Search results for "Inflammation."

showing 10 items of 2627 documents

Double Negative (IgG+IgD-CD27-) B Cells are Increased in a Cohort of Moderate-Severe Alzheimer’s Disease Patients and Show a Pro-Inflammatory Traffic…

2014

Alzheimer's disease (AD) is a progressive, irreversible, and debilitating disease for which no effective preventive or disease modifying therapies or treatments have so far been detected. The crucial step in AD pathogenesis is the production of amyloid-42 peptide, which causes chronic inflammation. Activated cells in the central nervous system (CNS) produce pro- inflammatory mediators that lead to the recruitment of myeloid or lymphocytic cells. As a consequence, the communication between the CNS and peripheral blood of AD subjects could influence the lymphocyte distribution and/or the expression of phenotypic markers. In the present paper, we show a significant decrease in total CD19 + B l…

MaleReceptors CCR6Receptors CCR7MyeloidLymphocyteB-Lymphocyte SubsetsC-C chemokine receptor type 7InflammationC-C chemokine receptor type 6Immunoglobulin DCD19Cohort StudiesAlzheimer DiseasemedicineHumansB cellAgedAged 80 and overSettore MED/04 - Patologia GeneralebiologyGeneral NeuroscienceGeneral MedicineImmunoglobulin DFlow CytometryTumor Necrosis Factor Receptor Superfamily Member 7Psychiatry and Mental healthClinical Psychologymedicine.anatomical_structurePhenotypeAlzheimer's Disease Inflammation B CellsImmunoglobulin GImmunologybiology.proteinFemaleSettore MED/26 - NeurologiaGeriatrics and Gerontologymedicine.symptomMental Status Schedule
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Co-option of Neutrophil Fates by Tissue Environments

2020

Classically considered short-lived and purely defensive leukocytes, neutrophils are unique in their fast and moldable response to stimulation. This plastic behavior may underlie variable and even antagonistic functions during inflammation or cancer, yet the full spectrum of neutrophil properties as they enter healthy tissues remains unexplored. Using a new model to track neutrophil fates, we found short but variable lifetimes across multiple tissues. Through analysis of the receptor, transcriptional, and chromatin accessibility landscapes, we identify varying neutrophil states and assign non-canonical functions, including vascular repair and hematopoietic homeostasis. Accordingly, depletion…

MaleReceptors CXCR4Transcription GeneticAngiogenesisNeutrophilsMedizinNeovascularization PhysiologicInflammationBiologyCXCR4General Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineSingle-cell analysismedicineNuclear Receptor Subfamily 4 Group A Member 1AnimalsCell LineageReceptorLung030304 developmental biology0303 health sciencesInnate immune systemChromatinChromatinCell biologyHematopoiesisIntestinesMice Inbred C57BLOrgan SpecificityFemalemedicine.symptomSingle-Cell AnalysisTranscriptome030217 neurology & neurosurgeryHomeostasisCell
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Exercise promotes IL-6 release from legs in older men with minor response to unilateral immobilization

2015

Physical inactivity is a major contributor to low-grade systemic inflammation. Most of the studies characterizing interleukin-6 (IL-6) and tumour necrosis factor-α (TNF-α) release from exercising legs have been done in young, healthy men, but studies on inactivity in older people are lacking. The impact of 14 days of one-leg immobilization (IM) on IL-6 and TNF-α release during exercise in comparison to the contralateral control (CON) leg was investigated. Fifteen healthy men (age 68.1 ± 1.1 year (mean ± SEM); BMI 27.0 ± 0.4 kg·m(2); VO2max 33.3 ± 1.6 ml·kg(‒1)·min(‒1)) performed 45 min of two-leg dynamic knee extensor exercise at 19.5 ± 0.9 W. Arterial and femoral venous blood samples from …

MaleRestraint Physical0301 basic medicinemedicine.medical_specialtyNecrosisPhysical Therapy Sports Therapy and RehabilitationThighSystemic inflammationCohort Studies03 medical and health sciences0302 clinical medicineHumansMedicineOrthopedics and Sports MedicineInterleukin 6ExerciseAgedLegKnee extensorsbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryGeneral MedicineVenous bloodBlood flowSurgery030104 developmental biologymedicine.anatomical_structureAnesthesiabiology.proteinmedicine.symptombusiness030217 neurology & neurosurgeryMinor ResponseEuropean Journal of Sport Science
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Anti-Inflammatory Treatment with Standardized Human Serum Protein Solution Reduces Local and Systemic Inflammatory Response after Hemorrhagic Shock

2005

<i>Objective:</i> Reperfusion after hemorrhagic shock leads to local and systemic inflammatory response. This study evaluates the effect of a short-term treatment with standardized human serum protein solution (SPS) on the local and systemic inflammatory response in the mesenteric microcirculation in the rat. <i>Methods:</i> Spontaneously breathing animals underwent median laparotomy and exteriorization of an ileal loop for intravital microscopy of the mesenteric microcirculation. Volume-controlled hemorrhagic shock was set by arterial blood withdrawal (2.5 ml/100 g body weight for 60 min), followed by reperfusion for 4 h. SPS (n = 10) or saline 0.9% (controls, n = 1…

