Search results for "Immune System"

showing 10 items of 2885 documents

Elevated levels of Bcl-3 inhibits Treg development and function resulting in spontaneous colitis

2017

Bcl-3 is an atypical NF-κB family member that regulates NF-κB-dependent gene expression in effector T cells, but a cell-intrinsic function in regulatory T (Treg) cells and colitis is not clear. Here we show that Bcl-3 expression levels in colonic T cells correlate with disease manifestation in patients with inflammatory bowel disease. Mice with T-cell-specific overexpression of Bcl-3 develop severe colitis that can be attributed to defective Treg cell development and function, leading to the infiltration of immune cells such as pro-inflammatory γδT cells, but not αβ T cells. In Treg cells, Bcl-3 associates directly with NF-κB p50 to inhibit DNA binding of p50/p50 and p50/p65 NF-κB dimers, t…

AdultMale0301 basic medicineP50ScienceGeneral Physics and AstronomyBiologyT-Lymphocytes RegulatoryInflammatory bowel diseaseArticleGeneral Biochemistry Genetics and Molecular BiologyYoung Adult03 medical and health sciences0302 clinical medicineImmune systemB-Cell Lymphoma 3 ProteinProto-Oncogene ProteinsGene expressionmedicineAnimalsHumansColitisMice KnockoutRegulation of gene expressionMultidisciplinaryEffectorHEK 293 cellsQNF-kappa BTranscription Factor RelANF-kappa B p50 SubunitGeneral ChemistryMiddle AgedColitismedicine.diseaseMice Inbred C57BLHEK293 Cells030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisImmunologyFemaleProtein BindingTranscription FactorsNature Communications
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Anti-Anisakis sp. antibodies in serum of healthy subjects. Relationship with αβ and γδ T cells

2016

Anisakiosis is nowadays one of the nematodoses more prevalent in Spain, with rates that oscillate between 0.43% in Galicia (N.W. Spain), and 15.7% and 22.1% in inland and southern regions, respectively. Likewise, it has been proved that Anisakis larvae have developed mechanisms to modulate the dichotomy of the host immune response for their own benefit. The experimental hypothesis of the present study was that Anisakis sp. larval products can be mediators of immune suppression and induce changes on the populations of αβ+ and γδ+ T cells. In the present study we determined the levels of anti-Anisakis antibodies in the serum of healthy people, and their relationship with the B and T cell subs…

AdultMale0301 basic medicineReceptors Antigen T-Cell alpha-betaT cellAntibodies HelminthAnisakiasisImmunoglobulin E03 medical and health sciencesImmune systemAntigenT-Lymphocyte SubsetsmedicineAnimalsHumansB cellAgedAged 80 and overbiologyReceptors Antigen T-Cell gamma-deltaMiddle Aged030108 mycology & parasitologyNatural killer T cellAnisakis030104 developmental biologymedicine.anatomical_structureSpainImmunologybiology.proteinFemaleParasitologyAntibodyCD8Acta Parasitologica
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Kinetics of torque teno virus DNA load in saliva and plasma following allogeneic hematopoietic stem cell transplantation

2018

Plasma torque teno virus (TTV) DNA load directly correlates with the degree of T-cell immune reconstitution early after allogeneic hematopoietic stem cell transplantation (allo-HSCT). Here, the kinetics of oral TTV DNA shedding was examined to assess whether quantitation of TTV DNA load in saliva may either replace or complement that in plasma for predicting lymphocyte (ALC) reconstitution after engraftment. This prospective observational study enrolled 38 nonconsecutive allo-HSCT recipients. Saliva and plasma specimens were collected at baseline (pretransplant) and at around days +30, +50, and +90 after allo-HSCT. TTV DNA was quantitated in both specimen types by real-time PCR. ALCs were m…

AdultMale0301 basic medicineTorque teno virusSalivaOral TTV DNA sheddingLymphocytemedicine.medical_treatmentTTV DNAemiaAllogeneic hematopoietic stem cell transplantation (allo-HSCT)Hematopoietic stem cell transplantationReal-Time Polymerase Chain ReactionTorque teno virus (TTV)Plasma03 medical and health sciences0302 clinical medicineImmune systemVirologymedicineHumansTransplantation HomologousProspective StudiesAllogeneic hematopoietic stem cell transplantation (allo-HSCT); Immune reconstitution; Oral TTV DNA shedding; Saliva; Torque teno virus (TTV); TTV DNAemia; Virology; Infectious DiseasesSalivaAgedTorque teno virusbusiness.industryHematopoietic Stem Cell TransplantationMiddle AgedImmune reconstitutionVirologyDNA Virus InfectionsTransplantation030104 developmental biologyInfectious Diseasesmedicine.anatomical_structureReal-time polymerase chain reactionDNA ViralFemalebusinessCytometry030215 immunologyJournal of Medical Virology
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Downregulation of miRNA17-92 cluster marks Vγ9Vδ2 T cells from patients with rheumatoid arthritis

