the treatment of cultured cells (HEK293) with a mixture of the most diffused environmental PCBs and, also with dihydrotestosterone (DHT), on the functional interaction between AR and Jarid1b. …

MaleJumonji Domain-Containing Histone DemethylasesJarid1bpolychlorinated biphenylsNuclear ProteinsEndocrine DisruptorsEpigenesis GeneticHistonesRepressor ProteinsReceptors Androgenandrogen receptorTumor Cells CulturedHumanshistone modificationFemaleenvironmentepigeneticResearch PaperEpigenetics
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Molecular mechanisms of NET formation and degradation revealed by intravital imaging in the liver vasculature

2015

AbstractNeutrophil extracellular traps (NETs) composed of DNA decorated with histones and proteases trap and kill bacteria but also injure host tissue. Here we show that during a bloodstream infection with methicillin-resistant Staphylococcus aureus, the majority of bacteria are sequestered immediately by hepatic Kupffer cells, resulting in transient increases in liver enzymes, focal ischaemic areas and a robust neutrophil infiltration into the liver. The neutrophils release NETs into the liver vasculature, which remain anchored to the vascular wall via von Willebrand factor and reveal significant neutrophil elastase (NE) proteolytic activity. Importantly, DNase although very effective at D…

MaleMethicillin-Resistant Staphylococcus aureusExtracellular TrapsProteasesHydrolasesKupffer CellsGeneral Physics and AstronomyBacteremiaBiologyHepatic Veinsmedicine.disease_causeExtracellular TrapsGeneral Biochemistry Genetics and Molecular BiologyHistonesMiceHepatic ArteryVon Willebrand factorProtein-Arginine Deiminase Type 4von Willebrand FactormedicineAnimalsMice KnockoutMultidisciplinaryDeoxyribonucleasesGeneral ChemistryNeutrophil extracellular trapsStaphylococcal Infectionsmedicine.disease3. Good healthCell biologyHistoneLiverNeutrophil InfiltrationStaphylococcus aureusNeutrophil elastaseImmunologybiology.proteinLeukocyte ElastaseInfiltration (medical)
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Phase ia/ii, two-arm, open-label, dose-escalation study of oral panobinostat administered via two dosing schedules in patients with advanced hematolo…

2013

Panobinostat is a potent oral pandeacetylase inhibitor that leads to acetylation of intracellular proteins, inhibits cellular proliferation and induces apoptosis in leukemic cell lines. A phase Ia/II study was designed to determine the maximum-tolerated dose (MTD) of daily panobinostat, administered on two schedules: three times a week every week or every other week on a 28-day treatment cycle in patients with advanced hematologic malignancies. The criteria for hematologic dose-limiting toxicities differed between patients with indications associated with severe cytopenias at baseline (leukemia and myeloid disorders) and those less commonly associated with baseline cytopenias (lymphoma and …

MaleOncologyCancer ResearchIndolesMyeloidhodgkin lymphomahydroxamic acidAdministration Oralresponse criteriaPharmacologyHydroxamic Acidst-cell lymphomaHistoneschemistry.chemical_compoundhemic and lymphatic diseasesAged 80 and overHematologyMiddle AgedLeukemiaTreatment Outcomemedicine.anatomical_structuremyelomaOncologyvorinostatHematologic NeoplasmsFemaleAdultmedicine.medical_specialtypanobinostatrefractory multiple-myelomaMaximum Tolerated DoseAntineoplastic AgentsmyelofibrosisNeutropeniahistone deacetylase inhibitorsmyelodysplastic disordersDrug Administration ScheduleYoung AdultInternal medicinePanobinostatmedicineHumansIn patientAdverse effectMyelofibrosisAgedNeoplasm Staginginternational-working-groupacetylationbusiness.industrymedicine.diseaseLymphomachemistryhistone deacetylasehypoxia-inducible factor-1-alphalbh589business
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Immunopositivity for histone macroH2A1 isoforms marks steatosisassociated hepatocellular carcinoma.

2012

BackgroundHepatocellular carcinoma (HCC) is one of the most common cancers worldwide. Prevention and risk reduction are important and the identification of specific biomarkers for early diagnosis of HCC represents an active field of research. Increasing evidence indicates that fat accumulation in the liver, defined as hepatosteatosis, is an independent and strong risk factor for developing an HCC. MacroH2A1, a histone protein generally associated with the repressed regions of chromosomes, is involved in hepatic lipid metabolism and is present in two alternative spliced isoforms, macroH2A1.1 and macroH2A1.2. These isoforms have been shown to predict lung and colon cancer recurrence but to ou…

