Search results for "Hydroquinones"

showing 10 items of 13 documents

Nitic oxide promotes strong cytotoxicity of phenolic compounds against escherichia coli. The influence of antioxidant defenses

2003

[EN] The induction of mutagenic and cytotoxic effects by simple phenolics, including catechol (CAT), 3,4dihydroxyphenylacetic acid (DOPAC), hydroquinone (HQ), and 2,5-dihydroxyphenylacetic (homogentisic) acid (HGA), appears to occur through an oxidative mechanism based on the ability of these compounds to undergo autoxidation, leading to quinone formation with the production of reactive oxygen species. This is supported by the detection of such adverse effects in plate assays using Escherichia coli tester strains deficient in the OxyR function, but not in OxyR(+) strains. The OxyR protein is a redox-sensitive regulator of genes encoding antioxidant enzymes including catalase and alkyl hydro…

AntioxidantUltraviolet Raysmedicine.medical_treatmentCatecholsOxidative toxicityFree radicalsOxidative phosphorylationNitric OxideBiochemistryAntioxidantschemistry.chemical_compoundCaffeic AcidsQUIMICA ORGANICASuperoxidesPhysiology (medical)medicineEscherichia coliBIOQUIMICA Y BIOLOGIA MOLECULARHydrogen peroxidechemistry.chemical_classificationMelaninsReactive oxygen speciesbiologyHydroquinoneAutoxidationDose-Response Relationship DrugPhenolEscherichia coli ProteinsNitric oxideHydrogen PeroxideCatalaseFlow CytometryQuinoneHydroquinonesDNA-Binding ProteinsOxygenRepressor ProteinschemistryBiochemistryCatalaseMutationbiology.proteinQuinoneOxyROxidation-ReductionDNA DamageMutagensTranscription Factors
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Effects of marine 2-polyprenyl-1,4-hydroquinones on phospholipase A2 activity and some inflammatory responses.

1995

Three 2-polyprenyl-1,4-hydroquinone derivatives (2-heptaprenyl-1,4-hydroquinone: IS1, 2-octaprenyl-1,4-hydroquinone: IS2 and 2-[24-hydroxy]-octaprenyl-1,4-hydroquinone: IS3) isolated from the Mediterranean sponge Ircinia spinosula, were evaluated for effects on phospholipase A2 activity of different origin (Naja naja venom, human recombinant synovial fluid and bee venom), as well as on human neutrophil function and mouse ear oedema induced by 12-O-tetradecanoylphorbol 13-acetate (TPA). IS1 interacted minimally with these responses. In contrast, IS2 and IS3 inhibited human recombinant synovial phospholipase A2 in a concentration-dependent manner, with minor effects on the rest of the enzymes…

Blood PlateletsLeukocyte migrationLeukotriene B4Cell SurvivalNeutrophilsPharmacologyBiologyIn Vitro TechniquesLeukotriene B4Phospholipases Achemistry.chemical_compoundMicePhospholipase A2SuperoxidesMicrosomesSynovial fluidAnimalsEdemaHumansPharmacologyPhospholipase AL-Lactate DehydrogenasePancreatic ElastaseAnti-Inflammatory Agents Non-SteroidalDegranulationBiological activityHydroquinonesPoriferaThromboxane B2Thromboxane B2Phospholipases A2Biochemistrychemistrybiology.proteinTetradecanoylphorbol AcetateEuropean journal of pharmacology
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Isoprenylhydroquinone glucoside: a new non-antioxidant inhibitor of peroxynitrite-mediated tyrosine nitration

2004

Abstract Three hydroquinone glucosides and four caffeoylquinic esters were examined for their effect on tyrosine nitration, as well as on the oxidation of dihydrorhodamine (DHR) 123 and cytochrome c 2+ induced by peroxynitrite. All these phenolics, which had previously been characterized as the active principles of the plant Phagnalon rupestre , were fairly active in preventing the oxidation of DHR 123, though inefficient in the cytochrome c test. While their antioxidant potency is associated with the presence of a caffeoyl moiety, not so an obvious chemical character was correlated to a greater activity against nitration of tyrosine. Here, the highest potency corresponded to 2-isoprenylhyd…

Cancer ResearchAntioxidantCytochromePhysiologymedicine.medical_treatmentClinical BiochemistryAsteraceaeBiochemistrychemistry.chemical_compoundGlucosidesGlucosidePeroxynitrous AcidNitrationmedicineTyrosineNitratesMolecular StructurebiologyHydroquinoneRhodaminesCytochrome cCytochromes cHydroquinoneschemistryBiochemistrybiology.proteinTyrosineOxidation-ReductionPeroxynitriteNitric Oxide
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Modulation of protein tyrosine nitration and inflammatory mediators by isoprenylhydroquinone glucoside.

