Search results for "Islet"

showing 10 items of 98 documents

Atorvastatin but not pravastatin impairs mitochondrial function in human pancreatic islets and rat β-cells. Direct effect of oxidative stress

2017

AbstractStatins are a class of drugs widely prescribed as frontline therapy for lowering plasma LDL-cholesterol in cardiovascular risk prevention. Several clinical reports have recently suggested an increased risk of type 2 diabetes associated with chronic use of these drugs. The pathophysiology of this effect remains to be fully elucidated but impaired β-cell function constitutes a potential mechanism. The aim of this study was to explore the effect of a chronic treatment with lipophilic and hydrophilic statins on β-cell function, using human pancreatic islets and rat insulin-secreting INS-1 cells; we particularly focused on the role of mitochondria and oxidative stress. The present study …

0301 basic medicinemedicine.medical_specialtyStatinmedicine.drug_classmedicine.medical_treatmentAtorvastatinPancreatic isletslcsh:MedicineType 2 diabetes030204 cardiovascular system & hematologyMitochondrionPharmacologymedicine.disease_causeArticle03 medical and health sciences0302 clinical medicineInternal medicinemedicinelcsh:ScienceMultidisciplinarybusiness.industryPancreatic isletsInsulinlcsh:RStatinmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurelcsh:Qlipids (amino acids peptides and proteins)Statins; Pancreatic isletsbusinessOxidative stressPravastatinmedicine.drug
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The neuropeptide 26RFa in the human gut and pancreas: potential involvement in glucose homeostasis

2019

Objective Recent studies performed in mice revealed that the neuropeptide 26RFa regulates glucose homeostasis by acting as an incretin and by increasing insulin sensitivity. However, in humans, an association between 26RFa and the regulation of glucose homeostasis is poorly documented. In this study, we have thus investigated in detail the distribution of 26RFa and its receptor, GPR103, in the gut and the pancreas, and determined the response of this peptidergic system to an oral glucose challenge in obese patients. Design and methods Distribution of 26RFa and GPR103 was examined by immunohistochemistry using gut and pancreas tissue sections. Circulating 26RFa was determined using a specif…

0301 basic medicinemedicine.medical_specialtyinsulinobesityEndocrinology Diabetes and Metabolismmedicine.medical_treatment[SDV]Life Sciences [q-bio]Incretinpancréas030209 endocrinology & metabolismlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceintestinGastric glandsInternal medicineInternal MedicineMedicineGlucose homeostasisglucose homeostasisFood and Nutritiongut;pancreas;glucose homeostasis;insulin;incretin;obesitypancreasglucoseComputingMilieux_MISCELLANEOUSinsulinehoméostasielcsh:RC648-665business.industryResearchStomachPancreatic isletsInsulindigestive oral and skin physiologyNeurosciencesmedicine.diseaseincretin[SDV] Life Sciences [q-bio]obésité030104 developmental biologymedicine.anatomical_structureEndocrinologyNeurons and CognitionAlimentation et NutritiongutbusinessPancreas
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Value of Quantitative DNA Analysis in Endocrine Tumors of the Pancreas

1997

The diagnosis of malignancy can be difficult in endocrine tumors of the pancreas. Moreover prognostically relevant factors are not available. The aim of this study was to evaluate retrospectively whether the DNA distribution pattern can differentiate between benign and malignant pancreatic endocrine tumors and secondly whether the DNA content of tumor cells gives prognostic information.Image cytometry of paraffin-embedded tumor material of 42 pancreatic endocrine tumors.In 27 benign endocrine pancreatic tumors (25 insulinomas, 2 benign nonfunctioning endocrine tumors) we could differentiate between 6 diploid, 15 hypotriploid and 6 triploid DNA histograms. In 15 malignant endocrine tumors of…

AdenomaAdultMaleCancer ResearchPathologymedicine.medical_specialtyPancreatic diseaseAdolescentBiologyMalignancychemistry.chemical_compoundmedicineHumansEndocrine systemEndocrine pancreatic tumorsAgedRetrospective StudiesPloidiesfungifood and beveragesDNA NeoplasmGeneral MedicineMiddle AgedAdenoma Islet CellPrognosismedicine.diseasePancreatic Neoplasmsmedicine.anatomical_structureOncologychemistryFemaleInsulinomaPancreasDNAOncology
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Long-term graft function of adult rat and human islets encapsulated in novel alginate-based microcapsules after transplantation in immunocompetent di…

2005

We describe the results of the first study to show that adult rat and human islets can be protected against xenogenic rejection in immunocompetent diabetic mice by encapsulating them in a novel alginate-based microcapsule system with no additional permselective membrane. Nonencapsulated islets lost function within 4–8 days after being transplanted into diabetic Balb/c mice, whereas transplanted encapsulated adult rat or human islets resulted in normoglycemia for >7 months. When rat islet grafts were removed 10 and 36 weeks after transplantation, the mice became immediately hyperglycemic, thus demonstrating the efficacy of the encapsulated islets. The explanted capsules showed only a …

AdultBlood GlucoseMaleendocrine systemmedicine.medical_specialtyTime FactorsRatónAlginatesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentIslets of Langerhans TransplantationCapsulesGraft functionIslets of LangerhansMiceGlucuronic AcidDiabetes mellitusInternal medicineInsulin SecretionInternal MedicineDiabetes MellitusMedicineAnimalsHumansInsulinInsulin secretiongeographyMice Inbred BALB Cgeography.geographical_feature_categorybusiness.industryHexuronic AcidsGraft SurvivalImmunosuppressionDiabetic mousemedicine.diseaseIsletRatsTransplantationEndocrinologybusinessDiabetes
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CD40 activation in human pancreatic islets and ductal cells.