MaleResuscitationCentral Venous PressureInflammatory responseAnti-Inflammatory AgentsHemodynamicsBlood PressureInflammationShock HemorrhagicMicrocirculationRats Sprague-DawleyHeart RateLeukocytesAnimalsMedicinebusiness.industryMicrocirculationHemodynamicsBlood ProteinsBlood proteinsRatsBlood pressureShock (circulatory)ImmunologySurgeryBlood Gas Analysismedicine.symptombusinessEuropean Surgical Research
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Impact of adenotonsillectomy on the evolution of inflammatory markers

2019

Background Tonsils are first-line host defence organs against pathogenic agents and participate in local and systemic immunity. Persistent increases in systemic inflammatory responses may contribute to associated morbidity. The aim of this study was to verify the short- and long-term impact of adenotonsillectomy on the evolution of inflammatory markers in 3- to 9-year-old children. Methods A prospective and longitudinal study was conducted over 1 year in 29 children who underwent tonsillectomy due to either chronic tonsillitis or adenotonsillar hypertrophy. Measurements of high-sensitivity C-reactive protein (hs-CRP) levels were taken. Levels of Th1-type cytokines [interleukin-1, interferon…

MaleRhinologymedicine.medical_specialtyLongitudinal studyTime Factorsmedicine.medical_treatmentAdhesion (medicine)GastroenterologyProinflammatory cytokineAdenoidectomy03 medical and health sciences0302 clinical medicineInternal medicineHumansMedicineLongitudinal StudiesProspective StudiesChild030223 otorhinolaryngologyTonsillectomyInflammationbusiness.industryHypertrophyHost defencemedicine.diseaseTonsillectomyTonsillitisC-Reactive ProteinOtorhinolaryngologyOtorhinolaryngologyChild Preschool030220 oncology & carcinogenesisChronic DiseaseCytokinesFemaleTumor necrosis factor alphabusinessBiomarkersClinical Otolaryngology
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Asthmatic bronchial epithelial cells have a deficient innate immune response to infection with rhinovirus

2005

Rhinoviruses are the major trigger of acute asthma exacerbations and asthmatic subjects are more susceptible to these infections. To investigate the underlying mechanisms of this increased susceptibility, we examined virus replication and innate responses to rhinovirus (RV)-16 infection of primary bronchial epithelial cells from asthmatic and healthy control subjects.Viral RNA expression and late virus release into supernatant was increased 50- and 7-fold, respectively in asthmatic cells compared with healthy controls. Virus infection induced late cell lysis in asthmatic cells but not in normal cells. Examination of the early cellular response to infection revealed impairment of virus induc…

MaleRhinovirusvirusesCHILDRENApoptosisResearch & Experimental MedicineINHALED CORTICOSTEROIDSmedicine.disease_causeVirus ReplicationImmunology and AllergyTRANSCRIPTIONCells CulturedCaspase 7Caspase 311 Medical And Health SciencesMiddle AgedMedicine Research & ExperimentalCaspasesRNA ViralFemalemedicine.symptomRhinovirusLife Sciences & BiomedicineEXPRESSIONAdultVIRUSESImmunologyInflammationBronchiBiologyAntiviral AgentsVirusArticleImmune systemINFLAMMATIONImmunitymedicineKINASELOWER AIRWAYSHumansInnate immune systemScience & TechnologyPicornaviridae InfectionsRECEPTOREpithelial CellsInterferon-betaAsthmaImmunity InnateEXACERBATIONSViral replicationGene Expression RegulationApoptosisImmunology
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Recent insights into the relationship between inflammatory liver diseases and atherosclerosis.

2011

Atherosclerosis is a dynamic process in the human body. Many studies have evaluated atherosclerosis and its relationship with other systems in the body. Our perception of its pathogenesis is evolving with the introduction of new players in the game. It is no longer possible to consider the atherosclerosis as an independent process, unaffected by the liver and its function. Although several tasks performed by the liver, such as lipid metabolism, have been implicated in the pathogenesis of atherosclerosis, the role of other disorders of the liver (autoimmune diseases, viral hepatitis, and cirrhosis) are not fully understood. In this review, the most commonly encountered inflammatory liver di…

MaleRiskHepatitis B virusCirrhosisInflammationHepacivirusBiologyGeneral Biochemistry Genetics and Molecular BiologyAutoimmune DiseasesHepatitisPathogenesisFibrosisPrevalencemedicineHumansInflammationHepatitisCholestasisLiver DiseasesResearchFatty liverLipid metabolismGeneral MedicineAtherosclerosismedicine.diseasehepatitis B hepatitis C hepatitis A inflammation fatty liver atherosclerosisFibrosisFatty LiverLiverImmunologyFemalemedicine.symptomViral hepatitis
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Soluble CD40L and Cardiovascular Risk in Asymptomatic Low-Grade Carotid Stenosis

