Search results for "Intestines"

showing 10 items of 177 documents

Intestinal helminth communities of the long-finned pilot whale (Globicephala melas) off the Faroe Islands.

1993

SUMMARYThe intestines of 170 long-finned pilot whales, Globicephala melas, caught off the Faroe Islands (N.E. Atlantic) were examined for helminth parasites. Eight species were detected but only 4 occurred in at least 10% of the sample. No core or recurrent group of species were identified and no correlations between abundances of species were significant. Diversity values were far below those reported for other endotherms. Colonization by helminths was random, whales not being readily colonized. These features point to largely unpredictable, isolationist infracommunities, there being little potential for inter-specific interactions. Older hosts tended to harbour more diverse infracommuniti…

DenmarkCetaceaPilot whaleAcanthocephalaHelminthsparasitic diseasesHelminthsAnimalsAtlantic OceanbiologyCommunityEcologyEcologyMarine habitatsWhalesSpecies diversitybiology.organism_classificationGlobicephala melasBiological EvolutionIntestinesInfectious DiseasesCestodaAnimal Science and ZoologyParasitologySpecies richnessTrematodaParasitology
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Congenital secretory diarrhoea caused by activating germline mutations in GUCY2C

2016

Objective Congenital sodium diarrhoea (CSD) refers to a form of secretory diarrhoea with intrauterine onset and high faecal losses of sodium without congenital malformations. The molecular basis for CSD remains unknown. We clinically characterised a cohort of infants with CSD and set out to identify disease-causing mutations by genome-wide genetic testing. Design We performed whole-exome sequencing and chromosomal microarray analyses in 4 unrelated patients, followed by confirmatory Sanger sequencing of the likely disease-causing mutations in patients and in their family members, followed by functional studies. Results We identified novel de novo missense mutations in GUCY2C, the gene encod…

DiarrheaMale0301 basic medicinemedicine.medical_specialtyReceptors PeptideColonGuanylinGuanosine MonophosphateMutation MissenseReceptors EnterotoxinGUANYLATE CYCLASEBiologyCHRONIC DIARRHOEAPathogenesis03 medical and health scienceschemistry.chemical_compoundsymbols.namesakeGermline mutationInternal medicineBACTERIAL ENTEROTOXINSmedicineHumansMissense mutationAbnormalities MultipleGenetic Predisposition to Disease1506Intestinal MucosaCyclic guanosine monophosphateSanger sequencingPAEDIATRIC DIARRHOEASodiumGastroenterologyInfantMolecular Reproduction Development & Genetics (formed by the merger of DBGL and CRBME)Molecular biologyIntestines030104 developmental biologyEndocrinologyIntestinal AbsorptionReceptors Guanylate Cyclase-CoupledchemistryINTESTINAL ION TRANSPORTsymbolsFemaleMetabolism Inborn ErrorsIntracellularUroguanylinGut
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Clostridium difficile heterogeneously impacts intestinal community architecture but drives stable metabolome responses

2015

Clostridium difficile-associated diarrhoea (CDAD) is caused by C. difficile toxins A and B and represents a serious emerging health problem. Yet, its progression and functional consequences are unclear. We hypothesised that C. difficile can drive major measurable metabolic changes in the gut microbiota and that a relationship with the production or absence of toxins may be established. We tested this hypothesis by performing metabolic profiling on the gut microbiota of patients with C. difficile that produced (n=6) or did not produce (n=4) toxins and on non-colonised control patients (n=6), all of whom were experiencing diarrhoea. We report a statistically significant separation (P-value o0…

DiarrheaMaleBacterial ToxinsDiseasePathogenesisGut floraMicrobiologyMicrobiologyFecesClostridiumMetabolomicsRNA Ribosomal 16SmedicineMetabolomeHumansMetabolomicsColitisEcology Evolution Behavior and SystematicsbiologyClostridioides difficileClostridium difficilebiology.organism_classificationmedicine.diseaseColitisIntestinesRNA BacterialDiarrheaClostridium InfectionsMetabolomeFemaleOriginal Articlemedicine.symptomBacterial infection
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Advanced strategy to exploit wine-making waste by manufacturing antioxidant and prebiotic fibre-enriched vesicles for intestinal health.

