Search results for "Intestine"

showing 10 items of 390 documents

Tolerance towards resident intestinal flora in mice is abrogated in experimental colitis and restored by treatment with interleukin-10 or antibodies …

1996

There is now increasing evidence that hyperresponsiveness towards intestinal flora is a crucial event in the pathogenesis of inflammatory bowel disease (IBD). In support of this hypothesis, we recently described in humans that tolerance exists towards indigenous intestinal flora but is broken in active IBD lesions. In the present study, we have attempted to transfer this model into mice from different genetic backgrounds (BALB/c, SJL/J, C3H/HeJ). We found that mononuclear cells from spleen, small bowel and large bowel of mice do not proliferate, i.e. are tolerant when exposed to bacterial sonicates derived from autologous intestine (BsA) but do proliferate, i.e. are immune when exposed to b…

ColonImmunologySpleenBiologyLymphocyte ActivationInflammatory bowel diseaseMicrobiologyMicePeyer's PatchesImmune systemCrohn DiseaseSpecies SpecificityImmunityIntestine SmallImmune TolerancemedicineAnimalsHumansImmunologic FactorsImmunology and AllergyColitisMice Inbred BALB CMice Inbred C3HBacteriaAntibodies MonoclonalInterleukinColitismedicine.diseaseInterleukin-12Recombinant ProteinsInterleukin-10RatsSpecific Pathogen-Free OrganismsIntestinesDisease Models AnimalInterleukin 10medicine.anatomical_structureTrinitrobenzenesulfonic AcidImmunologyLeukocytes MononuclearInterleukin 12SpleenEuropean Journal of Immunology
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Midgut microbiota and host immunocompetence underlie Bacillus thuringiensis killing mechanism

2016

Bacillus thuringiensis is a widely used bacterial entomopathogen producing insecticidal toxins, some of which are expressed in insect-resistant transgenic crops. Surprisingly, the killing mechanism of B. thuringiensis remains controversial. In particular, the importance of the septicemia induced by the host midgut microbiota is still debated as a result of the lack of experimental evidence obtained without drastic manipulation of the midgut and its content. Here this key issue is addressed by RNAi-mediated silencing of an immune gene in a lepidopteran host Spodoptera littoralis, leaving the midgut microbiota unaltered. The resulting cellular immunosuppression was characterized by a reduced …

Crops Agricultural0301 basic medicineHemocytesSerratiaBacillus thuringiensisSpodopteraSerratiaMicrobiologyHemolysin Proteins03 medical and health sciencesBacterial ProteinsInsect-pathogen interactionImmunityBacillus thuringiensisAnimalsPest Control Biologicalbioinsecticide | insect-pathogen interactions | insect biocontrol | pore-forming toxins | immunitySpodoptera littoralisRNA Double-StrandedClostridiumImmunosuppression TherapyPore-forming toxinMultidisciplinaryBacillus thuringiensis ToxinsInsect biocontrolbiologyHost (biology)MicrobiotafungiImmunityMidgutBiological Sciencesbiology.organism_classificationImmunity InnateBioinsecticideEndotoxinsIntestines030104 developmental biologyGene Expression RegulationLarvaPore-forming toxinInsect ProteinsRNA InterferenceImmunocompetenceProceedings of the National Academy of Sciences
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Definition of the single integration site of the pathogenicity locus in Clostridium difficile.

1996

We determined the nucleotide sequence 3.8 kb upstream and 5.2 kb downstream of the toxin genes A and B of Clostridium difficile. Nine ORFs were discovered. Based on PCR-directed approaches, two were attributed to the pathogenicity locus (PaLoc). The other seven were found in every C. difficile isolate obtained from the human gastrointestinal tract, respectless of their toxinogenicity. The ORFs cdu1 and cdu2/2' upstream of the PaLoc displayed similarity to repressors of Gram-positive bacteria (cdu1), and to an Na+/H+ antiporter described for Enterococcus hirae (cdu2/2'). Downstream of the locus a putative ABC transporter (cdd2-4) was identified. With a set of three paired primers used in pol…

DNA BacterialSequence analysisBacterial ToxinsMolecular Sequence DataVirulenceLocus (genetics)BiologyEnterotoxinsOpen Reading FramesBacterial ProteinsSpecies SpecificityGeneticsHumansAmino Acid SequenceORFSGeneGeneticsBase SequenceSequence Homology Amino AcidVirulenceClostridioides difficileNucleic acid sequenceGeneral MedicineMolecular biologyIntestinesTerminator (genetics)DNA Transposable ElementsATP-Binding Cassette TransportersMobile genetic elementsGene
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Metatranscriptomic Approach to Analyze the Functional Human Gut Microbiota

2011

The human gut is the natural habitat for a large and dynamic bacterial community that has a great relevance for health. Metagenomics is increasing our knowledge of gene content as well as of functional and genetic variability in this microbiome. However, little is known about the active bacteria and their function(s) in the gastrointestinal tract. We performed a metatranscriptomic study on ten healthy volunteers to elucidate the active members of the gut microbiome and their functionality under conditions of health. First, the microbial cDNAs obtained from each sample were sequenced using 454 technology. The analysis of 16S transcripts showed the phylogenetic structure of the active microbi…

