Search results for "Host-Pathogen Interaction"

showing 10 items of 189 documents

On the Age of Leprosy

2014

Leprosy is a chronic infection of the skin and nerves caused by Mycobacterium leprae and the newly discovered Mycobacterium lepromatosis. Human leprosy has been documented for millennia in ancient cultures. Recent genomic studies of worldwide M. leprae strains have further traced it along global human dispersals during the past ∼ 100,000 years. Because leprosy bacilli are strictly intracellular, we wonder how long humans have been affected by this disease-causing parasite. Based on recently published data on M. leprae genomes, M. lepromatosis discovery, leprosy bacilli evolution, and human evolution, it is most likely that the leprosy bacilli started parasitic evolution in humans or early h…

Immune defenselcsh:Arctic medicine. Tropical medicinelcsh:RC955-962EpidemiologyImmunologyReviewDermatologymedicine.disease_causeMicrobiologyMycobacteriumLeprosymedicineHumansMycobacterium lepraeBiologyPhylogenyMycobacterium lepromatosisClinical GeneticsbiologyHuman evolutionary geneticslcsh:Public aspects of medicinePublic Health Environmental and Occupational Healthlcsh:RA1-1270Genomicsbiology.organism_classificationmedicine.diseaseBiological EvolutionMycobacterium lepraeChronic infectionInfectious DiseasesHuman evolutionImmunologyHost-Pathogen InteractionsMedicineClinical ImmunologyLeprosyPublic HealthMycobacteriumPLoS Neglected Tropical Diseases
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Toll-like receptors – sentries in the B-cell response

2009

Summary Toll-like receptors (TLR) play a central role in the initiation of the innate immune response to pathogens. Upon recognition of molecular motifs specific for microbial molecules TLR mediate pro-inflammatory cytokine secretion and enhance antigen presentation; in B cells they further promote expansion, class switch recombination and immunoglobulin secretion. As a result of their adjuvant properties, TLR ligands have become an integral component of antimicrobial vaccines. In spite of this, little is known of the direct effects of TLR engagement on B-lymphocyte function. The scope of this review is to outline the differences in TLR expression and reactivity in murine and human B-cell s…

ImmunologyAntigen presentationReview ArticleBiologyImmunoglobulin secretionImmunomodulationMicemedicineImmunology and AllergyAnimalsHumansReceptorB cellB-LymphocytesInnate immune systemToll-Like ReceptorsImmunoglobulin Class SwitchingImmunity InnateCell biologymedicine.anatomical_structureImmunoglobulin class switchingImmunologyAntibody FormationHost-Pathogen InteractionsCytokine secretionFunction (biology)
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Complex Cellular Responses of Helicobacter pylori-Colonized Gastric Adenocarcinoma Cells ▿

2011

ABSTRACT Helicobacter pylori is an important class I carcinogen that persistently infects the human gastric mucosa to induce gastritis, gastric ulceration, and gastric cancer. H. pylori pathogenesis strongly depends on pathogenic factors, such as VacA (vacuolating cytotoxin A) or a specialized type IV secretion system (T4SS), which injects the oncoprotein CagA (cytotoxin-associated gene A product) into the host cell. Since access to primary gastric epithelial cells is limited, many studies on the complex cellular and molecular mechanisms of H. pylori were performed in immortalized epithelial cells originating from individual human adenocarcinomas. The aim of our study was a comparative anal…

ImmunologyImmunoblottingAdenocarcinomaMicrobiologyHelicobacter InfectionsStomach NeoplasmsCell Line TumormedicineGastric mucosaCell AdhesionCagAHumansImmunoprecipitationSecretionInterleukin 8Cell adhesionAuthor CorrectionbiologyHelicobacter pyloriReverse Transcriptase Polymerase Chain ReactionInterleukin-8Bacterial InfectionsHelicobacter pylorimedicine.diseasebiology.organism_classificationbacterial infections and mycosesdigestive system diseasesInfectious Diseasesmedicine.anatomical_structureCell cultureBacterial TranslocationImmunologyHost-Pathogen InteractionsCancer researchAdenocarcinomaParasitology
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Host–pathogen interactions in Vibrio vulnificus: responses of monocytes and vascular endothelial cells to live bacteria

