Search results for "Homeodomain"

showing 10 items of 137 documents

Early asymmetric cues triggering the dorsal/ventral gene regulatory network of the sea urchin embryo

2014

Dorsal/ventral (DV) patterning of the sea urchin embryo relies on a ventrally-localized organizer expressing Nodal, a pivotal regulator of the DV gene regulatory network. However, the inceptive mechanisms imposing the symmetry-breaking are incompletely understood. In Paracentrotus lividus, the Hbox12 homeodomain-containing repressor is expressed by prospective dorsal cells, spatially facing and preceding the onset of nodal transcription. We report that Hbox12 misexpression provokes DV abnormalities, attenuating nodal and nodal-dependent transcription. Reciprocally, impairing hbox12 function disrupts DV polarity by allowing ectopic expression of nodal. Clonal loss-of-function, inflicted by b…

Embryo NonmammalianTranscription GeneticEctodermp38 Mitogen-Activated Protein Kinasessymmetry breakingdorsal ventral axis sea urchin embryo nodal homeodomain repressor p38 MAPKAnimals Genetically ModifiedCell polarityMorphogenesisGene Regulatory NetworksBiology (General)ZebrafishSea urchinsea urchin embryoGeneticsbiologyGeneral NeuroscienceQRdorsal/ventral polarityCell PolarityGene Expression Regulation DevelopmentalEmbryoGeneral MedicineCell biologymedicine.anatomical_structureGene Knockdown Techniquesembryonic structuresParacentrotusMedicineCuesResearch Articleanimal structuresQH301-705.5Nodal ProteinScienceEmbryonic DevelopmentSettore BIO/11 - Biologia Molecolarep38 MAPKModels BiologicalGeneral Biochemistry Genetics and Molecular Biologybiology.animalEctodermmedicineAnimalsBody PatterningHomeodomain ProteinsGeneral Immunology and MicrobiologyotherCell Biologybiology.organism_classificationEmbryonic stem cellhomeodomain repressorRepressor ProteinsDevelopmental Biology and Stem CellsnodalNODALDevelopmental biologyeLife
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Inhibitory activities of short linear motifs underlie Hox interactome specificity in vivo

2015

Hox proteins are well-established developmental regulators that coordinate cell fate and morphogenesis throughout embryogenesis. In contrast, our knowledge of their specific molecular modes of action is limited to the interaction with few cofactors. Here, we show that Hox proteins are able to interact with a wide range of transcription factors in the live Drosophila embryo. In this context, specificity relies on a versatile usage of conserved short linear motifs (SLiMs), which, surprisingly, often restrains the interaction potential of Hox proteins. This novel buffering activity of SLiMs was observed in different tissues and found in Hox proteins from cnidarian to mouse species. Although th…

Embryo Nonmammalian[SDV]Life Sciences [q-bio]Amino Acid MotifsinteractomeInteractomeBimolecular fluorescence complementationMiceTARGET GENEDrosophila ProteinsCELL REGULATIONProtein Interaction MapsBiology (General)Hox genetranscription factorGeneticsD. melanogasterGeneral NeuroscienceQRINTERACTION MODULESGeneral MedicineREGIONSHoxTRANSCRIPTION FACTORSDrosophila melanogasterGenomics and Evolutionary BiologyOrgan Specificityembryonic structuresMedicineOligopeptidesProtein BindingResearch Articleanimal structuresQH301-705.5ScienceembryoContext (language use)Computational biology[SDV.BC]Life Sciences [q-bio]/Cellular BiologyCell fate determinationBiologyBinding CompetitiveGeneral Biochemistry Genetics and Molecular BiologyFluorescenceProtein–protein interactionEvolution MolecularStructure-Activity Relationship[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyAnimalsShort linear motif[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyBiFCTranscription factor[SDV.BC] Life Sciences [q-bio]/Cellular BiologydevelopmentHomeodomain ProteinsABDOMINAL-AGeneral Immunology and MicrobiologyBIMOLECULAR FLUORESCENCE COMPLEMENTATIONREPRESSIONDNAPROTEIN INTERACTIONSIntrinsically Disordered ProteinsDROSOPHILA-MELANOGASTERMutationeLife
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The RNA-binding protein ELAV regulates Hox RNA processing, expression and function within the Drosophila nervous system