DNA Complementarylcsh:MedicineGastroenterology and HepatologyGut floraPrevotellaceaeMicrobiologyMicrobiologyMicrobial EcologyMicrobial PhysiologyRNA Ribosomal 16SHumansMicrobiomeRNA Messengerlcsh:ScienceGeneBacteroidaceaeBiologyGeneticsMultidisciplinarybiologyBacteriaGene Expression ProfilingLachnospiraceaelcsh:RComputational BiologyGenomicsBiodiversitySequence Analysis DNAbiology.organism_classificationGastrointestinal TractMetagenomicsMedicineSmall IntestineMetagenomelcsh:QMetagenomicsGenome Expression AnalysisRuminococcaceaeResearch ArticlePLoS ONE
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Intestinal helminth communities of the long-finned pilot whale (Globicephala melas) off the Faroe Islands.

1993

SUMMARYThe intestines of 170 long-finned pilot whales, Globicephala melas, caught off the Faroe Islands (N.E. Atlantic) were examined for helminth parasites. Eight species were detected but only 4 occurred in at least 10% of the sample. No core or recurrent group of species were identified and no correlations between abundances of species were significant. Diversity values were far below those reported for other endotherms. Colonization by helminths was random, whales not being readily colonized. These features point to largely unpredictable, isolationist infracommunities, there being little potential for inter-specific interactions. Older hosts tended to harbour more diverse infracommuniti…

DenmarkCetaceaPilot whaleAcanthocephalaHelminthsparasitic diseasesHelminthsAnimalsAtlantic OceanbiologyCommunityEcologyEcologyMarine habitatsWhalesSpecies diversitybiology.organism_classificationGlobicephala melasBiological EvolutionIntestinesInfectious DiseasesCestodaAnimal Science and ZoologyParasitologySpecies richnessTrematodaParasitology
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Congenital secretory diarrhoea caused by activating germline mutations in GUCY2C

2016

Objective Congenital sodium diarrhoea (CSD) refers to a form of secretory diarrhoea with intrauterine onset and high faecal losses of sodium without congenital malformations. The molecular basis for CSD remains unknown. We clinically characterised a cohort of infants with CSD and set out to identify disease-causing mutations by genome-wide genetic testing. Design We performed whole-exome sequencing and chromosomal microarray analyses in 4 unrelated patients, followed by confirmatory Sanger sequencing of the likely disease-causing mutations in patients and in their family members, followed by functional studies. Results We identified novel de novo missense mutations in GUCY2C, the gene encod…

DiarrheaMale0301 basic medicinemedicine.medical_specialtyReceptors PeptideColonGuanylinGuanosine MonophosphateMutation MissenseReceptors EnterotoxinGUANYLATE CYCLASEBiologyCHRONIC DIARRHOEAPathogenesis03 medical and health scienceschemistry.chemical_compoundsymbols.namesakeGermline mutationInternal medicineBACTERIAL ENTEROTOXINSmedicineHumansMissense mutationAbnormalities MultipleGenetic Predisposition to Disease1506Intestinal MucosaCyclic guanosine monophosphateSanger sequencingPAEDIATRIC DIARRHOEASodiumGastroenterologyInfantMolecular Reproduction Development & Genetics (formed by the merger of DBGL and CRBME)Molecular biologyIntestines030104 developmental biologyEndocrinologyIntestinal AbsorptionReceptors Guanylate Cyclase-CoupledchemistryINTESTINAL ION TRANSPORTsymbolsFemaleMetabolism Inborn ErrorsIntracellularUroguanylinGut
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Clostridium difficile heterogeneously impacts intestinal community architecture but drives stable metabolome responses

2015

Clostridium difficile-associated diarrhoea (CDAD) is caused by C. difficile toxins A and B and represents a serious emerging health problem. Yet, its progression and functional consequences are unclear. We hypothesised that C. difficile can drive major measurable metabolic changes in the gut microbiota and that a relationship with the production or absence of toxins may be established. We tested this hypothesis by performing metabolic profiling on the gut microbiota of patients with C. difficile that produced (n=6) or did not produce (n=4) toxins and on non-colonised control patients (n=6), all of whom were experiencing diarrhoea. We report a statistically significant separation (P-value o0…

DiarrheaMaleBacterial ToxinsDiseasePathogenesisGut floraMicrobiologyMicrobiologyFecesClostridiumMetabolomicsRNA Ribosomal 16SmedicineMetabolomeHumansMetabolomicsColitisEcology Evolution Behavior and SystematicsbiologyClostridioides difficileClostridium difficilebiology.organism_classificationmedicine.diseaseColitisIntestinesRNA BacterialDiarrheaClostridium InfectionsMetabolomeFemaleOriginal Articlemedicine.symptomBacterial infection
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Advanced strategy to exploit wine-making waste by manufacturing antioxidant and prebiotic fibre-enriched vesicles for intestinal health.

