Search results for "Inflammatory cytokine"

showing 10 items of 464 documents

Release of Inflammatory Mediators (PGE2, IL-6) by Fenofibric Acid-Photosensitized Human Keratinocytes and Fibroblasts

1998

Ultraviolet-A radiation has weak effects on the release of inflammatory mediators by skin cells due to the poor overlap between UVA wavelengths and the absorption spectra of the relevant chromophores of key biomole-cules. However, this situation could be very different in the presence of a photosensitizing drug. To investigate this issue, we have irradiated human skin cells (keratinocytes and fibroblasts) in the presence of fenofibric acid (the active phototoxic metabolite of fenofibrate). The results of this research show a dual effect on the production/release of inflammatory mediators: the synthesis of the proinflammatory cytokine interleukin-6 becomes strongly inhibited at photosensitiz…

biologyMetaboliteHuman skinStimulationGeneral MedicineBiochemistryMolecular biologyProinflammatory cytokinechemistry.chemical_compoundBiochemistrychemistryIn vivobiology.proteinPhotosensitizerPhysical and Theoretical ChemistryInterleukin 6PhototoxicityPhotochemistry and Photobiology
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Pathogenesis of Atherosclerosis: The Alternative Hypothesis

1998

The concept that oxidation is the major single event underlying the transformation of LDL to a proinflammatory molecule dominates the world literature. An alternative hypothesis on the pathogenesis of atherosclerosis will be presented here. We have found that nonoxidative, enzymatic modification of LDL with ubiquitous enzymes also transforms the molecule to an atherogenic moiety. Enzymatically altered LDL (E-LDL) shares major properties in common with lipoproteins that have been isolated from atherosclerotic lesions. It activates complement and is recognized by a scavenger receptor on human macrophages, thus inducing foam cell formation. Uptake of E-LDL is accompanied by induction of MCP−1 …

biologybusiness.industryComplement systemCell biologyProinflammatory cytokinePathogenesisImmunologybiology.proteinExtracellularMedicinelipids (amino acids peptides and proteins)Radiology Nuclear Medicine and imagingSecretionScavenger receptorAntibodyCardiology and Cardiovascular MedicinebusinessFoam cellJournal of Interventional Cardiology
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Is it the time of seno-therapeutics application in cardiovascular pathological conditions related to ageing?

2021

It rates that in 2030, the cardiovascular diseases (CVD) will result in 40% of all deaths and rank as the leading cause. Thus, the research of appropriate therapies able to delay or retard their onset and progression is growing. Of particular interest is a new branch of the medical science, called anti-ageing medicine since CVD are the result of cardiovascular ageing. Senescent cells (SC) accumulate in cardiovascular system contributing to the onset of typical age-related cardiovascular conditions (i.e., atherosclerosis, medial aorta degeneration, vascular remodeling, stiffness). Such conditions progress in cardiovascular pathologies (i.e., heart failure, coronary artery disease, myocardial…

business.industryAnti-ageing medicineSenolyticsCVD Anti-ageing medicine Senotherapeutics Senolytics SenomorphicsReview ArticleDegeneration (medical)RM1-950medicine.diseaseBioinformaticsCVDPhenotypeProinflammatory cytokineCoronary artery diseaseAgeingSenotherapeuticsHeart failureGeneral Earth and Planetary SciencesMedicineSettore MED/05 - Patologia ClinicaMyocardial infarctionSenomorphicsTherapeutics. PharmacologybusinessPathologicalGeneral Environmental Science
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P68 A diet rich in wheat alpha-amylase/trypsin inhibitors (ATIs) enhances disease progression in the MRL-Fas(lpr) mouse model of systemic lupus eryth…