2008

Aims/hypothesis: CD40 expression on non-haematopoietic cells is linked to inflammation. We previously reported that CD40 is expressed on isolated human and non-human primate islets and its activation results in secretion of IL-8, macrophage inflammatory protein 1-beta (MIP-1β) and monocyte chemoattractant protein-1 (MCP-1) through nuclear factor-κB and extracellularly regulated kinases 1/2 pathways. The objective of this study was to identify the pattern of gene expression, and to study viability and functionality affected by CD40-CD40 ligand (CD40L) interaction in human islets. Furthermore, we have studied the CD40-mediated cytokine/chemokine profile in pancreatic ductal cells, as they are…

AdultChemokinemedicine.medical_specialtyDuctal cellsCell SurvivalEndocrinology Diabetes and Metabolismmedicine.medical_treatmentChemokine CXCL1CD40 Chemokines Cytokines Ductal cells Inflammation Insulin Islets of Langerhans Microarray Quantitative RT-PCRCD40 LigandEnzyme-Linked Immunosorbent AssayIslets of LangerhansYoung AdultInternal medicineInternal MedicinemedicineHumansCD40 AntigensMacrophage inflammatory proteinOligonucleotide Array Sequence AnalysisCD40biologySettore BIO/16 - Anatomia UmanaReverse Transcriptase Polymerase Chain ReactionPancreatic isletsPancreatic DuctsMiddle AgedFlow CytometryMolecular biologyCXCL1CXCL2Endocrinologymedicine.anatomical_structureCytokinebiology.proteinCytokinesChemokinesDiabetologia
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Autoimmune Diabetes Recurrence After Pancreas Transplantation: Diagnosis, Management, and Literature Review

2019

[EN] Background: Pancreas transplantation can be a viable treatment option for patients with type 1 diabetes mellitus (T1DM), especially for those who are candidates for kidney transplantation. T1DM may rarely recur after pancreas transplantation, causing the loss of pancreatic graft. The aim of this study was to describe the prevalence of T1DM recurrence after pancreas transplantation in our series. Material/Methods: Eighty-one patients transplanted from 2002 to 2015 were included. Autoantibody testing (GADA and IA-2) was performed before pancreas transplantation and during the follow-up. Results: The series includes 48 males and 33 females, mean age 37.4+5.7 years and mean duration of dia…

AdultGraft RejectionMaleReoperationmedicine.medical_specialtyendocrine system diseasesmedicine.medical_treatment030232 urology & nephrologyAutoimmunity030230 surgeryPancreas transplantationGastroenterologyTECNOLOGIA ELECTRONICA03 medical and health sciences0302 clinical medicineRecurrenceDiabetes mellitusInternal medicinemedicineHumansProspective StudiesKidney transplantationAutoantibodiesOriginal PaperTransplantationType 1 diabetesGlutamate Decarboxylasebusiness.industryPancreatic isletsnutritional and metabolic diseasesImmunosuppressionGeneral Medicinemedicine.diseaseKidney TransplantationTransplantationmedicine.anatomical_structuresurgical procedures operativeDiabetes Mellitus Type 1FemalePancreas TransplantationPancreasbusinessImmunosuppression
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Beyond islet trasplantation in diabetes cell terapy:from embryonic stem cells to transdifferentation of adult cells

2013

Exogenous insulin is, at the moment, the therapy of choice of diabetes, but does not allow tight regulation of glucose leading to long-term complications. Recently, pancreatic islet transplantation to reconstitute insulin-producing cells, has emerged as an alternative promising therapeutic approach. Unfortunately, the number of donor islets is too low compared with the high number of patients needing a transplantation leading to a search for renewable sources of high-quality -cells. This review, summarizes more recent promising approaches to the generation of new -cells from embryonic stem cells for transdifferentiation of adult cells, particularly a critical examination of the seminal work…

AdultIslets of Langerhans TransplantationBiologyCell therapyTherapeutic approachIslet transplantationdiabetes embryonic stem cellstransdifferentationSettore BIO/13 - Biologia ApplicataDiabetes mellitusDiabetes MellitusmedicineHumansEmbryonic Stem CellsTransplantationgeographygeography.geographical_feature_categoryTransdifferentiationCell Differentiationmedicine.diseaseIsletEmbryonic stem cellTransplantationSettore MED/18 - Chirurgia GeneraleImmunologyCancer researchSurgeryPancreatic islet transplantation
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INFLUENCE OF ISLET TRANSPORTATION ON PANCREATIC ISLET ALLOTRANSPLANTATION IN TYPE 1 DIABETIC PATIENTS WITHIN THE SWISS-FRENCH GRAGIL NETWORK

