Search results for "Immune system"

showing 10 items of 2885 documents

Immunosenescence and its hallmarks: How to oppose aging strategically? A review of potential options for therapeutic intervention

2019

Aging is accompanied by remodeling of the immune system. With time, this leads to a decline in immune efficacy, resulting in increased vulnerability to infectious diseases, diminished responses to vaccination, and a susceptibility to age-related inflammatory diseases. An age-associated immune alteration, extensively reported in previous studies, is the reduction in the number of peripheral blood naive cells, with a relative increase in the frequency of memory cells. These two alterations, together with inflamm-aging, are considered the hallmarks of immunosenescence. Because aging is a plastic process, it is influenced by both nutritional and pharmacological interventions. Therefore, the rol…

0301 basic medicinelcsh:Immunologic diseases. AllergyAgingImmunosenescenceT cellmedicine.medical_treatmentT-LymphocytesImmunologyNutritional StatusInflammationCell CountReviewImmunomodulation03 medical and health sciences0302 clinical medicineImmune systemmedicineImmunology and AllergyHumansAgedNutritionInflammationSettore MED/04 - Patologia Generalebusiness.industryGrowth factorInterleukin-7ImmunotherapyImmunosenescenceHematopoietic Stem CellsVaccination030104 developmental biologymedicine.anatomical_structureImmunologyImmunotherapymedicine.symptomSignal transductionbusinesslcsh:RC581-607Immunologic Memory030215 immunology
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CD32 ligation promotes the activation of CD4+T cells

2018

Low affinity receptors for the Fc portion of IgG (FcγRs) represent a critical link between innate and adaptive immunity. Immune complexes (ICs) are the natural ligands for low affinity FcγRs, and high levels of ICs are usually detected in both, chronic viral infections and autoimmune diseases. The expression and function of FcγRs in myeloid cells, NK cells and B cells have been well characterized. By contrast, there are controversial reports about the expression and function of FcγRs in T cells. Here, we demonstrated that ∼2% of resting CD4+ T cells express cell surface FcγRII (CD32). Analysis of CD32 expression in permeabilized cells revealed an increased proportion of CD4+CD32+ T cells (∼…

0301 basic medicinelcsh:Immunologic diseases. AllergyCD32CIENCIAS MÉDICAS Y DE LA SALUDmedicine.drug_classIgGproliferationImmunologyCellFc receptorT cellsInmunologíaMonoclonal antibodyACTIVATION03 medical and health sciences0302 clinical medicineImmune systemFcγRmedicineImmunology and AllergyReceptorbiologyChemistryIGGCYTOKINESFCΓRPROLIFERATION//purl.org/becyt/ford/3.1 [https]Acquired immune systemMolecular biologycytokinesMedicina Básica030104 developmental biologymedicine.anatomical_structureCytoplasm030220 oncology & carcinogenesisT CELLSbiology.proteinactivation//purl.org/becyt/ford/3 [https]lcsh:RC581-607
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Taste receptors, innate immunity and longevity: the case of TAS2R16 gene

2019

Abstract Background Innate immunity utilizes components of sensory signal transduction such as bitter and sweet taste receptors. In fact, empirical evidence has shown bitter and sweet taste receptors to be an integral component of antimicrobial immune response in upper respiratory tract infections. Since an efficient immune response plays a key role in the attainment of longevity, it is not surprising that the rs978739 polymorphism of the bitter taste receptor TAS2R16 gene has been shown to be associated with longevity in a population of 941 individuals ranging in age from 20 to 106 years from Calabria (Italy). There are many possible candidate genes for human longevity, however of the many…

0301 basic medicinelcsh:Immunologic diseases. AllergyCandidate geneAgingmedia_common.quotation_subjectPopulationImmunologyLongevityShort ReportCase control studyGenome-wide association studyBiologylcsh:Geriatrics03 medical and health sciences0302 clinical medicineImmune systemstomatognathic systemTaste receptorGWASReceptoreducationBitter taste receptormedia_commonSettore MED/04 - Patologia GeneraleGeneticsInnate immunityeducation.field_of_studyInnate immune systemLongevitylcsh:RC952-954.6030104 developmental biologyBitter taste receptors; Case control study; GWAS; Innate immunity; Longevity; TAS2R16 gene; Immunology; AgingTAS2R16 geneBitter taste receptorslcsh:RC581-607030215 immunologyImmunity & Ageing : I & A
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Nitration of Wheat Amylase Trypsin Inhibitors Increases Their Innate and Adaptive Immunostimulatory Potential