2020

Background Wheat alpha-amylase/trypsin inhibitors (ATIs) are the second most prevalent proteins in wheat (3–4% vs 80–90% for gluten) and potent activators of the innate immune system via the toll like receptor 4 (TLR4)-MD2-CD14 complex in cells of the mononuclear phagocyte system (Junker Y et al, J Exp Med 2012), triggering several autoimmune/inflammatory diseases. In contrast, pure gluten that is de-enriched of ATIs shows no stimulatory activity. MRL-Fas(lpr) mice develop progressive and spontaneous glomerular, tubulointerstitial and perivascular kidney disease, arthritis, lymphadenopathy, splenomegaly and circulating autoantibodies in a syndrome that resembles systemic lupus erythematosus…

business.industryAutoantibodyArthritisSpleenInflammationMononuclear phagocyte systemurologic and male genital diseasesmedicine.diseaseProinflammatory cytokinemedicine.anatomical_structureimmune system diseasesImmunologymedicineTumor necrosis factor alphaHemoglobinuriamedicine.symptomskin and connective tissue diseasesbusinessPoster presentations
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Importance of HLA-DR+ and CD1a+ Epidermal Cells for Cytokine Production in Psoriasis

1995

Psoriasis is an inflammatory skin disease characterised by marked hyperproliferation of keratinocytes in association with vascular expansion, leukocyte infiltration and lymphocyte activation1. Cytokines are thought to play a pivotal role in the pathogenesis. It is speculated that a dysregulation of the cytokine network involving tumor-necrosis-factor-a (TNF-α), interleukin-6 (IL-6) and interleukin-8 (IL-8) might be the basic mechanism of the psoriatic inflammatory response2. TNF-α is the prototype of a proinflammatory peptide exerting immunomodulatory effects. IL-6 is a multifunctional cytokine with a broad range of biological functions in both acute and chronic inflammatory reactions. IL-8…

business.industrymedicine.medical_treatmentLymphocyteChemotaxismedicine.diseaseProinflammatory cytokinePathogenesisCytokinemedicine.anatomical_structurePsoriasisImmunologymedicineHLA-DRbusinessInfiltration (medical)
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B cells assume the command

2015

A proinflammatory B cell cytokine activates autoimmunity, and B cell depletion treats multiple sclerosis (Li et al., this issue).

business.industrymedicine.medical_treatmentMultiple sclerosisGeneral Medicinemedicine.diseasemedicine.disease_causeProinflammatory cytokineInflammatory mediatorAutoimmunitymedicine.anatomical_structureB cell depletionCytokineGranulocyte macrophage colony-stimulating factorImmunologymedicinebusinessB cellmedicine.drugScience Translational Medicine
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Cannabis Influences the Putative Cytokines-Related Pathway of Epilepsy among Egyptian Epileptic Patients

2019

The study aims to investigate: (1) the prevalence of cannabis among epileptic patients seen at Mansoura University Hospital, (2) serum levels and gene expression of cytokines in epilepsy patients and the controls. and (3) the possibility that cannabis use affects the cytokine levels in epilepsy patients, triggering its future use in treatment. We recruited 440 epilepsy patients and 200 controls matched for age, gender, and ethnicity. Of the epileptic patients, 37.5% demonstrated lifetime cannabis use with a mean duration of 15 &plusmn

cannabismedicine.medical_specialtypurified cannabidiol (cbd)inflammatory cytokinesmedicine.medical_treatmenttreatment-resistant epilepsyArticlelcsh:RC321-571Proinflammatory cytokine03 medical and health sciencesEpilepsy0302 clinical medicineInternal medicinemedicineanticonvulsant drugslcsh:Neurosciences. Biological psychiatry. Neuropsychiatryseizures030304 developmental biology0303 health sciencesbiologybusiness.industryGeneral NeuroscienceInterleukintetrahydrocannabinol (thc)Cannabis useUniversity hospitalbiology.organism_classificationmedicine.diseaseCytokineepilepsyTumor necrosis factor alphaCannabisbusiness030217 neurology & neurosurgeryBrain Sciences
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Effects of n-3 Polyunsaturated Fatty Acids (ω-3) Supplementation on Some Cardiovascular Risk Factors with a Ketogenic Mediterranean Diet

