Search results for "Inhibition"

showing 10 items of 590 documents

Job skill discretion and emotion control strategies as antecedents of recovery from work

2014

Recovery from work protects employees’ health and well-being, and therefore it is important to understand its antecedents. The aim of this study conducted among 183 middle-aged participants drawn from the Finnish Jyvaskyla Longitudinal Study of Personality and Social Development was to examine whether job skill discretion and emotion control strategies (emotional rumination and emotional inhibition) are related to psychological aspects of recovery from work (subjective recovery evaluation, psychological detachment and relaxation). The results of hierarchical general linear models confirmed the hypothesis that job skill discretion is positively associated with subjective recovery evaluation …

Organizational Behavior and Human Resource ManagementLongitudinal studyjob skill discretionRelaxation (psychology)media_common.quotation_subjectControl (management)Social changeDiscretionemotional inhibitionemotional ruminationrecoveryjob resourcesRuminationwork-related stressmedicinePersonalitymedicine.symptomPsychologyEmotional exhaustionSocial psychologyApplied Psychologyta515media_commonEuropean Journal of Work and Organizational Psychology
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Phospho-p38 MAPK expression in COPD patients and asthmatics and in challenged bronchial epithelium

2015

<b><i>Background:</i></b> The role of mitogen-activated protein kinases (MAPK) in regulating the inflammatory response in the airways of patients with chronic obstructive pulmonary disease (COPD) and asthmatic patients is unclear. <b><i>Objectives:</i></b> To investigate the expression of activated MAPK in lungs of COPD patients and in bronchial biopsies of asthmatic patients and to study MAPK expression in bronchial epithelial cells in response to oxidative and inflammatory stimuli. <b><i>Methods:</i></b> Immunohistochemical expression of phospho (p)-p38 MAPK, p-JNK1 and p-ERK1/2 was measured in bronchial mucosa in pat…

P38 MAPKMaleMAPK/ERK pathwayAsthma phenotypeSMOKERespiratory SystemMitogen-activated protein kinases; p65; Pathology of chronic obstructive pulmonary disease; Chronic obstructive pulmonary disease phenotypes; Asthma phenotypesPathology of chronic obstructive pulmonary diseasep38 Mitogen-Activated Protein KinasesChronic obstructive pulmonary disease phenotypePulmonary Disease Chronic ObstructiveOXIDATIVE STRESSMACROPHAGESRespiratory systemMitogen-activated protein kinasesChronic obstructive pulmonary disease phenotypesMitogen-activated protein kinases; p65; pathology of chronic obstructive pulmonary disease phenotypes; asthma phenotypesCOPDp65KinaseAsthma phenotypes; Chronic obstructive pulmonary disease phenotypes; Mitogen-activated protein kinases; p65; Pathology of chronic obstructive pulmonary disease; Pulmonary and Respiratory MedicineACTIVATED PROTEIN-KINASEInterleukinMiddle AgedImmunohistochemistrypathology of chronic obstructive pulmonary disease phenotypesAsthma phenotypesFemaleLife Sciences & BiomedicinePulmonary and Respiratory Medicinep38 mitogen-activated protein kinasesBlotting WesternINHIBITIONSocio-culturaleBronchiRespiratory MucosaOBSTRUCTIVE PULMONARY-DISEASE1102 Cardiovascular Medicine And HaematologyCell LinemedicineHumansLymphocyte CountInterleukin 8AgedAsthmaScience & Technologybusiness.industryInterleukin-8Transcription Factor RelAPATHWAYSMitogen-activated protein kinasemedicine.diseaseAsthmarespiratory tract diseasesSEVERITYCase-Control StudiesCELLSImmunologybusiness
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MCC1019, a selective inhibitor of the Polo-box domain of Polo-like kinase 1 as novel, potent anticancer candidate

2019

Polo-like kinase (PLK1) has been identified as a potential target for cancer treatment. Although a number of small molecules have been investigated as PLK1 inhibitors, many of which showed limited selectivity. PLK1 harbors a regulatory domain, the Polo box domain (PBD), which has a key regulatory function for kinase activity and substrate recognition. We report on 3-bromomethyl-benzofuran-2-carboxylic acid ethyl ester (designated: MCC1019) as selective PLK1 inhibitor targeting PLK1 PBD. Cytotoxicity and fluorescence polarization-based screening were applied to a library of 1162 drug-like compounds to identify potential inhibitors of PLK1 PBD. The activity of compound MC1019 against the PLK1…

