Search results for "IMM"

showing 10 items of 18201 documents

IL-4 induces the formation of multinucleated giant cells and expression of ?5 integrin in central giant cell lesion

2017

Background It is now well established that IL-4 has a central role in the development of monocytes to multinucleated giant cells (MGCs) by inducing the expression of integrins on the surface of monocytes. The aim of this study was to investigate the potential role of IL-4 in induction of β5 integrin expression in the peripheral blood samples of patients with giant cell granuloma. Material and Methods Monocytes were isolated from peripheral blood samples of patients with central giant cell granuloma (CGCG) and healthy controls using human Monocyte Isolation Kit II. Isolated monocytes were then cultured in the absence or presence of IL-4 (10 and 20 ng/mL), and following RNA extraction and cDN…

0301 basic medicinePathologymedicine.medical_specialtyIntegrin beta ChainsIntegrinImmunocytochemistryGiant CellsMonocytes03 medical and health sciences0302 clinical medicineGranuloma Giant CellmedicineMacrophage fusionMacrophageHumansGeneral DentistryInterleukin 4Cells CulturedOral Medicine and PathologybiologyChemistryMonocyteResearch030206 dentistrymedicine.disease:CIENCIAS MÉDICAS [UNESCO]030104 developmental biologymedicine.anatomical_structureOtorhinolaryngologyGiant cellUNESCO::CIENCIAS MÉDICASbiology.proteinSurgeryInterleukin-4Central giant-cell granuloma
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Pulmonary Adenocarcinoma With Enteric Differentiation: Immunohistochemistry and Molecular Morphology

2018

Pulmonary adenocarcinoma with enteric differentiation (PAED) is a rare subtype of lung adenocarcinoma recently recognized in the WHO classification. It is defined as an adenocarcinoma in which the enteric component exceeds 50% and have to show the expression of at least 1 immunohistochemical marker of enteric differentiation. Although the definition of this tumor type is very important, above all in the differential diagnosis between a primary lung tumor and a metastasis of colorectal adenocarcinoma, this cancer still lacks a distinctive immunohistochemical and molecular signature. We recruited the largest series in the literature of PAEDs according to the morphology and the positivity for …

0301 basic medicinePathologymedicine.medical_specialtyLung NeoplasmsHistologyintestinal-type adenocarcinomaCellular differentiationDNA Mutational AnalysisThyroid Nuclear Factor 1AdenocarcinomaBiologymedicine.disease_causePathology and Forensic MedicineMetastasisDiagnosis DifferentialProto-Oncogene Proteins p21(ras)03 medical and health sciences0302 clinical medicineKRASBiomarkers TumormedicineHumansCDX2 Transcription FactorPathology Molecularenteric lung adenocarcinoma intestinal-type adenocarcinoma CDX-2 CDX2 KRASLungKeratin-7entericCancerCell DifferentiationPulmonary adenocarcinoma with enteric differentiation (PAED)lung adenocarcinomamedicine.diseaseCDX-2ImmunohistochemistryMedical Laboratory Technology030104 developmental biologymedicine.anatomical_structureCDX2Alveolar Epithelial Cells030220 oncology & carcinogenesisMutationAdenocarcinomaImmunohistochemistryKRASDifferential diagnosisColorectal Neoplasms
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Lack of NG2 exacerbates neurological outcome and modulates glial responses after traumatic brain injury

2015

Traumatic brain injury (TBI) is a major cause of death and disability. The underlying pathophysiology is characterized by secondary processes including neuronal death and gliosis. To elucidate the role of the NG2 proteoglycan we investigated the response of NG2-knockout mice (NG2-KO) to TBI. Seven days after TBI behavioral analysis, brain damage volumetry and assessment of blood brain barrier integrity demonstrated an exacerbated response of NG2-KO compared to wild-type (WT) mice. Reactive astrocytes and expression of the reactive astrocyte and neurotoxicity marker Lcn2 (Lipocalin-2) were increased in the perilesional brain tissue of NG2-KO mice. In addition, microglia/macrophages with acti…

0301 basic medicinePathologymedicine.medical_specialtyMicrogliaTraumatic brain injurybusiness.industryNeurotoxicityPoison controlBrain damagemedicine.diseaseBlood–brain barrier03 medical and health sciencesCellular and Molecular Neuroscience030104 developmental biologymedicine.anatomical_structurenervous systemNeurologyGliosisImmunologymedicineNeurogliamedicine.symptombusinessGlia
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Sca-1+cardiac fibroblasts promote development of heart failure

