Search results for "Inducer"

showing 10 items of 178 documents

Growth, interleukin-2 production, and responsiveness to IL-2 in T4- positive T Lymphocyte populations from malignant cutaneous T cell lymphoma (Sezar…

1984

Abstract Functional analysis and surface phenotyping using monoclonal antibodies have revealed that malignant T lymphocyte populations in the peripheral blood of patients with Sezary's syndrome resemble the T helper cell populations from normal individuals. In this article we have studied the effects of the immunosuppressive drug cyclosporine A (CsA) on growth, interleukin-2 (IL-2) production, and the induction of IL-2 responsiveness of peripheral blood monocytes (PBMs) from five patients with Sezary's syndrome in vitro, using the lectin phytohemagglutinin (PHA) and the phorbol ester phorbol myristate acetate (PMA) as stimuli. The following results were obtained: PHA-induced cell proliferat…

Interleukin 2medicine.medical_specialtyT cellImmunologyCyclosporinsBiologyLymphocyte ActivationBiochemistryCyclosporin aInternal medicinemedicineHumansSezary SyndromePhytohemagglutininsReceptorCell growthCutaneous T-cell lymphomaT-Lymphocytes Helper-InducerCell BiologyHematologyT helper cellT lymphocytemedicine.diseasePhenotypemedicine.anatomical_structureEndocrinologyInterleukin-2Tetradecanoylphorbol Acetatemedicine.drugBlood
researchProduct

Heat-stable antigen is expressed by murine keratinocytes and delivers costimulatory signals in T-cell activation.

1995

Heat-stable antigen (HSA), expressed by various antigen-presenting cells (APC), has been described as a costimulatory molecule for CD4+ T cells. Recently, we observed that HSA also serves as an important costimulatory molecule on epidermal Langerhans cells (LC). During these studies, low levels of HSA staining were also detected on normal murine keratinocytes (KC). To investigate whether HSA also is involved in T-cell activation by KC, normal murine KC or the spontaneously transformed KC cell-line PAM 212 were treated with PDB or PMA to induce HSA-expression. FACS analyses showed induction of HSA expression on normal murine KC, as well as PAM 212 cells. In functional assays PDB or PMA-treat…

Keratinocytesmedicine.drug_classT cellT-LymphocytesMolecular Sequence DataProtein Data Bank (RCSB PDB)DermatologyBiologyCleavage (embryo)Monoclonal antibodyLymphocyte ActivationBiochemistryMicePhosphoinositide Phospholipase CAntigenAntigens CDPhorbol EstersmedicineAnimalsInducerRNA MessengerMolecular BiologyCells CulturedMice Inbred BALB CMice Inbred C3HPhospholipase CBase SequencePhosphoric Diester HydrolasesPhosphatidylinositol Diacylglycerol-LyaseAntibodies MonoclonalMolecular biologyStainingbody regionsmedicine.anatomical_structureMolecular Probesembryonic structuresImmunizationLymph NodesExperimental dermatology
researchProduct

Distinct Roles for IL-1 Receptor Type I Signaling in Early Versus Established Leishmania major Infections

2006

IL-1alpha/beta released by infected dendritic cells (DC) plays a critical role in the development of protective immunity against Leishmania major. Previous studies demonstrated that treatment of susceptible BALB/c mice with IL-1alpha during T-cell priming (days 1-3 post-infection) induced T helper (Th)1-mediated protection. In contrast, we now demonstrate that prolonged treatment with IL-1alpha (for 3 weeks) worsened disease outcome. To characterize the receptor involved, L. major infections in IL-1 receptor type I (IL-1RI) knockout mice were studied. In C57BL/6 IL-1RI-/- mice, the IL-1alpha-mediated protective effect was abrogated. The course of high-dose infection (2 x 10(5) parasites) in…

