Search results for "Insulin"

showing 10 items of 1360 documents

Genetic ablation of macrohistone H2A1 leads to increased leanness, glucose tolerance and energy expenditure in mice fed a high-fat diet.

2015

Contains fulltext : 155347.pdf (Publisher’s version ) (Closed access) BACKGROUND/OBJECTIVES: In the context of obesity, epigenetic mechanisms regulate cell-specific chromatin plasticity, perpetuating gene expression responses to nutrient excess. MacroH2A1, a variant of histone H2A, emerged as a key chromatin regulator sensing small nutrients during cell proliferation and differentiation. Mice genetically ablated for macroH2A1 (knockout (KO)) do not show overt phenotypes under a standard diet. Our objective was to analyse the in vivo role of macroH2A1 in response to nutritional excess. METHODS: Twelve-week-old whole-body macroH2A1 KO male mice were given a high-fat diet (60% energy from lard…

EXPRESSIONCHROMATINNonalcoholic steatohepatitisModels Molecularmedicine.medical_specialtyHISTONE VARIANT MACROH2Amacrohistone H2A1 High fat diet obesity.Endocrinology Diabetes and MetabolismLIVER-DISEASE NAFLDTHERMOGENESISMedicine (miscellaneous)Adipose tissueBiologyDiet High-FatCell LineHistonesMiceINFLAMMATIONAdipose Tissue BrownThinnessInternal medicineBINDINGmedicineAnimalsGenetic ablationNutrition and DieteticsAdipogenesisNONALCOHOLIC STEATOHEPATITISTRANSCRIPTIONAL COREGULATOR PELP1medicine.diseaseNUTRITION&DIETETICSObesityDisease Models AnimalRenal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11]EndocrinologyEnergy expenditureFat dietOBESITYInsulin ResistanceEnergy MetabolismThermogenesisInternational journal of obesity (2005)
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Trace elements in glucometabolic disorders: an update.

2010

International audience; Many trace elements, among which metals, are indispensable for proper functioning of a myriad of biochemical reactions, more particularly as enzyme cofactors. This is particularly true for the vast set of processes involved in regulation of glucose homeostasis, being it in glucose metabolism itself or in hormonal control, especially insulin. The role and importance of trace elements such as chromium, zinc, selenium, lithium and vanadium are much less evident and subjected to chronic debate. This review updates our actual knowledge concerning these five trace elements. A careful survey of the literature shows that while theoretical postulates from some key roles of th…

Endocrinology Diabetes and Metabolism030209 endocrinology & metabolismReviewBioinformatics03 medical and health sciences0302 clinical medicineInsulin resistancemedicineInternal Medicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologySDV:BBMBiochemical reactionsGlucose homeostasisIn patient[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyBeneficial effects[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biologylcsh:RC620-627030304 developmental biology0303 health sciencesbusiness.industrymedicine.disease3. Good healthTrace (semiology)lcsh:Nutritional diseases. Deficiency diseasesbusiness
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Impaired glucose regulation, depressive symptoms, and health-related quality of life

2020

IntroductionThis study aims to investigate whether the associations between impaired glucose regulation and health-related quality of life are modified by severity or type of depressive symptoms.Research design and methodsFor this cross-sectional study, we included 1939 individuals (mean age 61.5 years) from the Helsinki Birth Cohort Study. Between 2001 and 2004, a standard 2-hour 75 g oral glucose tolerance test was applied to define normoglycemia, pre-diabetes, and newly diagnosed diabetes. Information on previously diagnosed diabetes was collected from national registers and questionnaires. Pre-diabetes was defined as having either impaired fasting glucose or impaired glucose tolerance. …

