Search results for "INTERFERON-GAMMA"

showing 10 items of 352 documents

Modulation of C1q mRNA Expression and Secretion by Interleukin-1,Interleukin-6, and Interferon-g in Resident and Stimulated Murine Peritoneal Macroph…

2002

The complement system plays an important role in the humoral immune response. Activation of the classical complement pathway is mediated by its subcomponent, C1q. Among the main C1q-synthesising tissues, macrophages have been attributed as a source of particular importance. We investigated the effects of cytokines (IL-1, IL-6 and Interferon-gamma) on local C1q mRNA expression and C1q secretion in resident and in thioglycollate-stimulated murine peritoneal macrophages in vitro. The macrophages were isolated from murine peritoneal lavage fluid, maintained in culture and incubated with the cytokines. Among the cytokines, only IL-6 had a stimulatory effect on C1q production (25% increase vs. co…

ImmunologyGene Expressionchemical and pharmacologic phenomenaIn Vitro TechniquesProinflammatory cytokineInterferon-gammaMiceClassical complement pathwayImmune systemmedicineAnimalsImmunology and AllergyMacrophageInterferon gammaRNA MessengerInterleukin 6Macrophage inflammatory proteinMice Inbred BALB CbiologyInterleukin-6ChemistryComplement C1qInterleukinHematologyMacrophage ActivationRecombinant ProteinsCell biologyThioglycolatesMacrophages Peritonealbiology.proteinInterleukin-1medicine.drugImmunobiology
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Major histocompatibility complex regulation of the class of the immune response: the H-2d haplotype determines poor interferon-γ response to several …

1990

The lymph node cells of CBA (H-2k), but not BALB/c (H-2d) mice, release interferon (IFN)-gamma into the supernatant when immunized with picryl chloride epicutaneously and then exposed to antigen (haptenized cells) in vitro 4 days later. The failure in IFN-gamma production maps to the major histocompatibility complex (MHC; H-2d) in the congenic BALB/c, BALB/k and BALB/b mice. The evidence that this is an MHC regulation of the class of response to a range of antigens and not a classical Ir gene effect is (a) the difference is seen with several antigens including picryl chloride, "oxazolone" and purified protein derivative of tuberculin and (b) BALB/c mice, which fail to produce IFN-gamma, sho…

ImmunologyMice Inbred StrainsDermatitis ContactMajor histocompatibility complexMajor Histocompatibility ComplexPicryl chlorideOxazoloneInterferon-gammaMicechemistry.chemical_compoundImmune systemH-2 AntigensAntigenInterferonmedicineAnimalsImmunology and AllergyInterferon gammaHistocompatibility Antigen H-2DbiologyH-2 AntigensImmunityHaplotypeschemistryImmunologybiology.proteinmedicine.drugEuropean Journal of Immunology
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Reciprocal IFN-gamma and TGF-beta responses regulate the occurrence of mucosal inflammation.

1997

The above new findings concerning the immunological mechanisms governing mucosa, immune responses and oral tolerance in TCR-transgenic mice, as well as those operative in mice with experimental colitis, greatly expand our understanding of the processes that normally control mucosal inflammation and possibly other types of inflammation as well (Fig. 1). They indicate that, in the nondiseased mouse, ingested proteins evoke a Th1-cell (IFN-gamma) response in the mucosal follicles that is quickly counter-regulated by induction of T-cell anergy/deletion, if this Th1-cell response is inhibited (experimentally by anti-IL-12), TGF beta-producing cells appear, and these are capable of active immune …

ImmunologyMucosal inflammationGastrointestinal inflammationExperimental colitisBiologyColitisProinflammatory cytokineInterferon-gammaInterferon γTransforming Growth Factor betaImmunologyAnimalsHumansIntestinal MucosaOral toleranceTransforming growth factorImmunology today
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Dextrans produced by lactic acid bacteria exhibit antiviral and immunomodulatory activity against salmonid viruses

2015

36 p.-7 fig.-1 tab.-1 fig. supl.

Infectious hematopoietic necrosis virusSpectrophotometry InfraredPolymers and PlasticsInfectious hematopoietic necrosis virusTroutIHNVSalmonid virusAquacultureIPNVAntiviral AgentsVirusCell LineMicrobiologyInterferon-gammaExopolysaccharideIn vivoLactobacillusLactic acid bacteriaMaterials ChemistryAnimalsImmunologic FactorsAntiviralDextranInfectious pancreatic necrosis virusbiologyOrganic Chemistryfood and beveragesDextransInfectious pancreatic necrosis virusbiology.organism_classificationImmunostimulantsIn vitroLactobacillus sakeiMolecular WeightLactobacillusBiochemistryLeuconostoc mesenteroidesInterferon Type ISalmonidaeCarbohydrate Polymers
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Updated insights into the mechanism of action and clinical profile of the immunoadjuvant QS-21: A review

