Search results for "Interleukin-6"

showing 10 items of 370 documents

IL-6 receptor independent stimulation of human gp130 by viral IL-6.

2000

Abstract The genome of human herpes virus 8, which is associated with Kaposi’s sarcoma, encodes proteins with similarities to cytokines and chemokines including a homologue of IL-6. Although the function of these viral proteins is unclear, they might have the potential to modulate the immune system. For viral IL-6 (vIL-6), it has been demonstrated that it stimulates IL-6-dependent cells, indicating that the IL-6R system is used. IL-6 binds to IL-6R, and the IL-6/IL-6R complex associates with gp130 which dimerizes and initiates intracellular signaling. Cells that only express gp130 but no IL-6R cannot be stimulated by IL-6 unless a soluble form of the IL-6R is present. This type of signaling…

MaleSTAT3 Transcription FactorChemokinemedicine.medical_treatmentImmunologyGenetic VectorsBiologylaw.inventionViral ProteinsImmune systemlawAntigens CDmedicineCytokine Receptor gp130Tumor Cells CulturedImmunology and AllergyAnimalsChemical PrecipitationHumansCloning MolecularPhosphorylationInterleukin 6Sarcoma KaposiAgedMembrane GlycoproteinsInterleukin-6Glycoprotein 130Receptors Interleukin-6Growth InhibitorsRecombinant ProteinsCell biologyDNA-Binding ProteinsCytokineInterleukin-6 receptorCOS CellsRecombinant DNAbiology.proteinTrans-ActivatorsIntracellularProtein BindingSignal TransductionJournal of immunology (Baltimore, Md. : 1950)
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Extracellular Nampt promotes macrophage survival via a nonenzymatic interleukin-6/STAT3 signaling mechanism.

2008

Macrophages play key roles in obesity-associated pathophysiology, including inflammation, atherosclerosis, and cancer, and processes that affect the survival-death balance of macrophages may have an important impact on obesity-related diseases. Adipocytes and other cells secrete a protein called extracellular nicotinamide phosphoribosyltransferase (eNampt; also known as pre-B cell colony enhancing factor or visfatin), and plasma levels of eNampt increase in obesity. Herein we tested the hypothesis that eNampt could promote cell survival in macrophages subjected to endoplasmic reticulum (ER) stress, a process associated with obesity and obesity-associated diseases. We show that eNampt potent…

MaleSTAT3 Transcription Factormedicine.medical_specialtyCell SurvivalNicotinamide phosphoribosyltransferaseApoptosisBiologyEndoplasmic ReticulumBiochemistryStat3 Signaling Pathwaychemistry.chemical_compoundParacrine signallingMicePiperidinesInternal medicinemedicineAnimalsSecretionAutocrine signallingSTAT3Nicotinamide PhosphoribosyltransferaseMolecular BiologyNicotinamide MononucleotideNicotinamide mononucleotideAcrylamidesInterleukin-6MacrophagesMechanisms of Signal TransductionCell BiologyCell biologyMice Inbred C57BLEndocrinologychemistryGene Expression Regulationbiology.proteinCytokinesSignal transductionSignal TransductionThe Journal of biological chemistry
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Subclinical atherosclerosis and fetuin-A plasma levels in essential hypertensive patients

2012

The intima-media thickness (IMT) is considered as a surrogate marker for atherosclerotic disease. The aim of this study was to analyze the relationship of carotid IMT with fetuin-A in patients with essential hypertension (EH) and normal renal function. The plasma levels of fetuin-A, interleukin 6 (IL-6), tumor necrosis factor-α (TNF-α) and the biomarker of oxidative stress 8-iso-PGF2alpha were assayed in samples from 105 untreated EH patients. Carotid IMT measurements were also performed. EH was studied overall and after dividing in EH with IMT ≥ and 0.9 mm (AUC (area under the curve) 0.738, P<0.0001). Our results suggest that in EH, fetuin-A is associated with the IMT independently of oxid…

