Search results for "Hsp"

showing 10 items of 557 documents

Die passivischen Ausdrücke der medizinischen Fachsprache

1998

PassivPassivparaphrasenFachsprachen
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Htid-1, the human homolog of the Drosophila melanogaster l(2)tid tumor suppressor, defines a novel physiological role of APC.

2007

Htid-1, the human counterpart of the Drosophila tumor suppressor gene lethal(2)tumorous imaginal discs (l(2)tid) encodes three splice forms translated into three cytosolic - Tid50, Tid48 and Tid46 - and three mitochondrial - Tid43, Tid40 and Tid38 - proteins. Here we provide evidence for the association of the endogenous Tid50/Tid48 proteins with the adenomatous polyposis coli (APC) tumor suppressor in normal colon epithelium, colorectal cancer cells and mouse NIH3T3 fibroblasts. Using the Glutathione S-transferase binding assay we show that the N-terminal region including the Armadillo domain (ARM) of APC is sufficient to bind the Tid molecules. Using immunoprecipitation and confocal micro…

Patched ReceptorsBeta-cateninTumor suppressor geneAdenomatous polyposis coliAdenomatous Polyposis Coli ProteinReceptors Cell SurfacePlasma protein bindingLigandsMitochondrial ProteinsMiceCytosolCell Line TumorAnimalsDrosophila ProteinsGuanine Nucleotide Exchange FactorsHumansIntestinal MucosaActinHeat-Shock Proteinsbeta CateninPatched ReceptorsbiologySequence Homology Amino AcidGene Expression ProfilingTumor Suppressor ProteinsWnt signaling pathwayGene Expression Regulation DevelopmentalCell BiologyHSP40 Heat-Shock ProteinsActin cytoskeletonMolecular biologyCell biologyMitochondriaDrosophila melanogasterras GTPase-Activating ProteinsMultiprotein Complexesbiology.proteinNIH 3T3 CellsRho Guanine Nucleotide Exchange FactorsProtein BindingCellular signalling
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Microsatellite Instability in Colorectal Cancer: Time to Stop Hiding!

2011

Colorectal cancer (CRC) is the second cause of cancer-related death worldwide. Surgery constitutes the primary therapy for these tumors, together with chemotherapy that is usually recommended in patients with metastatic primary CRC. Although molecularly distinct entities arising from different physiopathogenic mechanisms - microsatellite (MSI) and chromosomal instability (also called microsatellite stable, MSS) - have been characterized in CRC, there is still no specific therapeutic approach that takes into account disease’s molecular heterogeneity [1]. MSI is observed in 1015% of sporadic CRCs. MSI CRCs displayed particular morphologic features, with greater predilection for the right colo…

Pathologymedicine.medical_specialtyChemotherapyColorectal cancermedicine.medical_treatmentMicrosatellite instabilityDiseaseBiologymedicine.diseasedigestive system diseasesTherapeutic approachOncologyChromosome instabilityCancer researchmedicineMicrosatelliteAnimalsHumansIn patientMicrosatellite InstabilityHSP110 Heat-Shock ProteinsColorectal NeoplasmsneoplasmsEditorial CommentsOncotarget
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Analysis of the RET, GDNF, EDN3, and EDNRB genes in patients with intestinal neuronal dysplasia and Hirschsprung disease

2001

BACKGROUNDHirschsprung disease (HSCR) is a frequent congenital disorder with an incidence of 1 in 5000 live births, characterised by the absence of parasympathetic intramural ganglion cells in the hindgut resulting in intestinal obstruction in neonates and severe constipation in infants and adults. Intestinal neuronal dysplasia (IND) shares clinical features with HSCR but the submucosal parasympathetic plexus is affected. IND has been proposed as one of the most frequent causes of chronic constipation and is often associated with HSCR.METHODSWe examined 29 patients diagnosed with sporadic HSCR, 20 patients with IND, and 12 patients with mixed HSCR/IND for mutations in the coding regions of …

Pathologymedicine.medical_specialtyGlial Cell Line-Derived Neurotrophic Factor ReceptorsHirschsprung diseaseMUTATION ANALYSISNerve Tissue ProteinsTYROSINE KINASEEDNRBArticleExonGermline mutationProto-Oncogene ProteinsNEUROTROPHIC FACTOR GDNFmedicineGlial cell line-derived neurotrophic factorDrosophila ProteinsHumansGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsAlleleintestinal neuronal dysplasiaAllelesPolymorphism Single-Stranded ConformationalIntestinal neuronal dysplasiabiologyReceptors EndothelinSHAH-WAARDENBURG SYNDROMEProto-Oncogene Proteins c-retENDOTHELIN-B-RECEPTORMULTIGENIC INHERITANCEGastroenterologyReceptor Protein-Tyrosine KinasesSequence Analysis DNAGERMLINE MUTATIONSbiochemical phenomena metabolism and nutritionPROTOONCOGENEmedicine.diseasePHENOTYPIC-EXPRESSIONGDNFPedigreeProto-Oncogene Proteins c-retDysplasiaCase-Control StudiesMutationbiology.proteinLIGANDRETCongenital disorderEDN3
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Distinctive patterns of Hsp60 levels and localization in human colon mucosa.

