Search results for "Immune system"

showing 10 items of 2885 documents

Circulating CSF-1 Promotes Monocyte and Macrophage Phenotypes that Enhance Lupus Nephritis

2009

Macrophages mediate kidney disease and are prominent in a mouse model (MRL- Fas lpr ) of lupus nephritis. Colony stimulating factor-1 (CSF-1) is the primary growth factor for macrophages, and CSF-1 deficiency protects MRL- Fas lpr mice from kidney disease and systemic illness. Whether this renoprotection derives from a reduction of macrophages and whether systemic CSF-1, as opposed to intrarenal CSF-1, promotes macrophage-dependent lupus nephritis remain unclear. Here, we found that increasing systemic CSF-1 hastened the onset of lupus nephritis in MRL- Fas lpr mice. Using mutant MRL- Fas lpr strains that express high, moderate, or no systemic CSF-1, we detected a much higher tempo of kidne…

MaleMacrophage colony-stimulating factorMice Inbred MRL lprLupus nephritisMice TransgenicInflammationKidneyMonocytesMiceimmune system diseasesmedicineAnimalsHumansskin and connective tissue diseasesCell ProliferationInflammationMice Inbred BALB CMice Inbred C3HSystemic lupus erythematosusbiologyCD68business.industryMacrophage Colony-Stimulating FactorMacrophagesMonocyteGeneral MedicineMonocyte proliferationmedicine.diseaseLupus NephritisMice Inbred C57BLDisease Models AnimalBasic ResearchPhenotypemedicine.anatomical_structureIntegrin alpha MNephrologyImmunologybiology.proteinFemalemedicine.symptombusinessJournal of the American Society of Nephrology
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Human CD8+ T-cells Recognizing Peptides from Mycobacterium tuberculosis (Mtb) Presented by HLA-E Have an Unorthodox Th2-like, Multifunctional, Mtb In…

2015

Mycobacterial antigens are not exclusively presented to T-cells by classical HLA-class Ia and HLA-class II molecules, but also through alternative antigen presentation molecules such as CD1a/b/c, MR1 and HLA-E. We recently described mycobacterial peptides that are presented in HLA-E and recognized by CD8+ T-cells. Using T-cell cloning, phenotyping, microbiological, functional and RNA-expression analyses, we report here that these T-cells can exert cytolytic or suppressive functions, inhibit mycobacterial growth, yet express GATA3, produce Th2 cytokines (IL-4,-5,-10,-13) and activate B-cells via IL-4. In TB patients, Mtb specific cells were detectable by peptide-HLA-E tetramers, and IL-4 and…

MaleMacrophageQH301-705.5ImmunologyAntigen presentationBacterial ProteinMycobacterium tuberculosiHuman leukocyte antigenGATA3 Transcription FactorCD8-Positive T-LymphocytesMicrobiologyMicrobiologyMycobacterium tuberculosisImmune systemTh2 CellsGeneticHLA-EBacterial ProteinsVirologyGeneticsCytotoxic T cellHumansBiology (General)Th2 CellCytokineMolecular BiologyAntigen PresentationbiologyMacrophagesHistocompatibility Antigens Class ICD8-Positive T-LymphocyteMycobacterium tuberculosisRC581-607biology.organism_classificationBacterial Proteins; CD8-Positive T-Lymphocytes; Cytokines; Female; GATA3 Transcription Factor; Histocompatibility Antigens Class I; Humans; Macrophages; Male; Mycobacterium tuberculosis; Peptides; Th2 Cells; Antigen Presentation; Microbiology; Parasitology; Virology; Immunology; Genetics; Molecular BiologyPhenotypeVirology3. Good healthPeptideCytokinesParasitologyFemaleImmunologic diseases. AllergyPeptidesCD8HumanResearch ArticlePLoS Pathogens
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Autoimmune skin inflammation is dependent on plasmacytoid dendritic cell activation by nucleic acids via TLR7 and TLR9

2010

Lupus-prone mice develop a chronic inflammatory response to cutaneous injury that depends on the production of type I interferon, TLR7, and TLR9.

MaleMice 129 StrainImmunologyGene ExpressionInflammationchemical and pharmacologic phenomenaMice Inbred StrainsReceptor Interferon alpha-betaBiologySkin DiseasesArticleProinflammatory cytokinePathogenesisTLR9MiceAutoimmune skin inflammationimmune system diseasesNucleic AcidsmedicineImmunology and AllergyAnimalsLupus Erythematosus SystemicReceptorskin and connective tissue diseasesTLR7SkinAutoimmune skin inflammation; TLR7; TLR9; plasmacytoid dendritic cells.Mice KnockoutPlasmacytoid dendritic cell activationLupus erythematosusReverse Transcriptase Polymerase Chain ReactionTLR9virus diseaseshemic and immune systemsTLR7DNADendritic Cellsmedicine.diseaseFlow CytometryMice Inbred C57BLplasmacytoid dendritic cells.Toll-Like Receptor 7Toll-Like Receptor 9ImmunologyMyeloid Differentiation Factor 88CytokinesFemalemedicine.symptomThe Journal of Experimental Medicine
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Antigen-presenting cells of haematopoietic origin prime cytomegalovirus-specific CD8 T-cells but are not sufficient for driving memory inflation duri…