MaleSettore MED/09 - Medicina Internaalpha-2-HS-GlycoproteinPhysiologyDinoprostKidneyEssential hypertensionmedicine.disease_causeCarotid Intima-Media ThicknessGastroenterologyatherosclerosiRisk FactorsMedicinebiologyArea under the curveMiddle Agedmusculoskeletal systemHypertensioncardiovascular systemRegression AnalysisBiomarker (medicine)FemaleEssential HypertensionCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyRenal functionPredictive Value of TestsInternal medicineInternal MedicineHumanscardiovascular diseasesintima-media thicknessInterleukin 6Settore MED/14 - NefrologiaInterleukin-6Tumor Necrosis Factor-alphabusiness.industrySurrogate endpointAtherosclerosismedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareFetuinfetuin-AOxidative StressCross-Sectional StudiesEndocrinologyinflammationCase-Control Studiesbiology.proteinbusinessBiomarkersOxidative stressHypertension Research
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Bone marrow failure by cytomegalovirus is associated with an in vivo deficiency in the expression of essential stromal hemopoietin genes.

1997

Bone marrow (BM) failure associated with cytomegalovirus (CMV) infection is a feared complication after clinical BM transplantation. Experiments in long-term BM cultures have indicated that BM stromal cells (BMSC) are targets of productive CMV infection, but an in situ infection of BM stroma remained to be documented, and the pathomechanism is open to question. Here we describe a murine in vivo model of lethal CMV aplastic anemia (CMV-AA). The reconstitution of hematopoietic progenitor cells expressing stem cell factor (SCF) receptor was found to be defective in CMV-AA. While murine CMV replication in permissive parenchymal tissues is cytolytic, the hematopoietic cord was found to be a site…

MaleStromal cellImmunologyGene ExpressionStem cell factorBiologyHematopoietic Cell Growth FactorsMicrobiologyMiceBone MarrowVirologyGranulocyte Colony-Stimulating FactormedicineAnimalsRNA MessengerAplastic anemiaMice Inbred BALB CStem Cell FactorInterleukin-6Hematopoietic Cell Growth FactorsBone marrow failureAnemia Aplasticmedicine.diseaseHematopoiesisTransplantationHaematopoiesisProto-Oncogene Proteins c-kitmedicine.anatomical_structureInsect ScienceImmunologyCytomegalovirus InfectionsFemaleBone marrowResearch Article
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Immuno-inflammatory activation in acute cardio-embolic strokes in comparison with other subtypes of ischaemic stroke

2009

Few studies have examined the relationship between inflammatory biomarker blood levels, cardioembolic stroke subtype and neurological deficit. So the aim of our study is to evaluate plasma levels of immuno-inflammatory variables in patients with cardio-embolic acute ischaemic stroke compared to other diagnostic subtypes and to evaluate the relationship between immuno-inflammatory variables, acute neurological deficit and brain infarct volume. One hundred twenty patients with acute ischaemic stroke and 123 controls without a diagnosis of acute ischaemic stroke were evaluated. The type of acute ischaemic stroke was classified according to the TOAST classification. We evaluated plasma levels o…

MaleTOAST Classificationmedicine.medical_specialtySettore MED/09 - Medicina InternaHeart DiseasesEmbolismInterleukin-1betaSeverity of Illness IndexBrain Ischemiacytokines stroke cardioembolicCentral nervous system diseaseBrain ischemiaDisability EvaluationPredictive Value of TestsInternal medicineSeverity of illnessmedicineHumanscardiovascular diseasesStrokeAgedAged 80 and overInterleukin-6Tumor Necrosis Factor-alphaVascular diseasebusiness.industryCerebral infarctionCase-control studyHematologyMiddle Agedmedicine.diseaseSurgeryStrokeRisk Factors inflammation cytokines Stroke Cerebral infarct cerebrovascular disease cerebrovascular accidentCase-Control StudiesLinear ModelsCardiologyFemaleSettore MED/26 - NeurologiaInflammation MediatorsbusinessBiomarkers
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Obesity causes PGC‐1α deficiency in the pancreas leading to marked IL‐6 upregulation via NF‐κB in acute pancreatitis