2011

Pathologymedicine.medical_specialtyHsp60 colon mucosaGeneticsmedicineHSP60BiologyMolecular BiologyBiochemistryHuman colonBiotechnology
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Etiopathological aspects of achalasia: lessons learned with Hirschsprung's disease

2011

SUMMARY The etiology of primary esophageal achalasia is largely unknown. There is increasing evidence that genetic alterations might play an important but underestimated role. Current knowledge of the genetic base of Hirschsprung's disease in contrast is far more detailed. The two enteric neuropathies have several clinical features in common. This association may also exist on a cellular and molecular level. The aim of this review is to enlighten those etiopathogenetic concepts of Hirschsprung's disease that seem to be useful in uncovering the pathological processes causing achalasia. Three aspects are looked at: (i) the genetic base of Hirschsprung's disease, particularly its major suscept…

Pathologymedicine.medical_specialtybusiness.industryEnteric neuropathyGastroenterologyAchalasiaGeneral MedicineDiseaseBioinformaticsmedicine.diseasedigestive systemdigestive system diseasesMolecular levelmedicineEtiologyRearranged during transfectionbusinessPathologicalHirschsprung's diseaseDiseases of the Esophagus
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The expression of heat shock proteins 27 and 105 in squamous cell carcinoma of the tongue and relationship with clinicopathological index

2010

Introduction: In oral cavity, the tongue is the most common site prone to development of squamous cell carcinoma (SCC). Considering malignant transformation as a cellular stress, the expression of heat shock proteins (HSPs) may be affected in this process. In this study we assessed the expression of HSP105 and HSP27 as two of the most interested stress proteins and investigated their relationship with grade and stage of the tongue SCC. Material and Methods: Fifty-six specimens including 31 early and 25 advanced tongue SCC were gathered. All specimens were graded histologically from I to III. Sixteen sections of normal oral mucosa were used as control group. The cellularity and intensity of …

Pathologymedicine.medical_specialtymedicine.medical_treatmentHSP27 Heat-Shock ProteinsMalignant transformationLesionHsp27TongueHeat shock proteinmedicineCarcinomaHumansHSP110 Heat-Shock ProteinsGeneral DentistryGrading (tumors)Retrospective Studiesbiologybusiness.industryImmunotherapyMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseTongue Neoplasmsstomatognathic diseasesmedicine.anatomical_structureOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASCarcinoma Squamous Cellbiology.proteinSurgerymedicine.symptombusinessMedicina Oral Patología Oral y Cirugia Bucal
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Hsp60 and Hsp10 as antitumour molecular agents

2007

The molecular chaperones Hsp60 and Hsp10 are, according to recent reports, involved in cancer development and progression. We, for instance, have found that their expression varies with distinctive patterns in different malignancies: they are overexpressed in colorectal, exocervical and prostate carcinogenesis, and colorectal cancer progression, but they are downregulated during bronchial carcinogenesis. There is also evidence showing that Hsp60 and Hsp10 can be used as therapeutic agents, for example in rheumatoid arthritis. In view of these findings we want now to call attention to the potential of Hsp60 and Hsp10 in cancer therapy.

PharmacologyOncologyCancer Researchmedicine.medical_specialtyanimal structuresbusiness.industryColorectal cancermedicine.medical_treatmentfungiCancerImmunotherapyMouse model of colorectal and intestinal cancermedicine.diseasemedicine.disease_causeOncologyRheumatoid arthritisInternal medicineMolecular MedicineMedicineHSP60Cancer developmentbusinessCarcinogenesisCancer Biology & Therapy
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The upgrade of the ALICE TPC with GEMs and continuous readout

2020

Journal of Instrumentation 16(03), P03022 (2021). doi:10.1088/1748-0221/16/03/P03022

Physics - Instrumentation and DetectorsComputer sciencePhysics::Instrumentation and DetectorsFOS: Physical sciences61001 natural sciences114 Physical sciences030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicine0103 physical sciencesMicropattern gaseous detectors (MSGC GEM THGEM RETHGEM MHSP MICROPIC MICROMEGAS InGrid etc)Electronicsddc:610Detectors and Experimental TechniquesInstrumentationphysics.ins-detMathematical PhysicsCMOS readout of gaseous detectorsLarge Hadron Collider010308 nuclear & particles physicsbusiness.industryDetectorTime projection Chambers (TPC)Readout electronicsInstrumentation and Detectors (physics.ins-det)ChipUpgradeGaseous imaging and tracking detectorsGas electron multiplierALICE (propellant)businessComputer hardware
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Type V collagen regulates the expression of apoptotic and stress response genes by breast cancer cells.

2004

Type V collagen is a "minor" component of normal human breast stroma, which is subjected to over-deposition in cases of ductal infiltrating carcinoma (DIC). We reported that, if used as a culture substrate for the DIC cell line 8701-BC, it exhibited poorly-adhesive properties and restrained the proliferative and motile behavior of the cell subpopulation able to attach onto it. Moreover, this collagen species was able to trigger DNA fragmentation and impair survival of 8701-BC cells. In this study, we have extended our investigation with the aim to obtain further evidence that the death induced by type V collagen was of the apoptotic type by (i) microscopic detection and quantitation of Apop…

PhysiologyClinical BiochemistryCellApoptosisBreast NeoplasmsEnzyme activatorCell Line TumormedicineHumansSettore BIO/06 - Anatomia Comparata E CitologiaCaspaseHeat-Shock ProteinsbiologyCarcinoma Ductal BreastCell BiologyMolecular biologyIn vitroEnzyme ActivationGene Expression Regulation Neoplasticmedicine.anatomical_structurecollagen breast cancer gene expressionApoptosisCell cultureCaspasesbiology.proteinDNA fragmentationHSP60FemaleCollagen Type VJournal of cellular physiology
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