Endocrinology Diabetes and MetabolismelämänlaatuPsychosocial ResearchCohort StudiesImpaired glucose tolerancehenkinen hyvinvointi0302 clinical medicineQuality of lifeinsulin resistanceMedicine18661506030212 general & internal medicineDepression (differential diagnoses)POPULATIONMETABOLIC SYNDROMERISKeducation.field_of_studyCOMPLICATIONSDepressionASSOCIATIONMiddle Agedkeski-ikäiset3. Good healthPREVALENCEtype 2INVENTORY-IIdepressiondiabetes mellitusikääntyneetmasennusmedicine.medical_specialtyPopulationDiseases of the endocrine glands. Clinical endocrinology03 medical and health sciencesInsulin resistanceInternal medicineDiabetes mellitusHumanseducationbusiness.industryDIABETES-MELLITUSinsuliiniresistenssimedicine.diseaseImpaired fasting glucoseRC648-665Cross-Sectional StudiesGlucosePSYCHOMETRIC PROPERTIESquality of life3121 General medicine internal medicine and other clinical medicineMetabolic syndromebusinessfyysinen hyvinvointiaikuistyypin diabetes030217 neurology & neurosurgeryBMJ Open Diabetes Research & Care
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A combined approach for gene discovery identifies insulin-like growth factor-binding protein-related protein 1 as a new gene implicated in human endo…

2003

In the past, human endometrial receptivity has been investigated by chasing specific molecules throughout the menstrual cycle. Now the genomic approach allows us to investigate the hierarchical contribution of a high number of genes to a specific function. In this study, we analyzed differentially the gene expression pattern of 375 human cytokines, chemokines, and related factors, plus that of their receptors, in endometrial receptivity. To do this, we used a combined approach of human endometrium and cell lines. We have compared the gene expression pattern in receptive vs. prereceptive human endometria and contrasted the results with gene expression in the highly adhesive cell line (to JAR…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryBiologyEndometriumBiochemistryInsulin-like growth factor-binding proteinCell LineEndometriumMiceEndocrinologyPregnancyGene expressionmedicineCell AdhesionAnimalsHumansRNA MessengerReceptorGeneIn Situ HybridizationMenstrual CycleFluorescent DyesMessenger RNAReverse Transcriptase Polymerase Chain ReactionBiochemistry (medical)Epithelial CellsMolecular biologyImmunohistochemistryInsulin-Like Growth Factor Binding ProteinsCytokinemedicine.anatomical_structureBlastocystGene Expression RegulationCell culturebiology.proteinFemaleStromal CellsCarrier ProteinsThe Journal of clinical endocrinology and metabolism
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The therapeutic modulation of atherogenic dyslipidemia and inflammatory markers in the metabolic syndrome: what is the clinical relevance?

2008

The metabolic syndrome consists of a constellation of clinical and biochemical risk factors that cluster together and heighten the risk for atherogenesis, cardiovascular diseases, and diabetes. Established risk cardiovascular factors like hypertension, atherogenic dyslipidaemia, and glucose intolerance occur in the setting of insulin resistance and central adiposity, with genetic and environmental influences modulating the ultimate risk. Chronic insults to the endothelium take its toll in the form of silent as well as clinically evident cardiovascular events. The cellular and vascular accompaniments have shed some light into the underlying pathophysiology. Heightened, low-grade inflammatory…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentInflammationMetabolic syndrome Insulin resistance Dyslipidemia Atherosclerosis Adipocytokinesmedicine.disease_causeTargeted therapyEndocrinologyInsulin resistanceRisk FactorsDiabetes mellitusHeredityInternal MedicinemedicineHumansClinical significanceDyslipidemiasInflammationMetabolic Syndromebusiness.industryCholesterol LDLGeneral MedicineAtherosclerosismedicine.diseaseC-Reactive ProteinImmunologymedicine.symptomMetabolic syndromebusinessBiomarkersDyslipidemiaActa Diabetologica
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The effects of muscle mass and muscle quality on cardio-metabolic risk in peripubertal girls: a longitudinal study from childhood to early adulthood.

