Search results for "Inflammation."

showing 10 items of 2627 documents

Relationships of growth factors, proinflammatory cytokines, and anti-inflammatory cytokines with long-term clinical results of autologous bone marrow…

2017

Aim The aim of the study was to test the hypothesis suggesting that the pre-intervention levels of proinflammatory cytokines, anti-inflammatory cytokines, and angiogenic growth factors predict the long-term clinical results of autologous bone marrow-derived mononuclear cell (ABMMC) transplantation in patients with primary ST elevation myocardial infarction (STEMI). Methods and results From 2003 to 2006, a total of 62 patients with primary STEMI were enrolled in an open randomized study registered under the title ESTABOMA. Patients were randomized into two groups: group 1 included patients treated with percutaneous coronary intervention (PCI) and ABMMC transplantation (n = 28); group 2 compr…

Male0301 basic medicinePhysiologyCardiovascular ProceduresCell Transplantationmedicine.medical_treatmentMyocardial InfarctionSocial Scienceslcsh:Medicine030204 cardiovascular system & hematologyPathology and Laboratory MedicineVascular MedicineGastroenterologyAnginaEndocrinology0302 clinical medicineImmune PhysiologyMedicine and Health SciencesBlood and Lymphatic System ProceduresPsychologyMyocardial infarctionlcsh:ScienceImmune ResponseBone Marrow TransplantationInnate Immune SystemMultidisciplinaryCardiac TransplantationAnginaMiddle AgedBrain natriuretic peptidesurgical procedures operativeCytokinesIntercellular Signaling Peptides and ProteinsFemaleInflammation MediatorsResearch Articlemedicine.medical_specialtySocial PsychologyImmunologyCardiologySurgical and Invasive Medical ProceduresTransplantation AutologousProinflammatory cytokine03 medical and health sciencesSigns and SymptomsDiagnostic MedicineGrowth FactorsInternal medicineчрескожное коронарное вмешательствоmedicineHumanscardiovascular diseasesInflammationTransplantationEndocrine Physiologybusiness.industrylcsh:RBiology and Life SciencesPercutaneous coronary interventionOrgan TransplantationMolecular Developmentmedicine.diseaseпровоспалительные цитокиныинфаркт миокардаTransplantation030104 developmental biologyImmune SystemHeart failureImmunologyConventional PCIтрансплантация мононуклеарных клетокlcsh:Qbusinessаутологичная трансплантация костного мозгаDevelopmental BiologyStem Cell Transplantation
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Polyphenol intake and cardiovascular risk factors in a population with type 2 diabetes: The TOSCA.IT study

2017

Background: The role of polyphenol intake on cardiovascular risk factors is little explored, particularly in people with diabetes. Aim: To evaluate the association between the intake of total polyphenols and polyphenol classes with the major cardiovascular risk factors in a population with type 2 diabetes. Methods: Dietary habits were investigated in 2573 males and females participants of the TOSCA.IT study. The European Prospective Investigation on Cancer and Nutrition (EPIC) questionnaire was used to assess dietary habits. In all participants, among others, we assessed anthropometry, plasma lipids, blood pressure, C-reactive protein and HbA1c following a standard protocol. The USDA and Ph…