2018

Abstract Background We aimed to evaluate the phenotype, function, and microRNA (miRNA)17–92 cluster expression in Vγ9Vδ2 T-cell subsets and the correlation with immune response in rheumatoid arthritis (RA) patients. Methods Peripheral blood from 10 early RA untreated patients and 10 healthy donors (HD) was obtained. Polyclonal Vγ9Vδ2 T-cell lines were generated and analysed by flow cytometry. Analysis of miRNA17–92 cluster expression was performed by real-time polymerase chain reaction (RT-PCR), and expression of mRNA target genes was also studied. Results A remarkable change in the distribution of Vγ9Vδ2 T-cell functional subsets was observed in the peripheral blood of RA patients compared…

AdultMale0301 basic medicinemiRNA17–92lcsh:Diseases of the musculoskeletal systemInflammatory cytokineImmunologyDown-RegulationBiologyγδ T cellsProinflammatory cytokineFlow cytometryArthritis RheumatoidPathogenesis03 medical and health sciences0302 clinical medicineImmune systemRheumatologyT-Lymphocyte SubsetsInflammatory cytokines; miRNA17-92; Rheumatoid arthritis; γδ T cells; Rheumatology; Immunology and Allergy; ImmunologymicroRNAmedicineHumansImmunology and AllergyRheumatoid arthritisRheumatoid arthritiγδ T cellmedicine.diagnostic_testEffectorInterleukinMiddle AgedInflammatory cytokinesPhenotypemiRNA17-92MicroRNAsSettore MED/16 - Reumatologia030104 developmental biology030220 oncology & carcinogenesisImmunologyFemalelcsh:RC925-935Research Article
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Pattern analysis of human cutaneous mast cell populations by total body surface mapping.

2003

SummaryBackground Mast cells (MCs), critical effector cells in allergic inflammation and innate immunity to bacteria, are located in large numbers in tissues that interface the external environment, including the skin. However, little is known about the distribution and numbers of human skin MCs. Objectives To assess the influence of age, sex and skin region on size and spatial distribution of MC populations in normal human skin. Methods Biopsies of healthy skin were obtained from 150 male and female individuals (age range 10–86 years). MCs were quantified and mapped planimetrically by histomorphometry in 15 anatomical sites (abdomen, thorax, lower and upper back, lower and upper arm, lower…

AdultMaleAdolescentBody Surface AreaBiopsyHuman skinCell CountDermatologyBiologyImmune systemSex FactorsmedicineHumansMast CellsChildNoseAgedSkinBody surface areaAged 80 and overintegumentary systemAge FactorsAnatomyCheekMiddle AgedMast cellmedicine.anatomical_structureAbdomenFemaleTotal body surface areaThe British journal of dermatology
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Gentamicin, norfloxacin and lysozyme concentration in human tears: in vivo and in vitro study

1992

Hen's egg lysozyme (HEL) activity was measured in vitro with gentamicin and norfloxacin by a turbidimetric technique. Gentamicin at the concentration of 10(-3) M inhibited HEL activity by 39%, while 10(-3) M norfloxacin did not affect HEL activity. However, an in vivo study in healthy persons did not show any significant statistical difference in tear lysozyme activity when 0.3% gentamicin or 0.3% norfloxacin were topically applied.

AdultMaleAdolescentEgg lysozymechemical and pharmacologic phenomenaIn Vitro TechniquesgentamicinPharmacologyMicrobiologyCorneachemistry.chemical_compoundIn vivomedicineHumansIn vitro studyheterocyclic compounds[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory OrganslysozymeNorfloxacinocular surface[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyChemistryhemic and immune systemsGeneral Medicinebiochemical phenomena metabolism and nutritioninfectionIn vitroOphthalmology[SDV.MHEP.OS] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansTearsTearsMuramidaseGentamicinGentamicinsOphthalmic SolutionsLysozyme[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyNorfloxacinmedicine.drugActa Ophthalmologica
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Gluten Stimulation Induces an in vitro Expansion of Peripheral Blood Tγδ Cells from HLA-DQ2-Positive Subjects of Families of Patients with Celiac Dis…

1998

The intestinal gluten sensitivity formally known as celiac disease (CD) is characterized by an evident involvement of local immune response and it is associated with the expression of HLA-DQ2 allele. The major role in the disease seems to be played by the T lymphocyte population bearing gamma delta T cell receptor (T gamma delta cells) which are increased both in peripheral blood and intestinal mucosae of celiac patients. In this paper data on the effects of in vitro gluten stimulation on lymphocytes expressing the T gamma delta phenotype are reported. Gluten seems to be able to induce the expansion of the T gamma delta cell population both in CD patients and their HLA-DQ2-positive asymptom…

AdultMaleAdolescentGlutensT-LymphocytesImmunologyEnzyme-Linked Immunosorbent AssayStimulationDiseasePolymerase Chain ReactionImmune systemHLA-DQ AntigensGeneticsHumansMedicineReceptors ImmunologicAlleleChildCells CulturedGenetics (clinical)chemistry.chemical_classificationbusiness.industryHLA-DQ2nutritional and metabolic diseasesMiddle AgedGlutendigestive system diseasesIn vitroPeripheral bloodCeliac DiseasePhenotypechemistryChild PreschoolImmunologyLeukocytes MononuclearFemaleInterleukin-4businessExperimental and Clinical Immunogenetics
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Hypoxic macrophages impair autophagy in epithelial cells through Wnt1: relevance in IBD.

