Search results for "Immune system"

showing 10 items of 2885 documents

H89 Treatment Reduces Intestinal Inflammation and Candida albicans Overgrowth in Mice

2020

Deregulation of the dynamic crosstalk between the gut microbiota, intestinal epithelial cells, and immune cells is critically involved in the development of inflammatory bowel disease and the overgrowth of opportunistic pathogens, including the human opportunistic fungus Candida albicans. In the present study, we assessed the effect of N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinolinesulfonamide (H89), a protein kinase A inhibitor, on the migration of macrophages to C. albicans through dextran sulphate sodium (DSS)-challenged Caco-2 cells. We also investigated the impact of H89 on intestinal inflammation and C. albicans clearance from the gut, and determined the diversity of the gut microbio…

0301 basic medicineMicrobiology (medical)<i>Lactobacillus johnsonii</i>colitisH89030106 microbiologyInflammationGut floraMicrobiologydigestive systemArticleMicrobiology03 medical and health sciences<i>Enterococcus faecalis</i>Immune system[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesVirologyCandida albicansmedicineEscherichia coliEnterococcus faecalismicrobiotaColitisCandida albicanslcsh:QH301-705.5Lactobacillus johnsoniiLactobacillus johnsoniiDSSH89;Candida albicans;Escherichia coli;Enterococcus faecalis;Lactobacillus johnsonii;microbiota;DSS;colitis;protein kinase AInnate immune systembiology<i>Escherichia coli</i>[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterologymedicine.diseasebiology.organism_classificationCorpus albicans3. Good health<i>Candida albicans</i>030104 developmental biologylcsh:Biology (General)[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/Pharmacologyprotein kinase Amedicine.symptomMicroorganisms
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Anaplasma phagocytophilum Induces TLR- and MyD88-Dependent Signaling in In Vitro Generated Murine Neutrophils

2021

Anaplasma phagocytophilum is a tick-transmitted obligate intracellular Gram-negative bacterium that replicates in neutrophils. It elicits febrile disease in humans and in animals. In a mouse model, elimination of A. phagocytophilum required CD4+ T cells, but was independent of IFN-γ and other classical antibacterial effector mechanisms. Further, mice deficient for immune recognition and signaling via Toll-like receptor (TLR) 2, TLR4 or MyD88 were unimpaired in pathogen control. In contrast, animals lacking adaptor molecules of Nod-like receptors (NLR) such as RIP2 or ASC showed delayed clearance of A. phagocytophilum. In the present study, we investigated the contribution of further pattern…

0301 basic medicineMicrobiology (medical)ChemokineCLRanimal diseasesImmunologylcsh:QR1-502Microbiologylcsh:MicrobiologyNLR03 medical and health sciencesCellular and Infection Microbiology0302 clinical medicineImmune systemTLRparasitic diseasesNOD1cytokineddc:610ReceptorOriginal ResearchbiologychemokinefungiPattern recognition receptorSignal transducing adaptor proteinMyD88bacterial infections and mycosesbiology.organism_classificationAnaplasma phagocytophilumCell biologyiNOS030104 developmental biologyInfectious DiseasesTLR4biology.proteinbacteriaAnaplasma phagocytophilum030215 immunologyFrontiers in Cellular and Infection Microbiology
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The Anti-apoptotic Murine Cytomegalovirus Protein vMIA-m38.5 Induces Mast Cell Degranulation.

2020

Mast cells (MC) represent "inbetweeners" of the immune system in that they are part of innate immunity by acting as first-line sentinels for environmental antigens but also provide a link to adaptive immunity by secretion of chemokines that recruit CD8 T cells to organ sites of infection. An interrelationship between MC and cytomegalovirus (CMV) has been a blank area in science until recently when the murine model revealed a role for MC in the resolution of pulmonary infection by murine CMV (mCMV). As to the mechanism, MC were identified as a target cell type of mCMV. Infected MC degranulate and synthesize the CC-chemokine ligand-5 (CCL-5), which is released to attract protective virus-spec…

0301 basic medicineMicrobiology (medical)Chemokinebone marrow-derived mast cells (BMMC)Muromegalovirusmurine cytomegalovirus030106 microbiologyImmunologygene m38.5lcsh:QR1-502CytomegalovirusApoptosisInhibitor of apoptosisMicrobiologylcsh:MicrobiologyCell Degranulation03 medical and health sciencesMiceImmune systemCellular and Infection MicrobiologyCytotoxic T cellAnimalsperitoneal exudate-derived mast cells (PEMC)Mast CellsdegranulationInnate immune systembiologyDegranulationvirus diseasesTransfectionBrief Research ReportAcquired immune systemCell biologyvMIA030104 developmental biologyInfectious Diseasesbiology.proteinmast cell-specific Cre recombinationApoptosis Regulatory ProteinsFrontiers in cellular and infection microbiology
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Sex-dependent metabolism of nevirapine in rats: impact on plasma levels, pharmacokinetics and interaction with nortriptyline.

