Search results for "Intellect"

showing 10 items of 642 documents

NEXMIF encephalopathy: an X-linked disorder with male and female phenotypic patterns

2021

Contains fulltext : 231688.pdf (Publisher’s version ) (Closed access) PURPOSE: Pathogenic variants in the X-linked gene NEXMIF (previously KIAA2022) are associated with intellectual disability (ID), autism spectrum disorder, and epilepsy. We aimed to delineate the female and male phenotypic spectrum of NEXMIF encephalopathy. METHODS: Through an international collaboration, we analyzed the phenotypes and genotypes of 87 patients with NEXMIF encephalopathy. RESULTS: Sixty-three females and 24 males (46 new patients) with NEXMIF encephalopathy were studied, with 30 novel variants. Phenotypic features included developmental delay/ID in 86/87 (99%), seizures in 71/86 (83%) and multiple comorbidi…

MalePediatricsmedicine.medical_specialtyINTELLECTUAL DISABILITYAutism Spectrum DisorderEncephalopathyNerve Tissue ProteinsILAE COMMISSIONMOSAICISMEpilepsy/geneticsCLASSIFICATIONEpilepsyBrain Diseases/geneticsGenes X-LinkedSeizuresIntellectual disabilityGenotypemedicineHumansdevelopmental and epileptic encephalopathyMYOCLONIAAtonic seizureGenetics (clinical)Brain Diseasesddc:618Neurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]KIAA2022business.industryMUTATIONSmedicine.diseasePhenotypeAutism Spectrum Disorder/geneticsGenes X-Linked/geneticsAutism spectrum disorderintellectual disabilityNEXMIFAutismepilepsyFemaleINACTIVATIONHuman medicineSeizures/geneticsbusinessPOSITION PAPERGenetics in Medicine
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Survivors of childhood cancer for more than twenty years.

2001

Present health status, complications, and development of long-term survivors of childhood cancer followed for more than 20 years in a single institution were reviewed. The departmental database was searched to identify patients diagnosed with childhood cancer and consequently treated between 1965 and 1978. A total of 124 (77%) long-term survivors participated on a voluntary basis in the study. A semi-standardized interview consisted of measures evaluating the present health condition, sequelae of treatment, second malignancies, intellectual development and presence of offspring of the former patients. The majority of patients were treated with chemotherapy (82%). 67% received radiotherapy a…

MalePediatricsmedicine.medical_specialtyIntellectual developmentOffspringmedicine.medical_treatmentHealth StatusChildhood cancerIntelligenceNuclear FamilyCentral Nervous System NeoplasmsNeoplasmsmedicineHumansRadiology Nuclear Medicine and imagingSurvivorsNeoplasm MetastasisChildRetrospective StudiesChemotherapybusiness.industrySequelaHematologyGeneral Medicinemedicine.diseasePrimary tumorRadiation therapyTreatment OutcomeOncologyMarital statusEducational StatusFemalebusinessFollow-Up StudiesActa oncologica (Stockholm, Sweden)
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Kabuki make-up (Niikawa-Kuroki) syndrome: Clinical and radiological observations in two sicilian children

1991

The Authors describe two patients aged 5 and 8, a female and a male, affected by a condition of polymalformations known as Kabuki make-up or Niikawa-Kuroki syndrome, having a neonatal incidence of 1:32,000 in Japan. There are two hypothesis about the apparent rarity of the syndrome in the rest of the world, including the Asian Continent: the first is that it exists, but is infrequently recognized outside Japan and the second is that it is really more frequent in those parts of the world, where ethnic exchanges are uncommon, as it happens in Japan.