2014

The regulated head-to-tail expression of Hox genes provides a coordinate system for the activation of specific programmes of cell differentiation according to axial level. Recent work indicates that Hox expression can be regulated via RNA processing but the underlying mechanisms and biological significance of this form of regulation remain poorly understood. Here we explore these issues within the developing Drosophila central nervous system (CNS). We show that the pan-neural RNA-binding protein (RBP) ELAV (Hu antigen) regulates the RNA processing patterns of the Hox gene Ultrabithorax (Ubx) within the embryonic CNS. Using a combination of biochemical, genetic and imaging approaches we demo…

Embryo Nonmammaliananimal structuresNeurogenesisRNA-binding proteinCellular differentiationMolecular Sequence DataRNA-binding proteinBiologyAntennapediaNervous SystemMorphogenesisAnimalsDrosophila ProteinsRNA Processing Post-TranscriptionalELAV/HuHox geneMolecular BiologyTranscription factorPhylogenyResearch ArticlesUltrabithoraxHomeodomain ProteinsAlternative polyadenylation (APA)GeneticsBase SequenceAlternative splicingGenes HomeoboxGene Expression Regulation DevelopmentalSegment-specific apoptosisHoxCell biologyDrosophila melanogasterELAV ProteinsRNA processingCentral nervous systemembryonic structuresDrosophilaDrosophila ProteinTranscription FactorsAlternative splicingDevelopmental BiologyDevelopment
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Human exome and mouse embryonic expression data implicate ZFHX3, TRPS1, and CHD7 in human esophageal atresia

2020

Introduction Esophageal atresia with or without tracheoesophageal fistula (EA/TEF) occurs approximately 1 in 3.500 live births representing the most common malformation of the upper digestive tract. Only half a century ago, EA/TEF was fatal among affected newborns suggesting that the steady birth prevalence might in parts be due to mutational de novo events in genes involved in foregut development. Methods To identify mutational de novo events in EA/TEF patients, we surveyed the exome of 30 case-parent trios. Identified and confirmed de novo variants were prioritized using in silico prediction tools. To investigate the embryonic role of genes harboring prioritized de novo variants we perfor…

EmbryologyCandidate geneGene ExpressionTranscriptomeMiceDatabase and Informatics MethodsMedicine and Health SciencesExomeExomeExome sequencingGenetics0303 health sciencesMultidisciplinaryComputer-Aided Drug DesignQ030305 genetics & hereditySequence analysisRGenomicsCongenital AnomaliesDNA-Binding Proteinsembryonic structuresAmino Acid AnalysisMedicineTranscriptome AnalysisTracheoesophageal FistulaResearch ArticleDrug Research and DevelopmentBioinformaticsSequence analysisScienceIn silicoBiologyResearch and Analysis Methods03 medical and health sciencesExome SequencingGeneticsCongenital DisordersAnimalsHumansddc:610Molecular Biology TechniquesEsophageal AtresiaMolecular BiologyDNA sequence analysis030304 developmental biologyHomeodomain ProteinsPharmacologyMolecular Biology Assays and Analysis TechniquesGene Expression ProfilingEmbryosDNA HelicasesBiology and Life SciencesComputational BiologyEmbryo MammalianGenome AnalysisFANCBRepressor ProteinsGene expression profilingBiological DatabasesDrug DesignMutation DatabasesMutationDevelopmental Biology
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Highly restricted expression at the ectoderm–endoderm boundary of PIHbox 9, a sea urchin homeobox gene related to the human HB9 gene

1998

Abstract Characterisation of a sea urchin (P. lividus) homeobox gene PIHbox 9 is reported. The homeodomain of PIHbox9 is 95% identical to the homeodomain of the human HB9 gene, indicating that the two genes are highly related. Temporal expression analysis during sea urchin embryogenesis showed an absence of transcripts at early cleavage stages. At late gastrula stage, transcripts were barely detectable and reached the highest abundance at prism/early pluteus stages. By whole mount in situ hybridisation we observed a highly restricted expression in a few cells of the ectoderm–endoderm boundary of embryos at the prism stage. At pluteus stages, expression of PIHbox 9 was confined around the an…