2015

Background: the ketogenic diet (KD) has become a widely used nutritional approach for weight loss. Some of the KD’s positive effects on metabolism and cardiovascular risk factors are similar to those seen after n-3 polyunsaturated fatty acids (ω-3) supplementation. We hypothesized that a ketogenic Mediterranean diet with phytoextracts combined with ω-3 supplementation may have increased positive effects on cardiovascular risk factors and inflammation. Methods: We analyzed 34 male overweight subjects

cardiovascular risk factorsBlood GlucoseMaleMediterranean dietinflammatory cytokinesmedicine.medical_treatmentN-3 polyunsaturated fatty acidPharmaceutical ScienceOverweightDiet Mediterraneanchemistry.chemical_compoundRisk FactorsWeight lossDrug DiscoveryInsulinlcsh:QH301-705.5Pharmacology Toxicology and Pharmaceutics (miscellaneous)Adipositychemistry.chemical_classificationCardiovascular risk factors; Inflammatory cytokines; Ketogenic diet; N-3 polyunsaturated fatty acids; Omega-3; Weight loss; Drug Discovery3003 Pharmaceutical ScienceMiddle Agedn-3 polyunsaturated fatty acidsketogenic dietCardiovascular DiseasesCytokinesomega-3medicine.symptomDiet KetogenicPolyunsaturated fatty acidAdultmedicine.medical_specialtyInflammatory cytokineBiologyCardiovascular risk factorWeight loArticleInternal medicineFatty Acids Omega-3Weight LossmedicineHumansTriglyceridesAgedAdiponectinPlant ExtractsCholesterolDrug Discovery3003 Pharmaceutical Science<i>n</i>-3 polyunsaturated fatty acidsCholesterol LDLOverweightEndocrinologylcsh:Biology (General)chemistryDietary SupplementsUric acidKetogenic dietMarine Drugs
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Absorption of kininogen from human plasma by Streptococcus pyogenes is followed by the release of bradykinin.

1997

H-kininogen (high-molecular-mass kininogen, HK) is the precursor of the vasoactive peptide hormone bradykinin (BK). Previous work has demonstrated that HK binds to Streptococcus pyogenesthrough M-proteins, fibrous surface proteins and important virulence factors of these bacteria. Here we find that M-protein-expressing bacteria absorb HK from human plasma. The HK bound to the bacteria was found to be cleaved, and analysis of the degradation pattern suggested that the cleavage of HK at the bacterial surface is associated with the release of BK. Moreover, addition of activated plasma prekallikrein to bacteria preincubated with human plasma, resulted in BK release. This mechanism, by which a p…

chemistry.chemical_classificationKininogenbiologyKininogensStreptococcus pyogenesBradykininVirulencePeptideCell BiologyPlasma protein bindingbiology.organism_classificationmedicine.disease_causeBradykininBiochemistryMicrobiologyProinflammatory cytokinechemistry.chemical_compoundchemistryStreptococcus pyogenesmedicineHumansMolecular BiologyBacteriaProtein BindingResearch Article
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Antioxidant and Antiinflammatory Properties of Heme Oxygenase-1 in Osteoarthritic Articular Cells

2012

Heme oxygenase-1 (HO-1) is induced in cells by various stimuli as a defense system against oxidative stress. It is known that reactive oxygen species (ROS) participates in the initiation and progression of osteoarthritis (OA) and several antioxidant systems may protect cartilage components. HO-1 induction or CO release from CORM-2 counteracts oxidative stress and protects against proinflammatory and catabolic effects of interleukin-1β in OA chondrocytes, osteoblasts, and synoviocytes as well as in OA osteochondral explants. Both approaches have been able to downregulate the production of mediators such as reactive oxygen species, nitric oxide, matrix metalloproteinases, prostaglandin E2, cy…

chemistry.chemical_classificationReactive oxygen speciesCartilagemedicine.disease_causeProinflammatory cytokineCell biologyHeme oxygenasechemistry.chemical_compoundmedicine.anatomical_structurechemistrymedicineProstaglandin E2HemeOxidative stressAggrecanmedicine.drug
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