2007

The nitration of tyrosine caused by peroxynitrite and other reactive nitrogen species is clearly detrimental for some physiological processes; however, its signalling role is still open to controversy. Among the natural phenolics known for their ability to oppose free tyrosine nitration, isoprenylhydroquinone glucoside is investigated due to its unusual structure, which contains a simple hydroxybenzene alkylated by a hemiterpenoid moiety. This hydroquinone was shown to be an effective inhibitor of peroxynitrite-induced protein tyrosine nitration in 3T3 fibroblasts. When tested on bovine seroalbumin nitration, however, the potency was reduced by half and the effect was almost abolished in th…

LipopolysaccharidesCell SurvivalNeutrophilsBlotting WesternInterleukin-1betaPharmaceutical ScienceNitric Oxide Synthase Type IIHemeNitric oxidechemistry.chemical_compoundMiceGlucosideGlucosidesNitrationPeroxynitrous AcidAnimalsHumansTyrosineReactive nitrogen speciesCells CulturedNitritesNitratesbiologyCell-Free SystemReverse Transcriptase Polymerase Chain ReactionRhodaminesTumor Necrosis Factor-alphaNitrotyrosineSerum Albumin Bovine3T3 CellsHydrogen PeroxideFibroblastsStimulation ChemicalHydroquinonesNitric oxide synthasechemistryBiochemistrybiology.proteinTetradecanoylphorbol AcetateTyrosineInflammation MediatorsPeroxynitriteEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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New prenylhydroquinone glycosides from Phagnalon rupestre.

2001

Three new hydroquinone glycosides were isolated from the MeOH extract of the aerial parts of Phagnalon rupestre. Their structures were elucidated as 1-O-beta-glucopyranosyl-1,4-dihydroxy-2-(3',3'-dimethylallyl)benzene (1), 1-O-beta-glucopyranosyl-1,4-dihydroxy-2-(3'-hydroxymethyl-3'-methylallyl)benzene (2), and 1-O-(4' '-O-caffeoyl)-beta-glucopyranosyl-1,4-dihydroxy-2-(3',3'-dimethylallyl)benzene (3) by spectroscopic methods.

Magnetic Resonance SpectroscopySpectrophotometry InfraredStereochemistryPharmaceutical ScienceAsteraceaeAnalytical Chemistrychemistry.chemical_compoundDrug DiscoveryPhenolsGlycosidesBenzenePharmacologychemistry.chemical_classificationChromatographyPlants MedicinalHydroquinoneMolecular StructurePlant StemsOrganic ChemistryGlycosidePhagnalon rupestreHydroquinonesComplementary and alternative medicinechemistryAldoseSpainMolecular MedicineSpectrophotometry UltravioletJournal of natural products
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Suppression of leukotriene B4 and tumour necrosis factor alpha release in acute inflammatory responses by novel prenylated hydroquinone derivatives.

1998

A series of prenyl hydroquinone derivatives synthesized as structural analogs of marine products were tested for their effects on inflammatory responses in vitro and in vivo. 2-Prenyl-1,4-hydroquinone (H1), 2-diprenyl-1,4-hydroquinone (H2), 2-triprenyl-1,4-hydroquinone (H3) and 2-tetraprenyl-1,4-hydroquinone (H4) scavenged reactive oxygen species and inhibited 5-lipoxygenase (5-LO) activity in human neutrophils. The inhibition of 5-LO activity was demonstrated in vivo in the mouse air pouch injected with zymosan and arachidonic acid-induced ear inflammation. The four compounds suppressed the production of tumour necrosis factor alpha (TNFalpha) in J774 cells stimulated with lipopolysacchari…

MaleNecrosisLipopolysaccharideLeukotriene B4Anti-Inflammatory AgentsPharmacologyLeukotriene B4Dinoprostonechemistry.chemical_compoundMiceIn vivomedicineAnimalsEdemaHumansCells CulturedNitritesPharmacologyInflammationArachidonic AcidbiologyTumor Necrosis Factor-alphaZymosanGeneral MedicineHydroquinonesNitric oxide synthasechemistryBiochemistryDepression ChemicalArachidonate 5-lipoxygenaseLuminescent Measurementsbiology.proteinTumor necrosis factor alphamedicine.symptomNaunyn-Schmiedeberg's archives of pharmacology
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Formation of mono- and diglucuronides and other glycosides of benzo(a)pyrene-3,6-quinol by V79 cell-expressed human phenol UDP-glucuronosyltransferas…

1995

Glucuronidation of quinols of polycyclic aromatic hydrocarbons (PAHs) represents an important detoxication pathway preventing toxic quinone/quinol redox cycles. Therefore, mono- and diglucuronide formation of benzo(a)pyrene-3,6-quinol was investigated and compared to that of structurally related 3,6-dihydroxychrysene and simple phenols (1-naphthol and 4-methylumbelliferone) using V79 cell-expressed human UGT1.6 (= P1) and human UGT1.7 (= P4). Properties of human UGT1.6 were compared to those of the rat ortholog. Cofactors related to UDP-glucuronic acid such as UDP-galacturonic acid and UDP-glucose were also studied. It was found that rat and human UGT1.6 and human UGT1.7 catalyse monoglucur…