EmbryologyEmbryo Nonmammaliananimal structuresEctodermParacentrotus lividusbiology.animalEctodermmedicineAnimalsPluteusSea urchinIn Situ HybridizationHomeodomain ProteinsGeneticsbiologyEndodermEmbryogenesisGene Expression Regulation DevelopmentalGastrulabiology.organism_classificationCell biologyGastrulationmedicine.anatomical_structureSea Urchinsembryonic structuresHomeoboxEndodermTranscription FactorsDevelopmental BiologyMechanisms of Development
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Lymphatic endothelial progenitors bud from the cardinal vein and intersomitic vessels in mammalian embryos.

2012

Abstract The lymphatic vasculature preserves tissue fluid balance by absorbing fluid and macromolecules and transporting them to the blood vessels for circulation. The stepwise process leading to the formation of the mammalian lymphatic vasculature starts by the expression of the gene Prox1 in a subpopulation of blood endothelial cells (BECs) on the cardinal vein (CV) at approximately E9.5. These Prox1-expressing lymphatic endothelial cells (LECs) will exit the CV to form lymph sacs, primitive structures from which the entire lymphatic network is derived. Until now, no conclusive information was available regarding the cellular processes by which these LEC progenitors exit the CV without co…

EndotheliumMesenchymegovernment.form_of_governmentRecombinant Fusion ProteinsImmunologyEmbryonic DevelopmentMice TransgenicBiologyBiochemistryMiceMicroscopy Electron TransmissionCell MovementVascular BiologymedicineAnimalsLymph sacsProgenitor cellEmbryonic Stem CellsHomeodomain ProteinsMice KnockoutBuddingMembrane GlycoproteinsCommon cardinal veinsTumor Suppressor ProteinsfungiCell BiologyHematologyAnatomyAdherens JunctionsCadherinsEmbryo MammalianCoronary VesselsCell biologyPlatelet Endothelial Cell Adhesion Molecule-1Lymphatic Endotheliummedicine.anatomical_structureLymphatic systemgovernmentsense organsEndothelium LymphaticBlood
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Arsenic trioxide alters the differentiation of mouse embryonic stem cell into cardiomyocytes

2015

AbstractChronic arsenic exposure is associated with increased morbidity and mortality for cardiovascular diseases. Arsenic increases myocardial infarction mortality in young adulthood, suggesting that exposure during foetal life correlates with cardiac alterations emerging later. Here, we investigated the mechanisms of arsenic trioxide (ATO) cardiomyocytes disruption during their differentiation from mouse embryonic stem cells. Throughout 15 days of differentiation in the presence of ATO (0.1, 0.5, 1.0 μM) we analysed: the expression of i) marker genes of mesoderm (day 4), myofibrillogenic commitment (day 7) and post-natal-like cardiomyocytes (day 15); ii) sarcomeric proteins and their orga…

Fetal ProteinsSarcomeresMesodermTime FactorsCellular differentiationBlotting WesternConnexinFluorescent Antibody TechniqueGene ExpressionAntineoplastic AgentsActininBiologyArticleArsenicalsCell Linechemistry.chemical_compoundMiceArsenic TrioxideTroponin TSpheroids CellularGene expressionmedicineAnimalsActininMyocytes CardiacArsenic trioxideHomeodomain ProteinsSyncytiumMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionCell DifferentiationMouse Embryonic Stem CellsOxidesEmbryonic stem cellCell biologyBiomechanical PhenomenaGATA4 Transcription Factormedicine.anatomical_structurechemistryConnexin 43ImmunologyHomeobox Protein Nkx-2.5T-Box Domain ProteinsTroponin CTranscription FactorsScientific Reports
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Nanog Regulates Primordial Germ Cell Migration Through Cxcr4b

