Search results for "Immune system"

showing 10 items of 2885 documents

Inhibition of human allergic T-cell responses by IL-10–treated dendritic cells: Differences from hydrocortisone-treated dendritic cells

2001

Abstract Background: Dendritic cells (DCs) are able to induce human allergic T H 1 responses as well as T H 2 responses. Objective: In this study, we examined the effect of antiinflammatory agents such as IL-10 and hydrocortisone (HC) on the accessory function of DCs and the resulting T-cell response, especially that of T H 2 cells. Methods: Naive and memory CD4 + T cells from atopic donors were stimulated with autologous allergen-pulsed DCs generated from CD14 + monocytes by culture with GM-CSF/IL-4 and fully matured with IL-1β, TNF-α, and PGE 2 in the presence or absence of IL-10 or HC. Results: IL-10–treated DCs and, to a lesser extent, HC-treated DCs showed a decreased expression of MHC…

CD4-Positive T-LymphocytesHypersensitivity ImmediateHydrocortisoneT-LymphocytesCD14T cellImmunologyAntigen presentationAnti-Inflammatory Agentschemical and pharmacologic phenomenaBiologyInterferon-gammaTh2 CellsmedicineHumansImmunology and AllergyAntigen-presenting cellCD86Antigen PresentationModels Immunologicalhemic and immune systemsDendritic CellsDendritic cellT lymphocyteAllergensInterleukin-10Interleukin 10medicine.anatomical_structureImmunologyCytokinesInterleukin-4Interleukin-5Immunologic MemoryJournal of Allergy and Clinical Immunology
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Modification of the human allergic immune response by allergen-DNA-transfected dendritic cells in vitro.

2004

Abstract Background Atopic-allergic diseases are characterized by T H 2-dominated immune responses, resulting in IgE production. DNA-based immunotherapies have been shown to shift the immune response toward a T H 1-type response in animal models. Objective The aim of the study was to analyze whether dendritic cells (DCs) transfected with allergen-DNA conjugates are able to stimulate human autologous CD4 + T cells, CD8 + T cells, or both from atopic individuals to produce T H 1 cytokines instead of T H 2 cytokines. Methods For this purpose, human mature DCs from atopic donors were transfected with an adenovirus encoding the allergen Phl p 1. Autologous CD4 + and CD8 + T cells were stimulated…

CD4-Positive T-LymphocytesHypersensitivity Immediatemedicine.medical_treatmentImmunologyGenetic Vectorschemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationTransfectionInterleukin 21Interferon-gammaImmune systemmedicineImmunology and AllergyCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellCells CulturedPlant ProteinsAdenoviruses HumanDendritic cellDendritic CellsAllergensTh1 CellsMolecular biologyCytokineImmunologyCytokinesCD8The Journal of allergy and clinical immunology
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The Regulatory T Cell Family: Distinct Subsets and their Interrelations

2003

The immune system, a highly effective and dynamic cellular network, protects a host from pathogens. Therefore, the immune system must distinguish self from nonself structures, but also between harmful and innocuous foreign Ags to prevent nonessential and self-destructive immune responses. The

CD4-Positive T-LymphocytesImmunity CellularRegulatory T cellanimal diseasesImmunologyReceptors Interleukin-2chemical and pharmacologic phenomenaCell Communicationbiochemical phenomena metabolism and nutritionBiologyImmunity Innatemedicine.anatomical_structureImmune systemT-Lymphocyte SubsetsImmunityImmunologymedicineAnimalsHumansbacteriaImmunology and AllergyReceptorThe Journal of Immunology
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Cutting Edge: TGF-β Induces a Regulatory Phenotype in CD4+CD25− T Cells through Foxp3 Induction and Down-Regulation of Smad7

2004

Abstract CD4+CD25+ regulatory cells are a subpopulation of T lymphocytes of thymic origin. However, recent data suggest an alternative commitment of regulatory T cells in the periphery, although the precise mechanism is unknown. In the present work, we demonstrate that TGF-β is able to induce Foxp3 expression and subsequently a regulatory phenotype in CD4+CD25− peripheral murine T cells. Similarly, TGF-β induced Foxp3 in human CD4+CD25− T cells. Moreover, we show that the inhibitory Smad7 protein that is normally induced by TGF-β and limits TGF-β signaling, is strongly down-regulated by Foxp3 at the transcriptional level. Foxp3-mediated down-regulation of Smad7 subsequently rendered CD4+CD2…