Male0301 basic medicinePhysiologyType 2 diabetes030204 cardiovascular system & hematologyCritical Care and Intensive Care MedicineSettore MED/13 - Endocrinologiachemistry.chemical_compound0302 clinical medicineDietary polyphenolsFlavonoidsPhenolic acidsCardiovascular riskType 2 diabetesLOW-GRADE INFLAMMATIONBLOOD-PRESSUREDIETARY POLYPHENOLSDARK CHOCOLATEINSULIN SENSITIVITYRANDOMIZED-TRIALSDISEASEMETAANALYSISMETABOLISMCHOLESTEROLRisk FactorsSurveys and QuestionnairesHydroxybenzoatesProspective StudiesFood scienceCardiovascular risk; Dietary polyphenols; Flavonoids; Phenolic acids; Type 2 diabeteseducation.field_of_studyPhenolic acidNutrition and Dieteticsbiologyfood and beveragesType 2 diabetesDietary polyphenolMiddle AgedDietary polyphenols; Flavonoids; Phenolic acids; Cardiovascular risk; Type 2 diabetesEuropean Prospective Investigation into Cancer and NutritionDietary polyphenolsCardiovascular DiseasesFemalePhenolic acidsPopulationCardiovascular risk Dietary polyphenols Flavonoids Phenolic acids Type 2 diabetes03 medical and health sciencesDiabetes mellitusmedicineHumanseducationTriglyceridesAgedFlavonoids030109 nutrition & dieteticsDose-Response Relationship Drugbusiness.industryCholesterol HDLC-reactive proteinPolyphenolsCholesterol LDLAnthropometrymedicine.diseaseCardiovascular riskCardiovascular risk; Dietary polyphenols; Flavonoids; Phenolic acids; Type 2 diabetes; Nutrition and Dietetics; Critical Care and Intensive Care MedicineDietCross-Sectional StudiesNutrition AssessmentDiabetes Mellitus Type 2chemistryFlavonoidbiology.proteinGlycated hemoglobinbusinessBody mass index
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Identification of periplakin as a major regulator of lung injury and repair in mice

2018

IF 12.784 (2016); International audience; Periplakin is a component of the desmosomes that acts as a cytolinker between intermediate filament scaffolding and the desmosomal plaque. Periplakin is strongly expressed by epithelial cells in the lung and is a target antigen for autoimmunity in idiopathic pulmonary fibrosis. The aim of this study was to determine the role of periplakin during lung injury and remodeling in a mouse model of lung fibrosis induced by bleomycin. We found that periplakin expression was downregulated in the whole lung and in alveolar epithelial cells following bleomycin-induced injury. Deletion of the Ppl gene in mice improved survival and reduced lung fibrosis developm…

Male0301 basic medicinePulmonologylcsh:MedicineMouse modelsMiceIdiopathic pulmonary fibrosischemistry.chemical_compoundFibrosisPeriplakinMice KnockoutLung InjuryGeneral Medicinerespiratory system3. Good healthmedicine.anatomical_structureCytokinesmedicine.symptomSignal TransductionResearch ArticleCell signalingDown-RegulationInflammationRespiratory MucosaLung injuryBleomycinBleomycin03 medical and health sciencesmedicineAnimalsHumansInflammationLung030102 biochemistry & molecular biologybusiness.industryMacrophagesPlakinslcsh:Rmedicine.diseaseFibrosisIdiopathic Pulmonary Fibrosisrespiratory tract diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologychemistryAlveolar Epithelial CellsCancer researchbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyJCI Insight
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Blockade of the trans-sulfuration pathway in acute pancreatitis due to nitration of cystathionine β-synthase

2019

© 2019 Published by Elsevier B.V.

Male0301 basic medicineS-AdenosylmethionineHomocysteineClinical BiochemistryNitric Oxide Synthase Type IINitrosative stressBiochemistryMicechemistry.chemical_compound0302 clinical medicineEdemaMedicineAcute inflammationHomocysteinelcsh:QH301-705.5lcsh:R5-920biologyGlutathioneUp-Regulationmedicine.anatomical_structureAcute pancreatitismedicine.symptomPancreaslcsh:Medicine (General)CeruletideResearch Papermedicine.medical_specialtyCystathionine beta-Synthase03 medical and health sciencesCystathionineInternal medicineAnimalsCysteineCystathionine β-synthaseS-adenosylmethionineMethioninebusiness.industryOrganic ChemistryGlutathionemedicine.diseaseCystathionine beta synthaseDisease Models Animal030104 developmental biologyEndocrinologyPancreatitischemistrylcsh:Biology (General)biology.proteinbusiness030217 neurology & neurosurgeryCysteineRedox Biology
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Effect of a Prebiotic Formulation on Frailty Syndrome