2010

Abstract Gonadal development in vertebrates depends on the early determination of primordial germ cells (PGCs) and their correct migration to the sites where the gonads develop. Several genes have been implicated in PGC specification and migration in vertebrates. Additionally, some of the genes associated with pluripotency, such as Oct4 and Nanog, are expressed in PGCs and gonads, suggesting a role for these genes in maintaining pluripotency of the germ lineage, which may be considered the only cell type that perpetually maintains stemness properties. Here, we report that medaka Nanog (Ol-Nanog) is expressed in the developing PGCs. Depletion of Ol-Nanog protein causes aberrant migration of …

Fish ProteinsHomeobox protein NANOGChromatin ImmunoprecipitationReceptors CXCR4endocrine systemCell typeGenotypeOryziasBiologyNanogCxcr4bOpen Reading FramesCell MovementAnimalsPromoter Regions Genetic3' Untranslated RegionsGeneIn Situ Hybridizationreproductive and urinary physiologyHomeodomain ProteinsRegulation of gene expressionMessenger RNABinding SitesReverse Transcriptase Polymerase Chain Reactionurogenital systemThree prime untranslated regionPGCGene Expression Regulation DevelopmentalCell BiologyImmunohistochemistryPhenotypeMolecular biologyChemokine CXCL12MedakaGerm CellsPhenotypeGene Knockdown Techniquesembryonic structuresMolecular Medicinebiological phenomena cell phenomena and immunityChromatin immunoprecipitationDevelopmental BiologyStem Cells
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Nanog Regulates Proliferation During Early Fish Development

2009

Abstract Nanog is involved in controlling pluripotency and differentiation of stem cells in vitro. However, its function in vivo has been studied only in mouse embryos and various reports suggest that Nanog may not be required for the regulation of differentiation. To better understand endogenous Nanog function, more animal models should be introduced to complement the murine model. Here, we have identified the homolog of the mammalian Nanog gene in teleost fish and describe the endogenous expression of Ol-Nanog mRNA and protein during medaka (Oryzias latipes) embryonic development and in the adult gonads. Using medaka fish as a vertebrate model to study Nanog function, we demonstrate that …

Fish ProteinsHomeobox protein NANOGOryziasRex1ProliferationOryziasBiologyNanogPolymerase Chain ReactionGene expressionAnimalsRNA MessengerGonadsTranscription factorIn Situ Hybridizationreproductive and urinary physiologyCell ProliferationHomeodomain ProteinsRegulation of gene expressionCell CycleEmbryogenesisGene Expression Regulation DevelopmentalCell Biologybiology.organism_classificationImmunohistochemistryMolecular biologyMedakaDifferentiationembryonic structuresMolecular Medicinebiological phenomena cell phenomena and immunityStem cellDevelopmental BiologyStem Cells
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Mutations in LMX1B cause abnormal skeletal patterning and renal dysplasia in nail patella syndrome

1998

The LIM-homeodomain protein Lmxlb plays a central role in dorso-ventral patterning of the vertebrate limb1. Targeted disruption of Lmxlb results in skeletal defects including hypoplas-tic nails, absent patellae and a unique form of renal dysplasia (see accompanying manuscript by H. Chen et al.; ref. 2). These features are reminiscent of the dominantly inherited skeletal malformation nail patella syndrome (NFS). We show that LMX1B maps to the NFS locus and that three independent NFS patients carry de novo heterozygous mutations in this gene. Functional studies show that one of these mutations disrupts sequence-specific DNA binding, while the other two mutations result in premature terminatio…

HeterozygotePathologymedicine.medical_specialtyLIM-Homeodomain ProteinsMolecular Sequence DataLocus (genetics)BiologyKidneyBone and BonesMiceGene mappingNail-Patella SyndromeGeneticsmedicineAnimalsHumansAmino Acid SequenceGeneBody PatterningNail patella syndromeHomeodomain ProteinsGeneticsBase SequenceDysostosismedicine.diseasePhenotypeRenal dysplasiaMutationHomeotic geneTranscription FactorsNature Genetics
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