2018

The causative effect of GM-CSF produced by cardiac fibroblasts to development of heart failure has not been shown. We identified the pathological GM-CSF-producing cardiac fibroblast subset and the specific deletion of IL-17A signaling to these cells attenuated cardiac inflammation and heart failure. We describe here the CD45- CD31- CD29+ mEF-SK4+ PDGFRα+ Sca-1+ periostin+ (Sca-1+ ) cardiac fibroblast subset as the main GM-CSF producer in both experimental autoimmune myocarditis and myocardial infarction mouse models. Specific ablation of IL-17A signaling to Sca-1+ periostin+ cardiac fibroblasts (PostnCre Il17rafl/fl ) protected mice from post-infarct heart failure and death. Moreover, Postn…

0301 basic medicinePathologymedicine.medical_specialtyMyocarditisIschemic cardiomyopathymedicine.medical_treatmentImmunologyInflammationCD29030204 cardiovascular system & hematologyBiologyPeriostinmedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicineCytokineHeart failurecardiovascular systemmedicineImmunology and AllergyMyocardial infarctionmedicine.symptomEuropean Journal of Immunology
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Inflammatory polyradiculoneuropathies: Clinical and immunological aspects, current therapies, and future perspectives

2020

Inflammatory polyradiculoneuropathies are heterogeneous disorders characterized by immune-mediated leukocyte infiltration of peripheral nerves and nerve roots leading to demyelination or axonal degeneration or both. Inflammatory polyradiculoneuropathies can be divided into acute and chronic: Guillain–Barré syndrome and chronic inflammatory demyelinating polyneuropathy and their variants. Despite major advances in immunology and molecular biology have been made in the last years, the pathogenesis of these disorders is not completely understood. This review summarizes the current literature of the clinical features and pathogenic mechanisms of inflammatory polyradiculoneuropathies and focuses…

0301 basic medicinePathologymedicine.medical_specialtyNerve rootImmunologylcsh:MedicineChronic inflammatory demyelinating polyneuropathymedicine.disease_causeGuillain–Barré syndromeinflammatory neuropathiesAutoimmunity03 medical and health scienceschronic inflammatory demyelinating polyneuropathy0302 clinical medicineperipheral nervous systemmedicineImmunology and AllergyGuillain-Barre syndromebusiness.industryautoimmunitylcsh:Rmedicine.diseasePeripheral030104 developmental biologymedicine.anatomical_structurePeripheral nervous systemSettore MED/26 - NeurologianeurophysiologybusinessInfiltration (medical)030217 neurology & neurosurgeryEuropean Journal of Inflammation
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Defining Ewing and Ewing-like small round cell tumors (SRCT): The need for molecular techniques in their categorization and differential diagnosis. A…

2016

Abstract Background Differentiation of Ewing sarcoma family of tumors (ESFT) and Ewing-like tumors remains problematic. Certain ESFT with morphological and immunohistochemical (IHC) profiles lack the EWSR1-ETS transcript. To improve diagnostic accuracy we investigated the presence of several specific transcripts in 200 small round cell tumors (SRCT) displaying ESFT morphology and immunophenotype in which EWSR1 FISH analysis was non-informative or negative. Design 200 tumors (formalin-fixed, paraffin-embedded) were analyzed by RT-PCR. All tumors were tested for EWSR1-ETS , EWSR1 / WT1 , PAX3 / 7-FOX01 or SYT / SSX transcripts, and the negative tumors were subsequently analyzed for CIC / DUX4…

0301 basic medicinePathologymedicine.medical_specialtyOncogene Proteins FusionDesmoplastic small-round-cell tumorCD99Sarcoma EwingBiologyTranslocation GeneticPathology and Forensic MedicineDiagnosis DifferentialFusion gene03 medical and health sciences0302 clinical medicineImmunophenotypingBiomarkers TumormedicineHumansPathology MolecularIn Situ Hybridization FluorescenceRNA-Binding ProteinsGeneral Medicinemedicine.diseaseSynovial sarcoma030104 developmental biology030220 oncology & carcinogenesisSarcoma Small CellImmunohistochemistryCalmodulin-Binding ProteinsSarcomaRNA-Binding Protein EWSDifferential diagnosisAnnals of Diagnostic Pathology
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Detection of mast cells in ameloblastomas and odontogenic keratocysts

2020

Background MCs (MCs) have been ascribed to mediating several diseases, including malignant neoplasms. These cells can play a role in angiogenesis, tissue remodeling and immune modulation and favor neoplasm progression. Despite the studies analyzing the contribution of MCs in odontogenic lesions, its biological behavior in ameloblastomas (AMBs) and odontogenic keratocysts (OKCs) remains unclear. This study aims to detect MCs in OKCs and AMBs and clarify the role of MCs in these lesions. Material and methods A total of 40 odontogenic lesions were analyzed. This included 20 OKCs and 20 AMBs, 10 being the solid type and the other 10 being the unicystic type of AMB. All cases were histologically…