PBD Polo box domainMTD maximal tolerance doseCDC25 cell division cycle 25HIF-1α hypoxia-inducible factor 1 αMST microscale thermophoresisIC50 50% inhibition concentrationMFP M phase promoting factorPARP-1 poly(ADP-ribose) polymerase-10302 clinical medicineFOXO forkhead box ONec-1 necrostatin 1CDC2 cell division cycle protein 2 homologGeneral Pharmacology Toxicology and PharmaceuticsMitotic catastropheCDK cyclin-dependent kinase0303 health sciencesChemistryPolo-like kinaseMono-targeted therapyCell cycleBUBR1 budding uninhibited by benzimidazole-related 1Polo box domain030220 oncology & carcinogenesisPLK1 Polo-like kinaseNecroptosisSpindle damagePLK1IHC immunohistochemistryOriginal articleNecroptosisCell cyclePLK1APC/C anaphase-promoting complex/cyclosomePLK3ABC avidin-biotin complexPI propidium iodide03 medical and health sciencesFBS fetal bovine serumPDB Protein Data BankKd the dissociation constantKinase activity030304 developmental biologyAkt/PKB signaling pathwayCell growthlcsh:RM1-950LC3 light chain 3lcsh:Therapeutics. PharmacologyCancer researchDAPKs death-associated protein kinase3-MA 3-methyladenineDAPI 4′6-diamidino-2-phenylindoleSAC spindle assembly checkpointActa Pharmaceutica Sinica B
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Endoplasmic Reticulum Stress Inhibition Protects against Excitotoxic Neuronal Injury in the Rat Brain

2007

Elevated brain glutamate with activation of neuronal glutamate receptors accompanies neurological disorders, such as epilepsy and brain trauma. However, the mechanisms by which excitotoxicity triggers neuronal injury are not fully understood. We have studied the glutamate receptor agonist kainic acid (KA) inducing seizures and excitotoxic cell death. KA caused the disintegration of the endoplasmic reticulum (ER) membrane in hippocampal neurons and ER stress with the activation of the ER proteins Bip, Chop, and caspase-12. Salubrinal, inhibiting eIF2α (eukaryotic translation initiation factor 2 subunit α) dephosphorylation, significantly reduced KA-induced ER stress and neuronal deathin vivo…

PERKMaleKainic acidProgrammed cell deathcaspase-12ExcitotoxicityBiologymedicine.disease_causeEndoplasmic ReticulumHippocampusCalcium in biologyeIF2 alphaSalubrinalchemistry.chemical_compoundsalubrinalmedicineExcitatory Amino Acid AgonistsAnimalsRats WistarNeuronsKainic AcidhippocampuGeneral NeuroscienceEndoplasmic reticulumGlutamate receptorBrainNeural InhibitionArticlesCell biologyRatsOxidative StresschemistryUnfolded protein responseNeuroscience
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Full inhibition of enzymatic browning in the presence of thiol-functionalised silica nanomaterial

2018

[EN] Darkening processed fruits and vegetables is caused mainly by enzymatic browning through polyphenol oxidase (PPO) action. Accordingly, we explored the potential of four silica-based materials (MCM-41 nanometric size, MCM-41 micrometric size, UVM-7 and aerosil), non-functionalised and functionalised with thiol groups, to inhibit PPO activity in the model system and apple juice. All materials showed relevant performance when immobilising and inhibiting PPO in model systems, and support topology is a main factor for enzyme immobilisation and inhibition. Thiol-containing silica UVM7-SH showed the greatest inactivation, and similar browning values to those obtained by acidification. The enz…

PPOTECNOLOGIA DE ALIMENTOSApple juiceTyrosinaseModel systemUVM-7Polyphenol oxidaseAnalytical ChemistryNanomaterials0404 agricultural biotechnologyQUIMICA ORGANICAThiolsBrowningOrganic chemistrySulfhydryl CompoundsFumed silicaInhibitionchemistry.chemical_classificationQUIMICA INORGANICA04 agricultural and veterinary sciencesGeneral MedicineSilicon Dioxide040401 food scienceNanostructuresEnzymechemistryFruitMalusThiolTyrosinaseCatechol OxidaseFood Science
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Influence of the functionalisation of mesoporous silica material UVM-7 on polyphenol oxidase enzyme capture and enzymatic browning

2020

Polyphenol oxidase (PPO), also known as tyrosinase and catechol oxidase, is the enzyme responsible for enzymatic browning in foods. It causes undesirable organoleptic, nutritional and colour changes. Here, we report the preparation of five nanomaterials and a study of their ability to modulate PPO enzyme activity. The materials consist of UVM-7 supports (a mesoporous silica material) modified with diverse functional groups (i.e. amine, carboxylic acid, isocyanate, alkane and pyridine). We also studied the PPO immobilisation capability of the materials. All the materials, except the carboxylic acid functionalised one, offer high PPO loading capabilities and the immobilisation speed increases…