2016

Aging can result in major changes in the composition and metabolic activities of bacterial populations in the gastrointestinal system and result in impaired function of the immune system. We assessed the efficacy of prebiotic Darmocare Pre (R) (Bonusan Besloten Vennootschap (BV), Numansdorp, The Netherlands) to evaluate whether the regular intake of this product can improve frailty criteria, functional status and response of the immune system in elderly people affected by the frailty syndrome. The study was a placebo-controlled, randomized, double blind design in sixty older participants aged 65 and over. The prebiotic product was composed of a mixture of inulin plus fructooligosaccharides …

Male0301 basic medicineSarcopeniamedicine.medical_treatmentDiseaseGut floraDISEASElcsh:Chemistrychemistry.chemical_compoundCognition0302 clinical medicineleucocytesMedicinelcsh:QH301-705.5SpectroscopyAged 80 and overfatigue; biomarker; leucocytes; inflammation; agingHand StrengthbiologyGUT MICROBIOTAInulinPHYSICAL-ACTIVITY QUESTIONNAIREGeneral MedicineComputer Science ApplicationsBiomarker (medicine)biomarkerPOPULATIONSFemaleHEALTHmedicine.medical_specialtyFrail ElderlyInulinFrailty syndromePlaceboArticleCatalysisVALIDATIONDIETInorganic Chemistry03 medical and health sciencesDouble-Blind MethodINTESTINAL MICROBIOTAInternal medicineHumansOLDER ADULTSPhysical and Theoretical ChemistryGeriatric AssessmentMolecular BiologyAgedbusiness.industryPrebioticOrganic ChemistryagingCONSUMPTIONbiology.organism_classificationmedicine.diseaseClinical trialPrebiotics030104 developmental biologylcsh:Biology (General)lcsh:QD1-999chemistryinflammationPhysical therapyfatigueSleepbusiness030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Drug survival of anakinra and canakinumab in monogenic autoinflammatory diseases: observational study from the International AIDA Registry

2021

Abstract Objectives To investigate survival of IL-1 inhibitors in monogenic autoinflammatory disorders (mAID) through drug retention rate (DRR) and identify potential predictive factors of drug survival from a real-life perspective. Patients and methods Multicentre retrospective study analysing patients affected by the most common mAID treated with anakinra or canakinumab. Survival curves were analysed with the Kaplan-Meier method. Statistical analysis included a Cox-proportional hazard model to detect factors responsible for drug discontinuation. Results Seventy-eight patients for a total of 102 treatment regimens were enrolled. The mean treatment duration was 29.59 months. The estimated D…

Male0301 basic medicineTime FactorsSettore MED/16 - REUMATOLOGIAInterleukin-1beta0302 clinical medicineSettore MED/38 - Pediatria Generale E SpecialisticaMonoclonalPharmacology (medical)RegistriesHumanizedmedia_commonIL-1 anakinra canakinumab innovative biotechnologies monogenic autoinflammatory disorders personalized medicinepersonalized medicineMiddle AgedPenetranceTreatment OutcomeAnakinraAntirheumatic AgentsAutoinflammationIL-1; anakinra; canakinumab; innovative biotechnologies; monogenic autoinflammatory disorders; personalized medicine; Adult; Antibodies Monoclonal Humanized; Antirheumatic Agents; Female; Follow-Up Studies; Hereditary Autoinflammatory Diseases; Humans; Interleukin 1 Receptor Antagonist Protein; Interleukin-1beta; Male; Middle Aged; Retrospective Studies; Time Factors; Treatment Outcome; Young Adult; RegistriesFemalemedicine.drugAdultDrugmedicine.medical_specialtymedia_common.quotation_subjectAntibodies Monoclonal HumanizedcanakinumabAntibodiesYoung Adult03 medical and health sciencesinnovative biotechnologiesRheumatologyInternal medicinemedicineHumansAdverse effectSurvival analysismonogenic autoinflammatory disordersRetrospective Studies030203 arthritis & rheumatologyAnakinraIL-1business.industryHereditary Autoinflammatory DiseasesRetrospective cohort studyInterleukin 1 Receptor Antagonist ProteinCanakinumab030104 developmental biologyObservational studybusinessFollow-Up Studies
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Administration of all‐ trans retinoic acid after experimental traumatic brain injury is brain protective