MalePediatricsmedicine.medical_specialtyKabukiEthnic groupNiikawa-Kuroki SyndromeBone and BonesIntellectual DisabilitymedicineHumansAbnormalities MultipleRadiology Nuclear Medicine and imagingChildGrowth Disordersbusiness.industryIncidence (epidemiology)Syndromelanguage.human_languageRadiographyItalyEl NiñoChild PreschoolFaceRadiological weaponPediatrics Perinatology and Child HealthlanguageFemaleCongenital diseasebusinessSicilianPediatric Radiology
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Diagnostic approach to microcephaly in childhood: a two-center study and review of the literature.

2014

Aim: The aim of this study was to assess the diagnostic approach to microcephaly in childhood and to identify the prevalence of the various underlying causes/disease entities. Method: We conducted a retrospective study on a cohort of 680 children with microcephaly (399 males, 281 females; mean age at presentation 7-8mo, range 1mo-5y) from patients presenting to Charité - University Medicine Berlin (n=474) and University Hospital Dresden (n=206). Patient discharge letters were searched electronically to identify cases of microcephaly, and then the medical records of these patients were used to analyze parameters for distribution. Results: The putative aetiology for microcephaly was ascertain…

MalePediatricsmedicine.medical_specialtyMicrocephalyEye DiseasesGenetic counselingMedizinComorbidityGene mutationEpilepsyDevelopmental NeuroscienceGermanyIntellectual DisabilitymedicineHumansRetrospective StudiesEpilepsybusiness.industryInfantRetrospective cohort studymedicine.diseaseComorbidityChild PreschoolPediatrics Perinatology and Child HealthCohortPractice Guidelines as TopicEtiologyMicrocephalyFemaleNeurology (clinical)businessDevelopmental medicine and child neurology
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ADHD symptoms and learning behaviors in children with ASD without intellectual disability. A mediation analysis of executive functions

2018

In spite of its importance for education, the relationship between learning behaviors (LB), attention deficit hyperactivity disorder symptoms (ADHD) and executive functioning (EF) in children with autism spectrum disorder (ASD) has hardly been explored. The first objective of the present study was to compare children with ASD without intellectual disability and children with typical development (TD) on ADHD symptoms and learning behaviors: Motivation/ competence, attitude toward learning, persistence on the task, and strategy/flexibility. The second objective was to analyze the mediator role of behavioral regulation and metacognition components of EF between ADHD symptoms and learning behav…

MalePervasive Developmental DisordersAutism Spectrum Disorderlcsh:MedicineSocial SciencesFamiliesExecutive Function0302 clinical medicineLearning and MemorySociologyIntellectual disabilityMedicine and Health SciencesPsychologylcsh:ScienceChildChildrenMultidisciplinarySchools05 social sciencesExecutive functionsProfessionsNeurologyAutism spectrum disorderFemalemedicine.symptomPsychology050104 developmental & child psychologyClinical psychologyResearch ArticleMediation (statistics)MetacognitionNeuropsychiatric DisordersImpulsivitybehavioral disciplines and activitiesEducation03 medical and health sciencesHuman LearningMetacognition IndexDevelopmental NeuroscienceIntellectual Disabilitymental disordersMental Health and PsychiatrymedicineAttention deficit hyperactivity disorderLearningHumans0501 psychology and cognitive sciencesBehaviorMotivationlcsh:RCognitive PsychologyBiology and Life SciencesTeachersmedicine.diseaseAge GroupsNeurodevelopmental DisordersAttention Deficit Disorder with HyperactivityPeople and PlacesDevelopmental PsychologyCognitive Sciencelcsh:QPopulation GroupingsAdhd030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Disruption of the ASTN2 / TRIM32 locus at 9q33.1 is a risk factor in males for Autism Spectrum Disorders, ADHD and other neurodevelopmental phenotypes

2014

Rare copy number variants (CNVs) disrupting ASTN2 or both ASTN2 and TRIM32 have been reported at 9q33.1 by genome-wide studies in a few individuals with neurodevelopmental disorders (NDDs). The vertebrate-specific astrotactins, ASTN2 and its paralog ASTN1, have key roles in glial-guided neuronal migration during brain development. To determine the prevalence of astrotactin mutations and delineate their associated phenotypic spectrum, we screened ASTN2/TRIM32 and ASTN1 (1q25.2) for exonic CNVs in clinical microarray data from 89 985 individuals across 10 sites, including 64 114 NDD subjects. In this clinical dataset, we identified 46 deletions and 12 duplications affecting ASTN2. Deletions o…