Leishmaniasis CutaneousPriming (immunology)DermatologyReceptor typeBiochemistryInterferon-gammaMiceTh2 CellsmedicineAnimalsParasite hostingLeishmania majorL-SelectinReceptorMolecular BiologyLeishmania majorMice KnockoutReceptors Interleukin-1 Type IMice Inbred BALB CbiologyReceptors Interleukin-1LeishmaniasisT-Lymphocytes Helper-InducerCell BiologyTh1 Cellsbiology.organism_classificationmedicine.diseaseMice Inbred C57BLGene Expression RegulationCD4 AntigensImmunologyKnockout mouseDisease ProgressionInterleukin-4Ex vivoInterleukin-1Signal TransductionJournal of Investigative Dermatology
researchProduct

Regulation of T cells in asthma: implications for genetic manipulation

2004

PURPOSE OF THE REVIEW Allergic asthma is a disease characterized by airway hyperresponsiveness, inflammation and remodeling. In the past few decades it has become clear that the pathogenesis and development of this disease is controlled by cytokines released by CD4 T helper type 2 lymphocytes that develop under the influence of natural killer lymphocytes. At birth, T cell priming exhibits a T helper type 2 bias and the development of the T helper phenotype is determined in the first year of life by environmental exposure to virus or bacterial substances or environmental allergens in genetically predisposed individuals. Decreased exposure to infection in early childhood has thus been linked …

LipopolysaccharidesT-LymphocytesT cellImmunologyPriming (immunology)Receptors Cell SurfaceInflammationBiologyType 2 immune responseImmune systemAntigenHygiene hypothesismedicineHumansImmunology and AllergyGeneticsMembrane GlycoproteinsToll-Like ReceptorsT-Lymphocytes Helper-InducerEnvironmental exposureAsthmamedicine.anatomical_structureImmunologyCytokinesmedicine.symptomT-Box Domain ProteinsTranscription FactorsCurrent Opinion in Allergy and Clinical Immunology
researchProduct

Corynebacterium parvum (Propionibacterium acnes): an inducer of tumor necrosis factor-alpha in human peripheral blood mononuclear cells and monocytes…

1990

The present study investigates the potential capacity of the immunostimulant Corynebacterium parvum (C.p.) to induce tumor necrosis factor-alpha (TNF-alpha) in human peripheral blood mononuclear cells (PBMC) and blood monocytes (BMo) in vitro. Both at the mRNA and protein level, stimulation of PBMC and BMo upon C.p. induces TNF-alpha. Compared to the hitherto used TNF-alpha inducers in vitro such as Sendai virus, phytohemagglutinin or lipopolysaccharide the C.p. stimulus displayed a threefold stronger induction of TNF-alpha production (p less than 0.001). Using C.p. as an inducer it was possible to demonstrate that TNF-alpha production is regulated by prostaglandin E2; preincubation of the …

Lipopolysaccharidesmedicine.drug_classLymphocyteImmunologyEnzyme-Linked Immunosorbent AssayBiologyIn Vitro TechniquesPeripheral blood mononuclear cellImmunostimulantDinoprostoneMonocytesInterferon-gammamedicineImmunology and AllergyHumansInterferon gammaInducerPropionibacterium acnesProstaglandin E2Cells CulturedDose-Response Relationship DrugTumor Necrosis Factor-alphaMonocyteBlotting NorthernMolecular biologymedicine.anatomical_structureImmunologyLeukocytes MononuclearRNATumor necrosis factor alphaImmunizationDNA Probesmedicine.drugEuropean journal of immunology
researchProduct

INTERINDIVIDUAL VARIATIONS OF EPOXIDE HYDRATASE ACTIVITY IN HUMAN LIVER AND LUNG BIOPSIES, LYMPHOCYTES AND FIBROBLAST CULTURES

1980

Publisher Summary Most pharmaceuticals, toxic compounds, mutagens, and carcinogens undergo metabolism in the human body. Differences in drug metabolizing enzymes cause differences in susceptibility towards effects or side effects of these compounds. This chapter presents a comparison of epoxide hydratase (EH) activity in human individuals. Activities were measured in biopsy samples of liver, which is the main site of drug metabolism, and of lung, which is an organ that is continually exposed to potential enzyme inducers and to carcinogenic compounds. EH was studied in native lymphocytes, cultured lymphocytes, and fibroblasts in which environmental influences can be controlled and the geneti…

