Search results for "Huntington"

showing 10 items of 54 documents

Is TGF-β1 a Biomarker of Huntington’s Disease Progression?

2021

Huntington’s disease (HD) is an autosomal dominant genetic disease that can be divided into preclinical and symptomatic stages. Due to the diverse HD phenotype, there is an urgent need to identify markers that would independently assess its severity. The aim of this study was to evaluate the use of plasma levels of TGF-β1 in the assessment of HD severity. One hundred HD patients and 40 healthy volunteers were included in the study. All HD patients underwent neurological and cognitive function assessment. TGF-β1 levels were determined in the plasma of all patients. The correlations between TGF-β1 levels and clinical profile and HD severity were also investigated. In symptomatic patients, cog…

0301 basic medicinemedicine.medical_specialtymarkersDiseaseGastroenterologyArticle03 medical and health sciences0302 clinical medicineDisease severityHuntington's diseaseInternal medicineTGF-β1medicineCognitive declineStage (cooking)business.industryRGeneral MedicinePlasma levelsHuntington diseasemedicine.disease030104 developmental biologyBiomarker (medicine)Medicinebusiness030217 neurology & neurosurgeryTransforming growth factorJournal of Clinical Medicine
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The MID1 protein is a central player during development and in disease.

2015

Loss-of-function mutations in the MID1 gene cause a rare monogenic disorder, Opitz BBB/G syndrome (OS), which is characterized by malformations of the ventral midline. The MID1 gene encodes the MID1 protein, which assembles a large microtubule-associated protein complex. Intensive research over the past several years has shed light on the function of the MID1 protein as a ubiquitin ligase and regulator of mTOR signalling and translational activator. As a central player in the cell MID1 has been implicated in the pathogenesis of various other disorders in addition to OS including cancer and neurodegenerative diseases. Influencing the activity of the MID1 protein complex is a promising new st…

0301 basic medicinephysiopathology [Huntington Disease]CarcinogenesisUbiquitin-Protein LigasesRegulatorDiseaseBiologyBioinformaticsmedicine.disease_causephysiopathology [Alzheimer Disease]Congenital AbnormalitiesPathogenesis03 medical and health sciencesMiceAlzheimer Diseasephysiology [Nuclear Proteins]medicineAnimalsHumansgenetics [Microtubule Proteins]ddc:610GenePI3K/AKT/mTOR pathwayActivator (genetics)Nuclear Proteinsgenetics [Nuclear Proteins]genetics [Transcription Factors]physiology [Transcription Factors]Ubiquitin ligase030104 developmental biologyHuntington DiseaseMutationbiology.proteinMicrotubule Proteinsphysiology [Microtubule Proteins]CarcinogenesisMid1 protein humanTranscription FactorsFrontiers in bioscience (Landmark edition)
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Bush, Sharon y los suyos

2004

Se trata de la versión mecanografiada del artículo publicado en "El País" el 16 de abril de 2004, pero debe aclararse que el texto es exactamente igual al publicado, únicamente cambia el título: "Los terminator de la democracia".

11-MHuntingtonVidal-Beneyto JoséEditorialistasAznarEspañaIrakOcupaciónTerrorismoTerroristasEleccionesPublicaciones: Obra periodística: Columnas y artículos de opiniónPropaganda políticaInvasiónTerminatorAl QaedaAntiterrorismoMundo islámicoTropas ideológicasIDEOLOGÍAGUERRABushDemocraciaAtentadosSolidaridad mundialCiudadanía mundialVotoArmas de destrucción masivaZapateroSharonNaciones UnidasLucha
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Estudio del papel de la proteína activadora de la apoptosis apaf-1 en modelos celulares de la enfermedad de huntington