2004

The influence of islet transportation on pancreatic islet allotransplantation in type 1 diabetic patients was evaluated within the GRAGIL network.From December 2001 to April 2003, 16 human pancreatic islet transplants were performed in 9 type 1 diabetic patients with an established kidney graft (functioning for at least 6 months) in four centers of the GRAGIL network. Islet isolation was performed in a core laboratory in Geneva, and the islet preparations were shipped by ambulance to each center for transplantation. One month after transplantation, the efficiency of the graft was assessed according to islet transportation time (ITT): ITT less than 2 hours (group 1, n=5), and ITT greater tha…

AdultMaleOncologyendocrine systemmedicine.medical_specialtyTime FactorsTissue and Organ Procurementendocrine system diseasesTissue and Organ Procurement/methodsmedicine.medical_treatmentIslets of Langerhans TransplantationTransportationHemoglobin A Glycosylated/metabolismInternal medicineImmunopathologymedicineHumansGlycated HemoglobinAutoimmune diseaseTransplantationType 1 diabetesgeographygeography.geographical_feature_categoryddc:617C-Peptidebusiness.industryGraft SurvivalIslets of Langerhans Transplantation/methods/physiologyCreatinine/bloodMiddle Agedmedicine.diseaseIsletTransplantationDiabetes Mellitus Type 1EndocrinologyDiabetes Mellitus Type 1/surgeryCreatinineC-Peptide/bloodFemaleFrancebusinessSwitzerlandAllotransplantationTransplantation
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Prevalence of residual B-cell function related to age at onset and genetic profile in newly diagnosed type I diabetics.

1987

Patients with type I (insulin-dependent) diabetes mellitus maintain B-cell function for a varying period of time after onset. This is commonly held to account for post-initial remission. To estimate residual B-cell function we measured plasma and 24-h urinary C-peptide in 68 type I diabetic patients (age range 4-35 years, within 10-180 days of the onset of symptoms, typed for HLA-A, -B, -C and DR loci. A positive correlation (r = 0.26; p less than 0.05) was found between urinary C-peptide levels and the age of the patient. The analysis of variance of urinary C-peptide values on the basis of the presence or absence of DR3 and DR4 antigens revealed that the DR3-positive patients had reduced e…

AdultMalemedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismUrinary systemLate onsetGastroenterologyExcretionchemistry.chemical_compoundIslets of LangerhansEndocrinologyAntigenInternal medicineDiabetes mellitusInternal MedicinemedicineHumansChildB cellC-PeptideC-peptidebusiness.industryAge FactorsGeneral MedicineHLA-DR Antigensmedicine.diseaseEndocrinologymedicine.anatomical_structureDiabetes Mellitus Type 1chemistryChild PreschoolFemaleAnalysis of variancebusinessActa diabetologica latina
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Immunoregulatory T-lymphocyte subset deficiency in newly diagnosed Type 1 (insulin-dependent) diabetes mellitus

1984

Humoral and cell-mediated disorders in Type 1 (insulin-dependent) diabetes suggest that an imbalance of immunoregulatory T-cell subsets exists. In 23 newly diagnosed (onset less than 3 months) and 21 long-standing Type 1 diabetic patients, T lymphocyte subsets were analyzed using monoclonal antibodies (OKT3, OKT4, OKT8, OKM1). The newly diagnosed patients showed a reduction with a significant difference from healthy controls in total T cells (OKT3+: 58.1 +/- 8.5% versus 70.7 +/- 8.0%), helper/inducer cells (OKT4+: 33.8 +/- 7.0% versus 47.1 +/- 8.3%), suppressor/cytotoxic cells (OKT8+: 18.5 +/- 7.3% versus 32 +/- 6.8%) and monocytes (OKM1+: 11.5 +/- 3.8% versus 19.9 +/- 5.2%) (p less than 0.…

AdultMalemedicine.medical_specialtyAdolescentmedicine.drug_classT-LymphocytesEndocrinology Diabetes and Metabolismchemical and pharmacologic phenomenaNewly diagnosedBiologyMonoclonal antibodymedicine.disease_causeT-Lymphocytes RegulatoryMonocytesAutoimmunityPathogenesisIslets of LangerhansLeukocyte CountDiabetes mellitusInternal medicineInternal MedicinemedicineHumansCytotoxic T cellChildType 1 diabetesImmunologic Deficiency SyndromesAntibodies MonoclonalT-Lymphocytes Helper-InducerT lymphocytemedicine.diseaseDiabetes Mellitus Type 1EndocrinologyFemaleT-Lymphocytes CytotoxicDiabetologia
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