Search results for "Innate"

showing 10 items of 638 documents

Profiling the diversity of innate lymphoid cells

2015

Genome-wide transcriptional profiling of tissue-resident innate lymphoid cells (ILCs) has provided important insight not only into their developmental relationships and phenotypic plasticity but also into previously unknown functions.

Phenotypic plasticityImmunologyInnate lymphoid cellImmunology and AllergyProfiling (information science)BiologyCell biologyNature Immunology
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Thrombotic risk in paroxysmal nocturnal hemoglobinuria-like (PNH-like) phenotype

2020

The complement system is an essential component of the innate immune defence that, if overly activated, may damage organs and tissues. For this reason, there is a fine complement regulatory system. The complement modulation system includes two proteins with important regulatory activity, CD55 or decay accelerating factor (DAF) and CD59 or membrane inhibitor of reactive lysis (MIRL). The paroxysmal nocturnal hemoglobinuria (PNH) is a clonal and non-neoplastic disease characterized by intravascular haemolysis, occurrence of thrombosis and bone marrow failure. In clinical practice, in opposition to PNH, a variety of pathological conditions have been observed with an acquired and non-genetic de…

PhysiologyHemoglobinuria ParoxysmalDiseaseCD59030204 cardiovascular system & hematologyHemolysis030218 nuclear medicine & medical imagingSettore MED/15 - Malattie Del Sangue03 medical and health sciences0302 clinical medicinehemic and lymphatic diseasesPhysiology (medical)medicineHumansParoxysmal nocturnal hemoglobinuriaDecay-accelerating factorcomplement systemInnate immune systembusiness.industryThrombosisHematologythromboembolic riskmedicine.diseasePhenotypeComplement systemPhenotypemedicine.anatomical_structureImmunologyParoxysmal nocturnal hemoglobinuriaBone marrowCD55Cardiology and Cardiovascular MedicinebusinessCD59
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Localized versus systemic effect of arbuscular mycorrhizal fungi on defence responses to Phytophthora infection in tomato plants

2002

Development of biological control for plant diseases is accepted as a durable and environmentally friendly alternative for agrochemicals. Arbuscular mycorrhizal fungi (AMF), which form symbiotic associations with root systems of most agricultural, horticultural and hardwood crop species, have been suggested as widespread potential bioprotective agents. In the present study the ability of two AMF (Glomus mosseae and Glomus intraradices) to induce local or systemic resistance to Phytophthora parasitica in tomato roots have been compared using a split root experimental system. Glomus mosseae was effective in reducing disease symptoms produced by P. parasitica infection, and evidence points to …

Phytophthora0106 biological sciencesGlycoside HydrolasesPhysiologyPlant SciencePlant Roots01 natural sciences[SDV.BV.BOT] Life Sciences [q-bio]/Vegetal Biology/BotanicsSolanum lycopersicumSymbiosisBotanyMycorrhizaSymbiosisPhycomycetesComputingMilieux_MISCELLANEOUSGlomusPlant Diseases2. Zero hungerbiologySuperoxide Dismutasebeta-GlucosidaseChitinasesfungiFungifood and beveragesGlucan 13-beta-Glucosidase04 agricultural and veterinary sciences[SDV.BV.BOT]Life Sciences [q-bio]/Vegetal Biology/BotanicsPhytophthora nicotianaebiology.organism_classificationImmunity InnateIsoenzymesOxidative StressChitinase040103 agronomy & agriculturebiology.protein0401 agriculture forestry and fisheriesPhytophthoraSolanaceaeSignal TransductionCONTROLE DE MALADIES010606 plant biology & botanyJournal of Experimental Botany
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Model for Generation of Large Numbers of Primary, Inflammatory Skin-Derived Neutrophils, and Macrophages

2012

Tissue macrophages and inflammatory neutrophils represent important cells of the innate immune system responsible for various important tasks, i.e., elimination of pathogens and/or granuloma formation. Isolation of large numbers of primary phagocytes is vital for research with these cells. Within this protocol, we present a strategy for isolation of large numbers of inflammatory neutrophils and macrophages from murine skin that allows for follow-up in vivo or in vitro studies.