2017

Increased cardio-metabolic risk is well documented in children and adolescents with obesity and normal weight obesity (NWO). However, the associations of muscle mass and muscle quality with cardio-metabolic risk, independent of weight status from childhood to adulthood, has not been examined. A total of 236 girls were followed from pre-puberty to early adulthood. Fat mass (FM) and lean mass (LM) of the whole body were assessed by a dual-energy X-ray absorptiometry; muscle cross-sectional area (mCSA), muscle density (mDen; skeletal muscle fat content) of the lower leg by the peripheral quantitative computerized tomography; and blood glucose, insulin, triglycerides and high-density lipoprotei…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentlongitudinal researchMedicine (miscellaneous)030204 cardiovascular system & hematologyOverweightBody Mass Index0302 clinical medicineRisk FactorsLongitudinal StudiesChildrasva-arvotNutrition and DieteticsFramingham Risk Scoregirlsta3141tytötNormal weight obesitymedicine.anatomical_structuremuscle massAdipose TissueCardiovascular DiseasesFemalemedicine.symptommedicine.medical_specialtyAdolescentblood sugar030209 endocrinology & metabolismpitkittäistutkimus03 medical and health sciencesInternal medicinemedicinefatty acid levelsHumansObesityMuscle Skeletalkehonkoostumusbody compositionbusiness.industryInsulinSkeletal muscleOverweightmedicine.diseaseObesityEndocrinologylihasmassaverensokeriLean body massbusinessBody mass indexInternational journal of obesity (2005)
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Subtype-specific endothelin-A and endothelin-B receptor desensitization correlates with differential receptor phosphorylation.

1998

In the rat cardiovascular system endothelin-1 (ET-1) elicits prolonged physiologic responses mediated by the ET A receptor, whereas the effects mediated by the ET B receptor are transient. The molecular mechanisms for the subtype-specific responses are not yet clear. However, post-translational modifications such as phosphorylation and palmitoylation may play an important role. In Sf9 cells overexpressing the human ET A and ET B receptors, both subtypes are palmitoylated. However, only the ET B but not the ET A receptor is phosphorylated in a ligand-dependent manner. Because phosphorylation is believed to play an important role in ligand-dependent receptor inactivation, we analyzed whether …

Endothelin Receptor Antagonistsmedicine.medical_specialtyTropomyosin receptor kinase BCHO CellsBiologyEstrogen-related receptor alphaInternal medicineCricetinaemedicineEnzyme-linked receptorAnimalsHumansCloning MolecularPhosphorylationReceptorProtease-activated receptor 2Insulin-like growth factor 1 receptorPharmacologyReceptors EndothelinInterleukin-13 receptorReceptor Endothelin AReceptor Endothelin BCell biologyRatsInterleukin 10KineticsEndocrinologyCardiology and Cardiovascular MedicineSignal TransductionJournal of cardiovascular pharmacology
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Modulation of Diabetes by Natural Products and Medicinal Plants via Incretins

2019

Incretins are metabolic hormones released after a meal that increase insulin secretion from pancreatic β-cells. The two main incretins are the intestinal peptides glucagon-like peptide-1 and glucose-dependent insulinotropic polypeptide. Both induce a decrease in glycemia, slow down the absorption of nutrients, and are inactivated by the enzyme dipeptidyl peptidase-4. Recently, incretin-based therapies have become a useful tool to treat diabetic patients, and different studies have focused on the identification of glucagon-like peptide-1 receptor agonists, including those of natural origin. This review focuses on the new findings of medicinal plants and natural products as possible active ag…