2019

Obesity is associated with local and systemic complications in acute pancreatitis. PPARγ coactivator 1α (PGC-1α) is a transcriptional coactivator and master regulator of mitochondrial biogenesis that exhibits dysregulation in obese subjects. Our aims were: (1) to study PGC-1α levels in pancreas from lean or obese rats and mice with acute pancreatitis; and (2) to determine the role of PGC-1α in the inflammatory response during acute pancreatitis elucidating the signaling pathways regulated by PGC-1α. Lean and obese Zucker rats and lean and obese C57BL6 mice were used first; subsequently, wild-type and PGC-1α knockout (KO) mice with cerulein-induced pancreatitis were used to assess the inflam…

MaleTaurocholic Acid0301 basic medicinemedicine.medical_specialtyPGC-1αPathology and Forensic Medicine03 medical and health sciencesDownregulation and upregulationInternal medicineAnimalsMedicineObesityPhosphorylationInterleukin 6PancreasCeruletideMice KnockoutIL-6biologyp65Interleukin-6business.industryNF-kappa BTranscription Factor RelAmedicine.diseasePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaRats ZuckerUp-Regulation3. Good healthMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologymedicine.anatomical_structureMitochondrial biogenesisPancreatitisbiology.proteinPancreatitisAcute pancreatitisPPARGC1AbusinessPancreasCeruletideSignal TransductionThe Journal of Pathology
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Time-restricted eating effects on performance, immune function, and body composition in elite cyclists: a randomized controlled trial.

2020

Abstract Background Although there is substantial interest in intermittent fasting as a dietary approach in active individuals, information regarding its effects in elite endurance athletes is currently unavailable. The present parallel randomized trial investigated the effects of a particular intermittent fasting approach, called time-restricted eating (TRE), during 4 weeks of high-level endurance training. Methods Sixteen elite under-23 cyclists were randomly assigned either to a TRE group or a control group (ND). The TRE group consumed 100% of its estimated daily energy needs in an 8-h time window (from 10:00 a.m. to 6:00 p.m.) whilst energy intake in the ND group was distributed in 3 me…

MaleTime FactorsElite cyclists Endurance Immune system Inflammation Intermittent fastingPhysiologyIntermittent fastinglaw.inventionEnduranceLeukocyte Count0302 clinical medicineRandomized controlled trialWeight losslawIntermittent fastingElectric ImpedanceTestosteroneInsulin-Like Growth Factor INutrition and DieteticsFastingCreatinineBody Compositionmedicine.symptomlcsh:RC1200-1245lcsh:Nutrition. Foods and food supplySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieBioelectrical impedance analysisResearch ArticleElite cyclistslcsh:TX341-641030209 endocrinology & metabolismClinical nutritionAthletic Performance03 medical and health sciencesYoung AdultEndurance trainingWeight LossmedicineHumansLymphocyte Countlcsh:Sports medicineInflammationbusiness.industryInterleukin-6Elite cyclists; Endurance; Immune system; Inflammation; Intermittent fasting030229 sport sciencesTransforming Growth Factor alphaGas analyzerBicyclingDietSports Nutritional Physiological PhenomenaImmune systemAthletesBasal metabolic rateBasal MetabolismbusinessEnergy IntakeFood ScienceJournal of the International Society of Sports Nutrition
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The protective role ofOcimum basilicumL. (Basil) against aspirin-induced gastric ulcer in mice: Impact on oxidative stress, inflammation, motor defic…

2018

The gastroprotective effect of Ocimum basilicum L. (Basil) hexane extract (OBHE) in aspirin-induced gastric ulcers in mice and its ameliorative effect on behavioral alterations were determined. Pretreatment with OBHE (100 or 200 mg kg-1) or misoprostol (50 μg kg-1) alleviated the aspirin-induced oxidative stress by significantly decreasing (p < 0.001) gastric ulcer index scores (57, 76 and 79%), gastric TBARS (by 49, 51 and 52%), NO (21, 28 and 29%), H2O2 (24, 42 and 45%), and the serum pro-inflammatory mediator TNF-α (21, 53 and 53%) and IL-6 (29, 30 and 31%), as well as by markedly increasing gastric GSH (41, 61 and 70%), GSH-Px (21, 32 and 34%), GST (33, 63 and 70%), GR (90, 99 and 112%)…

