Search results for "Inflammation."

showing 10 items of 2627 documents

Circulating miR-21, miR-146a and Fas ligand respond to postmenopausal estrogen-based hormone replacement therapy--a study with monozygotic twin pairs.

2014

Biological aging is associated with physiological deteriorations and its’ remodeling, which are partly due to changes in the hormonal profile. MicroRNAs are known to post-transcriptionally regulate various cellular processes associated with cell senescence; differentiation, replication and apoptosis. Measured from the serum, microRNAs have the potential to serve as noninvasive markers for diagnostics/prognostics and therapeutic targets. We analysed the association of estrogen-based hormone replacement therapy (HRT) with selected microRNAs and inflammation markers from the serum, leukocytes and muscle tissue biopsy samples obtained from 54-62 year-old postmenopausal monozygotic twins (n=11 p…

SenescenceAdultmedicine.medical_specialtyAgingFas Ligand Proteinmedicine.drug_classmedicine.medical_treatmentMonozygotic twinInflammationApoptosisBiologyta3111Fas ligand“Inflamm-aging”Internal medicinemicroRNAmedicineestrogenHumansmicrornasMuscle SkeletalHormone therapyCellular SenescenceInflammationmicroRNAEstrogen Replacement TherapyapoptosisHormone replacement therapy (menopause)ta3141Cell DifferentiationEstrogenstwinsTwins MonozygoticMiddle AgedPostmenopauseAgeinghormone replacement therapyMicroRNAsEndocrinologyEstrogenFemalemedicine.symptomBiomarkersDevelopmental BiologyHormoneMechanisms of ageing and development
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Oxidative stress in environmental-induced carcinogenesis.

2009

Reactive oxygen species (ROS) are the more abundant free radicals in nature and have been related with a number of tissue/organ injuries induced by xenobiotics, ischemia, activation of leucocytes, UV exposition, etc. Oxidative stress is caused by an imbalance between ROS production and a biological system's ability to readily detoxify these reactive intermediates or easily repair the resulting damage. Thus, oxidative stress is accepted as a critical pathophysiological mechanism in different frequent human pathologies, including cancer. In fact ROS can cause protein, lipid, and DNA damage, and malignant tumors often show increased levels of DNA base oxidation and mutations. Different lifesty…

SenescenceAgingDNA damageHealth Toxicology and MutagenesisInflammationOxidative phosphorylationBiologymedicine.disease_causeModels BiologicalNeoplasmsGeneticsmedicineAnimalsHumansObesityLife StyleCarcinogenchemistry.chemical_classificationReactive oxygen speciesCarcinogens EnvironmentalOxidative StressCell Transformation NeoplasticBiochemistrychemistryCancer researchmedicine.symptomCarcinogenesisReactive Oxygen SpeciesOxidative stressMutation research
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Low Grade Inflammation as a Common Pathogenetic Denominator in Age-Related Diseases: Novel Drug Targets for Anti-Ageing Strategies and Successful Age…

2010

Nowadays, people are living much longer than they used to do, however they are not free from ageing. Ageing, an inexorable intrinsic process that affects all cells, tissues, organs and individuals, is a post-maturational process that, due to a diminished homeostasis and increased organism frailty, causes a reduction of the response to environmental stimuli and, in general, is associated to an increased predisposition to illness and death. However, the high incidence of death due to infectious, cardiovascular and cancer diseases underlies a common feature in these pathologies that is represented by dysregulation of both instructive and innate immunity. Several studies show that a low-grade s…

SenescenceAgingPathologymedicine.medical_specialtyAgeing age-related diseases diet drug inflammation longevity.LongevityPopulationInflammationDiseaseBioinformaticsSystemic inflammationDrug Delivery SystemsDegenerative diseaseDrug DiscoveryAnimalsHumansMedicineeducationInflammationPharmacologyeducation.field_of_studyInnate immune systemDose-Response Relationship Drugbusiness.industrymedicine.diseasePharmaceutical PreparationsAgeingInflammation Mediatorsmedicine.symptombusiness
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Hsp60 and human aging: Les liaisons dangereuses 

2013

Stressors can cause abnormal intracellular accumulation of Hsp60 and its localization in extramitochondrial sites, secretion, and circulation, with immune system activation. Dysfunction of chaperones associated with their quantitative and qualitative decline with aging (chaperonopathies of aging) characterizes senescence and is a potential causal factor in the physiological deterioration that occurs with it. The role of Hsp60 in aging is not easy to elucidate, because aging is accompanied by pathologies (e.g., cardiovascular and neurodegenerative disorders, osteoporosis, diabetes, cancer, etc.) in which Hsp60 has been implicated but, although those disorders are more frequent in the elderly…