2020

BACKGROUND AND PURPOSE: All‐trans retinoic acid (ATRA) is a vitamin A metabolite, important in the developing and mature brain. Pre‐injury ATRA administration ameliorates ischaemic brain insults in rodents. This study examined the effects of post‐traumatic ATRA treatment in experimental traumatic brain injury (TBI). EXPERIMENTAL APPROACH: Male adult mice were subjected to the controlled cortical impact model of TBI or sham procedure and killed at 7 or 30 days post‐injury (dpi). ATRA (10 mg kg−1, i.p.) was given immediately after the injury and 1, 2 and 3 dpi. Neurological function and sensorimotor coordination were evaluated. Brains were processed for (immuno‐) histological, mRNA and protei…

Male0301 basic medicineTraumatic brain injuryRetinoic acidTretinoinPharmacologyHippocampal formationHMGB1Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBrain Injuries TraumaticmedicineAnimalsInflammationPharmacologyMicrogliabiologybusiness.industryBrainmedicine.diseaseGranule cellResearch PapersAstrogliosis030104 developmental biologymedicine.anatomical_structurechemistryBlood-Brain BarrierApoptosisbiology.proteinbusiness030217 neurology & neurosurgeryBritish Journal of Pharmacology
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Pancreatic Protein Tyrosine Phosphatase 1B Deficiency Exacerbates Acute Pancreatitis in Mice

2016

Acute pancreatitis (AP) is a common and devastating gastrointestinal disorder that causes significant morbidity. The disease starts as local inflammation in the pancreas that may progress to systemic inflammation and complications. Protein tyrosine phosphatase 1B (PTP1B) is implicated in inflammatory signaling, but its significance in AP remains unclear. To investigate whether PTP1B may have a role in AP, we used pancreas PTP1B knockout (panc-PTP1B KO) mice and determined the effects of pancreatic PTP1B deficiency on cerulein- and arginine-induced acute pancreatitis. We report that PTP1B protein expression was increased in the early phase of AP in mice and rats. In addition, histological an…

Male0301 basic medicineWistarSystemic inflammationMedical and Health SciencesOral and gastrointestinalMicePathology2.1 Biological and endogenous factorsAetiologyNon-Receptor Type 1CancerMice KnockoutProtein Tyrosine Phosphatase Non-Receptor Type 1Pancreatitis Acute NecrotizingReverse Transcriptase Polymerase Chain ReactionRegular Articlemedicine.anatomical_structureAcute NecrotizingGastrointestinal disorderAcute pancreatitisTumor necrosis factor alphamedicine.symptomPancreashormones hormone substitutes and hormone antagonistsmedicine.medical_specialtyKnockoutInflammationPathology and Forensic MedicineProinflammatory cytokinePancreatic Cancer03 medical and health sciencesRare DiseasesInternal medicinemedicineAnimalsRats WistarAnimalbusiness.industrymedicine.diseaseRatsDisease Models Animal030104 developmental biologyEndocrinologyPancreatitisDisease ModelsPancreatitisProtein Tyrosine PhosphataseDigestive DiseasesbusinessThe American Journal of Pathology
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Mast cells' involvement in inflammation pathways linked to depression: evidence in mastocytosis