Enfermedad cardiovascularPharmaceutical SciencePharmacology01 natural sciencesAnalytical Chemistry//purl.org/becyt/ford/1 [https]Tratamiento médicoDPP-4Drug DiscoveryReceptorchemistry.chemical_classificationGIPGLUCAGON-LIKE PEPTIDE-1digestive oral and skin physiologyGlucagon-like peptide-1Diabetes mellitus tipo 2HomeopatíaINCRETINSMolecular MedicineCIENCIAS NATURALES Y EXACTAShormones hormone substitutes and hormone antagonistsendocrine systemOtras Ciencias BiológicasIncretinIncretinsCiencias BiológicasDiabetes mellitusDiabetes MellitusmedicineAnimalsHumansHypoglycemic Agents//purl.org/becyt/ford/1.6 [https]Dipeptidyl peptidase-4PharmacologyBiological ProductsPlants MedicinalGuar gum010405 organic chemistryOrganic ChemistryGLUCOSE-DEPENDENT INSULINOTROPIC POLYPEPTIDEmedicine.diseaseDIPEPTIDYL PEPTIDASE-40104 chemical sciences010404 medicinal & biomolecular chemistryEnzymeComplementary and alternative medicinechemistryGLP-1PhytotherapyHormone
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CLA-Enriched Diet Containing t10,c12-CLA Alters Bile Acid Homeostasis and Increases the Risk of Cholelithiasis in Mice

2011

International audience; Mice fed a mixture of CLA containing t10,c12-CLA lose fat mass and develop hyperinsulinemia and hepatic steatosis due to an accumulation of TG and cholesterol. Because cholesterol is the precursor in bile acid (BA) synthesis, we investigated whether t10,c12-CLA alters BA metabolism. In Expt. 1, female C57Bl/6J mice were fed a standard diet for 28 d supplemented with a CLA mixture (1 g/100 g) or not (controls). In Expt. 2, the feeding period was reduced to 4, 6, and 10 d. In Expt. 3, mice were fed a diet supplemented with linoleic acid, c9,t11-CLA, or t10,c12-CLA (0.4 g/100 g) for 28 d. In Expt. 1, the BA pool size was greater in CLA-fed mice than in controls and the …

Enterohepatic circulationmedicine.medical_specialtymedicine.drug_classLinoleic acid[SDV]Life Sciences [q-bio]Blotting WesternMedicine (miscellaneous)030209 endocrinology & metabolismCholesterol 7 alpha-hydroxylasePolymerase Chain ReactionBile Acids and SaltsMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsCholelithiasisInternal medicineHyperinsulinémiemedicineHyperinsulinemiaAnimalsHomeostasisEnterohepatic circulation030304 developmental biology0303 health sciencesNutrition and DieteticsBile acidintegumentary systemCholesterolalpha-Linolenic Acidfood and beveragesmedicine.diseaseDietary FatsBile Salt Export PumpMice Inbred C57BLCholesterol 7-alpha hydroxylaseCholesterolEndocrinologyMetabolismLiverchemistryNutrient physiologyFemalelipids (amino acids peptides and proteins)Steatosis[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Correction: DAPIT Over-Expression Modulates Glucose Metabolism and Cell Behaviour in HEK293T Cells

2015

Introduction Diabetes Associated Protein in Insulin-sensitive Tissues (DAPIT) is a subunit of mitochondrial ATP synthase and has also been found to associate with the vacuolar H+-ATPase. Its expression is particularly high in cells with elevated aerobic metabolism and in epithelial cells that actively transport nutrients and ions. Deletion of DAPIT is known to induce loss of mitochondrial ATP synthase but the effects of its over-expression are obscure. Results In order to study the consequences of high expression of DAPIT, we constructed a transgenic cell line that constitutively expressed DAPIT in human embryonal kidney cells, HEK293T. Enhanced DAPIT expression decreased mtDNA content and …

Epithelial-Mesenchymal Transitionmitochondrial metabolismBiolääketieteet - BiomedicineCellActive Transport Cell NucleusGene DosageRespiratory chainlcsh:MedicineGene ExpressionMitochondrionta3111glukoosiNeoplasmsmedicineHumansLactic Acidglucoselcsh:ScienceTranscription factorMultidisciplinaryATP synthasebiologyCell growthta1184lcsh:RHEK 293 cellsCorrectionMitochondrial Proton-Translocating ATPasesMitochondriaCell biologyHEK293 CellsDiabetes Associated Protein in Insulin-sensitive Tissuesmedicine.anatomical_structureCell culturebiology.proteinATP synthaselcsh:QResearch ArticlePLOS ONE
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