2011

Expansion of the CD8 T-cell memory pool, also known as ‘memory inflation’, for certain but not all viral epitopes in latently infected host tissues is a special feature of the immune response to cytomegalovirus. The Ld-presented murine cytomegalovirus (mCMV) immediate–early (IE) 1 peptide is the prototype of an epitope that is associated with memory inflation. Based on the detection of IE1 transcripts in latently infected lungs it was previously proposed that episodes of viral gene expression and antigenic activity due to desilencing of a limited number of viral genes may drive epitope-specific memory inflation. This would imply direct antigen presentation through latently infected host tis…

MaleMice Inbred BALB CMuromegalovirusbiologyAntigen presentationAntigen-Presenting CellsPriming (immunology)CD8-Positive T-LymphocytesVirologyEpitopeImmediate-Early ProteinsVirus LatencyEpitopesMiceImmune systemAntigenVirologyImmunologyMHC class Ibiology.proteinAnimalsCytotoxic T cellFemaleAntigen-presenting cellImmunologic MemoryJournal of General Virology
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Age-Dependent Association of Human Mannose-Binding Lectin Mutations With Susceptibility to Invasive Meningococcal Disease in Childhood

2007

Mannose-binding lectin (MBL) is an important factor of the innate immune system, and MBL-initiated complement activation is an important early defense mechanism against various bacterial infections, including invasive meningococcal disease.In a pediatric cohort (ages 2-215 months) with invasive meningococcal disease, we investigated the overall and age-stratified frequency of 3 MBL exon 1 variations (C154T, G161A, G170A), previously shown to result in markedly decreased MBL plasma concentrations, by allele specific fluorescent hybridization probe real-time PCR assays and direct sequencing. Healthy age-matched volunteers with the same ethnic background and no history of meningococcal disease…

MaleMicrobiology (medical)AgingAdolescentMannosechemical and pharmacologic phenomenaMeningococcal diseasemedicine.disease_causeMannose-Binding Lectinchemistry.chemical_compoundPrevalencemedicineHumansGenetic Predisposition to DiseaseChildMannan-binding lectinMutationInnate immune systembiologybusiness.industryInfantLectinbacterial infections and mycosesmedicine.diseaseComplement systemMeningococcal InfectionsInfectious DiseaseschemistryChild PreschoolMutationPediatrics Perinatology and Child HealthImmunologyCohortbiology.proteinFemalebusinessPediatric Infectious Disease Journal
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Immunological response against SARS-CoV-2 following full-dose administration of Comirnaty® COVID-19 vaccine in nursing home residents

2022

6 páginas, 2 figuras, 3 tablas. Se puede acceder al texto completo de este artículo desde PubMedCentral: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8490129 . Los datos de investigación utilizados en el mismo, se encuentran disponibles en: https://doi.org/10.1016/j.cmi.2021.09.031.

MaleMicrobiology (medical)medicine.medical_specialtyCOVID-19 VaccinesSARS-CoV-2-S antibodiesFingerstickSevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)Nursing home residentsCD8-Positive T-LymphocytesAntibodies ViralImmune systemComirnaty®COVID-19 vaccineInternal medicinemedicineHumansProspective StudiesProspective cohort studyAgedAged 80 and overVenipuncturebiologybusiness.industrySARS-CoV-2COVID-19General MedicineNursing HomesVaccinationInfectious Diseasesbiology.proteinFemaleOriginal ArticleAntibodybusinessCD8
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Dectin-1 Stimulation of Hematopoietic Stem and Progenitor Cells Occurs In Vivo and Promotes Differentiation Toward Trained Macrophages via an Indirec…

2020

Invasive candidiasis is an increasingly frequent cause of serious and often fatal infections. Understanding host defense is essential to design novel therapeutic strategies to boost immune protection against Candida albicans. In this article, we delve into two new concepts that have arisen over the last years: (i) the delivery of myelopoiesis-inducing signals by microbial components directly sensed by hematopoietic stem and progenitor cells (HSPCs) and (ii) the concept of “trained innate immunity” that may also apply to HSPCs. We demonstrate that dectin-1 ligation in vivo activates HSPCs and induces their differentiation to trained macrophages by a cell-autonomous indirect mechanism. This p…

MaleMyeloidbeta-Glucanshematopoietic stem and progenitor cellstlr2BiologyMicrobiologyHost-Microbe Biology03 medical and health sciencesMicetrained immunity0302 clinical medicineImmune systemVirologymedicineAnimalsLectins C-TypeProgenitor cell030304 developmental biologyMice Knockout0303 health sciencesInnate immune systemStem CellsCandidiasisCell DifferentiationHematopoietic Stem CellsImmunity InnateToll-Like Receptor 2QR1-502Cell biologymacrophagesTransplantationMice Inbred C57BLTLR2Haematopoiesismedicine.anatomical_structureMyeloid Differentiation Factor 88Femalecandida albicansBone marrowdectin-1030215 immunologyResearch ArticleSignal TransductionmBio
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Fasting-Mimicking Diet Is Safe and Reshapes Metabolism and Antitumor Immunity in Patients with Cancer.