2015

ABSTRACT  Aim: To demonstrate that Vibrio vulnificus, a sepsis-related aquatic pathogen, can provoke a strong pro-inflammatory reaction in blood-associated target cells. Materials & methods: We selected two strains of the two main phylogenetic lineages, two human cell lines, monocytes and vascular endothelial cells and designed an in vitro infection model simulating early septicemia. Results: Both strains caused a strong cell-specific pro-inflammatory response and produced a high degree of cell damage that ended with death by lysis (endothelial cells) or apoptosis/lysis (monocytes). The interaction with endothelial cells was stronger than expected and significantly different for both l…

InflammationMicrobiology (medical)LysisbiologyGene Expression ProfilingEndothelial CellsVibrio vulnificusbiology.organism_classificationmedicine.diseaseMicrobiologyVirologyMonocytesIn vitroMicrobiologySepsisStress PhysiologicalApoptosisHost-Pathogen InteractionsmedicineHumansVibrio vulnificusPathogenCell damageCells CulturedBacteriaFuture Microbiology
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Susceptibility of Spodoptera frugiperda and S. exigua to Bacillus thuringiensis Vip3Aa insecticidal protein

2011

The Vip3Aa protein is an insecticidal protein secreted by Bacillus thuringiensis during the vegetative stage of growth. The activity of this protein has been tested after different steps/protocols of purification using Spodoptera frugiperda as a control insect. The results showed that the Vip3Aa protoxin was stable and retained full toxicity after being subjected to common biochemical steps used in protein purification. Bioassays with the protoxin in S. frugiperda and S. exigua showed pronounced differences in LC(50) values when mortality was measured at 7 vs. 10d. At 7d most live larvae were arrested in their development. LC(50) values of "functional mortality" (dead larvae plus larvae rem…

InsecticidesLongevityBacillus thuringiensisInsect pest controlInsect proteaseBacterial ProteinSpodopteraSpodopteraMedian lethal doseMicrobiologyLethal Dose 50Vegetative insecticidal proteinBacterial ProteinsSpecies SpecificityBacillus thuringiensisparasitic diseasesExiguaProtein purificationBotanyAnimalsBacillus thuringiensiBioassayPest Control BiologicalInsecticideEcology Evolution Behavior and SystematicsbiologyAnimalfungiMidgutbiology.organism_classificationBioinsecticideHost-Pathogen InteractionMode of actionLarvaHost-Pathogen InteractionsInstarBiological AssayElectrophoresis Polyacrylamide GelDisease SusceptibilityJournal of Invertebrate Pathology
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Combining Hexanoic Acid Plant Priming with Bacillus thuringiensis Insecticidal Activity against Colorado Potato Beetle

2013

Interaction between insect herbivores and host plants can be modulated by endogenous and exogenous compounds present in the source of food and might be successfully exploited in Colorado potato beetle (CPB) pest management. Feeding tests with CPB larvae reared on three solanaceous plants (potato, eggplant and tomato) resulted in variable larval growth rates and differential susceptibility to Bacillus thuringiensis Cry3Aa toxin as a function of the host plant. An inverse correlation with toxicity was observed in Cry3Aa proteolytic patterns generated by CPB midgut brush-border membrane vesicles (BBMV) from Solanaceae-fed larvae, being the toxin most extensively proteolyzed on potato, followed…