CD4-Positive T-LymphocytesImmunologyDown-Regulationchemical and pharmacologic phenomenaThymus GlandBiologyImmunophenotypingSmad7 ProteinMiceInterleukin 21Downregulation and upregulationT-Lymphocyte SubsetsTransforming Growth Factor betaTGF beta signaling pathwayAnimalsHumansImmunology and AllergyCytotoxic T cellIL-2 receptorCells CulturedZAP70FOXP3Cell DifferentiationForkhead Transcription FactorsReceptors Interleukin-2hemic and immune systemsPhenotypeCell biologyDNA-Binding ProteinsTrans-ActivatorsSpleenSignal TransductionThe Journal of Immunology
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MHC class II tetramer guided detection of Mycobacterium tuberculosis-specific CD4+ T cells in peripheral blood from patients with pulmonary tuberculo…

2007

Novel diagnostic tools are needed to diagnose latent infection and to provide biologically meaningful surrogate markers to define cellular immune responses against Mycobacterium tuberculosis (MTB). Interferon gamma-based assays have recently been developed in addition to the more than 100-year-old tuberculin skin test (TST) for the immune diagnosis of MTB in blood. The advent of soluble MHC/peptide tetramer molecules allows to objectively enumerate antigen-specific T cells. We identified novel MHC class II-restricted MTB epitopes and used HLA-DR4 tetrameric complexes to visualize ex vivo CD4(+) T cells directed against the antigens Ag85B and the 19-kDa lipoprotein, shared between MTB and ot…

CD4-Positive T-LymphocytesImmunologyMolecular Sequence DataEpitopes T-Lymphocytechemical and pharmacologic phenomenaMajor histocompatibility complexEpitopeImmune systemAntigenMHC class IHumansAmino Acid SequenceTuberculosis PulmonaryMHC class IIAntigen PresentationAntigens BacterialbiologyHistocompatibility Antigens Class IICD28General MedicineMycobacterium tuberculosisrespiratory systembacterial infections and mycosesVirologyImmunologybiology.proteinCD8Scandinavian journal of immunology
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Differential Regulatory Capacity of CD25+ T Regulatory Cells and Preactivated CD25+ T Regulatory Cells on Development, Functional Activation, and Pro…

2004

Abstract CD25+ T regulatory (Treg) cells play a central role regarding the maintenance of peripheral tolerance via suppression of autoaggressive CD4+ T cells, CD8+ T cells, and Th1 cells. In this study we demonstrate that CD25+ Treg cells can also suppress the differentiation of murine conventional CD4+ T cells toward Th2 cells in a contact-dependent manner. However, the cytokine production and proliferation of established Th2 cells could not be inhibited by freshly isolated CD25+ Treg cells, whereas a strong inhibition of differentiated Th2 cells by in vitro preactivated CD25+ Treg cells could be observed. Inhibition of both conventional CD4+ T cells and Th2 cells is accompanied by a stron…

CD4-Positive T-LymphocytesImmunologySuccinimideschemical and pharmacologic phenomenaLymphocyte ActivationMiceInterleukin 21Th2 CellsT-Lymphocyte SubsetsAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellInterleukin 3Mice Inbred BALB CCD40biologyPeripheral toleranceForkhead Transcription FactorsReceptors Interleukin-2hemic and immune systemsFluoresceinsCell biologyDNA-Binding ProteinsMice Inbred C57BLbiology.proteinInterleukin 12CytokinesThe Journal of Immunology
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Asthmatic changes in mice lacking T-bet are mediated by IL-13

2005

Mice with a targeted deletion of the T-bet gene exhibit spontaneous airway hyperresponsiveness (AHR), airway inflammation, enhanced recovery of T(h)2 cytokines from bronchoalveolar lavage fluid, sub-epithelial collagen deposition and myofibroblast transformation. Here we analyze the mechanisms responsible for the chronic airway remodeling observed in these mice. CD4+ T cells isolated from the lung of T-bet-deficient mice were spontaneously activated CD44(high)CD69(high) memory T cells, with a typical T(h)2 cytokine profile. Neutralization of IL-13 but not IL-4 resulted in amelioration of AHR in airways of mice lacking T-bet. IL-13 blockade also led to reduced eosinophilia and decreased vime…