MaleReceptors Cell Surface/geneticsAutismChild Development Disorders Pervasive/geneticsGene ExpressionGenome-wide association studyMedical and Health SciencesTripartite Motif ProteinsRisk FactorsReceptors2.1 Biological and endogenous factorsProtein IsoformsNerve Tissue Proteins/geneticsCopy-number variationAetiologyChildGenetics (clinical)Sequence DeletionPediatricGenetics & HeredityGeneticseducation.field_of_studySingle NucleotideArticlesGeneral MedicineExonsBiological SciencesMental HealthPhenotypeAutism spectrum disorderOrgan SpecificityCerebellar cortexChild PreschoolCell SurfaceSpeech delayFemalemedicine.symptomTranscription Initiation SiteAttention Deficit Disorder with Hyperactivity/geneticsChromosomes Human Pair 9HumanPair 9AdultPediatric Research InitiativeChild Development DisordersAdolescentDNA Copy Number VariationsIntellectual and Developmental Disabilities (IDD)Ubiquitin-Protein LigasesPopulationTranscription Factors/geneticsNerve Tissue ProteinsReceptors Cell SurfaceBiologyPolymorphism Single NucleotideChromosomesYoung AdultClinical ResearchProtein Isoforms/geneticsBehavioral and Social ScienceGeneticsmedicineAttention deficit hyperactivity disorderHumansGenetic Predisposition to DiseasePolymorphismPreschooleducationMolecular BiologyGenetic Association StudiesPervasiveGlycoproteinsHuman GenomeNeurosciencesInfant NewbornGlycoproteins/geneticsInfantNewbornmedicine.diseaseBrain DisordersAttention Deficit Disorder with HyperactivityChild Development Disorders PervasiveCase-Control StudiesAutismTranscription Factors
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Parental Perceptions of the Use of Coercive Measures on Children with Developmental Disabilities

2016

Background Children with developmental disabilities who exhibit challenging behaviour are potentially subject to the use of coercive interventions. The aim of the study was to investigate the prevalence of the use of coercive measures by authorities, according to parents’ reports. Materials and Methods A postal survey was distributed, as a total population study, to 946 Finnish parents of children with developmental disabilities, between the ages of 5 and 15, and who were entitled to the highest disability allowance. Results Of the respondents, 54 (22%) answered ‘yes’ when asked whether their child had been subjected to coercive procedures by authorities. The parents had seldom approved the…

MaleRestraint Physical030506 rehabilitationAdolescentChallenging behaviourCoercionDevelopmental DisabilitiesPersons with Mental DisabilitiesPsychological interventionAllowance (money)restrictive interventionsEducationDevelopmental psychology03 medical and health sciencesIntervention (counseling)Surveys and QuestionnairesIntellectual disabilityDevelopmental and Educational PsychologymedicineSanctionsHumansta516Parental perceptionParent-Child RelationsChildFinlandProblem Behaviorchildren with disabilities4. Education05 social sciences050301 educationparentsChild Day Care Centers16. Peace & justicemedicine.diseasevanhemmatChild PreschoolFemalePerception0305 other medical scienceSeclusionPsychology0503 educationClinical psychologyJournal of Applied Research in Intellectual Disabilities
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Polysomnographic Findings in Fragile X Syndrome Children with EEG Abnormalities