SenescenceAginganimal structuresOsteoporosischemical and pharmacologic phenomenaInflammationDiseaseBiologycomplex mixturesMitochondrial ProteinsPathogenesisImmune systemDiabetes mellitusmedicineHumansCellular SenescenceAutoantibodiesHeart FailurefungiHSP 60 AGING CHAPERONES.Neurodegenerative DiseasesChaperonin 60Atherosclerosismedicine.diseaseMitochondriaImmune SystemImmunologyHSP60Arthropathy Neurogenicmedicine.symptomFrontiers in Bioscience
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Effects of Aging and Diet on Cardioprotection and Cardiometabolic Risk Markers.

2019

The prevalence of several diseases increases by age, including cardiovascular diseases, which are the leading cause of morbidity and mortality worldwide. Aging, as a complex process characterized by senescence, triggers various pathways, such as oxidative stress, systemic inflammation, metabolism dysfunction, telomere shortening, mitochondrial dysfunction and deregulated autophagy. A better understanding of the mechanisms underlying senescence may lead to the development of new therapeutic targets and strategies for age-related pathologies and extend the healthy lifespan. Modulating lifestyle risk factors and adopting healthy dietary patterns remain significant tools in delaying the aging …

SenescenceAgingmedia_common.quotation_subjectCardioprotectionDisease030204 cardiovascular system & hematologyDiabeteBioinformaticsSystemic inflammation03 medical and health sciences0302 clinical medicineRisk FactorsCardiovascular DiseaseDiabetes mellitusDrug DiscoveryMedicineHumansLife Style030304 developmental biologymedia_commonPharmacologyCardioprotection0303 health sciencesbusiness.industryRisk FactorLongevityCardiovascular riskmedicine.diseaseMetabolic syndromeDietCardiovascular DiseasesLife expectancyQuality of LifeNutraceuticalMetabolic syndromemedicine.symptombusinessHumanCurrent pharmaceutical design
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Inflammatory networks in ageing, age-related diseases and longevity

2006

Inflammation is considered a response set by the tissues in response to injury elicited by trauma or infection. It is a complex network of molecular and cellular interactions that facilitates a return to physiological homeostasis and tissue repair. The individual response against infection and trauma is also determined by gene variability. Ageing is accompanied by chronic low-grade inflammation state clearly showed by 2-4-fold increase in serum levels of inflammatory mediators. A wide range of factors has been claimed to contribute to this state; however, the most important role seems to be played by the chronic antigenic stress, which affects immune system thorough out life with a progress…

SenescenceAgingmedia_common.quotation_subjectLongevityInflammationDiseaseBiologyImmune systemGeneticmedicineAnimalsHumansSettore MED/05 - Patologia ClinicaCentenarianmedia_commonInflammationSettore MED/04 - Patologia GeneraleLongevityAgeingPharmacogenomicsAtherosclerosiImmunologyCentenarianmedicine.symptomPharmacogenomicsDevelopmental Biology
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Inflammation, ageing and cancer

2008

Cancer is generally recognized as an age-related disease. In fact, incidence and mortality rates of most human cancers increase consistently with age up to 90 years, but they plateau and decline thereafter. A low-grade systemic inflammation characterizes ageing and this pro-inflammatory status underlies biological mechanisms responsible for age-related inflammatory diseases. On the other hand, clinical and epidemiological studies show a strong association between chronic infection, inflammation and cancer and indicate that even in tumours not directly linked to pathogens, the microenvironment is characterized by the presence of a smouldering inflammation, fuelled primarily by stromal leukoc…

SenescenceAgingmedia_common.quotation_subjectLongevityInflammationDiseaseBiologySystemic inflammationGeneticNeoplasmsmedicineHumansSettore MED/05 - Patologia ClinicaAgedCancermedia_commonAged 80 and overInflammationSettore MED/04 - Patologia GeneraleIncidenceLongevityCancermedicine.diseaseAgeingChronic infectionAgeingImmunologymedicine.symptomDevelopmental BiologyMechanisms of Ageing and Development
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SGLT2i and GLP-1RA in Cardiometabolic and Renal Diseases: From Glycemic Control to Adipose Tissue Inflammation and Senescence