PPOTECNOLOGIA DE ALIMENTOSPyridinesSurface PropertiesApple juiceCarboxylic acidTyrosinaseCarboxylic AcidsUVM-701 natural sciencesPolyphenol oxidaseAnalytical Chemistry0404 agricultural biotechnologyQUIMICA ORGANICABrowningOrganic chemistryAminesCatechol oxidaseEdetic AcidInhibitionchemistry.chemical_classificationbiology010401 analytical chemistryQUIMICA INORGANICA04 agricultural and veterinary sciencesGeneral MedicineMesoporous silicaEnzymes ImmobilizedSilicon Dioxide040401 food scienceEnzyme assay0104 chemical sciencesNanostructuresFruit and Vegetable JuicesOxygenchemistryMalusbiology.proteinAmine gas treatingTyrosinaseOxidation-ReductionCatechol OxidaseFood Science
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Development of natural and synthetic compounds as kinase inhibitors targeting cancer cells and cancer stem cells

2023

Pancreatic ductal adenocarcinomaInhibition of migrationantibiotic resistanceImidazo[21-b][134]thiadiazole derivativeskinase inhibitorsanti-biofilm agentsAntiproliferative activityFAK inhibitionSettore CHIM/08 - Chimica Farmaceutica
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Modelling the spatial and temporal constrains of the GABAergic influence on neuronal excitability

2021

GABA (γ-amino butyric acid) is an inhibitory neurotransmitter in the adult brain that can mediate depolarizing responses during development or after neuropathological insults. Under which conditions GABAergic membrane depolarizations are sufficient to impose excitatory effects is hard to predict, as shunting inhibition and GABAergic effects on spatiotemporal filtering of excitatory inputs must be considered. To evaluate at which reversal potential a net excitatory effect was imposed by GABA (EGABAThr), we performed a detailed in-silico study using simple neuronal topologies and distinct spatiotemporal relations between GABAergic and glutamatergic inputs. These simulations revealed for GABAe…

Patch-Clamp TechniquesAction potentialPhysiologyAction PotentialsSynaptic TransmissionNervous SystemBiochemistryMiceNerve FibersAnimal CellsMedicine and Health SciencesGABAergic NeuronsBiology (General)gamma-Aminobutyric AcidNeuronsMembrane potentialEcologyChemistryPyramidal CellsDepolarizationNeurochemistryNeurotransmittersCA3 Region HippocampalElectrophysiologyReceptors GlutamateComputational Theory and MathematicsModeling and SimulationExcitatory postsynaptic potentialGABAergicAnatomyCellular TypesShunting inhibitionResearch Articlemedicine.drugQH301-705.5Models NeurologicalNeurophysiologyAMPA receptorMembrane Potentialgamma-Aminobutyric acidCellular and Molecular NeuroscienceGlutamatergicSpatio-Temporal AnalysisGeneticsmedicineAnimalsComputer SimulationReceptors AMPAReversal potentialMolecular BiologyEcology Evolution Behavior and SystematicsComputational BiologyBiology and Life SciencesNeural InhibitionDendritesCell BiologyNeuronal DendritesAxonsMice Inbred C57BLAnimals Newbornnervous systemCellular NeuroscienceSynapsesDepolarizationNeuroscienceNeurosciencePLOS Computational Biology
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Effect of depolarizing GABAA-mediated membrane responses on excitability of Cajal-Retzius cells in the immature rat neocortex

2011

In immature neurons activation of ionotropic GABA receptors induces depolarizing membrane responses due to a high intracellular Cl− concentration ([Cl−]i). However, it is difficult to draw conclusions about the functional consequences of subthreshold GABAergic depolarizations, since GABAergic membrane shunting and additional effects on voltage-dependent ion channels or action potential threshold must be considered. To systematically investigate factors that determine the GABAergic effect on neuronal excitability we performed whole cell patch-clamp recordings from Cajal-Retzius cells in immature rat neocortex, using [Cl−]i between 10 and 50 mM. The effect of focal GABA application was quant…

Patch-Clamp TechniquesPhysiologyModels NeurologicalAction PotentialsDifferential ThresholdNeocortexMembrane PotentialsGABA AntagonistsChloridesInterneuronsmedicineAnimalsPatch clampGABAergic NeuronsRats WistarReceptorgamma-Aminobutyric AcidNeocortexGABAA receptorChemistryGeneral NeuroscienceReceptors GABA-ARatsPyridazinesRheobasemedicine.anatomical_structureAnimals NewbornIon Channel GatingNeuroscienceShunting inhibitionIntracellularIonotropic effectJournal of Neurophysiology
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Pursuit of the emerging dialogue between psychoanalysis and neuroscience: clinical and research perspectives.

2005

Patient Care TeamBrain MappingPsychoanalysisInterprofessional RelationsEmotionsStatistics as TopicNeurosciencesBrainNeural InhibitionMagnetic Resonance ImagingPsychoanalysisDreamsPsychiatry and Mental healthClinical PsychologyBorderline Personality DisorderPsychoanalytic TheoryHumansNerve NetPsychologyPsychomotor PerformanceDefense MechanismsForecastingThe International journal of psycho-analysis
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