2017

Abstract Nevirapine (NVP) is a non-nucleoside reverse transcriptase inhibitor (NNRTI) widely used in the treatment of human immunodeficiency virus type 1 (HIV-1) and is the first-choice NNRTI during pregnancy. NVP shows a sex dimorphic profile in humans with sex differences in bioavailability, biotransformation and toxicity. In this study, sex differences in NVP metabolism and inhibition of NVP metabolism by the antidepressant nortriptyline (NT) were evaluated using rats as experimental animals. NVP was administered orally to male and female rats and sex differences in plasma levels and pharmacokinetic parameters were analysed. NVP plasma levels were higher in female compared with male rats…

0301 basic medicineMicrobiology (medical)Malemedicine.medical_specialtyNevirapineMetabolite030106 microbiologyCmaxNortriptylineAntidepressive Agents Tricyclic030226 pharmacology & pharmacy03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSex FactorsPharmacokineticsimmune system diseasesInternal medicinemedicineAnimalsPharmacology (medical)Drug InteractionsNevirapineRats WistarIC50Chemistryvirus diseasesGeneral MedicineBioavailabilityInfectious DiseasesEndocrinologyToxicityMicrosomes LiverReverse Transcriptase InhibitorsFemaleNortriptylinemedicine.drugInternational journal of antimicrobial agents
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Klebsiella pneumoniae Lipopolysaccharides Serotype O2afg Induce Poor Inflammatory Immune Responses Ex Vivo

2021

Currently, Klebsiella pneumoniae is a pathogen of clinical relevance due to its plastic ability of acquiring resistance genes to multiple antibiotics. During K. pneumoniae infections, lipopolysaccharides (LPS) play an ambiguous role as they both activate immune responses but can also play a role in immune evasion. The LPS O2a and LPS O2afg serotypes are prevalent in most multidrug resistant K. pneumoniae strains. Thus, we sought to understand if those two particular LPS serotypes were involved in a mechanism of immune evasion. We have extracted LPS (serotypes O1, O2a and O2afg) from K. pneumoniae strains and, using human monocytes ex vivo, we assessed the ability of those LPS antigens to in…

0301 basic medicineMicrobiology (medical)SerotypeChemokineQH301-705.5Klebsiella pneumoniae<i>Klebsiella pneumoniae</i>030106 microbiologyMicrobiologyArticleNF-κBMicrobiology03 medical and health scienceschemistry.chemical_compoundImmune systemAntigenVirologyantimicrobial resistanceBiology (General)Pathogenimmune evasionbiologylipopolysaccharideNF-κBSettore CHIM/06 - Chimica Organicalipopolysaccharidesbiology.organism_classificationKlebsiella pneumoniae030104 developmental biologychemistrynosocomial infectionbiology.proteinlipids (amino acids peptides and proteins)Ex vivoMicroorganisms
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'Secondary prevention' against female HPV infection: literature review of the role of carrageenan.

2020

Introduction: Human papillomaviruses (HPVs) are common sexually transmitted pathogens, causally associated with cervical cancer and other anogenital cancers, as well as approximately 20% of head and neck cancers. The HPV vaccine is an exceptional primary prevention tool, but the question of adequate secondary-prevention strategies remains open. The aim of this review is to better clarify the role of carrageenan in HPV prevention-strategies. Areas covered: A comprehensive literature search was performed (PubMed/MEDLINE, Embase, Google Scholar, Cochrane Databases) to identify articles on the use of carrageenan against HPV infection. The studies were identified using combinations of the search…

0301 basic medicineMicrobiology (medical)Settore MED/07 - Microbiologia E Microbiologia Clinicamedicine.medical_treatment030106 microbiologyCarrageenan papillomavirus prevention HPV microbicideMEDLINEAlphapapillomavirusBioinformaticsCarrageenanMicrobiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemVirologyMicrobicidemedicineSecondary PreventionAnimalsHumans030212 general & internal medicinePapillomavirus VaccinesCervical cancerbusiness.industryMechanism (biology)Papillomavirus InfectionsHPV infectionmedicine.diseaseSettore MED/40 - Ginecologia E OstetriciaCarrageenanInfectious DiseaseschemistryFemalebusinessAdjuvantExpert review of anti-infective therapy
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Extracellular Vesicles From the Helminth Fasciola hepatica Prevent DSS-Induced Acute Ulcerative Colitis in a T-Lymphocyte Independent Mode

2018

The complexity of the pathogenesis of inflammatory bowel disease (ulcerative colitis and Crohn's disease) has led to the quest of empirically drug therapies, combining immunosuppressant agents, biological therapy and modulators of the microbiota. Helminth parasites have been proposed as an alternative treatment of these diseases based on the hygiene hypothesis, but ethical and medical problems arise. Recent reports have proved the utility of parasite materials, mainly excretory/secretory products as therapeutic agents. The identification of extracellular vesicles on those secreted products opens a new field of investigation, since they exert potent immunomodulating effects. To assess the ef…

