Search results for "Innate"

showing 10 items of 638 documents

Eliminating Factor H-Binding Activity of Borrelia burgdorferi CspZ Combined with Virus-Like Particle Conjugation Enhances Its Efficacy as a Lyme Dise…

2018

The spirochete Borrelia burgdorferi is the causative agent of Lyme disease, the most common tick-borne disease in the U.S and Europe. No potent human vaccine is currently available. The innate immune complement system is vital to host defense against pathogens, as complement activation on the surface of spirochetes results in bacterial killing. Complement system is inhibited by the complement regulator factor H. To escape killing, B. burgdorferi produces an outer surface protein CspZ that binds factor H to inhibit complement activation on the cell surface. Immunization with CspZ alone does not protect mice from infection, which we speculate is because factor H-binding cloaks potentially pro…

Malelcsh:Immunologic diseases. Allergy0301 basic medicine030106 microbiologyImmunologySerum Bactericidal Antibody Assayvirus-like particlesEpitopeMicrobiologyMice03 medical and health sciencesAntigenvaccineBorreliaAnimalsLyme diseaseImmunology and AllergyVaccines Virus-Like Particleddc:610Borrelia burgdorferiOriginal ResearchInnate immune systembiologyBorreliaImmunogenicityImmunization PassiveLyme Disease Vaccinesfactor Hbiology.organism_classificationAntibodies Bacterial3. Good healthComplement systemCspZ030104 developmental biologyBorrelia burgdorferiComplement Factor Hbiology.proteinAntibodylcsh:RC581-607Bacterial Outer Membrane ProteinsFrontiers in Immunology
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Prognostic Value of Complement Properdin in Cancer

2021

© 2021 Mangogna, Varghese, Agostinis, Alrokayan, Khan, Stover, Belmonte, Martorana, Ricci, Bulla and Kishore. The complement system is readily triggered by the presence of damage-associated molecular patterns on the surface of tumour cells. The complement alternative pathway provides rapid amplification of the molecular stress signal, leading to complement cascade actvation to deal with pathogens or malignant cells. Properdin is the only known positive regulator of the alternative pathway. In addition, properdin promotes the phagocytic uptake of apoptotic T cells by macrophages and dendritic cells without activating the complement system, thus, establishing its ability to recognize “altered…

Malelcsh:Immunologic diseases. Allergybioinformatics analysisNeutrophilsComplement Pathway AlternativeImmunologyDatasets as TopicInflammationchemical and pharmacologic phenomenaBiologyLymphocytes Tumor-InfiltratingImmune systemNeoplasmsTumor Microenvironmentmedicinebioinformatics; cancer; complement; innate immunity; prognosis; properdinData MiningHumansImmunology and AllergyCytotoxic T cellcancercomplementRNA MessengerRNA Neoplasminnate immunityOriginal ResearchTumor microenvironmentInnate immune systembioinformaticMacrophagesDendritic CellsbioinformaticsLymphocyte SubsetsComplement systemproperdinAlternative complement pathwayCancer researchProperdinFemaleprognosismedicine.symptomTranscriptomelcsh:RC581-607prognostic markerprognosi
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A trade-off between sexual signalling and immune function in a natural population of the drumming wolf spider Hygrolycosa rubrofasciata.

2005

The field of ecological immunology is ultimately seeking to address the question ‘Why is there variation in immune function?’ Here, we provide experimental evidence that costs of ubiquitous sexual signals are a significant source of variation in immune function. In the mating season, males of the wolf spider Hygrolycosa rubrofasciata drum against dry leaves while wandering around the habitat searching for receptive females. According to a previous study, the male metabolic rate during the drumming increases 22-fold compared to the resting metabolic rate. In the present study, we examined whether investment in costly courtship drumming decreases male immune function in a wild population of H…

Malemedia_common.quotation_subjectPopulationWolf spiderZoologyCourtshipSexual Behavior AnimalHemolymphSeasonal breederAnimalsAnimal communicationeducationEcology Evolution Behavior and SystematicsFinlandmedia_commoneducation.field_of_studybiologyEcologySpidersbiology.organism_classificationImmunity InnateAnimal CommunicationNatural population growthMate choiceSexual selectionFemaleEnergy MetabolismAntimicrobial Cationic PeptidesJournal of evolutionary biology
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TLR7 and TLR8 ligands and antiphospholipid antibodies show synergistic effects on the induction of IL-1beta and caspase-1 in monocytes and dendritic …

2009

TLRs represent the first line of defense against invading pathogens in the innate immune system. Certain cytokines are important mediators and essentially necessary to assure an appropriately regulated immune response. Recent data gave initial evidence that IL-1beta is one of the most relevant members of these regulating cytokines. We investigated the induction of IL-1beta production in monocytes and pDCs stimulated with ligands for TLR7 and TLR8 and with antiphospholipid antibodies (aPL). Using human monocytes and pDCs for stimulation with specific TLR7 and TLR8 ligands such as resiquimod (R848) and single stranded RNA (RNA42) as well as with a human monoclonal aPL HL5B resulted in a speci…

