Search results for "Immunity"

showing 10 items of 1537 documents

Molecular Characterization of Virus-induced Autoantibody Responses

2004

Here we present a comprehensive molecular mapping of virus-induced autoimmune B cell responses obtained by serological identification of antigens by recombinant expression cloning analysis. Immunoscreening of cDNA expression libraries of various organs (lung, liver, and spleen) using sera from mice infected with cytopathic (vaccinia virus [VV]) or noncytopathic (lymphocytic choriomeningitis virus [LCMV]) viruses revealed a broad specificity of the elicited autoantibody response. Interestingly, the majority of the identified autoantigens have been previously described as autoantigens in humans. We found that induction of virus-induced autoantibodies of the immunoglobulin G class largely depe…

DNA ComplementaryTime FactorsautoantibodiesT-LymphocytesvirusesCD40 LigandImmunologyVaccinia virusBiologyLymphocytic choriomeningitisArticleImmunoglobulin GVirusMice03 medical and health sciences0302 clinical medicineAntigenmedicineAnimalsHumansLymphocytic choriomeningitis virusImmunology and AllergyTissue DistributionCD40 AntigensB cellGene Library030304 developmental biologyB-Lymphocytes0303 health sciencesvirus-induced immunopathologyAutoantibodyAntiviral antibodySEREXbiology.organism_classificationmedicine.diseaseVirologytumor immunity3. Good healthMice Inbred C57BLmedicine.anatomical_structureDatabases as TopicVesicular stomatitis virusImmunoglobulin GImmunologybiology.proteinAlgorithms030215 immunologyJournal of Experimental Medicine
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CD95 death-inducing signaling complex formation and internalization occur in lipid rafts of type I and type II cells

2004

We investigated the membrane localization of CD95 in type I and type II cells, which differ in their ability to recruit and activate caspase-8. We found that CD95 was preferentially located in lipid rafts of type I cells, while it was present both in raft and non-raft plasma membrane sub-domains of type II cells. After stimulation, CD95 located in phospholipid-rich plasma membrane was recruited to lipid rafts in both types of cells. Similarly, CD95 cross-linking resulted in caspase-independent translocation of FADD/MORT1 and caspase-8 to the lipid rafts, which was prevented by a death domain-defective receptor. CD95 internalization was then rapid in type I and delayed in type II cells and s…

Death Domain Receptor Signaling Adaptor ProteinsEndosomeT-Lymphocytesmedia_common.quotation_subjectImmunologyApoptosisReceptors Tumor Necrosis FactorCell LineMembrane MicrodomainsSettore MED/04 - PATOLOGIA GENERALECell Line TumorReceptorsHumansImmunology and Allergyfas ReceptorFADDInternalizationLipid raftLipid raftsDeath domainmedia_commonTumorbiologyVesicleFas receptorEndocytosisCell biologyProtein TransportCholesterolCD95 death-inducing signaling complexCaspasesCD95biology.proteinlipids (amino acids peptides and proteins)biological phenomena cell phenomena and immunityCaspase-8Tumor Necrosis FactorCaspase-8; CD95; Lipid rafts; Apoptosis; Caspases; Cell Line Tumor; Cholesterol; Death Domain Receptor Signaling Adaptor Proteins; Humans; Membrane Microdomains; Protein Binding; Protein Transport; Receptors Tumor Necrosis Factor; T-Lymphocytes; fas Receptor; Endocytosis; Signal Transduction; Immunology and Allergy; ImmunologyProtein BindingSignal TransductionEuropean Journal of Immunology
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Regalismo e inmunidad eclesiástica en la España del siglo XVIII: la resistencia del clero valenciano a la imposición del estanco del tabaco

2007

The charters abolition decree of the Kingdoms of Valencia and Aragon kept the traditional regulation on the jurisdiction and ecclesiastic immunity, and this exception was ratified in the Royal Order the 7th of September in 1707. As the members of this class thought that it was implying a reinforcement of their privileges, they presented an intense resistance to the imposition of the tobacco monopoly, which the monarchy considered as a «well-known royalty» inherent in its sovereignty. Initially, the opposition was assumed by the ecclesiastic hierarchy and it generated jurisdictional conflicts so serious that Philip V proceeded to the emission of dispositions in favour of his royal prerogativ…