2014

A defective induction of epithelial autophagy may have a role in the pathogenesis of inflammatory bowel diseases. This process is regulated mainly by extracellular factors such as nutrients and growth factors and is highly induced by diverse situations of stress. We hypothesized that epithelial autophagy is regulated by the immune response that in turn is modulated by local hypoxia and inflammatory signals present in the inflamed mucosa. Our results reveal that HIF-1 alpha and Wnt1 were co-localized with CD68 in cells of the mucosa of IBD patients. We have observed increased protein levels of beta-catenin, phosphorylated mTOR, and p62 and decreased expression of LC3II in colonic epithelial …

AdultMaleAdolescentImmunologyWnt1 ProteinBiologyYoung AdultImmune systemAutophagyExtracellularHumansImmunology and AllergyIntestinal MucosaWNT1Wnt Signaling PathwayPI3K/AKT/mTOR pathwayRegulation of gene expressionCD68MacrophagesTOR Serine-Threonine KinasesAutophagyWnt signaling pathwayEpithelial CellsMiddle AgedHypoxia-Inducible Factor 1 alpha SubunitInflammatory Bowel DiseasesCell HypoxiaCell biologyGene Expression RegulationFemale
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Regulation of the neutralizing anti-hepatitis B surface (HBs) antibody response in vitro in HBs vaccine recipients and patients with acute or chronic…

1996

Abstract Antibodies directed to the HBs antigen indicate viral clearance and the development of life-long immunity in patients that recovered from HBV infection. In HBs antigen vaccine recipients anti-HBs antibodies provide protective immunity. However, little is known about the regulation of this HBs-specific antibody response. The existence of anti-HBs-secreting B cells was demonstrated using the highly sensitive ELISPOT technique compared with conventional ELISA in serum and cell culture supernatants. In the peripheral blood of patients with acute self-limited hepatitis B, HBs-specific B cells were demonstrated with a high frequency despite undetectable anti-HBs serum antibodies. HBV-imm…

AdultMaleAdolescentT-LymphocytesImmunologymedicine.disease_cause03 medical and health sciences0302 clinical medicineImmune systemNeutralization TestsImmunityHumansImmunology and AllergyMedicineHepatitis B VaccinesHepatitis B AntibodiesRapid PublicationsB cellAgedHepatitis Chronic030304 developmental biologyHepatitis B virusB-LymphocytesImmunity Cellular0303 health sciencesHepatitis B Surface Antigensbiologybusiness.industryELISPOTvirus diseasesMiddle AgedHepatitis BHepatitis Bmedicine.diseasebiology.organism_classificationVirologydigestive system diseases3. Good healthmedicine.anatomical_structureHepadnaviridaeAcute DiseaseImmunologybiology.proteinFemale030211 gastroenterology & hepatologyAntibodybusinessClinical and Experimental Immunology
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Distinct single-component adjuvants steer human DC-mediated T-cell polarization via Toll-like receptor signaling toward a potent antiviral immune res…

2021

Significance Vaccines profit from the addition of adjuvants to better and more specifically initiate, amplify, and shape immune responses. Although the number of adjuvant candidates has steadily increased, peaking in the current SARS-CoV-2 pandemic, little is known about their inherent mode of action. Using human blood immune cells, we established a multilayer method to systematically assess the adjuvants’ effects on innate and adaptive immune cells. By employing a multiplex analysis with cells from 30 different donors, we determined important patterns of adjuvant function. Moreover, we demonstrate correlates of an antiviral immune response using a Toll-like receptor 7/8 ligand adjuvant and…

AdultMaleAdolescentT-LymphocytesMonophosphoryl Lipid ALipid Achemistry.chemical_compoundImmunology and InflammationImmune systemAdjuvants ImmunologicInterferonTLRmedicineHumansprimary human cellsAgedImmunity CellularToll-like receptorMultidisciplinarySARS-CoV-2ChemistryToll-Like ReceptorsImidazolesCOVID-19Dendritic CellsTLR7biochemical phenomena metabolism and nutritionBiological SciencesMiddle AgedCOVID-19 ; TLR ; primary human cells ; adjuvants ; mRNA vaccines420Cell biologymRNA vaccinesLipid AadjuvantsTLR4[SDV.IMM]Life Sciences [q-bio]/ImmunologyFemaleResiquimodmedicine.drugProceedings of the National Academy of Sciences
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