Malefood.ingredientmedicine.drug_classAnxietyMotor ActivityPharmacologyProtective AgentsUlcer indexmedicine.disease_cause030226 pharmacology & pharmacyAnxiolyticMice03 medical and health sciences0302 clinical medicinefoodTBARSAnimalsHumansMedicineStomach UlcerMisoprostolAspirinAspirinBehavior AnimalbiologyInterleukin-6Plant ExtractsTumor Necrosis Factor-alphabusiness.industryBasilicumGeneral MedicineOcimumbiology.organism_classificationPlant LeavesOxidative StressOcimum basilicumFemaleInterleukin-4business030217 neurology & neurosurgeryOxidative stressFood Sciencemedicine.drugFood &amp; Function
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Increased interleukin-6 and C-reactive protein levels after instrumented lumbar spine fusion in older patients

2019

Purpose: Interleukin 6 (IL-6) and the acute phase C-reactive protein (CRP) blood concentrations after lumbar spine fusion may be affected by age. The purpose of this prospective observational study was to assess postoperative serum levels of pro-inflammatory IL-6 and CRP after instrumented lumbar spine fusion surgery. We hypothesized that older patients would have increased levels of IL-6 and CRP after surgery. Methods: IL-6 and high-sensitive CRP biochemical marker levels were measured before instrumented spinal fusion, and postoperatively at 1 and 3 days, 6 weeks, and 3 months. The 49 patients in this sample were divided into two groups: age ≤ 60 years (n = 23) and age > 60 years (n = 26)…

MalefusionGastroenterologyleikkaushoitoDISEASEsurgery0302 clinical medicinePostoperative Complicationslcsh:Orthopedic surgeryOlder patientsMARKERSMedicinePostoperative PeriodProspective Studieskirurgiata315Aged 80 and over030222 orthopedicsLumbar VertebraebiologyAge FactorsMiddle AgedRECOVERYFemaleSpinal Diseasesmedicine.symptommusculoskeletal diseasesAdultmedicine.medical_specialtyLumbar spine fusionInflammationspinelannerankaC-reactive protein03 medical and health sciencesLumbarAGEselkärankaINFLAMMATIONc-reaktiivinen proteiiniInternal medicineINJURYHumansQUALITYInterleukin 6lumbarAgedbusiness.industryinterleukin-6DISABILITYC-reactive protein030229 sport sciences3126 Surgery anesthesiology intensive care radiologylcsh:RD701-811Spinal Fusionbiology.proteinbusinessBiomarkers
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Effects of levosimendan on hemodynamics, local cerebral blood flow, neuronal injury, and neuroinflammation after asphyctic cardiac arrest in rats.

2014

Despite advances in cardiac arrest treatment, high mortality and morbidity rates after successful cardiopulmonary resuscitation are still a major clinical relevant problem. The post cardiac arrest syndrome subsumes myocardial dysfunction, impaired microcirculation, systemic inflammatory response, and neurological impairment. The calcium-sensitizer levosimendan was able to improve myocardial function and initial resuscitation success after experimental cardiac arrest/cardiopulmonary resuscitation. We hypothesized that levosimendan exerts beneficial effects on cerebral blood flow, neuronal injury, neurological outcome, and inflammation 24 hours after experimental cardiac arrest/cardiopulmonar…

Malemedicine.medical_specialtyCardiotonic AgentsHemodynamicsGene ExpressionEnzyme-Linked Immunosorbent AssayCritical Care and Intensive Care MedicineHippocampusRats Sprague-DawleyInternal medicineMedicineAnimalsNeuroinflammationSimendanCerebral CortexNeuronsAnalysis of Variancebusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionHemodynamicsHydrazonesLevosimendanCardiopulmonary ResuscitationHeart ArrestRatsSprague dawleyPyridazinesCerebral blood flowAnesthesiaCerebrovascular CirculationCardiologybusinessmedicine.drugCritical care medicine
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