2019

Background Vaccine adjuvants are compounds that significantly enhance/prolong the immune response to a co-administered antigen. The limitations of the use of aluminium salts that are unable to elicite cell responses against intracellular pathogens such as those causing malaria, tuberculosis, or AIDS, have driven the development of new alternative adjuvants such as QS-21, a triterpene saponin purified from Quillaja saponaria. Purpose The aim of this review is to attempt to clarify the mechanism of action of QS-21 through either receptors or signaling pathways in vitro and in vivo with special emphasis on the co-administration with other immunostimulants in new adjuvant formulations, called a…

InflammasomesT-Lymphocytesmedicine.medical_treatmentHerpes zosterPharmaceutical ScienceMonophosphoryl Lipid AAPCs antigen presenting cellsMiceCMI cell mediated immunity0302 clinical medicineDrug DiscoveryHerpes Zoster VaccineMedicineNSCLC non small cell lung carcinomaCancerImmunity CellularVaccines Synthetic0303 health sciencesImmunogenicityIl-2 interleukine 2HIV human immunodeficiency virusLipid A030220 oncology & carcinogenesisCytokinesMolecular MedicineDCs dendritic cellsNK natural killerAdjuvantTLR Toll-like receptorHerpes Zoster VaccineCD cluster of differentiationAntigen-Presenting CellsCTL cytotoxic T lymphocytesHZ herpes zosterMPL 3-deacylated monophosphoryl lipidVaccine adjuvantImmunoadjuvantArticleVZV varicella zoster virus03 medical and health sciencesImmune systemAdjuvants ImmunologicAntigenPAMPs pathogen-associated molecular patternsMalaria VaccinesPRRs pathogen recognition receptorsQS-21 Quillaja saponaria Molina-fraction 21AnimalsMHC major histocompatibility complexMtb Mycobacterium tuberculosis bacteriaSARS severe acute respiratory syndromeAntigen-presenting cellIFN-γ interferon-gamma030304 developmental biologyPharmacologybusiness.industryA-β amyloid-betaTNF-α tumor necrosis factor-alphaSaponinsQS-21MalariaQuillaja saponariaComplementary and alternative medicineTCR T-cell receptorLiposomesImmunologyKLH keyhole limpet hemocyaninbusinessdLN draining lymph nodesMAPK mitogen activated protein kinasePhytomedicine
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Correction to: Depletion of regulatory T cells increases T cell brain infiltration, reactive astrogliosis, and interferon-γ gene expression in acute …

2019

Traumatic brain injury (TBI) is a major cause of death and disability. T cells were shown to infiltrate the brain during the first days after injury and to exacerbate tissue damage. The objective of this study was to investigate the hitherto unresolved role of immunosuppressive, regulatory T cells (Tregs) in experimental TBI."Depletion of regulatory T cell" (DEREG) and wild type (WT) C57Bl/6 mice, treated with diphtheria toxin (DTx) to deplete Tregs or to serve as control, were subjected to the controlled cortical impact (CCI) model of TBI. Neurological and motor deficits were examined until 5 days post-injury (dpi). At the 5 dpi endpoint, (immuno-) histological, protein, and gene expressio…

InflammationGeneral NeuroscienceImmunologyCorrectionBrainT-Lymphocytes Regulatorylcsh:RC346-429Lymphocyte DepletionCellular and Molecular NeuroscienceDisease Models AnimalInterferon-gammaMiceNeurologyAstrocytesBrain Injuries TraumaticAnimalsGliosislcsh:Neurology. Diseases of the nervous systemJournal of Neuroinflammation
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Immunologic Effects of Interferon

1990

Interferons can be defined as a family of induced proteins sharing the capacity to exert pleiotropic effects on cell functions and to render cells resistant to virus infection. They are activating genes coding for a number of enzymes, most of which have not yet been characterized, and also by enhancing the synthesis of cell surface components. This enables interferons to modulate the immune response at different levels. This article will focus on the effects of interferon on antigen presentation, regulation of the immune response, activation of macrophage functions, and on its role in the pathogenesis of some diseases.