2018

Amylase trypsin inhibitors (ATI) can be found in all gluten containing cereals and are, therefore, ingredient of basic foods like bread or pasta. In the gut ATI can mediate innate immunity via activation of the Toll-like receptor 4 (TLR4) on immune cells residing in the lamina propria, promoting intestinal, as well as extra-intestinal, inflammation. Inflammatory conditions can induce formation of peroxynitrite (ONOO-) and, thereby, endogenous protein nitration in the body. Moreover, air pollutants like ozone (O3) and nitrogen dioxide (NO2) can cause exogenous protein nitration in the environment. Both reaction pathways may lead to the nitration of ATI. To investigate if and how nitration mo…

0301 basic medicinelcsh:Immunologic diseases. AllergyCell SurvivalT cellnon-celiac wheat sensitivityImmunologyInflammationAdaptive ImmunityImmunophenotyping03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemprotein nitrationT-Lymphocyte SubsetsNitrationCell Line TumorwheatmedicineImmunology and AllergyHumansamylase trypsin inhibitorsTriticumPlant ProteinsOriginal ResearchInnate immune systemMacrophagesfood and beveragesDendritic CellsTetranitromethaneallergyImmunity InnateToll-Like Receptor 4030104 developmental biologymedicine.anatomical_structurechemistryBiochemistryAmylasesTLR4Cytokinesmedicine.symptomlcsh:RC581-607Trypsin InhibitorsPeroxynitriteBiomarkers030215 immunologyFrontiers in immunology
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The cAMP pathway as therapeutic target in autoimmune and inflammatory diseases

2016

Nucleotide signaling molecules contribute to the regulation of cellular pathways. In the immune system, cyclic adenosine monophosphate (cAMP) is well established as a potent regulator of innate and adaptive immune cell functions. Therapeutic strategies to interrupt or enhance cAMP generation or effects have immunoregulatory potential in autoimmune and inflammatory disorders. Here, we provide an overview of the cyclic AMP axis and its role as a regulator of immune functions and discuss the clinical and translational relevance of interventions with these processes.

0301 basic medicinelcsh:Immunologic diseases. AllergyCell signalingT regulatory cellsImmunologyRegulatorT cellsTregsInflammationAutoimmunityReviewmedicine.disease_causeAutoimmunity03 medical and health scienceschemistry.chemical_compoundImmune systemmedicineCyclic AMPImmunology and AllergyCyclic adenosine monophosphateTregs; T regulatory CellsInflammationbusiness.industryCellular pathwaystargeted therapiesCell biology030104 developmental biologychemistryImmunologycAMP-dependent pathwaymedicine.symptombusinesslcsh:RC581-607Frontiers in Immunology
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Human Vδ1+ T Cells in the Immune Response to Plasmodium falciparum Infection

2019

Naturally acquired protective immunity to Plasmodium falciparum malaria is mainly antibody-mediated. However, other cells of the innate and adaptive immune system also play important roles. These include so-called unconventional T cells, which express a γδ T-cell receptor (TCR) rather than the αβ TCR expressed by the majority of T cells-the conventional T cells. The γδ T-cell compartment can be divided into distinct subsets. One expresses a TCR involving Vγ9 and Vδ2, while another major subset uses instead a TCR composed of Vδ1 paired with one of several types of γ chains. The former of these subsets uses a largely semi-invariant TCR repertoire and responds in an innate-like fashion to pyro…

0301 basic medicinelcsh:Immunologic diseases. AllergyCell typeImmunologyPlasmodium falciparummalariaVdelta1 gamma delta T cells03 medical and health sciences0302 clinical medicineImmune systemAntigenparasitic diseasesImmunology and AllergyReceptorinnate immunityInnate immune systembiologyT-cell receptorgamma-delta (γ/δ) T lymphocytesPlasmodium falciparumAcquired immune systembiology.organism_classificationacquired immunity030104 developmental biologyImmunologylcsh:RC581-607030215 immunologyFrontiers in Immunology
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γδ T Cells and Tumor Microenvironment: From Immunosurveillance to Tumor Evasion.

2018

gamma delta T cells possess cytotoxic antitumor activity mediated by production of proinflammatory cytokines, direct cytotoxic activity, and regulation of the biological functions of other cell types. Hence, these features have prompted the development of therapeutic strategies in which gamma delta T cells agonists or ex vivo-expanded gamma delta T cells are administered to tumor patients. Several studies have shown that gamma delta T cells are an important component of tumor-infiltrating lymphocytes in patients affected by different types of cancer and a recent analysis of similar to 18,000 transcriptomes from 39 human tumors identified tumor-infiltrating.d T cells as the most significant …

