Search results for "L1"

showing 10 items of 831 documents

Nano-Enhanced Cancer Immunotherapy: Immunology Encounters Nanotechnology

2020

Cancer immunotherapy utilizes the immune system to fight cancer and has already moved from the laboratory to clinical application. However, and despite excellent therapeutic outcomes in some hematological and solid cancers, the regular clinical use of cancer immunotherapies reveals major limitations. These include the lack of effective immune therapy options for some cancer types, unresponsiveness to treatment by many patients, evolving therapy resistance, the inaccessible and immunosuppressive nature of the tumor microenvironment (TME), and the risk of potentially life-threatening immune toxicities. Given the potential of nanotechnology to deliver, enhance, and fine-tune cancer immunothera…

0301 basic medicinePD-L1medicine.medical_treatmentimmune checkpoint inhibitorNanotechnologyReviewmacrophage03 medical and health sciencesMice0302 clinical medicineImmune systemDrug Delivery SystemsCancer immunotherapyPD-L1NeoplasmsPD-1MedicineAnimalsHumansNanotechnologytumor microenvironmentTreatment resistanceAdverse effecttoll like receptor (TLR)lcsh:QH301-705.5Tumor microenvironmentbiologybusiness.industryCancerGeneral Medicinemedicine.diseaseCombined Modality TherapyImmune therapy030104 developmental biologylcsh:Biology (General)030220 oncology & carcinogenesissiRNAbiology.proteinCAR T cell therapymyeloid derived suppressor cells (MDSC)Immunotherapybusinessbi-specific antibody therapyCells
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Gene Expression (mRNA) Markers for Differentiating between Malignant and Benign Follicular Thyroid Tumours

2017

Distinguishing between follicular thyroid cancer (FTC) and follicular thyroid adenoma (FTA) constitutes a long-standing diagnostic problem resulting in equivocal histopathological diagnoses. There is therefore a need for additional molecular markers. To identify molecular differences between FTC and FTA, we analyzed the gene expression microarray data of 52 follicular neoplasms. We also performed a meta-analysis involving 14 studies employing high throughput methods (365 follicular neoplasms analyzed). Based on these two analyses, we selected 18 genes differentially expressed between FTA and FTC. We validated them by quantitative real-time polymerase chain reaction (qRT-PCR) in an independe…

0301 basic medicinePathologyMicroarrayThyroid Glandlaw.inventionlawFollicular phaseGene expressionAdenocarcinoma Follicularfollicular thyroid adenoma; follicular thyroid cancer; gene expression; microarray; meta-analysisSpectroscopyPolymerase chain reactionOligonucleotide Array Sequence Analysisfollicular thyroid cancerGeneral MedicineCANCERComputer Science ApplicationsGene Expression Regulation NeoplasticCARCINOMASFUSION ONCOGENEmicroarrayNEOPLASMSmedicine.medical_specialtyMOLECULAR MARKERSASPIRATIONBiologyCatalysisCLASSIFICATIONArticleInorganic Chemistry03 medical and health sciencesNODULESADENOMASmedicineBiomarkers TumorHumansRNA MessengerThyroid NeoplasmsPhysical and Theoretical ChemistryFollicular thyroid cancerMolecular BiologyGeneGene Expression ProfilingOrganic ChemistryThyroid adenomaACVRL1medicine.diseaseMODELmeta-analysis030104 developmental biologyfollicular thyroid adenomaMutationgene expressionInternational Journal of Molecular Sciences
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Unusual presentation of blastic plasmacytoid dendritic cell neoplasm: Pitfalls in other hematolymphoid neoplasms