2019

Fragile X syndrome (FXS) is a genetic syndrome with intellectual disability due to the loss of expression of the FMR1 gene located on chromosome X (Xq27.3). This mutation can suppress the fragile X mental retardation protein (FMRP) with an impact on synaptic functioning and neuronal plasticity. Among associated sign and symptoms of this genetic condition, sleep disturbances have been already described, but few polysomnographic reports in pediatric age have been reported. This multicenter case-control study is aimed at assessing the sleep macrostructure and at analyzing the presence of EEG abnormalities in a cohort of FXS children. We enrolled children with FXS and, as controls, children wit…

MaleSleep Wake Disorderscongenital hereditary and neonatal diseases and abnormalitiesPediatricsmedicine.medical_specialtyAdolescentArticle SubjectPolysomnographyNeurosciences. Biological psychiatry. NeuropsychiatryFragile X Mental Retardation Protein03 medical and health sciences0302 clinical medicinechildrenIntellectual disabilitymedicineHumansIctalCircadian rhythmChildEEG abnormalitiesPathologicalPSG030304 developmental biology0303 health sciencesNeuronal PlasticityFragile X syndrome; intellectual disability; polysomnographicbusiness.industryCase-control studyNeuropsychologyElectroencephalographyGeneral Medicinemedicine.diseasepolysomnographicFragile X syndromeNeuropsychology and Physiological PsychologyNeurologyintellectual disabilityCase-Control StudiesFragile X SyndromeCohortFemaleNeurology (clinical)FXSSleepbusiness030217 neurology & neurosurgeryRC321-571Research ArticleBehavioural Neurology
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Intragenic deletions of IL1RAPL1: Report of two cases and review of the literature

2010

IL1RAPL1 (interleukin-1 receptor accessory protein-like 1) located at Xp21.3-22.1 has repeatedly been shown to be deleted in patients with a contiguous gene syndrome also affecting neighboring genes, in particular DMD (dystrophin), DAX-1 (NR0B1, nuclear receptor subfamily 0, group B, member 1), and GK (glycerol kinase). In contrast, intragenic deletions of IL1RAPL1 or other mutations or cytogenetic aberrations affecting IL1RAPL1 have only rarely been identified. Up to date, they have mostly been associated with nonspecific mental retardation (MRX). We report on two nonrelated patients with MR and additional dysmorphic features who both show intragenic deletions of IL1RAPL1, one of them bein…

MaleSubfamilyMicroarrayBiologyPolymerase Chain ReactionContiguous gene syndromeExonIntellectual DisabilityGeneticsmedicineHumansAbnormalities MultipleGeneIn Situ Hybridization FluorescenceGenetics (clinical)X chromosomeSequence DeletionGeneticsKaryotypeMicroarray Analysismedicine.diseasePhenotypePedigreePhenotypeKaryotypingInterleukin-1 Receptor Accessory ProteinAmerican Journal of Medical Genetics Part A
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Further Delineation of Duplications of ARX Locus Detected in Male Patients with Varying Degrees of Intellectual Disability

2022

The X-linked gene encoding aristaless-related homeobox (ARX) is a bi-functional transcription factor capable of activating or repressing gene transcription, whose mutations have been found in a wide spectrum of neurodevelopmental disorders (NDDs); these include cortical malformations, paediatric epilepsy, intellectual disability (ID) and autism. In addition to point mutations, duplications of the ARX locus have been detected in male patients with ID. These rearrangements include telencephalon ultraconserved enhancers, whose structural alterations can interfere with the control of ARX expression in the developing brain. Here, we review the structural features of 15 gain copy-number variants …

MaleTranscription FactorUltraconserved enhancersIntellectual disability3D structureCatalysisInorganic ChemistryMiceAnimalsHumansPhysical and Theoretical ChemistryChildMolecular BiologySpectroscopyHomeodomain ProteinsAnimalKDM5C-SYN1 axiOrganic ChemistryKDM5C-SYN1 axisGenes HomeoboxHomeodomain ProteinGeneral MedicineXp21.3 duplicationComputer Science ApplicationsUltraconserved enhancerSettore MED/03 - Genetica MedicaMutationARXHumanTranscription Factors
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