Insecticidesmedicine.disease_causeMass Spectrometrylcsh:Chemistrychemistry.chemical_compoundHemolysin ProteinsPlant Growth RegulatorsCysteine ProteasesBacillus thuringiensisPlant defense against herbivoryColorado potato beetleElectrophoresis Gel Two-Dimensionallcsh:QH301-705.5SpectroscopySolanaceaeHexanoic acidbiologyfood and beveragesGeneral MedicineComputer Science ApplicationsColeopterasurgical procedures operativeBiochemistryLarvaHost-Pathogen Interactionsplant hormonesInsect ProteinsSolanaceaeproteolysisColoradoMolecular Sequence DataBacillus thuringiensisCatalysisArticleMicrobiologyCry3Aa toxinInorganic Chemistryintestain proteasesBacterial Proteinsplant defensemedicineAnimalsAmino Acid SequencePhysical and Theoretical ChemistryprimingMolecular BiologyCaproatesSolanum tuberosumBacillus thuringiensis ToxinsToxinOrganic ChemistryColorado potato beetlefungiBody WeightMidgutColorado potato beetle;<i> Bacillus thuringiensis</i>; Cry3Aa toxin; intestain proteases; proteolysis; Solanaceae; hexanoic acid; priming; plant defense; plant hormonesFeeding Behaviorbiology.organism_classificationDietEndotoxinsPapainchemistrylcsh:Biology (General)lcsh:QD1-999hexanoic acidPeptidesDigestive SystemSequence AlignmentInternational Journal of Molecular Sciences
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Phenolic-glycolipid-1 and lipoarabinomannan preferentially modulate TCR- and CD28-triggered proximal biochemical events, leading to T-cell unresponsi…

2012

Abstract Background Advanced stages of leprosy show T cell unresponsiveness and lipids of mycobacterial origin are speculated to modulate immune responses in these patients. Present study elucidates the role of phenolicglycolipid (PGL-1) and Mannose-capped lipoarabinomannan (Man-LAM) on TCR- and TCR/CD28- mediated signalling. Results We observed that lipid antigens significantly inhibit proximal early signalling events like Zap-70 phosphorylation and calcium mobilization. Interestingly, these antigens preferentially curtailed TCR-triggered early downstream signalling events like p38 phosphorylation whereas potentiated that of Erk1/2. Further, at later stages inhibition of NFAT binding, IL-2…

LipopolysaccharidesEndocrinology Diabetes and MetabolismT-LymphocytesClinical BiochemistryPGL-1Man-LAMGene ExpressionLymphocyte ActivationJurkat cellsJurkat CellsEndocrinologyT-cell activationIL-2 receptorPhosphorylationExtracellular Signal-Regulated MAP KinasesPromoter Regions Geneticlcsh:RC620-627Protein Kinase CImmunity CellularZAP-70 Protein-Tyrosine KinaseCD28hemic and immune systemsCell biologyMycobacterium lepraelcsh:Nutritional diseases. Deficiency diseasesmedicine.anatomical_structureHost-Pathogen InteractionsProtein BindingMAP Kinase Signaling SystemT cellReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyImmune systemCD28 AntigensLeprosymedicineHumansSecretionCalcium SignalingCell ProliferationBiochemistry medicalAntigens BacterialLipoarabinomannanNFATC Transcription FactorsResearchBiochemistry (medical)T-cell receptorInterleukin-2 Receptor alpha SubunitMycobacteriaGene Expression RegulationAnergyImmunologyLeukocytes MononuclearInterleukin-2GlycolipidsLipids in Health and Disease
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Serological Tests Do Not Predict Residual Fibrosis in Hepatitis C Cirrhotics with a Sustained Virological Response to Interferon

2016

BACKGROUND AND AIM: Liver biopsy (LB) has lost popularity to stage liver fibrosis in the era of highly effective anti-hepatitis C virus (HCV) therapy, yet diagnosis of persistent cirrhosis may have important implications following HCV eradication. As performance of serological non-invasive tests (NITs) to predict residual fibrosis in non-viremic HCV patients is unknown, we investigated accuracy of NITs to predict residual fibrosis in cirrhotics after a sustained virological response (SVR) to interferon (IFN). METHODS: Thirty-eight patients with a pre-treatment histological diagnosis of cirrhosis and a 48–104 months post-SVR LB were tested with APRI, CDS, FIB-4, FibroQ, Forns Score, GUCI Ind…