DecreeHistoryRoyal prerogativeEcclesiastical immunityRegalismoOpposition (politics)Social SciencesEighteenth centurysiglo xviiiValencianlcsh:Social SciencesHMonarchySovereigntyfraudeFraudeConflictividadTobaccoInmunidad eclesiásticaSociologyTax systemFiscalidadInmunidad eclesiástica; Regalismo; Fiscalidad; Fraude; Conflictividad; Tabaco; Valencia; siglo XVIIIJurisdictionFraudRoyal prerogativeconflictividadregalismofiscalidadlanguage.human_languagetabacolcsh:HConflict situationsTabacoLawlanguageValenciavalenciaEcclesiastical immunity; Royal prerogative; Tax system; Fraud; Conflict situations; Tobacco; Valencia; Eighteenth centuryAdministration (government)siglo XVIIIinmunidad eclesiásticaHispania : Revista española de historia
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Suppression of humoral immune response against herpes simplex virus induced by defective strains, ts- and TK- mutants.

1988

Suppression of humoral antibody formation against HSV is not only induced by replicating Herpes simplex virus type 2 (HSV-2) but also by the defective strain ANG and the deletion mutant 1301 of Herpes simplex virus type 1 (HSV-1). Moreover, ts-mutants A, H, K, S, 1201 and 1208 of HSV-1 as well as some ts-mutants of HSV-2 and “defective-interfering” particles of HSV-1 after high multiplicity of infection-passages induced suppression. Treatment of infected mice with ACG reduced antibody-formation but did not result in suppression. UV-irradiation of the antibody producing strain Len of HSV-1 strongly reduces antibody formation and induces suppression. Experiments using a series of intertypic r…

Deletion mutantGenes ViralvirusesMutantBiologymedicine.disease_causeAntibodies ViralVirus ReplicationGenomeThymidine KinaseMiceImmune systemVirologyViral InterferencemedicineImmune ToleranceAnimalsSimplexvirusRecombination GeneticDefective VirusesGeneral MedicineVirologyHerpes simplex virusHumoral immunityMutationbiology.proteinViral diseaseAntibodyArchives of virology
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Gene therapy for type 1 diabetes: is it ready for the clinic?

1999

This review, in addition to updating the growing list of type 1 diabetes- relevant gene therapies, offers an outline of short-term objectives that can readily be met to move, at least, adenoviral and adeno-associated viral-based protocols into the clinic, first as a means of facilitating islet allografts as well as platforms with which to introduce immunoregulatory transgenes. A wide array of genes have been tested to restore insulin production, to drive the differentiation of insulin-producing progenitors, and to confer immunosuppression in an antigen- and tissue-restricted manner.

Diabetes; Gene therapy; Immunotherapy; Autoimmunity.medicine.medical_treatmentGenetic enhancementTransgeneImmunologyGenetic VectorsAutoimmunity.BioinformaticsDiabeteAdenoviridaeGene therapyAntigenmedicineAnimalsHumansProgenitor cellgeographyType 1 diabetesgeography.geographical_feature_categorybusiness.industryInsulinGene Transfer TechniquesImmunosuppressionGenetic TherapyIsletmedicine.diseaseDiabetes Mellitus Type 1Immunotherapybusiness
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Liver fibrosis: Common mechanisms and antifibrotic therapies

2015

Liver fibrosis and in particular cirrhosis have become major endpoints in clinical trials of patients with chronic liver diseases. Here, viral hepatitis, alcoholic and non-alcoholic steatohepatitis have become the major etiologies. We have made great progress in our understanding of the mechanisms and the cell biology of liver fibrosis and have already made the transition from preclinical testing of antifibrotic agents and strategies towards clinical translation. There continues to be an urgent need for specific antifibrotic therapies, despite the advent of highly potent antiviral agents that can even induce regression of advanced fibrosis. This review addresses central mechanisms and cells…