2020

Grape extract-loaded fibre-enriched vesicles, nutriosomes, were prepared by combining antioxidant extracts obtained from grape pomaces and a prebiotic, soluble fibre (Nutriose®FM06). The nutriosomes were small in size (from ∼140 to 260 nm), homogeneous (polydispersity index < 0.2) and highly negative (∼ −79 mV). The vesicles were highly stable during 12 months of storage at 25 °C. When diluted with warmed (37 °C) acidic medium (pH 1.2) of high ionic strength, the vesicles only displayed an increase of the mean diameter and a low release of the extract, which were dependent on Nutriose concentration. The formulations were highly biocompatible and able to protect intestinal cells (Caco-2) fro…

Dietary FiberAntioxidantmedicine.medical_treatmentWine02 engineering and technologyGut flora01 natural sciencesAntioxidantsMiceColloid and Surface ChemistryPhospholipid vesiclesFood scienceMice Inbred BALB CSoluble fibre010304 chemical physicsbiologyChemistryVesiclefood and beveragesSurfaces and InterfacesGeneral Medicine021001 nanoscience & nanotechnologyGrape pomaceIntestinal cellsIntestinesHomogeneousFemale0210 nano-technologyBiotechnologyPhospholipid vesiclesCell SurvivalSurface PropertiesGut microbiotaIn vivo studiesAntioxidant activity0103 physical sciencesmedicineAnimalsHumansPrebiotic activityPhysical and Theoretical ChemistryParticle SizeWineWaste ProductsPrebioticfungibiology.organism_classificationGastrointestinal MicrobiomeOxidative StressPrebioticsNutriosomesCaco-2 CellsColloids and surfaces. B, Biointerfaces
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N-Glycosylation modification of proteins is an early marker of the enterocytic differentiation process of HT-29 cells

1990

International audience; The human colon cancer cell line HT-29 remains totally undifferentiated when glucose is present in the culture medium (HT-29 Glc+), while the same cells may undergo typical enterocytic differentiation after reaching confluence when grown in glucose-deprived medium (HT-29 Glc-). Recently, we demonstrated a deficiency in the overall N-glycan processing in confluent undifferentiated cells, whereas differentiated cells follow a classical pattern of N-glycosylation. The main changes in N-glycosylation observed in confluent undifferentiated cells may be summarised as follows: 1) the conversion of high mannose into complex glycopeptides is greatly decreased; 2) this decreas…

EmbryologyGlycosylationGrowth phaseCellular differentiationMedicine (miscellaneous)macromolecular substancesBiology03 medical and health sciences0302 clinical medicineN-linked glycosylationPolysaccharides[ CHIM.ORGA ] Chemical Sciences/Organic chemistry[SDV.BDD] Life Sciences [q-bio]/Development BiologyTumor Cells CulturedHumansProcess (anatomy)[SDV.BDLR] Life Sciences [q-bio]/Reproductive Biology030304 developmental biologychemistry.chemical_classification0303 health sciences[CHIM.ORGA]Chemical Sciences/Organic chemistryProteinsCell Differentiation[CHIM.ORGA] Chemical Sciences/Organic chemistryGlycopeptideIntestinescarbohydrates (lipids)Human colon cancer[SDV.AEN] Life Sciences [q-bio]/Food and NutritionGlucoseReproductive MedicineBiochemistrychemistryCell culture030220 oncology & carcinogenesisColonic Neoplasmslipids (amino acids peptides and proteins)Animal Science and ZoologyGlycoproteinMannoseCell DivisionDevelopmental BiologyFood Science
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Immunodetection of the microvillous cytoskeleton molecules villin and ezrin in the parasitophorous vacuole wall of Cryptosporidium parvum (Protozoa: …

1999

Microvilli - actin - villin - ezrin - Cryptosporidium parvum The sporozoites and merozoites of the Apicomplexan protozoan Cryptosporidium parvum (C. parvum) invade the apical side of enterocytes and induce the formation of a parasitophorous vacuole which stays in the brush border area and disturbs the distribution of microvilli. The vacuole is separated from the apical cytoplasm of the cell by an electron-dense layer of undetermined composition. In order to characterize the enterocyte cytoskeleton changes that occur during C. parvum invasion and development, we used both confocal immunofluorescence and immunoelectron microscopy to examine at the C.parvum-enterocyte interface the distributio…

Feces/microbiologyIntestines/parasitologyMicrofilament Proteins/ analysisVacuoleddc:616.07Actins/analysisRats Sprague-DawleyFecesMiceEzrinCarrier Proteins/ analysisCryptosporidium/ chemistry/pathogenicity/ultrastructureCytoskeletonMicroscopy ImmunoelectronCytoskeletonMice Inbred BALB CMicroscopy ConfocalbiologyMicrovilliMicrofilament ProteinsCytoskeleton/ chemistryGeneral MedicineCell biologyIntestinesCryptosporidium parvumFemaleVillinHistologyImmunoelectron microscopyVacuoles/ultrastructurePhosphoproteins/ analysisCryptosporidiummacromolecular substancesPathology and Forensic Medicineparasitic diseasesAnimalsApical cytoplasmActinCell Biologybiology.organism_classificationPhosphoproteinsActinsRatsMicrovilli/ chemistryCytoskeletal ProteinsMicroscopy ElectronVacuolesbiology.proteinCarrier ProteinsEuropean journal of cell biology
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Anti-acids lead to immunological and morphological changes in the intestine of BALB/c mice similar to human food allergy

