Search results for "Innate"

showing 10 items of 638 documents

Specificity of human natural antibodies referred to as anti-Tn

2020

International audience; To understand the role of human natural IgM known as antibodies against the carbohydrate epitope Tn, the antibodies were isolated using GalNAcα−Sepharose affinity chromatography, and their specificity was profiled using microarrays (a glycan array printed with oligosaccharides and bacterial polysaccharides, as well as a glycopeptide array), flow cytometry, and inhibition ELISA. The antibodies bound a restricted number of GalNAcα-terminated oligosaccharides better than the parent monosaccharide, e.g., 6-O-Su-GalNAcα and GalNAcα1−3Galβ1−3(4)GlcNAcβ. The binding with several bacterial polysaccharides that have no structural resemblance to the affinity ligand GalNAcα was…

0301 basic medicineGlycanGlycansImmunologyTn antigenAntibody Affinity[SDV.CAN]Life Sciences [q-bio]/CancerAnti-Glycan antibodiesEpitopeAntigen-Antibody ReactionsEpitopesJurkat Cells03 medical and health sciences0302 clinical medicineAffinity chromatography[SDV.CAN] Life Sciences [q-bio]/CancerAntibody SpecificityNeoplasmsTn antigenHumansAntigens Tumor-Associated CarbohydrateAmino Acid SequenceMolecular BiologyPeptide sequenceCancerbiologyChemistryBacterial polysaccharideImmunity Innate3. Good health030104 developmental biologyCarbohydrate SequenceImmunoglobulin MBiochemistryNatural antibodiesbiology.proteinParatopeAntibody030215 immunology
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Efficacy of interleukin 10 gene hydrofection in pig liver vascular isolated ‘in vivo’ by surgical procedure with interest in liver transplantation

2019

AIM Liver transplantation is the only curative strategy for final stage liver diseases. Despite the great advances achieved during the last 20 years, the recipient immune response after transplantation is not entirely controlled. This results in high rates of acute cell rejection and, approximately, 10% of early mortality. Therapeutic treatment could be improved by efficiently transfecting genes that encode natural immunosuppressant proteins, employing safe procedures that could be transferred to clinical setting. In this sense, interleukin 10 plays a central role in immune tolerance response by acting at different levels. METHODS hIL10 gene was hydrofected by retrograde hydrodynamic inject…

0301 basic medicineGraft RejectionCardiovascular ProceduresSwinePhysiologymedicine.medical_treatmentGene TransferVascular SurgeryLiver transplantationPharmacologyImmune tolerance0302 clinical medicineImmune PhysiologyMedicine and Health SciencesMammalsInnate Immune SystemMultidisciplinaryQRGene Transfer TechniquesEukaryotaBlood proteinsRecombinant ProteinsInterleukin-10Interleukin 10LiverVertebratesModels AnimalMedicineCytokines030211 gastroenterology & hepatologyFemaleAnatomyResearch ArticlePlasmidsScienceImmunologyGenetic VectorsSurgical and Invasive Medical ProceduresResearch and Analysis MethodsInjectionsEnd Stage Liver Disease03 medical and health sciencesDigestive System ProceduresGene DeliveryImmune systemIn vivomedicineGene Expression and Vector TechniquesGeneticsImmune ToleranceAnimalsHumansMolecular Biology TechniquesMolecular BiologyTransplantationMolecular Biology Assays and Analysis Techniquesbusiness.industryOrganismsBiology and Life SciencesOrgan TransplantationGenetic TherapyMolecular DevelopmentLiver TransplantationTransplantation030104 developmental biologyImmune SystemAmniotesHydrodynamicsLiver functionbusinessDevelopmental BiologyPLoS ONE
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Echinodermata: The complex immune system in echinoderms

