Search results for "Inflammation."

showing 10 items of 2627 documents

Role of Redox Signaling and Inflammation in Skeletal Muscle Adaptations to Training

2016

The inflammatory response to exercise-induced muscle damage has been extensively described. Exercise has important modulatory effects on immune function. These effects are mediated by diverse factors including pro-inflammatory cytokines, classical stress hormones, and hemodynamic effects leading to cell redistribution. As has been reported regarding oxidative stress, inflammation can have both detrimental and beneficial effects in skeletal muscle. In this review we will address the role of inflammation on protein metabolism in skeletal muscle. Specifically, we will review studies showing that treatment with cyclooxygenase-inhibiting drugs modulate the protein synthesis response to one bout …

0301 basic medicinemedicine.medical_specialtyprotein synthesisPhysiologyClinical BiochemistryInflammationReviewBiologyBioinformaticsmedicine.disease_causeBiochemistryProinflammatory cytokineMuscle hypertrophyprostaglandins03 medical and health sciences0302 clinical medicineImmune systemInternal medicinemedicineoxidative stressMolecular Biologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950Skeletal muscle030229 sport sciencesCell Biology030104 developmental biologyEndocrinologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistryanti-inflammatoriesmedicine.symptomhypertrophyOxidative stressHormoneAntioxidants
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New insights about the putative role of myokines in the context of cardiac rehabilitation and secondary cardiovascular prevention.

2017

Exercise training prevents the onset and the development of many chronic diseases, acting as an effective tool both for primary and for secondary prevention. Various mechanisms that may be the effectors of these beneficial effects have been proposed during the past decades: some of these are well recognized, others less. Muscular myokines, released during and after muscular contraction, have been proposed as key mediators of the systemic effects of the exercise. Nevertheless the availability of an impressive amount of evidence regarding the systemic effects of muscle-derived factors, few studies have examined key issues: (I) if skeletal muscle cells themselves are the main source of cytokin…

0301 basic medicinemedicine.medical_specialtyregular exercisemedicine.medical_treatmentInflammationBioinformaticsCachexiaCoronary artery disease03 medical and health sciencesMyokineMyokineReview Article on Myokines Exercise and Cardiovascular DiseasemedicineRehabilitationMyokines; cardiac rehabilitation; cardiovascular disease (CVD); cerebrovascular disease; regular exercisecardiovascular disease (CVD)business.industrySkeletal muscleGeneral Medicinemedicine.diseasecerebrovascular diseasecardiac rehabilitation030104 developmental biologyCytokinemedicine.anatomical_structureSarcopeniaPhysical therapymedicine.symptombusiness
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Blockade of Pannexin-1 Channels and Purinergic P2X7 Receptors Shows Protective Effects Against Cytokines-Induced Colitis of Human Colonic Mucosa

2018

Introduction: The pannexin-1 (Panx1) channels are found in many cell types, and ATP released from these channels can act on nearby cells activating purinergic P2X7 receptors (P2X7R) which lead to inflammation. Although Panx1 and P2X7R are implicated in the process of inflammation and cell death, few studies have looked at the role they play in inflammatory bowel disease in human. Hence, the aim of the present study was to investigate the function of Panx1 and P2X7R in an ex vivo colitis model developed from human colonic mucosal explants.Materials and Methods: Healthy human colonic mucosal strips (4 × 10 mm) were incubated in carbogenated culture medium at 37°C for 16 h. Proinflammatory cyt…

0301 basic medicinemedicine.medical_specialtytissue explantsCryptInflammationInflammatory bowel diseasecolonic inflammationProinflammatory cytokine03 medical and health sciencesInternal medicinemedicinePharmacology (medical)ColitisOriginal ResearchPharmacologyTight junctionChemistrylcsh:RM1-950Purinergic receptorpannexin-1medicine.diseaselcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologyP2X7 receptorhuman colitisTumor necrosis factor alphamedicine.symptomFrontiers in Pharmacology
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Roles of NGAL and MMP-9 in the tumor microenvironment and sensitivity to targeted therapy.

2016

Various, diverse molecules contribute to the tumor microenvironment and influence invasion and metastasis. In this review, the roles of neutrophil gelatinase-associated lipocalin (NGAL) and matrix metalloproteinase-9 (MMP-9) in the tumor microenvironment and sensitivity to therapy will be discussed. The lipocalin family of proteins has many important functions. For example when NGAL forms a complex with MMP-9 it increases its stability which is important in cancer metastasis. Small hydrophobic molecules are bound by NGAL which can alter their entry into and efflux from cells. Iron transport and storage are also influenced by NGAL activity. Regulation of iron levels is important for survival…