2005

Background and Purpose— We investigated whether soluble CD40L (sCD40L) may predict the risk of cardiovascular (CV) events in patients with asymptomatic carotid plaques. Methods— Forty-two patients with asymptomatic low-grade carotid stenosis (ALCS) and 21 controls without any carotid stenosis were enrolled. All subjects had at least a major cardiovascular risk factor (CRF). Plasma levels of C-reactive protein (CRP), IL-6, and sCD40L were measured. Subjects were reviewed every 12 months (median follow-up, 8 years). Results— ALCS patients had higher ( P <0.0001) CRP, IL-6, and sCD40L than controls. Fourteen patients experienced a CV event. Cox regression analysis showed that only high sCD…

MaleRiskmedicine.medical_specialtySoluble CD40LCD40 LigandAsymptomaticatherosclerosiPredictive Value of TestsInternal medicineHumansrisk factorsMedicineRisk factorStrokeProportional Hazards ModelsAdvanced and Specialized NursingbiologyInterleukin-6business.industryProportional hazards modelVascular diseaseC-reactive proteinMiddle Agedmedicine.diseaseSurgeryStrokecarotid stenosiStenosisC-Reactive ProteinSolubilityinflammationPredictive value of testsbiology.proteinCardiologycarotid stenosisFemaleNeurology (clinical)atherosclerosismedicine.symptomatherosclerosis; carotid stenosis; inflammation; risk factorsCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesStroke
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Metabolic effects of the obstructive sleep apnea syndrome and cardiovascular risk

2008

The obstructive sleep apnea syndrome (OSAS) is characterized by collapse of the upper airway during sleep, recurring apneas, intermittent hypoxemia and daytime somnolence. OSAS is often associated with obesity, and its prevalence is expected to rise due to the obesity epidemics worldwide. OSAS is associated with increased cardiovascular risk which appears to be normalized by treatment with nasal continuous positive airway pressure (nCPAP) during sleep, suggesting an independent role of OSAS in accelerating atherosclerosis. Insulin resistance (IR) and the metabolic syndrome (MetS) are often found in OSAS patients, but the relative role played by OSAS and obesity is still unclear. Both OSAS a…

MaleRiskmedicine.medical_specialtySympathetic nervous systemPhysiologymedicine.medical_treatmentSettore MED/10 - Malattie Dell'Apparato RespiratorioGlucose metabolism dyslipidemia sleep loss animal models children adipocyteHypoxemiaInsulin resistancestomatognathic systemPhysiology (medical)Internal medicineAdipocytesmedicineAnimalsHumansObesityContinuous positive airway pressureChildInflammationSleep Apnea Obstructivebusiness.industryGeneral Medicinemedicine.diseaseObesitynervous system diseasesrespiratory tract diseasesObstructive sleep apneaDisease Models AnimalGlucosemedicine.anatomical_structureEndocrinologyAdipose TissueCardiovascular DiseasesCardiologyMetabolic syndromemedicine.symptombusinessDyslipidemiaArchives of Physiology and Biochemistry
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Interruption of Macrophage-Derived IL-27(p28) Production by IL-10 during Sepsis Requires STAT3 but Not SOCS3

2014

Abstract Severe sepsis and septic shock are leading causes of morbidity and mortality worldwide. Infection-associated inflammation promotes the development and progression of adverse outcomes in sepsis. The effects of heterodimeric IL-27 (p28/EBI3) have been implicated in the natural course of sepsis, whereas the molecular mechanisms underlying the regulation of gene expression and release of IL-27 in sepsis are poorly understood. We studied the events regulating the p28 subunit of IL-27 in endotoxic shock and polymicrobial sepsis following cecal ligation and puncture. Neutralizing Abs to IL-27(p28) improved survival rates, restricted cytokine release, and reduced bacterial burden in C57BL/…

MaleSTAT3 Transcription Factormedicine.medical_treatmentImmunologySuppressor of Cytokine Signaling ProteinsInflammationSpleenBiologyArticleSepsisMiceSepsismedicineAnimalsHumansImmunology and AllergyReceptors CytokineAntibodies BlockingCecumCells CulturedMice KnockoutSeptic shockInterleukinsMacrophagesReceptors Interleukinmedicine.diseaseBacterial LoadInterleukin-10Mice Inbred C57BLToll-Like Receptor 4Adaptor Proteins Vesicular TransportDisease Models AnimalOxidative StressInterleukin 10Cytokinemedicine.anatomical_structureIntegrin alpha MSuppressor of Cytokine Signaling 3 ProteinMyeloid Differentiation Factor 88ImmunologyTLR4biology.proteinmedicine.symptomJournal of Immunology
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