2020

Abstract Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare CD4+/CD56+ hematological malignancy with aggressive clinical course and poor prognosis. Histologically, BPDCN is characterized by a diffuse, monomorphous infiltration of cutaneous, subcutaneous, and sometimes other tissues such as lymph nodes and bone marrow, by medium-sized neoplastic cells with blastoid morphology. Typically, there is absence of lymphocytic infiltrate. Diagnosis relies on immunophenotypic expression of CD4, CD56, and the more specific markers of plasmacytoid dendritic cells CD123, CD303/BDCA2, and TCL1. We report a case of a 57-year-old man who presented a 4 cm-long solitary, erythemateous lesion on t…

0301 basic medicinePathologymedicine.medical_specialtyMyeloidMYCASXL1BlastoidPathology and Forensic MedicineLymphocytic Infiltrate03 medical and health sciences0302 clinical medicineBlastic plasmacytoid dendritic cell neoplasmT-cells infiltrationlcsh:PathologyMedicineTET2biologybusiness.industryETV6medicine.diseasebiology.organism_classificationLymphomaETV6030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisInterleukin-3 receptorBone marrowbusinessInfiltration (medical)lcsh:RB1-214Human Pathology: Case Reports
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CXCL5, CXCL8, and CXCL10 regulation by bacteria and mechanical forces in periodontium.

2021

Made available in DSpace on 2021-06-25T10:46:01Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-03-01 Objective: The aim of the present study was to evaluate the expressions of CXCL5, CXCL8, and CXCL10 in periodontal cells and tissues in response to microbial signals and/or biomechanical forces. Methods: Human gingival biopsies from inflamed and healthy sites were used to examine the chemokine expressions and protein levels by real-time PCR and immunohistochemistry. The chemokines were also investigated in gingival biopsies from rats submitted to experimental periodontitis and/or tooth movement. Furthermore, chemokine levels were determined in human periodontal fibroblasts stimulated…

0301 basic medicinePeriodontiumChemokineChemokine CXCL5Periodontal LigamentGingiva03 medical and health sciencesGingivitisstomatognathic systemOrthodontic tooth movementmedicineCXCL10AnimalsHumansInterleukin 8Periodontitis610 Medicine & healthPeriodontitisbiologyFusobacterium nucleatumbusiness.industryInterleukin-8General MedicinePeriodontiummedicine.diseasebiology.organism_classificationGingivitisRatsChemokine CXCL10stomatognathic diseases030104 developmental biologyCXCL5Immunologybiology.protein030101 anatomy & morphologyStress MechanicalAnatomyFusobacterium nucleatummedicine.symptombusinessDevelopmental Biology
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Cocaine-induced changes in CX3CL1 and inflammatory signaling pathways in the hippocampus: Association with IL1β

2020

Cocaine induces neuroinflammatory response and interleukin-1 beta (IL1β) is suggested a final effector for many cocaine-induced inflammatory signals. Recently, the chemokine fractalkine (CX3CL1) has been reported to regulate hippocampus-dependent neuroinflammation and synaptic plasticity via CX3C-receptor 1 (CX3CR1), but little is known about the impact of cocaine. This study is mainly focused on the characterization of CX3CL1, IL1β and relevant inflammatory signal transduction pathways in the hippocampus in acute and repeated cocaine-treated male mice. Complementarily, the rewarding properties of cocaine were also assessed in Cx3cr1-knockout (KO) mice using a conditioned place preference (…

0301 basic medicinePharmacologyChemokinemedicine.medical_specialtybiologyChemistryHippocampusCREBConditioned place preference03 medical and health sciencesCellular and Molecular Neuroscience030104 developmental biology0302 clinical medicineEndocrinologyInternal medicineCX3CR1Synaptic plasticitybiology.proteinmedicineCX3CL1030217 neurology & neurosurgeryNeuroinflammationNeuropharmacology
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MYC and MCL1 Cooperatively Promote Chemotherapy-Resistant Breast Cancer Stem Cells via Regulation of Mitochondrial Oxidative Phosphorylation