2008

Abstract We have shown that anti-acid medication for treating dyspeptic disorders can block protein digestion and induce a higher risk for food sensitization. This mechanism was confirmed in human and animal studies on the humoral as well as the cellular level. Here we aimed to investigate the outcome of the treatment with the anti-acid drug sucralfate on the intestine in our murine model, assuming that morphological and immunological changes will occur. BALB/c mice were fed codfish extract plus sucralfate. Antibodies were examined in ELISA, RBL assay and Western blot. Quantitative morphological analysis of the intestine was performed by design-based stereology, focussing on epithelium, lam…

Fish ProteinsPathologymedicine.medical_specialtyCD3 ComplexProtein digestionSucralfateBlotting WesternEnzyme-Linked Immunosorbent AssayBiologyToxicologyImmunoglobulin EPathology and Forensic MedicineMiceCecumTh2 CellsmedicineAnimalsHumansMice Inbred BALB CLamina propriaGoblet cellCell BiologyGeneral MedicineAllergensImmunoglobulin EEosinophilMolecular biologyIntestinesSucralfatemedicine.anatomical_structureDuodenumbiology.proteinFemaleAntacidsFood Hypersensitivitymedicine.drugExperimental and Toxicologic Pathology
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In-vitro antioxidant capacity and cytoprotective/cytotoxic effects upon Caco-2 cells of red tilapia (Oreochromis spp.) viscera hydrolysates.

2019

Abstract The antioxidant capacity of red tilapia viscera hydrolysates (RTVH) with different degrees of hydrolysis (DH) as well as their ultrafiltration membrane fractions, were analyzed using different chemical assays. Their protective effects against oxidative stress were evaluated using H2O2-stressed human intestinal differentiated Caco-2. The highest antioxidant capacity was obtained with a DH of 42.5% (RTVH-A) and its

Fish Proteinsfood.ingredient030309 nutrition & dieteticsCell SurvivalProtein HydrolysatesUltrafiltrationmedicine.disease_causeHydrolysateAntioxidants03 medical and health sciencesHydrolysis0404 agricultural biotechnologyfoodFunctional FoodmedicineAnimalsHumansFood science0303 health sciencesbiologyChemistryHydrolysisCell CycleTilapia04 agricultural and veterinary sciencesbiology.organism_classification040401 food scienceGlutathioneIn vitroIntestinesOreochromisOxidative StressVisceraCaco-2Caco-2 CellsReactive Oxygen SpeciesOxidative stressFood ScienceTilapiaFood research international (Ottawa, Ont.)
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Presystemic metabolism and intestinal absorption of antipsoriatic fumaric acid esters.

2003

Psoriasis is a chronic inflammatory skin disease. Its treatment is based on the inhibition of proliferation of epidermal cells and interference in the inflammatory process. A new systemic antipsoriasis drug, which consists of dimethylfumarate and ethylhydrogenfumarate in the form of their calcium, magnesium and zinc salts has been introduced in Europe with successful results. In the present study, a homologous series of mono- and diesters of fumaric acid has been studied with respect to the sites and kinetics of presystemic ester degradation using pancreas extract, intestinal perfusate, intestinal homogenate and liver S9 fraction. In addition, intestinal permeability has been determined usi…

Fumaric acidCell Membrane PermeabilitySwineDimethyl FumaratePharmaceutical ScienceBiological AvailabilityPancreatic ExtractsIntestinal absorptionchemistry.chemical_compoundIntestinal mucosaFumaratesmedicineAnimalsHumansPsoriasisPharmacology (medical)Enzyme InhibitorsIntestinal MucosaCells CulturedPharmacologyIntestinal permeabilityDimethyl fumarateMicrovilliGeneral MedicineMetabolismmedicine.diseasePropranololIntestineschemistryBiochemistryS9 fractionAtenololIntestinal AbsorptionLipophilicityCaco-2 CellsLiver ExtractsBiopharmaceuticsdrug disposition
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Gene transfer approaches for the treatment of inflammatory bowel disease.

2003

The pathogenesis of Crohn's disease and ulcerative colitis, the two major forms of inflammatory bowel disease, involves a complex interplay between certain genetic, environmental and immunological factors. Considerable research progress in the last decade defined key inflammatory pathways in the inflamed gut and identified new potential therapeutic targets. Since the current medical treatment with corticosteroids and anti-inflammatory drugs is often associated with undesired side effects and cannot completely cure IBD, these current advances in our understanding of intestinal pathology may now allow the development of new biologic treatment strategies including gene therapy. In this review,…

Genetic enhancementGenetic VectorsGene ExpressionGene transferDiseaseInflammatory bowel diseaseAdenoviridaePathogenesisCrohn DiseaseIntestinal inflammationGeneticsMedicineAnimalsHumansMolecular BiologyMedical treatmentbusiness.industryBacterial InfectionsGenetic Therapymedicine.diseaseInflammatory Bowel DiseasesUlcerative colitisIntestinesDisease Models AnimalImmunologyMolecular MedicineCytokinesColitis UlcerativeImmunotherapybusinessStem Cell TransplantationGene therapy
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