2018

View references (418) The Echinodermata are an ancient phylum of benthic marine invertebrates with a dispersal-stage planktonic larva. These animals have innate immune systems characterized initially by clearance of foreign particles, including microbes, from the body cavity of both larvae and adults, and allograft tissue rejection in adults. Immune responsiveness is mediated by a variety of adult coelomocytes and larval mesenchyme cells. Echinoderm diseases from a range of pathogens can lead to mass die-offs and impact aquaculture, but some individuals can recover. Genome sequences of several echinoderms have identified genes with immune function, including expanded families of Toll-like r…

0301 basic medicineImmunoglobulin geneProteomicsSea CucumbersAntimicrobial peptidesDiseasesImmune responsesBiologySenescenceImmune development03 medical and health sciences0302 clinical medicineImmune systemAsteroideaAsteroidea Brittle stars Coelomocytes Crinoidea Diseases Echinoidea Genomics Holothuroidea Immune development Immune responses Immuno-toxicology Larval immune cells Ophiuroidea Proteomics Sea cucumbers Sea lilies Sea stars Sea urchins SenescenceApostichopus JaponicusSea cucumbersAsteroidea; Brittle stars; Coelomocytes; Crinoidea; Diseases; Echinoidea; Genomics; Holothuroidea; Immune development; Immune responses; Immuno-toxicology; Larval immune cells; Ophiuroidea; Proteomics; Sea cucumbers; Sea lilies; Sea stars; Sea urchins; SenescenceCrinoideaSea starsHolothuroideaOphiuroideaSea urchinsInnate immune systemCoelomocytesfungiLarval immune cellsSea liliesChemotaxisEchinoideaMarine invertebratesGenomicsbiology.organism_classificationComplement systemCell biology030104 developmental biologyEchinodermBrittle starsCoelomocytes Apostichopus Japonicus Sea CucumbersImmuno-toxicology030217 neurology & neurosurgery
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EBI2 in splenic and local immune responses and in autoimmunity

2017

Abstract The seven transmembrane G protein-coupled receptor EBV-induced gene 2 (EBI2), also known as GPR183, is expressed in particular in immune cells. Activated by its endogenous ligands, which are a group of oxysterols, it functions as a chemo-attractant receptor, mediating cell migration. In coordination with other receptors, EBI2 plays important roles in controlling the migration of immune cells during the course of a T-dependent Ab response in the spleen. In recent years, it has become clear that EBI2 also has other roles to play in the immune system. Thus, EBI2 seems to be involved in innate immune responses, such as those mediated by TLR signaling, and it has been implicated in regi…

0301 basic medicineImmunologyAutoimmunitySpleenBiologymedicine.disease_causeReceptors G-Protein-CoupledAutoimmunity03 medical and health sciences0302 clinical medicineImmune systemmedicineAnimalsHumansImmunology and AllergyReceptorG protein-coupled receptorInnate immune systemGPR183Cell migrationCell Biologybiochemical phenomena metabolism and nutritionImmunity Innate030104 developmental biologymedicine.anatomical_structureImmunologybacteriaSpleen030217 neurology & neurosurgeryJournal of Leukocyte Biology
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Targeting cellular fatty acid synthesis limits T helper and innate lymphoid cell function during intestinal inflammation and infection

2019

CD4+ T cells contribute critically to a protective immune response during intestinal infections, but have also been implicated in the aggravation of intestinal inflammatory pathology. Previous studies suggested that T helper type (Th)1 and Th17 cells depend on de novo fatty acid (FA) synthesis for their development and effector function. Here, we report that T-cell-specific targeting of the enzyme acetyl-CoA carboxylase 1 (ACC1), a major checkpoint controlling FA synthesis, impaired intestinal Th1 and Th17 responses by limiting CD4+ T-cell expansion and infiltration into the lamina propria in murine models of colitis and infection-associated intestinal inflammation. Importantly, pharmacolog…

0301 basic medicineImmunologyBiologyMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemRAR-related orphan receptor gammamedicineAnimalsImmunology and AllergyFatty acid synthesisBarrier functionLamina propriaEffectorFatty AcidsInnate lymphoid cellT-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 3ColitisInflammatory Bowel DiseasesImmunity InnateBiosynthetic PathwaysDisease Models Animal030104 developmental biologymedicine.anatomical_structurechemistryImmunologyLipogenesisBiomarkersAcetyl-CoA Carboxylase030215 immunologyMucosal Immunology
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Rheostatic Functions of Mast Cells in the Control of Innate and Adaptive Immune Responses

2017

Mast cells are evolutionarily ancient cells, endowed with a unique developmental, phenotypic, and functional plasticity. They are resident cells that participate in tissue homeostasis by constantly sampling the microenvironment. As a result of their large repertoire of receptors, they can respond to multiple stimuli and selectively release different types and amounts of mediator. Here, we present and discuss the recent mast cell literature, focusing on studies that demonstrate that mast cells are more than a switch that is turned ‘off’ when in the resting state and ‘on’ when in the degranulating state. We propose a new vision of mast cells in which, by operating in a ‘rheostatic 

0301 basic medicineImmunologyBiologymedicine.disease_causeAutoimmunityImmunomodulation03 medical and health sciencesMediatorImmune systemImmunityMAST CELLmedicineAnimalsHomeostasisHumansADAPTIVE IMMUNITYImmunology and AllergyMast CellsReceptorTissue homeostasisImmunology and Allergy; ImmunologyMAST CELL INNATE IMMUNITY ADAPTIVE IMMUNITYMast cellAcquired immune systemImmunity InnateCell biologySelf Tolerance030104 developmental biologymedicine.anatomical_structureCellular MicroenvironmentOrgan SpecificityImmunologyINNATE IMMUNITYTrends in Immunology
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Danger signals: Chemotherapy enhancers?

2017

IF 9.614; International audience; Endogenous danger signals are molecules normally present in a given cell compartment that are rapidly released following cell stress and induce immune responses. We and others have shown that dying tumor cells treated with some chemotherapies are able to induce anticancer immune responses, which rely on their release of danger signals such as the nuclear protein HMGB1. DNA can also be released from chemotherapy-treated tumor cells, act as a danger signal, and boost anticancer immunity. While the immunostimulatory properties of DNA have been identified for decades, the recent discovery of a novel family of receptors, cytosolic DNA sensors, has provided a nov…

0301 basic medicineImmunologyCelleducationBiologyHMGB1CD8+ T cellschemotherapyCancer Vaccines03 medical and health sciences0302 clinical medicineImmune systemDrug TherapyNeoplasmsmedicineImmunology and AllergyCytotoxic T cellAnimalsHumanscancer[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMolecular Targeted TherapyNuclear proteinHMGB1 ProteinReceptorinnate immunityInnate immune systemDNAadaptive immunityAcquired immune systemCombined Modality TherapyImmunity Innate3. Good health030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinCancer research[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapy030215 immunologySignal TransductionSTINGImmunological Reviews
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Group 3 Innate Lymphoid Cells Program a Distinct Subset of IL-22BP-Producing Dendritic Cells Demarcating Solitary Intestinal Lymphoid Tissues.

2019

Solitary intestinal lymphoid tissues such as cryptopatches (CPs) and isolated lymphoid follicles (ILFs) constitute steady-state activation hubs containing group 3 innate lymphoid cells (ILC3) that continuously produce interleukin (IL)-22. The outer surface of CPs and ILFs is demarcated by a poorly characterized population of CD11c+ cells. Using genome-wide single-cell transcriptional profiling of intestinal mononuclear phagocytes and multidimensional flow cytometry, we found that CP- and ILF-associated CD11c+ cells were a transcriptionally distinct subset of intestinal cDCs, which we term CIA-DCs. CIA-DCs required programming by CP- and ILF-resident CCR6+ ILC3 via lymphotoxin-β receptor sig…