Primary (chemistry)Granuloma formationInnate immune systemIn vivoImmunologyMurine skinBiologyIsolation (microbiology)In vitro
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Cellular Models and Assays to Study NLRP3 Inflammasome Biology

2020

The NLRP3 inflammasome is a multi-protein complex that initiates innate immunity responses when exposed to a wide range of stimuli, including pathogen-associated molecular patterns (PAMPs) and danger-associated molecular patterns (DAMPs). Inflammasome activation leads to the release of the pro-inflammatory cytokines interleukin (IL)-1β and IL-18 and to pyroptotic cell death. Over-activation of NLRP3 inflammasome has been associated with several chronic inflammatory diseases. A deep knowledge of NLRP3 inflammasome biology is required to better exploit its potential as therapeutic target and for the development of new selective drugs. To this purpose, in the past few years, several tools have…

Programmed cell death2019-20 coronavirus outbreakInflammasomesInterleukin-1betaReviewBiologyBiochemical assaysModels BiologicalCatalysisInflammasomelcsh:ChemistryInorganic ChemistryNLRP3NLR Family Pyrin Domain-Containing 3 ProteinPyroptosismedicineDeep knowledgeAlarminsAnimalsHumansPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyInnate immune systemintegumentary systemCell modelsPathogen-Associated Molecular Pattern MoleculesOrganic ChemistryInterleukin-18InterleukinInflammasomeGeneral MedicineBiophysical assaysImmunity InnateComputer Science ApplicationsCell biologyNLRP3 inhibitorslcsh:Biology (General)lcsh:QD1-999Mechanism of actionRead-outsmedicine.symptomInflammasome complexSignal Transductionmedicine.drugInternational Journal of Molecular Sciences
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Acute organ failure following the loss of anti-apoptotic cellular FLICE-inhibitory protein involves activation of innate immune receptors

2014

Apoptosis signaling is involved in both physiological tissue homeostasis and acute and chronic diseases. The role of regulatory apoptosis signaling molecules and their organ-specific functions are less defined. Therefore, we investigated the loss of the anti-apoptotic cellular FLICE-inhibitory protein (cFLIP) and the mechanisms of the resulting lethal organ failure in vivo using inducible knockout mice. These were generated by crossing floxed cFLIP mice to a tamoxifen inducible Rosa26-creERT2 mouse strain. Death following global loss of cFLIP resulted from liver failure, accumulation of M1-polarized macrophages and accompanying hepatic cell death and inflammation. Apoptosis was also promine…

Programmed cell deathCASP8 and FADD-Like Apoptosis Regulating ProteinMice TransgenicInflammationBiologyMiceImmune systemmedicineAnimalsMolecular BiologyTissue homeostasisOriginal PaperInnate immune systemMacrophagesMembrane ProteinsCell BiologyLiver Failure AcuteImmunity InnateCell biologyToll-Like Receptor 4TransplantationApoptosisToll-Like Receptor 9Stimulator of interferon genesHepatocytesmedicine.symptomCell Death & Differentiation
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Cell cooperation in coelomocyte cytotoxic activity of Paracentrotus lividus coelomocytes

2007

The coelomic fluid from the sea urchin Paracentrotus lividus contains several coelomocyte types including amoebocytes and uncoloured spherulocytes involved in immune defences. In the present paper, we show a Ca(2+)-dependent cytotoxic activity for the unfractionated coelomocytes assayed in vitro, with rabbit erythrocytes and the K562 tumour cell line. In a plaque-forming assay, whole coelomocyte preparations as well as density gradient separated coelomocyte populations revealed that cell populations enriched in uncoloured spherulocytes, exerted high cytotoxic activity by releasing lysins in the presence of amoebocytes. This cooperative effect could be dependent on soluble factors released b…