0301 basic medicinelcsh:Immunologic diseases. AllergyCell typegamma delta T cellmedicine.medical_treatmentImmunologyReviewBiologycyototxicityProinflammatory cytokineTranscriptome03 medical and health sciences0302 clinical medicineImmune systemmedicineImmunology and AllergyCytotoxic T cellgamma delta T cellstumor microenvironmentTumor microenvironmentimmunosuppressionImmunotherapyImmunosurveillance030104 developmental biologyCancer researchimmunotherapylcsh:RC581-607030215 immunologyFrontiers in immunology
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Update of Immunosenescence in Cerebral Small Vessel Disease

2020

Aging of the central nervous system (CNS) is closely associated with chronic sterile low-grade inflammation in older organisms and related immune response. As an amplifier for neuro-inflammaging, immunosenescence remodels and deteriorates immune systems gradually with the passage of time, and finally contributes to severe outcomes like stroke, dementia and neurodegeneration in elderly adults. Cerebral small vessel disease (CSVD), one of the major causes of vascular dementia, has an intensive connection with the inflammatory response and immunosenescence plays a crucial role in the pathology of this disorder. In this review, we discuss the impact of immunosenescence on the development of CSV…

0301 basic medicinelcsh:Immunologic diseases. AllergyImmunologyInflammationDiseaseReview03 medical and health sciences0302 clinical medicineImmune systemmedicineDementiaAnimalsHumansImmunology and AllergyVascular dementiaStrokeimmunosenescenceMechanism (biology)business.industrycerebral small vessel diseasepathogenesisagingImmunosenescencemedicine.disease030104 developmental biologyCerebral Small Vessel DiseasesImmunologyinflammagingmedicine.symptombusinesslcsh:RC581-607030217 neurology & neurosurgeryFrontiers in Immunology
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Enhanced protection of C57 BL/6 vs Balb/c mice to melanoma liver metastasis is mediated by NK cells.

2017

ABSTRACT The B16F10 murine melanoma cell line displays a low expression of MHC class I molecules favoring immune evasion and metastases in immunocompetent C57 BL/6 wild-type mice. Here, we generated metastases to the liver, an organ that is skewed towards immune tolerance, by intrasplenic injection of B16F10 cells in syngeneic C57 BL/6 compared to allogeneic Balb/c mice. Surprisingly, Balb/c mice, which usually display a pronounced M2 macrophage and Th2 T cell polarization, were ∼3 times more susceptible to metastasis than C57 BL/6 mice, despite a much higher M1 and Th1 T cell immune response. The anti-metastatic advantage of C57 BL/6 mice could be attributed to a more potent NK-cell mediat…

0301 basic medicinelcsh:Immunologic diseases. AllergyImmunologyNK cellsMajor histocompatibility complexcancer immunologyliverlcsh:RC254-282BALB/cImmune toleranceMetastasis03 medical and health sciencesImmune systemMHC class ImedicineImmunology and Allergymetastasisinnate immunityOriginal ResearchInnate immune systembiologybiology.organism_classificationmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmacrophages030104 developmental biologyOncologyCancer cellCancer researchbiology.proteinlcsh:RC581-607Oncoimmunology
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RNase H2 Loss in Murine Astrocytes Results in Cellular Defects Reminiscent of Nucleic Acid-Mediated Autoinflammation

2018

Aicardi-Goutières syndrome (AGS) is a rare early onset childhood encephalopathy caused by persistent neuroinflammation of autoimmune origin. AGS is a genetic disorder and >50% of affected individuals bear hypomorphic mutations in ribonuclease H2 (RNase H2). All available RNase H2 mouse models so far fail to mimic the prominent CNS involvement seen in AGS. To establish a mouse model recapitulating the human disease, we deleted RNase H2 specifically in the brain, the most severely affected organ in AGS. Although RNase H2δGFAPmice lacked the nuclease in astrocytes and a majority of neurons, no disease signs were apparent in these animals. We additionally confirmed these results…

0301 basic medicinelcsh:Immunologic diseases. AllergyMaleEncephalomyelitis Autoimmune ExperimentalAicardi–Goutières syndromeRNase PDNA damageImmunologyRibonuclease HFluorescent Antibody TechniqueAicardi-goutières Syndrome ; Cellular Senescence ; Dna Damage ; Interferon Signature ; Rnase H2BiologyNervous System MalformationsAutoimmune Diseases03 medical and health sciencesMiceAutoimmune Diseases of the Nervous SystemNucleic AcidsmedicineImmunology and Allergycellular senescenceAnimalsRibonucleaseNeuroinflammationCells CulturedOriginal ResearchInflammationMice KnockoutInnate immune systemBrainmedicine.diseaseMolecular biologyImmunohistochemistryDisease Models Animal030104 developmental biologymedicine.anatomical_structurePhenotypeinterferon signatureAstrocytesKnockout mousebiology.proteinAicardi–Goutières syndromeDNA damageFemalelcsh:RC581-607RNase H2BiomarkersAstrocyteFrontiers in Immunology
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