0301 basic medicineMicrobiology (medical)lcsh:QR1-502MACROPHAGE ACTIVATIONMicrobiologyInflammatory bowel diseaselcsh:MicrobiologyINNATE IMMUNE-SYSTEMCOLONIZATIONPathogenesis03 medical and health sciences0302 clinical medicineImmune systemHygiene hypothesisColitis ulcerosainflammatory bowel diseaseINFECTIONmedicineColitisSODIUM-INDUCED COLITISIN-VIVOOriginal ResearchCrohn's diseaseInnate immune systembusiness.industryDSS-ulcerative colitisFasciola hepaticamedicine.diseaseUlcerative colitis3. Good healthMICE030104 developmental biologyEnfermedad inflamatoria intestinal030220 oncology & carcinogenesisImmunologyCELLSSistema digestivobusinessextracellular vesiclesEnfermedadINFLAMMATORY-BOWEL-DISEASERESPONSES
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Gut dysbiosis and adaptive immune response in diet-induced obesity vs. Systemic inflammation

2017

A mutual interplay exists between adaptive immune system and gut microbiota. Altered gut microbial ecosystems are associated with the metabolic syndrome, occurring in most obese individuals. However, it is unknown why 10-25% of obese individuals are metabolically healthy, while normal weight individuals can develop inflammation and atherosclerosis. We modelled these specific metabolic conditions in mice fed with a chow diet, an obesogenic but not inflammatory diet - mimicking healthy obesity, or Paigen diet - mimicking inflammation in the lean subjects. We analysed a range of markers and cytokines in the aorta, heart, abdominal fat, liver and spleen, and metagenomics analyses were performed…

0301 basic medicineMicrobiology (medical)medicine.medical_specialtyAdaptive immune systemlcsh:QR1-502SpleenInflammationGut microbiotaGut floraSystemic inflammationMicrobiologylcsh:Microbiology03 medical and health sciencesImmune systemInternal medicineErysipelotrichiamedicineObesityOriginal ResearchInflammationbiologymedicine.diseaseAcquired immune systembiology.organism_classification030104 developmental biologymedicine.anatomical_structureEndocrinologyAdaptive immune system; Gut microbiota; Inflammation; Obesity; Microbiology; Microbiology (medical)ImmunologyMetabolic syndromemedicine.symptom
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An Inflammatory Profile Correlates With Decreased Frequency of Cytotoxic Cells in Coronavirus Disease 2019

2020

Abstract Increased production of inflammatory cytokines and myeloid-derived suppressor cells occurs in patients with coronavirus disease 2019. These inversely correlated with perforin-expressing natural killer (NK) and CD3+ T cells. We observed a lower number of perforin-expressing NK cells in intensive care unit (ICU) patients compared with non-ICU patients, suggesting an impairment of the immune cytotoxic arm as a pathogenic mechanism.

0301 basic medicineMicrobiology (medical)medicine.medical_treatmentMDSCInflammationchemical and pharmacologic phenomenaNK cellsProinflammatory cytokineNatural killer cell03 medical and health sciences0302 clinical medicineImmune systemmedicineCytotoxic T cellcytotoxic cellcytotoxic cellsbiologybusiness.industryCOVID-19COVID-19; cytotoxic cells; inflammation; MDSC; NK cells030104 developmental biologymedicine.anatomical_structureCytokineInfectious DiseasesPerforinSettore MED/38 - PEDIATRIA GENERALE E SPECIALISTICAinflammation030220 oncology & carcinogenesisImmunologybiology.proteinMyeloid-derived Suppressor Cellmedicine.symptombusinessClinical Infectious Diseases
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Systemic Candidiasis and TLR2 Agonist Exposure Impact the Antifungal Response of Hematopoietic Stem and Progenitor Cells.

2018

We have previously demonstrated that Candida albicans induces differentiation of hematopoietic stem and progenitor cells (HSPCs) toward the myeloid lineage both in vitro and in vivo in a TLR2- and Dectin-1-dependent manner, giving rise to functional macrophages. In this work, we used an ex vivo model to investigate the functional consequences for macrophages derived from HSPCs in vivo-exposed to Pam3CSK4 (a TLR2 agonist) or C. albicans infection. Short in vivo treatment of mice with Pam3CSK4 results in a tolerized phenotype of ex vivo HSPC-derived macrophages, whereas an extended Pam3CSK4 treatment confers a trained phenotype. Early during candidiasis, HSPCs give rise to macrophages trained…

0301 basic medicineMicrobiology (medical)medicine.medical_treatmenthematopoietic stem and progenitor cellsImmunologylcsh:QR1-502Colony Count MicrobialBiologyKidneyMicrobiologylcsh:Microbiology03 medical and health sciencesLipopeptidesMiceCandida albicansmedicineTLR2host-pathogen interactionsMacrophageAnimalsProgenitor cellCandida albicansinnate immunityInnate immune systemMacrophagesCandidiasisCell Differentiationbiology.organism_classificationmedicine.diseaseHematopoietic Stem CellsToll-Like Receptor 2Haematopoiesis030104 developmental biologyInfectious DiseasesCytokineImmunologySystemic candidiasisEx vivoSpleenFrontiers in cellular and infection microbiology
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