LungHuman livermedicine.diagnostic_testbiologyMetabolismmedicine.anatomical_structureBiochemistryBiopsymedicinebiology.proteinEpigeneticsEnzyme inducerCarcinogenDrug metabolism
researchProduct

The nuclear receptor PPARγ selectively inhibits Th17 differentiation in a T cell–intrinsic fashion and suppresses CNS autoimmunity

2009

T helper cells secreting interleukin (IL)-17 (Th17 cells) play a crucial role in autoimmune diseases like multiple sclerosis (MS). Th17 differentiation, which is induced by a combination of transforming growth factor (TGF)-beta/IL-6 or IL-21, requires expression of the transcription factor retinoic acid receptor-related orphan receptor gamma t (ROR gamma t). We identify the nuclear receptor peroxisome proliferator-activated receptor gamma (PPAR gamma) as a key negative regulator of human and mouse Th17 differentiation. PPAR gamma activation in CD4(+) T cells selectively suppressed Th17 differentiation, but not differentiation into Th1, Th2, or regulatory T cells. Control of Th17 differentia…

MESH: Nuclear Receptor Subfamily 1 Group F Member 3Helper-InducerReceptors Retinoic AcidT-LymphocytesMESH: Interleukin-17Cellular differentiationRetinoic AcidPeroxisome proliferator-activated receptorNeurodegenerativeInbred C57BLMedical and Health SciencesMiceInterleukin 210302 clinical medicineGroup FRAR-related orphan receptor gammaMESH: Nuclear Receptor Co-Repressor 2Receptors2.1 Biological and endogenous factorsThyroid HormoneImmunology and AllergyMESH: AnimalsAetiologyEncephalomyelitisPromoter Regions Geneticchemistry.chemical_classificationOrphan receptor0303 health sciencesReceptors Thyroid HormoneInterleukin-17Cell DifferentiationT-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 33. Good healthCell biologyDNA-Binding Proteinsmedicine.anatomical_structureMESH: Repressor Proteins[SDV.IMM]Life Sciences [q-bio]/ImmunologyInterleukin 17MESH: Cell Differentiationmedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisNuclear Receptor Subfamily 1Member 31.1 Normal biological development and functioningT cellImmunologyBiologyAutoimmune DiseasePromoter RegionsExperimental03 medical and health sciencesGeneticUnderpinning researchMESH: Mice Inbred C57BLInternal medicineMESH: Promoter Regions GeneticGeneticsmedicineAnimalsHumansNuclear Receptor Co-Repressor 2MESH: Receptors Thyroid HormoneMESH: T-Lymphocytes Helper-InducerMESH: Encephalomyelitis Autoimmune ExperimentalMESH: Mice030304 developmental biologyMESH: Receptors Retinoic AcidMESH: HumansInflammatory and immune systemNeurosciencesBrief Definitive ReportCorrectionMESH: Multiple SclerosisBrain DisordersMice Inbred C57BLPPAR gammaRepressor ProteinsEndocrinologyMESH: PPAR gammaNuclear receptorchemistryMESH: DNA-Binding Proteins030217 neurology & neurosurgeryAutoimmuneJournal of Experimental Medicine
researchProduct

The role of Interleukin-2 during the activation of cytotoxic T-lymphocytes

1983

Der vorliegende Artikel stellt eine Ubersicht dar Uber die derzeitigen Vorstellungen des Zusammenwirkens zellularer und humoraler Faktoren, die zu T-Zell-vermittelten zytotoxischen Immunreaktionen fuhren. Da im Ablauf derartiger Immunreaktionen hormonahnliche Wachstumsfaktoren (interleukine) eine entscheidende Rolle spielen, liegt der Schwerpunkt der Diskussion auf der Beschreibung dieser Mediatoren; insbesondere wird die Bedeutung von Interleukin-2 (Il-2) diskutiert. Il-2 ist ein losliches, nicht antigenspezifisches Glykoprotein mit einem Molekulargewicht von 15 000 Dalton (humanes Il-2) bzw. 30 000 Dalton (murines Il-2). Es wird in vitro von T-Helfer-Lymphozyten sezerniert, die in der Mau…