0301 basic medicinemedicine.medical_treatmentDrug resistance; Iron transport; Lcn2; Lipocalins; MMP-9; NGAL; SiderocalinsAcute-Phase ProteinLipocalinLipocalinMetastasisTargeted therapyAntineoplastic Agent0302 clinical medicineNeoplasmsTumor MicroenvironmentNeoplasm MetastasisNGALProto-Oncogene ProteinMedicine (all)SiderocalinsLipocalinsNeoplasm MetastasiMatrix Metalloproteinase 9030220 oncology & carcinogenesismedicine.symptomSignal transductionMMP-9HumanProtein BindingSignal TransductionSiderocalinAntineoplastic AgentsInflammationBiologyModels Biological03 medical and health sciencesLcn2Lipocalin-2Proto-Oncogene ProteinsmedicineHumansIron transportMolecular BiologyTumor microenvironmentInnate immune systemCell Biologymedicine.disease030104 developmental biologyDrug resistanceCancer cellImmunologyCancer researchNeoplasmAcute-Phase Proteins
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Immunomodulatory Therapy of Inflammatory Liver Disease Using Selectin-Binding Glycopolymers

2017

Immunotherapies have the potential to significantly advance treatment of inflammatory disease and cancer, which are in large part driven by immune cells. Selectins control the first step in immune cell adhesion and extravasation, thereby guiding leukocyte trafficking to tissue lesions. We analyzed four different highly specific selectin-binding glycopolymers, based on linear poly(2-hydroxypropyl)-methacrylamide (PHPMA) polymers. These glycopolymers contain either the tetrasaccharide sialyl-LewisX (SLeX) or the individual carbohydrates fucose, galactose, and sialic acids mimicking the complex SLeX binding motive. The glycopolymers strongly bind to primary human macrophages, without activatin…

0301 basic medicinemedicine.medical_treatmentGeneral Physics and Astronomy02 engineering and technologyFucoseImmunomodulationMice03 medical and health scienceschemistry.chemical_compoundImmune systemPolysaccharidesmedicineAnimalsHumansGeneral Materials ScienceCell adhesionCells CulturedInflammationBinding SitesbiologyChemistryLiver DiseasesGeneral EngineeringImmunotherapy021001 nanoscience & nanotechnologyDynamic Light ScatteringExtravasationIn vitro3. Good healthMice Inbred C57BLDisease Models Animal030104 developmental biologyBiochemistryConcanavalin ASelectinsbiology.proteinCancer researchCytokines0210 nano-technologySelectinACS Nano
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Immunosuppression, peripheral inflammation and invasive infection from endogenous gut microbiota activate retinal microglia in mouse models

2016

Although its actual role in the progression of degenerative processes is not fully known, the persistent activated state of retinal microglia and the concurrent secretion of inflammatory mediators may contribute to neuronal death and permanent vision loss. Our objective was to determine whether non-ocular conditions (immunosuppression and peripheral inflammation) could lead to activation of retinal microglia. Mouse models of immunosuppression induced by cyclophosphamide and/or peripheral inflammation by chemically induced sublethal colitis in C57BL/6J mice were used. Retinal microglia morphology, spatial distribution and complexity, as well as MHCII and CD11b expression levels were determin…

0301 basic medicinemedicine.medical_treatmentImmunologyInflammationMicrobiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationVirologymedicineColitisMicrogliabiologyImmunosuppressionRetinalmedicine.diseaseTLR2030104 developmental biologymedicine.anatomical_structureIntegrin alpha MchemistryImmunologybiology.proteinmedicine.symptom030217 neurology & neurosurgeryMicrobiology and Immunology
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Famotidine inhibits toll-like receptor 3-mediated inflammatory signaling in SARS-CoV-2 infection

2021

Apart from prevention using vaccinations, the management options for COVID-19 remain limited. In retrospective cohort studies, use of famotidine, a specific oral H2 receptor antagonist (antihistamine), has been associated with reduced risk of intubation and death in patients hospitalized with COVID-19. In a case series, nonhospitalized patients with COVID-19 experienced rapid symptom resolution after taking famotidine, but the molecular basis of these observations remains elusive. Here we show using biochemical, cellular, and functional assays that famotidine has no effect on viral replication or viral protease activity. However, famotidine can affect histamine-induced signaling processes i…

0301 basic medicinemedicine.medical_treatmentPharmacologyVirus ReplicationBiochemistrychemistry.chemical_compoundChemokine CCL2Coronavirus 3C ProteasesResearch ArticlesToll-like receptorbiologyNF-kappa BFamotidineMolecular Docking SimulationCytokine release syndromeCytokinemedicine.symptomSignal transductionHistaminemedicine.drugProtein BindingSignal TransductionHistamine AntagonistsInflammation03 medical and health sciencesToll-like receptormedicineHumansInterleukin 6Molecular BiologyBinding Sites030102 biochemistry & molecular biologybusiness.industryInterleukin-6SARS-CoV-2Cell Biologymedicine.diseasehistamineToll-Like Receptor 3Famotidine030104 developmental biologychemistryA549 CellsSARS-CoV2biology.proteinanti-viral signalingInterferon Regulatory Factor-3Caco-2 CellsbusinessHeLa Cells
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Specific inflammatory response of Anemonia sulcata (Cnidaria) after bacterial injection causes tissue reaction and enzymatic activity alteration