2021

Background. Over the last few years, the use of sodium-glucose cotransporter 2 inhibitors (SGLT2i) and glucagon-like peptide 1 receptor agonists (GLP-1RA) has increased substantially in medical practice due to their documented benefits in cardiorenal and metabolic health. In this sense, and in addition to being used for glycemic control in diabetic patients, these drugs also have other favorable effects such as weight loss and lowering blood pressure, and more recently, they have been shown to have cardio and renoprotective effects with anti-inflammatory properties. Concerning the latter, the individual or associated use of these antihyperglycemic agents has been linked with a decrease in p…

SenescenceEndocrinology Diabetes and MetabolismAdipose tissueInflammationGlycemic ControlReview ArticleDiseaseBioinformaticsDiseases of the endocrine glands. Clinical endocrinologyGlucagon-Like Peptide-1 ReceptorProinflammatory cytokineEndocrinologySodium-glucose cotransporter 2 inhibitors (SGLT2i)Weight lossDiabetes mellitusmedicineHumansSodium-Glucose Transporter 2 InhibitorsCellular SenescenceGlycemicInflammationMetabolic SyndromeManagement of diabetesRonyonsDiabetisbusiness.industryRC648-665medicine.diseaseCor MalaltiesAdipose TissueantihyperglycemicPatients obesity and cardiorenal compromiseglycemic controlmedicine.symptombusinessGlucagon-like peptide 1 receptor agonists (GLP-1RA)
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Small extracellular vesicles from young mice prevent frailty, improve healthspan and decrease epigenetic age in old mice

2021

SummaryAging is associated with an increased risk of frailty, disability, comorbidities, institutionalization, falls, fractures, hospitalization, and mortality. Searching for strategies to delay the degenerative changes associated with aging and frailty is interesting. We treated old animals intravenously with small extracellular vesicles (sEVs) derived from adipose mesenchymal stem cells (ADSCs) of young animals, and we found an improvement of several functional parameters usually altered with aging, such as motor coordination, grip strength, fatigue resistance, fur regeneration, and renal function. Frailty index analysis showed that 40% of old control mice were frail, whereas none of the …

Senescencemedicine.medical_specialtyKidneybusiness.industryMesenchymal stem cellAdipose tissueInflammationmedicine.disease_causeGrip strengthEndocrinologymedicine.anatomical_structureInternal medicineMetabolomeMedicinemedicine.symptombusinessOxidative stress
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Imbalance between sympathetic and sensory innervation in peritoneal endometriosis

2011

To investigate possible mechanisms of pain pathophysiology in patients with peritoneal endometriosis, a clinical study on sensory and sympathetic nerve fibre sprouting in endometriosis was performed. Peritoneal lesions (n= 40) and healthy peritoneum (n= 12) were immunostained and analysed with anti-protein gene product 9.5 (PGP 9.5), anti-substance P (SP) and anti-tyrosine hydroxylase (TH), specific markers for intact nerve fibres, sensory nerve fibres and sympathetic nerve fibres, respectively, to identify the ratio of sympathetic and sensory nerve fibres. In addition, immune cell infiltrates in peritoneal endometriotic lesions were analysed and the nerve growth factor (NGF) and interleuki…

Sensory and sympathetic innervationPathologySympathetic nervous systemSympathetic Nervous SystemInterleukin-1betaEndometriosisSubstance PSubstance PSensory Receptor CellBehavioral Neurosciencechemistry.chemical_compoundNerve FibersGAP-43 ProteinGanglia SpinalNerve Growth FactorAscitic FluidMedicineEndometriosis; Neurotransmitters; Sensory and sympathetic innervationEndometriosiNGFImmunity CellularGanglia SympatheticNeurotransmittersMiddle AgedImmunohistochemistrymedicine.anatomical_structureNerve FiberIL-1βPain generation of endometriosis-related symptomFemaleUbiquitin ThiolesteraseHumanSensory nerveAdultmedicine.medical_specialtySensory Receptor CellsTyrosine 3-MonooxygenaseNeuriteImmunologyEndometriosisYoung AdultImmune cell infiltratePeritoneumInternal medicineHumansNerve Growth FactorsNeurotransmitterCell ProliferationInflammationEndocrine and Autonomic Systemsbusiness.industryStromal Cellmedicine.diseaseSensory Receptor CellsNerve growth factorEndocrinologychemistryLaparoscopyStromal CellsbusinessChronic pelvic painBrain, Behavior, and Immunity
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