MacrophagesLymphocyte CooperationDrug DiscoveryHumansInterleukin-2Molecular MedicineT-Lymphocytes Helper-InducerGeneral MedicineReceptors ImmunologicModels BiologicalGenetics (clinical)Interleukin-1T-Lymphocytes CytotoxicKlinische Wochenschrift
researchProduct

Nandrolone decanoate interferes with testosterone biosynthesis altering blood-testis barrier components

2017

Abstract The aim of this study was to investigate whether nandrolone decanoate (ND) use affects testosterone production and testicular morphology in a model of trained and sedentary mice. A group of mice underwent endurance training while another set led a sedentary lifestyle and were freely mobile within cages. All experimental groups were treated with either ND or peanut oil at different doses for 6 weeks. Testosterone serum levels were measured via liquid chromatography–mass spectrometry. Western blot analysis and quantitative real‐time PCR were utilized to determine gene and protein expression levels of the primary enzymes implicated in testosterone biosynthesis and gene expression leve…

Male0301 basic medicinemedicine.medical_specialtyNandrolone decanoateStimulationEndogenyMUC1BiologyMale infertilityMice03 medical and health sciencesAnabolic AgentsWestern blotPhysical Conditioning AnimalInternal medicineTestisGene expressionmedicineAnimalsNandroloneBlood-testis barrier; MMP-2; MMP-9; MUC1; Nandrolone decanoate; Testosterone; TJP1; Molecular Medicine; Cell Biologyblood–testis barrierInducerTestosteroneTJP1TestosteroneBlood-testis barrierBlood–testis barrierMMP‐9medicine.diagnostic_testMMP-2Mucin-1Tissue Inhibitor of MetalloproteinasesOriginal ArticlesCell Biologymedicine.diseaseProtein Transport030104 developmental biologyEndocrinologyGene Expression RegulationMatrix Metalloproteinase 9Zonula Occludens-1 ProteinMatrix Metalloproteinase 2Molecular MedicineOriginal ArticleMMP‐2Sedentary BehaviorMMP-9Signal Transduction
researchProduct

A human relevance investigation of PPARα-mediated key events in the hepatocarcinogenic mode of action of propaquizafop in rats

2018

Propaquizafop is an herbicide with demonstrated hepatocarcinogenic activity in rodents. A rodent-specific mode of action (MOA) in the liver via activation of peroxisome proliferator-activated receptor α (PPARα) has been postulated based on existing data. Experience with PPARα-inducing pharmaceuticals indicates a lack of human relevance of this MOA. The objective of the present investigation was to evaluate the dependency of early key events leading to liver tumors on PPARα activation in wildtype (WT) compared to PPARα-knockout (KO) rats following 2 weeks exposure to 75, 500 and 1000 ppm propaquizafop in the diet. In WT rats, both WY-14643 (50 mg/kg bw/day) and propaquizafop (dose-dependentl…

Male0301 basic medicinemedicine.medical_specialtyPeroxisome proliferator-activated receptor010501 environmental sciencesBiologyToxicologyRisk Assessment01 natural sciencesMuscle hypertrophyRats Sprague-Dawley03 medical and health sciencesCytochrome P-450 Enzyme SystemInternal medicinemedicineAnimalsHumansAcyl-CoA oxidasePPAR alphaRelevance (information retrieval)Enzyme inducerReceptorMode of actionCarcinogen0105 earth and related environmental scienceschemistry.chemical_classificationGlutathione PeroxidaseHerbicidesGlutathione peroxidaseLiver NeoplasmsOrgan SizeGeneral MedicineGlutathioneDiet030104 developmental biologyEndocrinologyLiverchemistrybiology.proteinKey (cryptography)Acyl-CoA OxidasePropionatesRats TransgenicNeuroscienceToxicology Letters
researchProduct