2016

International audience; Converging sources of evidence point to a role for inflammation in the development of depression, fatigue and cognitive dysfunction. More precisely, the tryptophan (TRP) catabolism is thought to play a major role in inflammation-induced depression. Mastocytosis is a rare disease in which chronic symptoms, including depression, are related to mast cell accumulation and activation. Our objectives were to study the correlations between neuropsychiatric features and the TRP catabolism pathway in mastocytosis in order to demonstrate mast cells' potential involvement in inflammation-induced depression. Fifty-four patients with mastocytosis and a mean age of 50.1 years were…

Male0301 basic medicine[SHS.PSY]Humanities and Social Sciences/PsychologyKynurenic Acidchemistry.chemical_compound0302 clinical medicineKynurenic acidMast CellsIndoleamine 23-dioxygenaseAcute stressQuinolinic acidKynurenineDepression (differential diagnoses)DepressionTryptophanMiddle AgedMast cellRat-brain3. Good healthPsychiatry and Mental healthmedicine.anatomical_structure[ SCCO.NEUR ] Cognitive science/NeuroscienceFemalemedicine.symptomMastocytosisSerotoninmedicine.medical_specialtyInflammationAryl-hydrocarbon receptorCentral-nervous-system[ SHS.PSY ] Humanities and Social Sciences/Psychology03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicinemedicineHumansIndoleamine-Pyrrole 23-Dioxygenase[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyMolecular BiologyInflammationPsychiatric Status Rating ScalesDepressive Disorder Majorbusiness.industry[SCCO.NEUR]Cognitive science/NeuroscienceBeck Depression InventoryInterferon-alphaMammalian brain030104 developmental biologyEndocrinologyImmune-systemchemistryImmunologyIndoleamine 2?3-dioxygenasebusinessStress Psychological030217 neurology & neurosurgeryKynurenineQuinolinic acid
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Fungal Dysbiosis and Intestinal Inflammation in Children With Beta-Cell Autoimmunity

2020

Although gut bacterial dysbiosis is recognized as a regulator of beta-cell autoimmunity, no data is available on fungal dysbiosis in the children at the risk of type 1 diabetes (T1D). We hypothesized that the co-occurrence of fungal and bacterial dysbiosis contributes to the intestinal inflammation and autoimmune destruction of insulin-producing beta-cells in T1D. Fecal and blood samples were collected from 26 children tested positive for at least one diabetes-associated autoantibody (IAA, GADA, IA-2A or ICA) and matched autoantibody-negative children with HLA-conferred susceptibility to T1D (matched for HLA-DQB1 haplotype, age, gender and early childhood nutrition). Bacterial 16S and funga…

Male0301 basic medicinebeta-Defensinstype 1 diabetessuolistomikrobistoAutoimmunityGut floramedicine.disease_causeautoimmuniteettiAutoimmunityFeces0302 clinical medicineautoimmuunisairaudetInsulin-Secreting CellsHLA-DQ beta-ChainsImmunology and AllergyMedicineChildFinlandOriginal ResearchCandida2. Zero hungerRISKMUCOSAtulehdusbiologyGUT MICROBIOTAdysbiosisFungal antigen3. Good healthChild PreschoolgutCATHELICIDIN LL-37Femalemedicine.symptomlcsh:Immunologic diseases. AllergyAdolescentImmunologyInflammationIMMUNITY03 medical and health sciencesmycobiomeSaccharomycesSEROCONVERSIONHumansPERMEABILITYAntibodies FungalTYPE-1AutoantibodiesType 1 diabetesbusiness.industrynuoruustyypin diabetesAutoantibodymedicine.diseasebiology.organism_classificationDiabetes Mellitus Type 1030104 developmental biologyMycoseshiivasienetinflammation3121 General medicine internal medicine and other clinical medicineImmunologyANTIBODIESONSET3111 BiomedicineCalprotectinbusinesslcsh:RC581-607Dysbiosis030215 immunology
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