CD4-Positive T-LymphocytesImmunologychemical and pharmacologic phenomenaVimentinLymphocyte ActivationSmad7 ProteinMiceTransforming Growth Factor betamedicineAnimalsVimentinImmunology and AllergyEosinophiliaSmad3 ProteinLungCells CulturedMice KnockoutInterleukin-13Lungbiologymedicine.diagnostic_testChemistryCD69hemic and immune systemsGeneral MedicineTransforming growth factor betaFibroblastsrespiratory systemActinsAsthmarespiratory tract diseasesDNA-Binding ProteinsMice Inbred C57BLBronchoalveolar lavagemedicine.anatomical_structureInterleukin 13ImmunologyTrans-Activatorsbiology.proteinCytokinesInterleukin-4medicine.symptomT-Box Domain ProteinsImmunologic MemoryMyofibroblastTranscription FactorsInternational Immunology
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Inhibitors of β-catenin affect the immuno-phenotype and functions of dendritic cells in an inhibitor-specific manner

2015

Many tumors are characterized by mutation-induced constitutive activation of β-catenin which promotes tumor growth and survival. Consequently, the development of specific β-catenin inhibitors for tumor therapy has come into the focus of drug development. β-Catenin was also shown to contribute to the tolerance-promoting function of unstimulated dendritic cells (DCs). In response to activation, DCs acquire potent T cell stimulatory capacity and induce profound tumor antigen-specific immune responses. Here we asked for effects of pre-clinically established β-catenin inhibitors (CCT-031374, iCRT-5, PNU-75654) on mouse bone marrow-derived (BM)DCs. All three inhibitors moderately increased surfac…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineCell SurvivalOvalbuminT cellImmunologyPopulationAntineoplastic AgentsBone Marrow CellsMice Transgenicchemical and pharmacologic phenomena03 medical and health sciencesImmune systemmedicineAnimalsImmunology and AllergyeducationCells Culturedbeta CateninCell ProliferationPharmacologyCD86education.field_of_studyCD40biologyFollicular dendritic cellsCell growthhemic and immune systemsDendritic CellsCell biologyMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structurebiology.proteinCytokinesCD80International Immunopharmacology
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Ci8 short, a novel LPS-induced peptide from the ascidian Ciona intestinalis,modulates responses of the human immune system

2017

The selective modulation of immunity is an emerging concept driven by the vast advances in our understanding of this crucial host defense system. Invertebrates have raised researchers’ interest as potential sources of new bioactive molecules owing to their antibacterial, anticancer and immunomodulatory activities. A LipoPolySaccharide (LPS) challenge in the ascidian Ciona intestinalis generates the transcript, Ci8 short, with cisregulatory elements in the 3′ UTR region that are essential for shaping innate immune responses. The derived amino acidic sequence in silico analysis showed specific binding to human Major Histocompatibility Complex (MHC) Class I and Class II alleles. The role of Ci…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineUntranslated regionImmunologyReceptors Antigen T-CellLymphocyte ActivationMajor histocompatibility complexInterferon-gamma03 medical and health sciences0302 clinical medicineImmune systemAnimalsHumansImmunology and AllergyCiona intestinalisClonal Selection Antigen-Mediated3' Untranslated RegionsCells CulturedCell ProliferationGeneticsZAP-70 Protein-Tyrosine KinaseInnate immune systembiologyThree prime untranslated regionT-cell receptorHematologyAcquired immune systembiology.organism_classificationHuman PBMCs Adaptive immunityT cellsImmunity InnateCiona intestinalisCell biology030104 developmental biologyLeukocytes Mononuclearbiology.proteinAntimicrobial Cationic Peptides030215 immunology
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Glycoprotein 96-activated dendritic cells induce a CD8-biased T cell response.

2005

Heat shock proteins (Hsps) are able to induce protective immune responses against pathogens and tumors after injection into immunocompetent hosts. The activation of components of the adaptive immune system, including cytotoxic T lymphocytes specific for pathogen- or tumor-derived peptides, is crucial for the establishment of immuno- protection. Hsps acquire these peptides during intracellular protein degradation and when released during necrotic cell death, facilitate their uptake and Minor Histocompatibility Complex (MHC)-restricted representation by professional antigen-presenting cells (APCs). In addition, the interaction of Hsps with APCs, including the Endoplasmatic Reticulum (ER)-resi…

CD4-Positive T-LymphocytesLipopolysaccharidesAntigen-Presenting CellsBone Marrow CellsMice TransgenicReceptors Cell SurfaceBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexLymphocyte ActivationBiochemistryMiceImmune systemHeat shock proteinCytotoxic T cellAnimalsHumansAntigen-presenting cellCells CulturedMembrane GlycoproteinsToll-Like ReceptorsCell DifferentiationCell BiologyDendritic cellDendritic CellsOriginal ArticlesAcquired immune systemLymphocyte SubsetsCell biologyMice Inbred C57BLToll-Like Receptor 4biology.proteinInflammation MediatorsCD8Signal TransductionCell stresschaperones
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