InflammationInterleukin 2Immunity CellularMacrophagesCellAntigen presentationCell BiologyDermatologyBiologyBiochemistryInterferon-gammamedicine.anatomical_structureImmune systemInterferonImmune SystemImmunologymedicineAnimalsHumansMacrophageTumor necrosis factor alphaInterferonsMolecular Biologymedicine.drugInterferon regulatory factorsJournal of Investigative Dermatology
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Drug-induced expansion and differentiation of Vγ9Vδ2 T cells in vivo: The role of exogenous IL-2

2005

Human Vgamma9Vdelta2 T cells recognize nonpeptidic Ags generated by the 1-deoxy-d-xylulose 5-phosphate (many eubacteria, algae, plants, and Apicomplexa) and mevalonate (eukaryotes, archaebacteria, and certain eubacteria) pathways of isoprenoid synthesis. The potent Vgamma9Vdelta2 T cell reactivity 1) against certain cancer cells or 2) induced by infectious agents indicates that therapeutic augmentations of Vgamma9Vdelta2 T cell activities may be clinically beneficial. The functional characteristics of Vgamma9Vdelta2 T cells from Macaca fascicularis (cynomolgus monkey) are very similar to those from Homo sapiens. We have found that the i.v. administration of nitrogen-containing bisphosphonat…

Injections SubcutaneousT cellImmunologyCD4-CD8 RatioPamidronateBiologyPharmacologyInterferon-gammaInterleukin 21HemiterpenesOrganophosphorus CompoundsT-Lymphocyte SubsetsmedicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigensAntigen-presenting cellCells CulturedCell ProliferationInterleukin 323-DiphosphoglycerateDiphosphonatesZAP70Cell DifferentiationReceptors Antigen T-Cell gamma-deltaTh1 CellsNatural killer T cellDiphosphatesMacaca fascicularismedicine.anatomical_structureInjections IntravenousImmunologyEpoxy CompoundsInterleukin-2Immunologic Memory
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Multifunctional CD4(+) T cells correlate with active Mycobacterium tuberculosis infection.

2010

Th1 CD4(+) T cells and their derived cytokines are crucial for protection against Mycobacterium tuberculosis. Using multiparametic flow cytometry, we have evaluated the distribution of seven distinct functional states (IFN-gamma/IL-2/TNF-alpha triple expressors, IFN-gamma/IL-2, IFN-gamma/TNF-alpha or TNF-alpha/IL-2 double expressors or IFN-gamma, IL-2 or TNF-alpha single expressors) of CD4(+) T cells in individuals with latent M. tuberculosis infection (LTBI) and active tuberculosis (TB). We found that triple expressors, while detectable in 85-90%TB patients, were only present in 10-15% of LTBI subjects. On the contrary, LTBI subjects had significantly higher (12- to 15-fold) proportions of…

Interleukin 2AdultCD4-Positive T-LymphocytesMaleTuberculosisSettore MED/17 - Malattie InfettiveImmunologyCell SeparationBiologyLymphocyte ActivationFlow cytometryMycobacterium tuberculosis03 medical and health sciences0302 clinical medicineImmune systemBacterial ProteinsCD4(+) T cells Cytokines Mycobacterium tuberculosis infection Tuberculosis disease interferon-gamma immunological memory disease responses protection cytokine immunity bcg vaccination virusmedicineImmunology and AllergyDistribution (pharmacology)HumansCytokineTuberculosis Pulmonary030304 developmental biologyTuberculosis disease.Settore MED/04 - Patologia Generale0303 health sciencesAntigens Bacterialmedicine.diagnostic_testMycobacterium tuberculosis infectionMycobacterium tuberculosisMiddle Agedbiology.organism_classificationmedicine.diseaseFlow CytometryPhenotypeVirologyCD4+ T cellsBacterial Load3. Good healthImmunologyAcute DiseaseChronic DiseaseCytokinesTumor necrosis factor alphaAcyltransferases030215 immunologymedicine.drugEuropean journal of immunology
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Abnormalities in serum concentrations of interleukin-2, interferon-alpha and interferon-gamma in schizophrenia not detected.

1992

The hypothesis of an immunological defect in schizophrenia has been supported by reports on abnormal production of interleukin-2 (IL-2) and interferons (IFNs) in schizophrenic patients. In the present study we determined the serum concentrations of IL-2, IFN-alpha and IFN-gamma in 10 first onset, neuroleptic-naive schizophrenics, in 6 pretreated patients who were drug free (1 week to 2 years) at the time of the investigation and in 15 matched healthy controls. No IFN-alpha was detected in schizophrenics' and in control sera. No differences were found in IL-2 and IFN-gamma levels between schizophrenics and controls. Thus the present study failed to support the hypothesis of an immunological …

Interleukin 2AdultMalePsychosisAlpha interferonInterferon-gammaReference ValuesInterferon αmedicineImmune ToleranceHumansInterferon gammaBiological PsychiatryInterferon alfaPsychiatric Status Rating ScalesSchizophrenia Paranoidbusiness.industryInterferon-alphaSerum concentrationmedicine.diseasePsychiatry and Mental healthSchizophreniaImmunologySchizophreniaInterleukin-2FemaleSchizophrenic Psychologybusinessmedicine.drugSchizophrenia research
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