Malemedicine.drug_classImmunologyInterleukin-1betaCaspase 1Enzyme-Linked Immunosorbent AssayCell SeparationBiologyRegulatory Sequences Nucleic AcidMonoclonal antibodyLigandsMonocytesProinflammatory cytokinechemistry.chemical_compoundImmune systemmedicineImmunology and AllergyHumansInnate immune systemCaspase 1ImidazolesHematologyTLR7Dendritic CellsTLR8Oligonucleotides AntisenseAntiphospholipid SyndromeFlow CytometrychemistryToll-Like Receptor 7Toll-Like Receptor 8Enzyme InductionImmunologyAntibodies AntiphospholipidRNAFemaleResiquimodImmunobiology
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Anti-aging activity of the Ink4/Arf locus.

2009

The proteins encoded by the Ink4/Arf locus, p16Ink4a, p19Arf and p15Ink4b are major tumour suppressors that oppose aberrant mitogenic signals. The expression levels of the locus are progressively increased during aging and genome-wide association studies have linked the locus to a number of aging-associated diseases and frailty in humans. However, direct measurement of the global impact of the Ink4/Arf locus on organismal aging and longevity was lacking. In this work, we have examined the fertility, cancer susceptibility, aging and longevity of mice genetically modified to carry one (Ink4/Arf-tg) or two (Ink4/Arf-tg/tg) intact additional copies of the locus. First, increased gene dosage of …

Malemedicine.medical_specialtyAgingDNA damageTransgenemedia_common.quotation_subjectLongevityGene DosageLocus (genetics)Mice TransgenicBiologyGene dosagelaw.inventionMicelawInternal medicineNeoplasmsmedicineAnimalsGenes Tumor SuppressorSpermatogenesisGeneCyclin-Dependent Kinase Inhibitor p16Infertility Malemedia_commonCell ProliferationCell growthLongevityCell BiologyImmunity InnateMice Inbred C57BLEndocrinologyCancer researchSuppressorTumor Suppressor Protein p53Aging cell
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TLR4 polymorphisms and ageing: implications for the pathophysiology of age-related diseases.

2009

Innate immunity provides a first line of host defense against infection by recognizing and killing microbes while simultaneously activating an instructive immune response. Toll-like receptors (TLRs) are principal mediators of rapid microbial recognition and function mainly by detection of pathogen-associated molecular patterns that do not exist in the host. Recognition of their ligands leads to a series of signaling events resulting in acute host responses, involved in killing pathogens. Discussion We describe the involvement of TLR4 polymorphisms in ageing, and in particular in age-related diseases, suggesting the crucial role of molecules of innate immunity in pathophysiology of these dis…

Malemedicine.medical_specialtyAgingImmunologyLongevitySNPBiologyPolymorphism Single NucleotideatherosclerosiImmune systemMedical microbiologyAlzheimer DiseasemedicinecancerImmunology and AllergyHumansGenetic Predisposition to DiseaseTLR4Receptorinnate immunityAllelesSettore MED/04 - Patologia GeneraleInnate immune systemHost (biology)Prostatic Neoplasmsmedicine.diseaseImmunity InnateToll-Like Receptor 4AgeingCardiovascular DiseasesImmunologyTLR4FemaleAlzheimer's diseaseAlzheimer’s diseaseFunction (biology)Journal of clinical immunology
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Langerin+ DCs regulate innate IL-17 production in the oral mucosa during Candida albicans-mediated infection

2018

The opportunistic fungal pathogen Candida albicans frequently causes diseases such as oropharyngeal candidiasis (OPC) in immunocompromised individuals. Although it is well appreciated that the cytokine IL-17 is crucial for protective immunity against OPC, the cellular source and the regulation of this cytokine during infection are still a matter of debate. Here, we directly visualized IL-17 production in the tongue of experimentally infected mice, thereby demonstrating that this key cytokine is expressed by three complementary subsets of CD90+ leukocytes: RAG-dependent αβ and γδ T cells, as well as RAG-independent ILCs. To determine the regulation of IL-17 production at the onset of OPC, we…