Liver CirrhosisMalePathologyCirrhosisBiopsylcsh:MedicineHepacivirusPathology and Laboratory MedicineGastroenterology0302 clinical medicineEsophageal varicesFibrosisOutcome Assessment Health CareMedicine and Health Scienceslcsh:ScienceSerodiagnosisMultidisciplinarymedicine.diagnostic_testLiver DiseasesHepatitis CMiddle AgedHepatitis C3. Good healthSerologyCirrhosisLiver030220 oncology & carcinogenesisLiver biopsyHepatocellular carcinomaHost-Pathogen InteractionsLiver FibrosisElasticity Imaging Techniques030211 gastroenterology & hepatologyFemaleAnatomyResearch Articlemedicine.medical_specialtyHistologySurgical and Invasive Medical ProceduresGastroenterology and HepatologyAntiviral Agents03 medical and health sciencesDiagnostic MedicineInternal medicineBiopsymedicineHumansSerologic TestsAspartate AminotransferasesAgedbusiness.industryPlatelet Countlcsh:RBiology and Life Sciencesmedicine.diseaseFibrosisROC Curvelcsh:QInterferonsTransient elastographybusinessBiomarkersDevelopmental BiologyPLoS ONE
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Candida albicans Yeast and Hyphae are Discriminated by MAPK Signaling in Vaginal Epithelial Cells

2011

We previously reported that a bi-phasic innate immune MAPK response, constituting activation of the mitogen-activated protein kinase (MAPK) phosphatase MKP1 and c-Fos transcription factor, discriminates between the yeast and hyphal forms of Candida albicans in oral epithelial cells (ECs). Since the vast majority of mucosal Candida infections are vaginal, we sought to determine whether a similar bi-phasic MAPK-based immune response was activated by C. albicans in vaginal ECs. Here, we demonstrate that vaginal ECs orchestrate an innate response to C. albicans via NF-κB and MAPK signaling pathways. However, unlike in oral ECs, the first MAPK response, defined by c-Jun transcription factor acti…

MAPK/ERK pathwaylcsh:MedicineYeast and Fungal ModelsPathogenesisSignal transductionMolecular cell biologyCandida albicansGranulocyte Colony-Stimulating FactorCandida albicanslcsh:ScienceImmune Response0303 health sciencesMultidisciplinarybiologyCandidiasisNF-kappa BSignaling cascadesObstetrics and GynecologyCorpus albicansInnate ImmunityHost-Pathogen InteractionInfectious DiseasesVaginaCytokinesMedicineFemaleSignal transductionCandidalysinResearch ArticleMAPK signaling cascadesMAP Kinase Signaling SystemUrologyImmunologySexually Transmitted DiseasesHyphaeMycologyMicrobiologyMicrobiologyImmune Activation03 medical and health sciencesModel OrganismsHumansTranscription factorBiology030304 developmental biologyInnate immune systemChemokine CCL20030306 microbiologyGenitourinary InfectionsInterleukin-6lcsh:RImmunityFungiMouth MucosaImmune DefenseEpithelial Cellsbiology.organism_classificationImmunity InnateCCL20Immune Systemlcsh:QClinical ImmunologyPLoS ONE
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Multi-infections of feminizing Wolbachia strains in natural populations of the terrestrial isopod Armadillidium vulgare.

2013

7 pages; International audience; Maternally inherited Wolbachia (α-Proteobacteria) are widespread parasitic reproductive manipulators. A growing number of studies have described the presence of different Wolbachia strains within a same host. To date, no naturally occurring multiple infections have been recorded in terrestrial isopods. This is true for Armadillidium vulgare which is known to harbor non simultaneously three Wolbachia strains. Traditionally, such Wolbachia are detected by PCR amplification of the wsp gene and strains are characterized by sequencing. The presence of nucleotide deletions or insertions within the wsp gene, among these three different strains, provides the opportu…

Male0106 biological sciencesGenotypeRickettsiaceae Infectionslcsh:MedicinePolymerase Chain Reaction010603 evolutionary biology01 natural scienceslaw.invention03 medical and health scienceslaw[ SDV.MP ] Life Sciences [q-bio]/Microbiology and ParasitologyGenotypeparasitic diseasesPrevalence[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalslcsh:ScienceGeneGenotypingPolymerase chain reactionreproductive and urinary physiology030304 developmental biologyArmadillidium vulgareGenetics0303 health sciencesMultidisciplinarybiologyHost (biology)Strain (biology)lcsh:RReproducibility of Resultsbiology.organism_classification[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyHost-Pathogen InteractionsbacteriaFemalelcsh:QWolbachiaFranceWolbachiaIsopodaResearch Article[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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