2015

The evolution of multicellular organisms was marked by adaptations to protect against pathogens. The mechanisms for discriminating the ’’self’’ from ’’non-self” have evolved into a long history of cellular and molecular strategies, from damage repair to the co-evolution of host-pathogen interactions. We investigated the inflammatory response in Anemonia sulcata (Cnidaria: Anthozoa) following injection of substances that varied in type and dimension, and observed clear, strong and specific reactions, especially after injection of Escherichia coli and Vibrio alginolyticus. Moreover, we analyzed enzymatic activity of protease, phosphatase and esterase, showing how the injection of different ba…

0301 basic medicinemedicine.medical_treatmentPhosphatasemedicine.disease_causeEsteraseMicrobiology03 medical and health sciences0302 clinical medicineEscherichia colimedicineAnimals030212 general & internal medicineEscherichia coliInflammation Anemonia sulcata Cnidaria Bacterial injection Esterases PhosphatasesVibrio alginolyticusEcology Evolution Behavior and SystematicsInflammationchemistry.chemical_classificationVibrio alginolyticusProteasebiologyFibrinolysisEsterasesFibrinogenAlkaline Phosphatasebiology.organism_classificationPhosphoric Monoester HydrolasesSea Anemones030104 developmental biologyEnzymechemistryHost-Pathogen InteractionsGelatinAlkaline phosphataseElectrophoresis Polyacrylamide GelBacteriaDensitometryPeptide HydrolasesJournal of Invertebrate Pathology
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Molecular Pathways Mediating Immunosuppression in Response to Prolonged Intensive Physical Training, Low-Energy Availability, and Intensive Weight Lo…

2019

Exercise and exercise-induced weight loss have a beneficial effect on overall health, including positive effects on molecular pathways associated with immune function, especially in overweight individuals. The main aim of our study was to assess how energy deprivation (i.e., "semi-starvation") leading to substantial fat mass loss affects the immune system and immunosuppression in previously normal weight individuals. Thus, to address this hypothesis, we applied a high-throughput systems biology approach to better characterize potential key pathways associated with immune system modulation during intensive weight loss and subsequent weight regain. We examined 42 healthy female physique athle…

0301 basic medicinemedicine.medical_treatmentPhysiologyliikuntaOverweightSystemic inflammationLeukocyte Countphysical training0302 clinical medicineWeight lossLeukocytesImmunology and AllergyMedicineOXIDATIVE STRESSta315DIETARY RESTRICTIONSport and Fitness SciencesOriginal Research2. Zero hungerimmunosuppressionIdrottsvetenskapbioinformatiikkaImmunosuppressionbioinformaticslow energy availability3. Good healthimmuunivasteIMMUNE FUNCTIONOBESITYChemokine secretionFemalemedicine.symptomfyysinen aktiivisuusAdultlcsh:Immunologic diseases. AllergyImmunologyEXERCISEInflammationYoung Adult03 medical and health sciencesLEPTINImmune systemINFLAMMATIONImmune ToleranceHumansimmunosuppression ; low energy availability ; physical training ; bioinformatics ; weight lossCell Proliferationbusiness.industrylaihdutusCYTOKINESmedicine.diseaseObesityDietenergiansaanti030104 developmental biologyHEMATOPOIETIC STEMImmunoglobulin G3121 General medicine internal medicine and other clinical medicineCELLS3111 Biomedicineweight lossEnergy IntakeTranscriptomelcsh:RC581-607business030215 immunologyFrontiers in Immunology
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Clinical Features at Onset and Genetic Characterization of Pediatric and Adult Patients with TNF-α Receptor—Associated Periodic Syndrome (TRAPS): A S…

2020

This study explores demographic, clinical, and therapeutic features of tumor necrosis factor receptor-associated periodic syndrome (TRAPS) in a cohort of 80 patients recruited from 19 Italian referral Centers. Patients’ data were collected retrospectively and then analyzed according to age groups (disease onset before or after 16 years) and genotype (high penetrance (HP) and low penetrance (LP) TNFRSF1A gene variants). Pediatric- and adult-onset were reported, respectively, in 44 and 36 patients; HP and LP variants were found, respectively, in 32 and 44 cases. A positive family history for recurrent fever was reported more frequently in the pediatric group than in the adult group (p<0.05…

0301 basic medicinemyalgiaMaleAbdominal painSettore MED/16 - REUMATOLOGIATNFRSF1AGene mutationGastroenterology0302 clinical medicinePathologyMedicineRB1-214PericarditisChildPrognosisPenetranceInflamacióFamilial Mediterranean FeverAIDA networkEstudi de casosReceptors Tumor Necrosis Factor Type IChild PreschoolAutoinflammationFemalemedicine.symptomResearch ArticleAdultmedicine.medical_specialtyArticle SubjectAdolescentGenotypetumor necrosis factorImmunologyContext (language use)Asymptomatic03 medical and health sciencesYoung AdultInternal medicineAnimalsHumansRetrospective Studies030203 arthritis & rheumatologyInflammationbusiness.industrytumor necrosis factor TRAPS AIDA networkTumor Necrosis Factor-alphaInfantTRAPSCell BiologyMyalgiaBiological productmedicine.disease030104 developmental biologyMutationCase studiesbusinessKidney diseaseMediators of Inflammation
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