0301 basic medicinePathologymedicine.medical_specialtyOral Medicine and Pathology030102 biochemistry & molecular biologybusiness.industryResearchanimal diseasesSignificant differenceImmune modulation:CIENCIAS MÉDICAS [UNESCO]humanitiesOdontogenic03 medical and health sciencesNeoplasm progression0302 clinical medicineTissue remodeling030220 oncology & carcinogenesisUNESCO::CIENCIAS MÉDICASMedicinebusinessGeneral Dentistry
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CD73 a novel marker for the diagnosis of benign and malignant salivary gland tumors

2019

Background Ecto-5’-nucleotidase (CD73) plays an important role in the development of several types of cancer; however, its prognostic significance in salivary gland tumors remains unknown. The current study was conducted to investigate the expression of CD73 in such tumors. Material and Methods In this retrospective study, immunohistochemical expression of CD73 was evaluated in 25 pleomorphic adenomas, 20 mucoepidermoid carcinomas and 20 adenoid cystic carcinomas using the Envision technique. Labeling indices of CD73 expression were calculated and compared between lesions. Results Immunohistochemical analysis demonstrated that the CD73 expression was significantly higher in salivary gland t…

0301 basic medicinePathologymedicine.medical_specialtyOral Medicine and PathologySalivary glandbusiness.industryResearchCancer:CIENCIAS MÉDICAS [UNESCO]Adenoidmedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureSalivary Gland Tissue030220 oncology & carcinogenesisNucleotidaseUNESCO::CIENCIAS MÉDICASmedicineBiomarker (medicine)ImmunohistochemistrybusinessGeneral Dentistry
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Distribution of macrophages and plasma cells in apical periodontitis and their relationship with clinical and image data

2017

BACKGROUND Macrophages and plasma cells play a key role in the regulation of innate and adaptive immunity. The aim of this study was to assess the presence of these cells in apical periodontitis and their distribution comparing with clinical and image data. MATERIAL AND METHODS Thirty-three lesions were selected and divided in two groups (17 periapical cysts and 16 periapical granulomas). Immunoreactions using anti-CD68 and anti-CD138 antibodies were carried out; image analysis was performed with an optical microscope and 5 high-power fields from each slide were evaluated leading to an average score of immunoexpression. This mean score was compared between the two groups and correlated with…

0301 basic medicinePathologymedicine.medical_specialtyPeriapical cystPeriapical GranulomaOperative Dentistry and Endodontics03 medical and health sciences0302 clinical medicineMedicineDistribution (pharmacology)General DentistryPeriodontitisbiologybusiness.industryCD68ResearchSignificant difference030206 dentistry:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseAcquired immune system030104 developmental biologyUNESCO::CIENCIAS MÉDICASbiology.proteinAntibodybusinessJournal of Clinical and Experimental Dentistry
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In Situ Activation of Pituitary-Infiltrating T Lymphocytes in Autoimmune Hypophysitis

2017

AbstractAutoimmune hypophysitis (AH) is a chronic inflammatory disease characterized by infiltration of T and B lymphocytes in the pituitary gland. The mechanisms through which infiltrating lymphocytes cause disease remain unknown. Using a mouse model of AH we assessed whether T lymphocytes undergo activation in the pituitary gland. Infiltrating T cells co-localized with dendritic cells in the pituitary and produced increased levels of interferon-γ and interleukin-17 upon stimulation in vitro. Assessing proliferation of CD3- and B220-postive lymphocytes by double immunohistochemistry (PCNA-staining) and flow cytometry (BrdU incorporation) revealed that a discrete proportion of infiltrating …

0301 basic medicinePathologymedicine.medical_specialtyPituitary glandT-LymphocytesT cellCD3Lymphocyte ActivationArticleImmunophenotypingFlow cytometryMice03 medical and health sciences0302 clinical medicineImmunophenotypingParenchymamedicineAnimalsAutoimmune HypophysitisLymphocyte CountCell ProliferationB-LymphocytesMultidisciplinarymedicine.diagnostic_testbiologybusiness.industryDendritic Cellsmedicine.diseaseImmunohistochemistry3. Good healthDisease Models Animal030104 developmental biologymedicine.anatomical_structurePituitary Gland030220 oncology & carcinogenesisbiology.proteinAutoimmune hypophysitisCytokinesFemaleInflammation MediatorsbusinessInfiltration (medical)BiomarkersScientific Reports
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