Programmed cell deathErythrocytesPhysiologyCytotoxicityCell CommunicationCell SeparationBiochemistryParacentrotus lividusbiology.animalCentrifugation Density GradientAnimalsHumansCytotoxic T cellCytotoxicityMolecular BiologySea urchinCoelomocyteCoelomocyte cooperationInnate immunityCell DeathEchinodermbiologyAnatomybiology.organism_classificationIn vitroCell biologyParacentrotus lividusCell cultureParacentrotusRabbitsCoelomocyteK562 CellsComparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology
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Iron Metabolism in the Tumor Microenvironment-Implications for Anti-Cancer Immune Response

2021

New insights into the field of iron metabolism within the tumor microenvironment have been uncovered in recent years. Iron promotes the production of reactive oxygen species, which may either trigger ferroptosis cell death or contribute to malignant transformation. Once transformed, cancer cells divert tumor-infiltrating immune cells to satisfy their iron demand, thus affecting the tumor immunosurveillance. In this review, we highlight how the bioavailability of this metal shapes complex metabolic pathways within the tumor microenvironment and how this affects both tumor-associated macrophages and tumor-infiltrating lymphocytes functions. Furthermore, we discuss the potentials as well as th…

Programmed cell deathIronReviewMalignant transformationImmune systemNeoplasmsTumor MicroenvironmentmedicineHumanscanceriron metabolismlcsh:QH301-705.5chemistry.chemical_classificationReactive oxygen speciesTumor microenvironmentInnate immune systemImmunityCancerGeneral Medicinemedicine.diseaseferroptosisferroptosiadaptive immune response tumor microenvironmentlcsh:Biology (General)chemistryCancer cellCancer researchinnate immune response
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Zinc accelerates respiratory burst termination in human PMN

2021

The respiratory burst of phagocytes is essential for human survival. Innate immune defence against pathogens relies strongly on reactive oxygen species (ROS) production by the NADPH oxidase (NOX2). ROS kill pathogens while the translocation of electrons across the plasma membrane via NOX2 depolarizes the cell. Simultaneously, protons are released into the cytosol. Here, we compare freshly isolated human polymorphonuclear leukocytes (PMN) to the granulocytes-like cell line PLB 985. We are recording ROS production while inhibiting the charge compensating and pH regulating voltage-gated proton channel (HV1). The data suggests that human PMN and the PLB 985 generate ROS via a general mechanism,…

Programmed cell deathMedicine (General)PhagocyteQH301-705.5NeutrophilsClinical BiochemistryBiochemistryIon ChannelsFlow cytometryR5-920medicineHumansPhagocyte ; Zinc ; Zinkstoffwechsel ; pH ; H<sub>v</sub>1 ; ROSBiology (General)HV1Respiratory Burstchemistry.chemical_classificationReactive oxygen speciesNADPH oxidaseInnate immune systembiologymedicine.diagnostic_testChemistrypHOrganic ChemistryNADPH OxidasesROSCell biologyRespiratory burstCytosolZincmedicine.anatomical_structurePhagocytebiology.proteinReactive Oxygen SpeciesResearch Paper
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Caspase-8 regulates TNF-alpha induced epithelial necroptosis and terminal ileitis

2011

Two groups identify the regulation of death-receptor-induced necroptosis as an epithelial intrinsic mechanism that is important for the maintenance of immune homeostasis and the prevention of intestinal inflammation in mice. Welz et al. describe an unexpected physiological function for FADD (Fas-associated protein with death domain), an adaptor protein required for death-receptor-induced apoptosis. Mice with intestinal epithelial specific knockout of FADD develop severe colon inflammation due to increased death of FADD-deficient colonic epithelial cells. Gunther et al. report a novel and unexpected function of caspase-8 in maintaining immune homeostasis in the gut. Caspase-8 expression by g…

Programmed cell deathPaneth CellsNecroptosisInflammationApoptosisBiologyIn Vitro Techniquesdigestive systemArticle03 medical and health sciencesMiceNecrosis0302 clinical medicineCrohn DiseasemedicineAnimalsHumansFADD030304 developmental biology0303 health sciencesCaspase 8MultidisciplinaryInnate immune systemTumor Necrosis Factor-alphaColitisIntestinal epithelium3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesPaneth cellImmunologybiology.proteinCancer researchTumor necrosis factor alphaGoblet Cellsmedicine.symptomGene DeletionNature
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