MaleUridine Diphosphate GlucoseGlucuronosyltransferaseStereochemistryGlucuronidationGlucuronatesmacromolecular substancesBiochemistryIsozymeSubstrate Specificitychemistry.chemical_compoundGlucosidesAnimalsHumansPhenolsBenzopyrenesGlucuronosyltransferaseRats WistarCarcinogenPharmacologychemistry.chemical_classificationbiologyGlycosideHydroquinonesRatsQuinonechemistryBenzo(a)pyreneBiochemistryUridine Diphosphate Glucuronic Acidbiology.proteinBiochemical Pharmacology
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Inhibition of Xanthine Oxidase by Phenolic Conjugates of Methylated Quinic Acid

2003

The caffeoyl conjugates of prenylhydroquinone glucoside and of quinic acid, either in the carboxyl-free or carboxymethyl forms, isolated from Phagnalon rupestre (Asteraceae), showed inhibitory activity on lipid peroxidation induced by Fe 2+/ascorbate and by CCl4/NADPH in rat liver microsomes, with IC50 values ranging from 3 to 11 microM. After having demonstrated their effect on the xanthine oxidase-regulated superoxide production, the active compounds were tested for the direct inhibition of this enzyme. Methylated dicaffeoylquinic conjugates competitively inhibited the enzyme and the highest potency was obtained for the 4,5-diester, with an IC50 value of 3.6 microM, nearly ten times lower…

MaleXanthine OxidaseAntioxidantStereochemistrymedicine.medical_treatmentQuinic AcidPharmaceutical ScienceAsteraceaeAntioxidantsAnalytical ChemistryLipid peroxidationInhibitory Concentration 50chemistry.chemical_compoundPhenolsGlucosideDrug DiscoverymedicineAnimalsEnzyme InhibitorsRats WistarXanthine oxidasePharmacologybiologyPlant ExtractsSuperoxideOrganic ChemistryQuinic acidXanthineHydroquinonesRatsComplementary and alternative medicinechemistryBiochemistryEnzyme inhibitorMicrosomes Liverbiology.proteinMolecular MedicineLipid PeroxidationPhytotherapyPlanta Medica
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Phenylpropanoid and phenylisoprenoid metabolites from Asteraceae species as inhibitors of protein carbonylation.

2011

Abstract Three phenolic antioxidant and anti-inflammatory compounds: 7-methylaromadendrin, isoprenylhydroquinone glucoside, and 3.5-dicaffeoylquinic acid methyl ester, all isolated from Western Mediterranean Asteraceae species, have been studied for their inhibitory activity against protein carbonylation, a harmful post-translational modification of peptide chains associated with degenerative diseases. All compounds have proven to be effective, with 50% inhibitory concentration (IC 50 ) values in the micromolar range, against bovine serum albumin carbonylation caused by hypochlorite, peroxynitrite, and phorbol ester-induced leukocyte oxidative burst.

NeutrophilsProtein CarbonylationLeukocyte oxidative burstHypochloritePlant ScienceHorticultureAsteraceaeBiochemistryAntioxidantsProtein Carbonylationchemistry.chemical_compoundInhibitory Concentration 50GlucosideGlucosidesPhenolsPeroxynitrous AcidPhorbol EstersHumansBovine serum albuminMolecular BiologyRespiratory BurstFlavonoidsbiologyPhenylpropanoidCell-Free SystemSerum Albumin BovineGeneral MedicineHydroquinonesHypochlorous AcidchemistryBiochemistryFlavanonesbiology.proteinChlorogenic AcidCarbonylationProtein Processing Post-TranslationalPeroxynitritePhytochemistry
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Exogenous ochronosis and striae atrophicae following the use of bleaching creams.

2005

Exogenous ochronosis is a paradoxical hyper-pigmentation of the skin caused by the long-term use of hydroquinone-containing bleaching creams. Ochronosis is an uncommon condition characterized by yellow-brown pigmented deposits in the dermis. We report two cases of exogenous ochronosis in two female patients of the sub-Saharan African population. The lesions were characterized by an asymptomatic hyper-pigmentation of the face with gradually progressive blue-black macular patches, and in case no. 2, in addition to dyschromic lesions, striae atrophicae were present. This phenomenon is the outcome of the use of skin care products containing high concentrations of hydroquinone- and glucocorticoi…

Pathologymedicine.medical_specialtyDermatology.DermisHyperpigmentationFemale patientmedicineHumansGlucocorticoidsPigmentation disorderStriae atrophicaeSkinSkin careOchronosisExogenous ochronosisbusiness.industryMiddle Agedmedicine.diseaseHydroquinonesmedicine.anatomical_structureAfrican populationFemalesense organsDermatologic AgentsAtrophybusinessOchronosisInternational journal of dermatology
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