Malemedicine.medical_treatment2405 ParasitologyPathology and Laboratory Medicine10263 Institute of Experimental ImmunologyMonocytesMice0302 clinical medicineAnimal CellsCandida albicansBiology (General)Candida albicansMononuclear Phagocyte SystemFungal PathogensInnate Immune Systemeducation.field_of_studyEukaryotaMononuclear phagocyte systemFlow CytometryCorpus albicans3. Good healthSpectrophotometryMedical MicrobiologyCytokinesCytophotometryCellular Types10244 Institute of VirologyQH301-705.5Immune CellsImmunologyMicrobiology03 medical and health sciences1311 GeneticsGenetics1312 Molecular BiologyeducationMicrobial PathogensMolecular BiologyMouth2403 ImmunologyBlood CellsOrganismsBiology and Life SciencesDendritic CellsMolecular DevelopmentYeastMice Inbred C57BLMannose-Binding Lectins030104 developmental biologyImmunologyThy-1 Antigens570 Life sciences; biologyParasitologyImmunologic diseases. AllergyDigestive SystemDevelopmental Biology0301 basic medicineNeutrophilsPhysiologyInterleukin-1betaYeast and Fungal ModelsInterleukin-23White Blood CellsSpectrum Analysis TechniquesCandidiasis OralImmune PhysiologyLeukocytesMedicine and Health SciencesCandidaStainingbiologyInterleukin-172404 MicrobiologyCell StainingSpecific Pathogen-Free OrganismsInfectious DiseasesCytokineExperimental Organism SystemsAntigens SurfaceFemaleAnatomyPathogensResearch ArticleLangerinPopulationMycologyOpportunistic InfectionsResearch and Analysis MethodsTongueImmunityVirologymedicineAnimalsLectins C-TypeInterleukin 6Interleukin-6Mouth MucosaFungiCell BiologyRC581-607biology.organism_classificationSpecimen Preparation and TreatmentImmune Systembiology.protein2406 VirologySpleen030215 immunology
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Genome-Wide Expression Profiles in Very Low Birth Weight Infants With Neonatal Sepsis

2014

BACKGROUND: Bacterial sepsis is associated with high morbidity and mortality in preterm infants. However, diagnosis of sepsis and identification of the causative agent remains challenging. Our aim was to determine genome-wide expression profiles of very low birth weight (VLBW) infants with and without bacterial sepsis and assess differences. METHODS: This was a prospective observational double-cohort study conducted in VLBW (<1500 g) infants with culture-positive bacterial sepsis and non-septic matched controls. Blood samples were collected as soon as clinical signs of sepsis were identified and before antibiotics were initiated. Total RNA was processed for genome-wide expression an…

Malemedicine.medical_treatmentInfant Premature DiseasesCohort StudiesSepsisSepsisGene expressionHumansInfant Very Low Birth WeightMedicineProspective StudiesGeneGram-Positive Bacterial InfectionsPrincipal Component AnalysisNeonatal sepsisTumor Necrosis Factor-alphabusiness.industryInfant NewbornBacterial Infectionsmedicine.diseaseImmunity InnateReverse transcription polymerase chain reactionLow birth weightEarly DiagnosisCytokinePediatrics Perinatology and Child HealthImmunologyCytokinesFemaleTumor necrosis factor alphamedicine.symptomGram-Negative Bacterial InfectionsTranscriptomebusinessGenome-Wide Association StudySignal TransductionPediatrics
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DNA Methylation in Inflammatory Pathways Modifies the Association between BMI and Adult-Onset Non-Atopic Asthma

2019

A high body mass (BMI) index has repeatedly been associated with non-atopic asthma, but the biological mechanism linking obesity to asthma is still poorly understood. We aimed to test the hypothesis that inflammation and/or innate immunity plays a role in the obesity-asthma link. DNA methylome was measured in blood samples of 61 non-atopic participants with asthma and 146 non-atopic participants without asthma (non-smokers for at least 10 years) taking part in the Swiss Cohort Study on Air Pollution and Lung and Heart Diseases in Adults (SAPALDIA) study. Modification by DNA methylation of the association of BMI or BMI change over 10 years with adult-onset asthma was examined at each CpG sit…

MaleobesityNon-atopic asthmaHealth Toxicology and Mutagenesislcsh:MedicineToxicologyBody Mass IndexCohort StudiesMice0302 clinical medicineMedicineinnate immunitynon-atopic asthmaInnate immunity0303 health sciencesDNA methylationNF-kappa Bepigenome-wide association study3. Good healthCpG siteDNA methylationFemaleEpigeneticsmedicine.symptomGlucocorticoidmedicine.drugAdultMAP Kinase Signaling SystemInflammationArticle03 medical and health sciencesEpigenome-wide association studyMD MultidisciplinaryAnimalsHumansObesityEpigeneticsadult-onset asthmaPI3K/AKT/mTOR pathway030304 developmental biologyAsthmaInflammationepigeneticsbusiness.industrylcsh:RPublic Health Environmental and Occupational Healthmedicine.diseaseObesityAsthmarespiratory tract diseasesPPAR gamma030228 respiratory systeminflammationImmunologybusinessAdult-onset asthmaInternational Journal of Environmental Research and Public Health
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The immune mediators in echinoderms as souce of novel AMPs against microbial biofilms

2012

Marine invertebrate coelomocytes innate immunity staphylococcal biofilmsSettore BIO/19 - Microbiologia Generale
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