MalePathologyMouseBiological Markers/metabolismEpidemiologyTumor Microenvironment/geneticsColorectal cancerGene ExpressionHepatocytes/metabolism/pathologyNonalcoholic SteatohepatitisHistonesFatty Liver/chemically induced/complications/genetics/metabolismMice0302 clinical medicineGastrointestinal CancersTumor MicroenvironmentPathologyProtein IsoformsDiethylnitrosamineSettore MED/49 - Scienze Tecniche Dietetiche ApplicateMice KnockoutRegulation of gene expression0303 health sciencesMultidisciplinaryProtein Isoforms/genetics/metabolismbiologyLiver DiseasesPTEN Phosphohydrolase/deficiency/geneticshepatocellular carcinoma biomarker histone variant steatosis epigeneticsLiver NeoplasmsQFatty liverRHistone ModificationAnimal ModelsImmunohistochemistry3. Good healthHistoneOncology030220 oncology & carcinogenesisHepatocellular carcinomaMedicineEpigeneticsCarcinoma Hepatocellular/etiology/genetics/metabolism/pathologyResearch ArticleGene isoformmedicine.medical_specialtyCarcinoma HepatocellularHistologyClinical Research DesignScienceGastroenterology and HepatologyDiet High-Fat03 medical and health sciencesModel OrganismsDiagnostic MedicineGastrointestinal TumorsGeneticsCancer GeneticsCancer Detection and DiagnosisEarly DetectionmedicineAnimalsHumansAnimal Models of DiseaseObesityddc:612BiologyHistones/genetics/metabolismNutrition030304 developmental biologyCell NucleusCell Nucleus/genetics/metabolism/pathologyTumor microenvironmentbusiness.industryPTEN PhosphohydrolaseCancers and NeoplasmsHepatocellular Carcinomamedicine.diseasedigestive system diseasesFatty LiverBiomarker EpidemiologyGene Expression RegulationHepatocytesbiology.proteinLiver Neoplasms/etiology/genetics/metabolism/pathologySteatosisbusinessBiomarkersGeneral Pathology
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Comparative Analysis of Chromatin-Delivered Biomarkers in the Monitoring of Sepsis and Septic Shock: A Pilot Study

2021

Sepsis management remains one of the most important challenges in modern clinical practice. Rapid progression from sepsis to septic shock is practically unpredictable, hence the critical need for sepsis biomarkers that can help clinicians in the management of patients to reduce the probability of a fatal outcome. Circulating nucleoproteins released during the inflammatory response to infection, including neutrophil extracellular traps, nucleosomes, and histones, and nuclear proteins like HMGB1, have been proposed as markers of disease progression since they are related to inflammation, oxidative stress, endothelial damage, and impairment of the coagulation response, among other pathological…

MalePilot Projectslaw.inventionCohort StudiesHistonessepsisMicelawHMGB1 ProteinBiology (General)SpectroscopyImmunoassayHMGB1biologyCommunicationAntibodies MonoclonalGeneral MedicineMiddle AgedIntensive care unitShock SepticChromatinComputer Science ApplicationsChemistryCohortFemaleELISAmedicine.symptomcirculating histonesmedicine.medical_specialtyQH301-705.5InflammationHMGB1CatalysisInorganic ChemistrySepsismedicineAnimalsHumansPhysical and Theoretical ChemistryIntensive care medicineMolecular BiologyPathologicalQD1-999Septic shockbusiness.industryOrganic ChemistrybiomarkersNeutrophil extracellular trapsmedicine.diseaseNucleoproteinsnucleosomesbiology.proteinCitrullineseptic shockbusinessInternational Journal of Molecular Sciences
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Genetic identification of a network of factors that functionally interact with the nucleosome remodeling ATPase ISWI.

2008

Nucleosome remodeling and covalent modifications of histones play fundamental roles in chromatin structure and function. However, much remains to be learned about how the action of ATP-dependent chromatin remodeling factors and histone-modifying enzymes is coordinated to modulate chromatin organization and transcription. The evolutionarily conserved ATP-dependent chromatin-remodeling factor ISWI plays essential roles in chromosome organization, DNA replication, and transcription regulation. To gain insight into regulation and mechanism of action of ISWI, we conducted an unbiased genetic screen to identify factors with which it interacts in vivo. We found that ISWI interacts with a network o…

MaleProteomicsCancer Researchlcsh:QH426-470Histone Deacetylase 1BiologySettore MED/08 - Anatomia PatologicaChromosomesHistone DeacetylasesChromatin remodelingHistonesHistone H403 medical and health sciences0302 clinical medicineGenetics and Genomics/EpigeneticsGeneticsAnimalsDrosophila ProteinsNucleosomeMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and Systematics030304 developmental biologyAdenosine TriphosphatasesGenetics0303 health sciencesNuclear ProteinsAcetylationChromatin Assembly and DisassemblyChromatinNucleosomesChromatiniswi drosophilaRepressor ProteinsChromatin epigeneticsHDAC Chromatin RemodellingSin3 Histone Deacetylase and Corepressor Complexlcsh:GeneticsDrosophila melanogasterHistoneHistone deacetylase complexbiology.proteinFemaleHistone deacetylaseHistone deacetylase activity030217 neurology & neurosurgeryResearch ArticleTranscription Factors
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