0301 basic medicineImmunologyPopulationCD11cGene ExpressionMice TransgenicC-C chemokine receptor type 6BiologyFlow cytometryImmunophenotyping03 medical and health sciencesMicePeyer's Patches0302 clinical medicineRNA Small CytoplasmicmedicineImmunology and AllergyAnimalsIntestinal Mucosaeducationeducation.field_of_studymedicine.diagnostic_testGene Expression ProfilingInnate lymphoid cellInterleukinDendritic CellsReceptors InterleukinLipid MetabolismImmunity InnateLymphocyte SubsetsCell biology030104 developmental biologyInfectious DiseasesLymphotoxinGene Expression Regulation030220 oncology & carcinogenesisHomeostasisBiomarkersSignal TransductionImmunity
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Primary sjogren syndrome: Focus on innate immune cells and inflammation

2020

Primary Sjogren Syndrome (pSS) is a complex, multifactorial rheumatic disease that mainly targets salivary and lacrimal glands, inducing epithelitis. The cause behind the autoimmunity outbreak in pSS is still elusive; however, it seems related to an aberrant reaction to exogenous triggers such as viruses, combined with individual genetic pre-disposition. For a long time, autoantibodies were considered as the hallmarks of this disease; however, more recently the complex interplay between innate and adaptive immunity as well as the consequent inflammatory process have emerged as the main mechanisms of pSS pathogenesis. The present review will focus on innate cells and on the principal mechani…

0301 basic medicineImmunologyinnate lymphoid cellslcsh:MedicineIFN signatureInflammationDiseaseReviewmedicine.disease_causeAutoimmunityPathogenesis03 medical and health sciences0302 clinical medicinestomatognathic systemDrug DiscoverymedicineInnate lymphoid cellPharmacology (medical)Sjogren syndromeCytokine030203 arthritis & rheumatologyPharmacologyInflammationInnate immunityInnate immune systemSjogren syndrome.business.industryInnate lymphoid celllcsh:RAutoantibodyAcquired immune systemcytokinesstomatognathic diseases030104 developmental biologyInfectious DiseasesImmunologymedicine.symptombusiness
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Regulation of kynurenine biosynthesis during influenza virus infection.

2017

Influenza A viruses (IAVs) remain serious threats to public health because of the shortage of effective means of control. Developing more effective virus control modalities requires better understanding of virus–host interactions. It has previously been shown that IAV induces the production of kynurenine, which suppresses T-cell responses, enhances pain hypersensitivity and disturbs behaviour in infected animals. However, the regulation of kynurenine biosynthesis during IAV infection remains elusive. Here we showed that IAV infection induced expression of interferons (IFNs), which upregulated production of indoleamine-2,3-dioxygenase (IDO1), which catalysed the kynurenine biosynthesis. Furt…

0301 basic medicineIndoleshost-pathogen interactionViral Nonstructural Proteinsmedicine.disease_causeVirus ReplicationBiochemistryinfluenza viruschemistry.chemical_compoundMiceInfluenza A Virus H1N1 SubtypeInterferonOximesinnate immunityLungOxazolesKynurenineRegulation of gene expressionMice Inbred BALB CSulfonamidesTryptophaninterferon3. Good healthHost-Pathogen InteractionsFemaleMetabolic Networks and Pathwaysmedicine.drugHost–pathogen interaction030106 microbiologyPrimary Cell CultureBiologyta3111Antiviral AgentsVirus03 medical and health sciences3-dioxygenase (IDO1)Orthomyxoviridae InfectionsmedicineAnimalsHumansImmunologic FactorsIndoleamine-Pyrrole 23-DioxygenasePyrrolesMolecular BiologyInnate immune systemta1184Macrophagesta1183ta1182Cell BiologyVirologyindoleamine-pyrrole 2Thiazoles030104 developmental biologyHerpes simplex virusViral replicationchemistryGene Expression RegulationInterferonsTranscriptomeKynurenineThe FEBS journal
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