2012

La apoptosis es un proceso biológico regulado a través de interacciones proteína-proteína que conforman diversos complejos. Estos complejos representan puntos interesantes de intervenciones químicas en diversas enfermedades neurodegenerativas como la enfermedad de Huntington (HD). Esta enfermedad se presenta debido a la expansión de poliglutaminas en el extremo N-terminal de la proteína huntingtina (Htt). En la actualidad, una de las características más estudiadas de esta patología es la formación de agregados de proteínas en el espacio citosólico y nuclear que incluyen a fragmentos N-terminal de la proteína huntingtina mutante (mHtt). El papel de los agregados en la patología es aún discut…

:CIENCIAS DE LA VIDA::Bioquímica [UNESCO]:CIENCIAS DE LA VIDA::Biología celular [UNESCO]enfermedad de Huntingtonagregados de poliglutamina:CIENCIAS DE LA VIDA::Neurociencias::Neuroquímica [UNESCO]apoptosisUNESCO::CIENCIAS DE LA VIDA::BioquímicaUNESCO::CIENCIAS DE LA VIDA::Biología celularUNESCO::CIENCIAS DE LA VIDA::Neurociencias::Neuroquímica
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New options in assisted reproduction technology: the Cryotop method of oocyte vitrification

2008

The Cryotop vitrification method has been shown to be a very useful tool for oocyte cryopreservation, giving excellent results regarding survival and clinical outcome. There are several clinical situations in which oocyte cryopreservation provides solutions that have not been available to date. This report describes three of these situations: (i) a low-responder patient who needed a single gene diagnosis due to the presence of a genetic disease; (ii) a patient undergoing endometrial bleeding on the day of oocyte retrieval who was also affected by a genetic disorder; and (iii) a patient who failed to become pregnant after the donation of vitrified oocytes and subsequently had the re-vitrifie…

AdultMaleReproductive Techniques Assistedmedia_common.quotation_subjectSingle geneBiologyPreimplantation genetic diagnosisPolymerase Chain ReactionEmbryo Culture TechniquesAndrologyEndometriumPregnancySurplus embryosEmbryo Culture TechniquemedicineHumansVitrificationPreimplantation Diagnosismedia_commonCryopreservationPregnancy OutcomeObstetrics and GynecologyOocyte cryopreservationOocyteHuntington Diseasemedicine.anatomical_structureReproductive MedicineOocytesFemaleReproductionPolycystic Ovary SyndromeDevelopmental BiologyReproductive BioMedicine Online
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Benefits of nonlinear analysis indices of walking stride interval in the evaluation of neurodegenerative diseases.

2021

Indices characterising the long-range temporal structure of walking stride interval (SI) variability such as Hurst exponent (H) and fractal dimension (D) may be used in addition to indices measuring the amount of variability like the coefficient of variation (CV). We assess the added value of the former indices in a clinical neurological context. Our aim is to demonstrate that they provide a clinical significance in aging and in frequent neurodegenerative diseases such as Parkinson's disease, Huntington, and amyotrophic lateral sclerosis. Indices assessing the temporal structure of variability are mainly dependent on SI time series length and algorithms used, making quantitative comparisons…

AdultMalemedicine.medical_specialtyAgingCoefficient of variationBiophysicsSTRIDEExperimental and Cognitive PsychologyContext (language use)DiseaseWalkingPhysical medicine and rehabilitationmedicineHumansOrthopedics and Sports MedicineClinical significanceAmyotrophic lateral sclerosisGaitHurst exponentPrincipal Component Analysisbusiness.industryNeurodegenerative DiseasesParkinson DiseaseGeneral MedicineMiddle Agedmedicine.diseaseFractalsHuntington DiseaseGait analysisFemalebusinessGait AnalysisAlgorithmsHuman movement science
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A novel function of Huntingtin in the cilium and retinal ciliopathy in Huntington's disease mice

2015

Huntington's disease (HD) is a neurodegenerative disorder caused by the toxic expansion of polyglutamine in the Huntingtin (HTT) protein. The pathomechanism is complex and not fully understood. Increasing evidence indicates that the loss of normal protein function also contributes to the pathogenesis, pointing out the importance of understanding the physiological roles of HTT. We provide evidence for a novel function of HTT in the cilium. HTT localizes in diverse types of cilia — including 9 + 0 non-motile sensory cilia of neurons and 9 + 2 motile multicilia of trachea and ependymal cells — which exert various functions during tissue development and homeostasis. In the photoreceptor cilium,…