2021

Abstract In tumor-bearing mice, cyclic fasting or fasting-mimicking diets (FMD) enhance the activity of antineoplastic treatments by modulating systemic metabolism and boosting antitumor immunity. Here we conducted a clinical trial to investigate the safety and biological effects of cyclic, five-day FMD in combination with standard antitumor therapies. In 101 patients, the FMD was safe, feasible, and resulted in a consistent decrease of blood glucose and growth factor concentration, thus recapitulating metabolic changes that mediate fasting/FMD anticancer effects in preclinical experiments. Integrated transcriptomic and deep-phenotyping analyses revealed that FMD profoundly reshapes antican…

MaleMyeloidmedicine.medical_treatmentAntineoplastic AgentsBreast NeoplasmsPharmacologyTranscriptomeImmune systemmedicineCytotoxic T cellHumansProspective Studiesbusiness.industryGrowth factorCancerMetabolismFastingMiddle Agedmedicine.diseaseClinical trialmedicine.anatomical_structureTreatment OutcomeOncologyFemalebusinessColorectal NeoplasmsCancer discovery
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SARS‐CoV‐2‐reactive interferon‐γ‐producing CD8+ T cells in patients hospitalized with coronavirus disease 2019

2020

Abstract There is limited information on severe acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2) T‐cell immune responses in patients with coronavirus disease 2019 (COVID‐19). Both CD4+ and CD8+ T cells may be instrumental in resolution of and protection from SARS‐CoV‐2 infection. Here, we tested 25 hospitalized patients either with microbiologically documented COVID‐19 (n = 19) or highly suspected of having the disease (n = 6) for presence of SARS‐CoV‐2‐reactive CD69+ expressing interferon‐γ (IFN‐γ) producing CD8+ T cells using flow‐cytometry for intracellular cytokine staining assay. Two sets of overlapping peptides encompassing the SARS‐CoV‐2 Spike glycoprotein N‐terminal 1 to 643 am…

MaleMyeloma proteinCD8-Positive T-LymphocytesAntibodies ViralLymphocyte ActivationCD8+ T cellsSARS‐CoV‐2Blood cell03 medical and health sciencesInterferon-gamma0302 clinical medicineImmune systemAntigenCOVID‐19Intensive careVirologyCytotoxic T cellMedicineHumans030212 general & internal medicineResearch ArticlesAgedAged 80 and overbiologybusiness.industryfungiCOVID-19T‐cell immunityMiddle AgedVirologyHospitalizationmedicine.anatomical_structureInfectious DiseasesImmunoglobulin GSpike Glycoprotein Coronavirusbiology.protein030211 gastroenterology & hepatologyFemaleAntibodybusinessCD8Preliminary DataResearch ArticleJournal of Medical Virology
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Pharmacokinetic Interaction between Nevirapine and Nortriptyline in Rats: Inhibition of Nevirapine Metabolism by Nortriptyline

2014

ABSTRACTOne of the most frequent comorbidities of HIV infection is depression, with a lifetime prevalence of 22 to 45%. Therefore, it was decided to study a potential pharmacokinetic interaction between the nonnucleoside reverse transcriptase inhibitor nevirapine (NVP) and the tricyclic antidepressant nortriptyline (NT). NVP and NT were administered to rats either orally, intraduodenally, or intravenously, and the changes in plasma levels and pharmacokinetic parameters were analyzed. Experiments with rat and human hepatic microsomes were carried out to evaluate the inhibitory effects of NT on NVP metabolism. NVP plasma concentrations were significantly higher when this drug was coadminister…

MaleNevirapineAnti-HIV AgentsAdministration OralNortriptylineAntidepressive Agents TricyclicPharmacologyPharmacokineticsimmune system diseasesIn vivomedicineAnimalsHumansPharmacology (medical)NevirapineRats WistarBiotransformationPharmacologyDose-Response Relationship DrugReverse-transcriptase inhibitorbusiness.industryvirus diseasesRatsDose–response relationshipInfectious DiseasesArea Under CurveInjections IntravenousMicrosomes LiverMicrosomeReverse Transcriptase InhibitorsNortriptylinebusinessDrug AntagonismDrug metabolismmedicine.drugAntimicrobial Agents and Chemotherapy
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