2020

Grape extract-loaded fibre-enriched vesicles, nutriosomes, were prepared by combining antioxidant extracts obtained from grape pomaces and a prebiotic, soluble fibre (Nutriose®FM06). The nutriosomes were small in size (from ∼140 to 260 nm), homogeneous (polydispersity index < 0.2) and highly negative (∼ −79 mV). The vesicles were highly stable during 12 months of storage at 25 °C. When diluted with warmed (37 °C) acidic medium (pH 1.2) of high ionic strength, the vesicles only displayed an increase of the mean diameter and a low release of the extract, which were dependent on Nutriose concentration. The formulations were highly biocompatible and able to protect intestinal cells (Caco-2) fro…

Dietary FiberAntioxidantmedicine.medical_treatmentWine02 engineering and technologyGut flora01 natural sciencesAntioxidantsMiceColloid and Surface ChemistryPhospholipid vesiclesFood scienceMice Inbred BALB CSoluble fibre010304 chemical physicsbiologyChemistryVesiclefood and beveragesSurfaces and InterfacesGeneral Medicine021001 nanoscience & nanotechnologyGrape pomaceIntestinal cellsIntestinesHomogeneousFemale0210 nano-technologyBiotechnologyPhospholipid vesiclesCell SurvivalSurface PropertiesGut microbiotaIn vivo studiesAntioxidant activity0103 physical sciencesmedicineAnimalsHumansPrebiotic activityPhysical and Theoretical ChemistryParticle SizeWineWaste ProductsPrebioticfungibiology.organism_classificationGastrointestinal MicrobiomeOxidative StressPrebioticsNutriosomesCaco-2 CellsColloids and surfaces. B, Biointerfaces
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Regional Intestinal Drug Permeability and Effects of Permeation Enhancers in Rat

2020

Sufficient colonic absorption is necessary for all systemically acting drugs in dosage forms that release the drug in the large intestine. Preclinically, colonic absorption is often investigated using the rat single-pass intestinal perfusion model. This model can determine intestinal permeability based on luminal drug disappearance, as well as the effect of permeation enhancers on drug permeability. However, it is uncertain how accurate the rat single-pass intestinal perfusion model predicts regional intestinal permeability and absorption in human. There is also a shortage of systematic in vivo investigations of the direct effect of permeation enhancers in the small and large intestine. In …

DrugKetoprofenmedia_common.quotation_subjectlcsh:RS1-441Pharmaceutical ScienceGastroenterology and Hepatology02 engineering and technologyPharmacology030226 pharmacology & pharmacyArticleDosage formlcsh:Pharmacy and materia medicaJejunumPharmaceutical Sciences03 medical and health sciences0302 clinical medicineabsorption-modifying excipientsintestinal perfusionIn vivoGastroenterologimedicineLarge intestineregional intestinal permeabilitymedia_commonIntestinal permeabilityChemistrypermeation enhancersPermeationFarmaceutiska vetenskaper021001 nanoscience & nanotechnologymedicine.diseasepharmaceutical developmentmedicine.anatomical_structureoral peptide delivery0210 nano-technologymedicine.drugPharmaceutics
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N-Glycosylation modification of proteins is an early marker of the enterocytic differentiation process of HT-29 cells

1990

International audience; The human colon cancer cell line HT-29 remains totally undifferentiated when glucose is present in the culture medium (HT-29 Glc+), while the same cells may undergo typical enterocytic differentiation after reaching confluence when grown in glucose-deprived medium (HT-29 Glc-). Recently, we demonstrated a deficiency in the overall N-glycan processing in confluent undifferentiated cells, whereas differentiated cells follow a classical pattern of N-glycosylation. The main changes in N-glycosylation observed in confluent undifferentiated cells may be summarised as follows: 1) the conversion of high mannose into complex glycopeptides is greatly decreased; 2) this decreas…

EmbryologyGlycosylationGrowth phaseCellular differentiationMedicine (miscellaneous)macromolecular substancesBiology03 medical and health sciences0302 clinical medicineN-linked glycosylationPolysaccharides[ CHIM.ORGA ] Chemical Sciences/Organic chemistry[SDV.BDD] Life Sciences [q-bio]/Development BiologyTumor Cells CulturedHumansProcess (anatomy)[SDV.BDLR] Life Sciences [q-bio]/Reproductive Biology030304 developmental biologychemistry.chemical_classification0303 health sciences[CHIM.ORGA]Chemical Sciences/Organic chemistryProteinsCell Differentiation[CHIM.ORGA] Chemical Sciences/Organic chemistryGlycopeptideIntestinescarbohydrates (lipids)Human colon cancer[SDV.AEN] Life Sciences [q-bio]/Food and NutritionGlucoseReproductive MedicineBiochemistrychemistryCell culture030220 oncology & carcinogenesisColonic Neoplasmslipids (amino acids peptides and proteins)Animal Science and ZoologyGlycoproteinMannoseCell DivisionDevelopmental BiologyFood Science
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