AxonemeMalecongenital hereditary and neonatal diseases and abnormalitiesHuntingtinCentrioleMice TransgenicNerve Tissue ProteinsBiologyMicrotubulesPhotoreceptor cellRetinalcsh:RC321-571MiceHuntington's diseaseIntraflagellar transportmental disordersmedicineAnimalsHumansPhotoreceptor CellsHuntingtinCilialcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSHuntingtin ProteinPhotoreceptorCiliumNuclear ProteinsHuntington's diseasemedicine.diseaseCell biologyCiliopathyDisease Models Animalmedicine.anatomical_structureHEK293 CellsHuntington DiseaseNeurologyFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organs
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Kaukasia geopoliittisen toiminnan näyttämönä : Kaukasian alueen geopoliittista tarkastelua läntisten, venäläisten, turkkilaisten ja iranilaisten intr…

2003

Brzezinski ZbigniewsydänmaaKaukasiaGeorgiapuskurivyöhykeetupiiritgeopolitiikkaTšetšeniaArmeniaIranreunavaltiotislamilainen kulttuuriVenäjäTurkkiAzerbaidžanHuntington Samuel PeurasianismiMackinder Halford John
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Degeneration of the Cerebellum in Huntington's Disease (HD): Possible Relevance for the Clinical Picture and Potential Gateway to Pathological Mechan…

2012

Huntington's disease (HD) is a polyglutamine disease and characterized neuropathologically by degeneration of the striatum and select layers of the neo- and allocortex. In the present study, we performed a systematic investigation of the cerebellum in eight clinically diagnosed and genetically confirmed HD patients. The cerebellum of all HD patients showed a considerable atrophy, as well as a consistent loss of Purkinje cells and nerve cells of the fastigial, globose, emboliform and dentate nuclei. This pathology was obvious already in HD brains assigned Vonsattel grade 2 striatal atrophy and did not correlate with the extent and distribution of striatal atrophy. Therefore, our findings sug…

CerebellumPathologymedicine.medical_specialtyAtaxiaGeneral NeuroscienceNeurodegenerationmedicine.diseasePathology and Forensic MedicinePathogenesisDysarthriamedicine.anatomical_structureAtrophynervous systemHuntington's diseasemedicineCorticobasal degenerationNeurology (clinical)medicine.symptomPsychologyNeuroscienceBrain Pathology
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A systematic comparison of kinetic modelling methods generating parametric maps for [11C]-(R)-PK11195

2006

[(11)C]-(R)-PK11195 is presently the most widely used radiotracer for the monitoring of microglia activity in the central nervous system (CNS). Microglia, the resident immune cells of the brain, play a critical role in acute and chronic diseases of the central nervous system and in host defence against neoplasia. The purpose of this investigation was to evaluate the reliability and sensitivity of five kinetic modelling methods for the formation of parametric maps from dynamic [(11)C]-(R)-PK11195 studies. The methods we tested were the simplified reference tissue model (SRTM), basis pursuit, a simple target-to-reference ratio, the Logan plot and a wavelet based Logan plot. For the reliabilit…

Correlation coefficientComputer scienceCognitive NeuroscienceBasis pursuitKinetic energySensitivity and SpecificityWaveletAlzheimer DiseaseModelling methodsComputer GraphicsImage Processing Computer-AssistedCluster AnalysisHumansPharmacokineticsCarbon RadioisotopesMathematical ComputingParametric statisticsBrain Mappingbusiness.industryBrainIsoquinolinesReceptors GABA-ALogan plotHuntington DiseaseNeurologyPositron-Emission TomographyMicrogliaNuclear medicinebusinessNeuroImage
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