Diagnostic ImagingLiver CirrhosisCirrhosisHepatologybusiness.industryLiver fibrosisGastroenterologyAdaptive Immunitymedicine.diseaseBioinformaticsAcquired immune systemImmunity InnateClinical trialBiomarkerFibrosisImmunologymedicineAnimalsHumansSteatohepatitisViral hepatitisbusinessBiomarkersClinics and Research in Hepatology and Gastroenterology
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Homotypic Protection Against Rotavirus-Induced Diarrhea in Infant Mice Breast-Fed by Dams Immunized with the Recombinant VP8* Subunit of the VP4 Caps…

2000

The outer capsid proteins VP4 and VP7 induce neutralizing antibody against rotavirus. We have investigated in a mouse model the protection mediated by immunization with VP8*, the amino-terminal tryptic fragment of VP4. BALB/c female mice immunized with simian rotavirus SA11 VP6 and VP8* proteins expressed in Escherichia coli were mated with seronegative males. Litters were orally challenged with the SA11 strain (P5B[2], G3) or with the murine rotavirus strain EDIM (P10[16], G3) to verify the degree of protection against diarrhea induced in the newborns. Only those pups born to dams immunized with VP8* did not develop diarrhea after having been orally challenged with the SA11 strain. Pups bo…

DiarrheaRotavirusRecombinant Fusion ProteinsvirusesImmunologyViral Nonstructural ProteinsAntibodies Viralmedicine.disease_causeRotavirus InfectionsMiceCapsidAntigenNeutralization TestsImmunityVirologyRotavirusEscherichia colimedicineAnimalsNeutralizing antibodyAntigens ViralGlutathione TransferaseMice Inbred BALB CbiologyVaccinationImmunization PassiveRotavirus VaccinesRNA-Binding Proteinsvirus diseasesVirologyDiarrheaBreast FeedingMilkAnimals NewbornImmunizationVaccines Subunitbiology.proteinMolecular MedicineCapsid ProteinsFemalemedicine.symptomAntibodyImmunity Maternally-AcquiredBreast feedingViral Immunology
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Influenza control in the 21st century: Optimizing protection of older adults

2009

Older adults (> or =65 years of age) are particularly vulnerable to influenza illness. This is due to a waning immune system that reduces their ability to respond to infection, which leads to more severe cases of disease. The majority ( approximately 90%) of influenza-related deaths occur in older adults and, in addition, catastrophic disability resulting from influenza-related hospitalization represents a significant burden in this vulnerable population. Current influenza vaccines provide benefits for older adults against influenza; however, vaccine effectiveness is lower than in younger adults. In addition, antigenic drift is also a concern, as it can impact on vaccine effectiveness due t…

DiseaseVaccines AttenuatedAntigenic driftDNA vaccinationAdjuvants ImmunologicCost of IllnessImmunityInfluenza HumanHumansMedicineAntigens ViralAgedAttenuated vaccineGeneral VeterinaryGeneral Immunology and Microbiologybusiness.industryHealth PolicyImmunogenicityVaccinationPublic Health Environmental and Occupational HealthVaccines VirosomeVaccinationInfectious DiseasesImmunizationInfluenza VaccinesImmunologyMolecular MedicinebusinessVaccine
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Frequency and Characterization of Movement Disorders in Anti-IgLON5 Disease

2021

Background and ObjectivesAnti-IgLON5 disease is a recently described neurologic disease that shares features of autoimmunity and neurodegeneration. Abnormal movements appear to be frequent and important but have not been characterized and are underreported. We describe the frequency and types of movement disorders in a series of consecutive patients with this disease.MethodsIn this retrospective, observational study, the presence and phenomenology of movement disorders were assessed with a standardized clinical questionnaire. Available videos were centrally reviewed by 3 experts in movement disorders.ResultsSeventy-two patients were included. In 41 (57%), the main reason for initial consult…

DystoniaPediatricsmedicine.medical_specialtyAtaxiaMovement disordersbusiness.industryautoimmunityAnti-IgLON5neurodegenerationChoreamedicine.diseaseAkathisianervous system diseasesmedicineBody regionHuman medicineNeurology (clinical)Myokymiamedicine.symptombusinessMyoclonusResearch Article
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A procedure to evaluate the electromagnetic immunity degree of a data acquisition system

2007

EMCimmunity
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