2017

Summary Most patients with advanced triple-negative breast cancer (TNBC) develop drug resistance. MYC and MCL1 are frequently co-amplified in drug-resistant TNBC after neoadjuvant chemotherapy. Herein, we demonstrate that MYC and MCL1 cooperate in the maintenance of chemotherapy-resistant cancer stem cells (CSCs) in TNBC. MYC and MCL1 increased mitochondrial oxidative phosphorylation (mtOXPHOS) and the generation of reactive oxygen species (ROS), processes involved in maintenance of CSCs. A mutant of MCL1 that cannot localize in mitochondria reduced mtOXPHOS, ROS levels, and drug-resistant CSCs without affecting the anti-apoptotic function of MCL1. Increased levels of ROS, a by-product of a…

0301 basic medicinePhysiologyMice NudeTriple Negative Breast NeoplasmsOxidative phosphorylationTumor initiationMitochondrionBiologyOxidative PhosphorylationArticleProto-Oncogene Proteins c-myc03 medical and health sciencesCancer stem cellCell Line TumorAnimalsHumansMCL1Molecular BiologyTriple-negative breast cancerchemistry.chemical_classificationReactive oxygen speciesCell BiologyMitochondria030104 developmental biologychemistryDrug Resistance NeoplasmNeoplastic Stem CellsCancer researchMyeloid Cell Leukemia Sequence 1 ProteinFemaleStem cellReactive Oxygen SpeciesCell Metabolism
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Prickly Pear By-Product in the Feeding of Livestock Ruminants: Preliminary Investigation

2020

Simple Summary The question of sustainability of animal nutrition has become a popular topic. The gains made by recycling safe, yet otherwise valueless, by-products and wastes from human food and fiber production (green feeds) may lessen the competition between humans and animals for crops and decrease the environmental impact of food production. In this context, prickly pear by-product, which results from Opuntia ficus-indica (L.) Mill. fruits processed for juice extraction, could be an optimum by-product to ruminant feeding. This study evaluates the nutritional characteristics and its stability during storage using potassium metabisulfite as a preservative agent. This preliminary investig…

0301 basic medicinePotassium metabisulfitePreservativeSettore AGR/19 - Zootecnica Specialeengineering.materialArticlestorage03 medical and health scienceschemistry.chemical_compoundAnimal scienceRuminantlcsh:ZoologyBy-productchemical compositionDry matterSettore AGR/18 - Nutrizione E Alimentazione Animalelcsh:QL1-991PEAR030109 nutrition & dieteticslcsh:Veterinary medicineGeneral VeterinarybiologyChemistryPulp (paper)0402 animal and dairy scienceprickly pear by-product04 agricultural and veterinary sciencesbiology.organism_classification040201 dairy & animal scienceengineeringlcsh:SF600-1100Animal Science and ZoologyFermentationAnimals
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The expression and prognostic relevance of programmed cell death protein 1 in tongue squamous cell carcinoma

2020

Background Programmed cell death protein 1 (PD‐1) is an immune checkpoint receptor which plays an important role in a patient´s immune responses to microbial and cancer antigens. It is expressed in tumor infiltrating lymphocytes (TILs) with many different malignancies. The aim of the study was to evaluate PD‐1 expression and its prognostic value in tongue cancer. Methods The data of tongue squamous cell carcinoma (TSCC) patients (N=81) treated in Tampere University Hospital between 1999‐2013 was used. Control data consisted of patients with non‐malignant tongue mucous membrane lesions (N=48). The formalin‐fixed paraffin‐embedded samples were stained immunohistochemically and scanned via dig…

0301 basic medicineProgrammed Cell Death 1 Receptorbiomarkkerittongue squamous cell carcinomaLYMPHOCYTES0302 clinical medicineImmunology and AllergyEPIDEMIOLOGYReceptorDISSECTIONAged 80 and over11832 Microbiology and virologyLIGAND 1 PD-L1Mucous membranemolekyylitGeneral MedicineMiddle AgedCANCER3. Good healthTongue Neoplasmsmedicine.anatomical_structure030220 oncology & carcinogenesisimmunohistochemistryCarcinoma Squamous CellSURVIVALImmunohistochemistrysyöpätauditProgrammed cell death protein 1 (PD-1)Microbiology (medical)AdultAdolescentPathology and Forensic Medicine03 medical and health sciencesYoung AdultImmune systemAntigenTonguePOOR-PROGNOSISmedicineBiomarkers TumorHumansNECKAgedmolecular markerbusiness.industryHUMAN-PAPILLOMAVIRUSCancerennusteetprogrammed cell death protein 1 (PD‐1)medicine.diseaseImmune checkpoint030104 developmental biologyCancer researchT-CELLSprognosis3111 Biomedicinebusiness
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CXCL10 and CCL21 Promote Migration of Pancreatic Cancer Cells Toward Sensory Neurons and Neural Remodeling in Tumors in Mice, Associated With Pain in…

2018

Background & Aims Pancreatic ductal adenocarcinoma (PDAC) is frequently accompanied by excruciating pain, which has been associated with attraction of cancer cells and their invasion of intrapancreatic sensory nerves. Neutralization of the chemokine CCL2 reduced cancer-associated pain in a clinical trial, but there have been no systematic analyses of the highly diverse chemokine families and their receptors in PDAC. Methods We performed an open, unbiased RNA-interference screen of mammalian chemokines in co-cultures of mouse PDAC cells (K8484) and mouse peripheral sensory neurons, and confirmed findings in studies of DT8082 PDAC cells. We studied the effects of chemokines on migration of PD…

0301 basic medicineReceptors CCR7ChemokineReceptors CXCR3Sensory Receptor Cellsendocrine system diseasesC-C chemokine receptor type 7CXCR303 medical and health sciencesChemokine receptor0302 clinical medicineCell MovementCell Line TumorGanglia SpinalPancreatic cancermedicineAnimalsHumansCXCL10AnalgesicsChemokine CCL21Hepatologybiologybusiness.industryGastroenterologyCancer Painmedicine.diseaseAntibodies NeutralizingCoculture Techniquesdigestive system diseasesChemokine CXCL10Mice Inbred C57BLPancreatic Neoplasms030104 developmental biologyCancer cellCancer researchbiology.protein030211 gastroenterology & hepatologybusinessCarcinoma Pancreatic DuctalSignal TransductionCCL21Gastroenterology
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p53 and p53-related mediators PAI-1 and IGFBP-3 are downregulated in peripheral blood mononuclear cells of HIV-patients exposed to non-nucleoside rev…

2019

The improved effectiveness and safety of the combined antiretroviral therapy (cART) has largely diminished mortality and AIDS-defining morbidity of HIV-patients. Nevertheless, chronic age-related diseases in these individuals are more common and their underlying pathogenic mechanisms of these actions seem to involve accelerated aging and enhanced inflammation. The present study explores markers of these processes in a heterogenous Spanish HIV cohort using peripheral blood samples of HIV-patients and matched uninfected controls. We isolated periheral blood mononuclear cells (PBMCs) and i) compared the expression of a panel of 14 genes related to inflammation and senescence in PBMCs of HIV-pa…

0301 basic medicineSenescenceAdultMaleAnti-HIV Agents030106 microbiologyDown-RegulationInflammationHIV InfectionsCCL2Peripheral blood mononuclear cell03 medical and health sciencesVirologyAntiretroviral Therapy Highly ActivePlasminogen Activator Inhibitor 1medicineCXCL10HumansCellular SenescencePharmacologyInflammationbusiness.industryInterleukin-6Interleukin-18virus diseasesMiddle AgedCCL20Chemokine CXCL10030104 developmental biologyInsulin-Like Growth Factor Binding Protein 3ImmunologyLeukocytes MononuclearReverse Transcriptase InhibitorsInterleukin 18Tumor necrosis factor alphaFemalemedicine.symptomTumor Suppressor Protein p53businessAntiviral research
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