Search results for "LEPROSY"

showing 10 items of 22 documents

Association between C′3 phenotypes and various diseases

1972

The distribution of C′3 phenotypes in leprosy, rheumatism, diabetes, hyperlipedemia and hepatitis were studied. There was a significant excess of FF-phenotypes in patients with rheumatic factor while in hepatitis the SS-phenotype was significantly lower and a relatively high frequency of FF was stated. The results are discussed.

Hepatitisbusiness.industrymedicine.diseasePhenotypeSex chromatinDiabetes mellitusImmunologyGeneticsmedicineIn patientLeprosyMetabolic diseasebusinessGenetics (clinical)RheumatismHuman Genetics
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On the Age of Leprosy

2014

Leprosy is a chronic infection of the skin and nerves caused by Mycobacterium leprae and the newly discovered Mycobacterium lepromatosis. Human leprosy has been documented for millennia in ancient cultures. Recent genomic studies of worldwide M. leprae strains have further traced it along global human dispersals during the past ∼ 100,000 years. Because leprosy bacilli are strictly intracellular, we wonder how long humans have been affected by this disease-causing parasite. Based on recently published data on M. leprae genomes, M. lepromatosis discovery, leprosy bacilli evolution, and human evolution, it is most likely that the leprosy bacilli started parasitic evolution in humans or early h…

Immune defenselcsh:Arctic medicine. Tropical medicinelcsh:RC955-962EpidemiologyImmunologyReviewDermatologymedicine.disease_causeMicrobiologyMycobacteriumLeprosymedicineHumansMycobacterium lepraeBiologyPhylogenyMycobacterium lepromatosisClinical GeneticsbiologyHuman evolutionary geneticslcsh:Public aspects of medicinePublic Health Environmental and Occupational Healthlcsh:RA1-1270Genomicsbiology.organism_classificationmedicine.diseaseBiological EvolutionMycobacterium lepraeChronic infectionInfectious DiseasesHuman evolutionImmunologyHost-Pathogen InteractionsMedicineClinical ImmunologyLeprosyPublic HealthMycobacteriumPLoS Neglected Tropical Diseases
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LEPROSY: A CASE REPORT FROM SICILY, ITALY

2009

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LEPROSYSettore MED/26 - Neurologia
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Phenolic-glycolipid-1 and lipoarabinomannan preferentially modulate TCR- and CD28-triggered proximal biochemical events, leading to T-cell unresponsi…

2012

Abstract Background Advanced stages of leprosy show T cell unresponsiveness and lipids of mycobacterial origin are speculated to modulate immune responses in these patients. Present study elucidates the role of phenolicglycolipid (PGL-1) and Mannose-capped lipoarabinomannan (Man-LAM) on TCR- and TCR/CD28- mediated signalling. Results We observed that lipid antigens significantly inhibit proximal early signalling events like Zap-70 phosphorylation and calcium mobilization. Interestingly, these antigens preferentially curtailed TCR-triggered early downstream signalling events like p38 phosphorylation whereas potentiated that of Erk1/2. Further, at later stages inhibition of NFAT binding, IL-2…

LipopolysaccharidesEndocrinology Diabetes and MetabolismT-LymphocytesClinical BiochemistryPGL-1Man-LAMGene ExpressionLymphocyte ActivationJurkat cellsJurkat CellsEndocrinologyT-cell activationIL-2 receptorPhosphorylationExtracellular Signal-Regulated MAP KinasesPromoter Regions Geneticlcsh:RC620-627Protein Kinase CImmunity CellularZAP-70 Protein-Tyrosine KinaseCD28hemic and immune systemsCell biologyMycobacterium lepraelcsh:Nutritional diseases. Deficiency diseasesmedicine.anatomical_structureHost-Pathogen InteractionsProtein BindingMAP Kinase Signaling SystemT cellReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyImmune systemCD28 AntigensLeprosymedicineHumansSecretionCalcium SignalingCell ProliferationBiochemistry medicalAntigens BacterialLipoarabinomannanNFATC Transcription FactorsResearchBiochemistry (medical)T-cell receptorInterleukin-2 Receptor alpha SubunitMycobacteriaGene Expression RegulationAnergyImmunologyLeukocytes MononuclearInterleukin-2GlycolipidsLipids in Health and Disease
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The HLA system and leprosy in Thailand

1978

To investigate immunogenetics of leprosy, 205 leprosy patients (26 with tuberculoid, 57 with borderline-tuberculoid, 21 with borderline, 31 with borderline-lepromatous, and 70 with lepromatous leprosy) have been typed for HLA antigens, and compared with 183 healthy controls from the same region (Northern Thailand). There was no significant difference between the overall group of leprosy patients or the three borderline classes and the controls. The two polar forms, tuberculoid and lepromatous leprosy, however, showed significant associations: HLA-A2 is decreased and HLA-Bw17 is increased in tuberculoid leprosy; HLA-B7 is increased in lepromatous leprosy. When both polar forms are compared w…

MaleLepromatous leprosyRacial GroupsSignificant differenceTuberculoid leprosyImmunogeneticsHuman leukocyte antigenBiologyThailandbacterial infections and mycosesmedicine.diseaseSex FactorsGene FrequencyGenesHLA AntigensLeprosyImmunologyGeneticsmedicineHumansFemaleIn patientLeprosyGenetics (clinical)Human Genetics
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Phenotypical and functional analysis of memory and effector human CD8 T cells specific for mycobacterial antigens

2006

Abstract Mycobacterium tuberculosis infects one-third of the global population and claims two million lives every year. Because memory CD8 T cells exhibit a high heterogeneity in terms of phenotype and functional characteristic, we investigated the frequency, phenotype, and functional properties of Ag85A epitope-specific HLA-A*0201 CD8 T cells in children affected by tuberculosis (TB) before and 4 mo after chemotherapy and healthy contact children. Using Ag85A peptide/HLA-A*0201 pentamer, we found a low frequency of blood peptide-specific CD8 T cells in tuberculous children before therapy, which consistently increased after therapy to levels detected in healthy contacts. Ex vivo analysis of…

MalePore Forming Cytotoxic ProteinsLEPROSYImmunologyEpitopes T-LymphocyteCD8-Positive T-LymphocytesBiologyTuberculinTUBERCULOSISEpitopeImmunophenotypingInterferon-gammaInterleukin 21Immune systemImmunophenotypingAntigenT-Lymphocyte SubsetsHLA-A2 AntigenHumansBACILLE CALMETTE-GUERINImmunology and AllergyCytotoxic T cellLymphocyte CountChildTuberculosis PulmonaryAntigens BacterialMembrane GlycoproteinsIFN-GAMMACOMPLEXHLA-A AntigensPerforinHIGH-FREQUENCIESMycobacterium tuberculosisINTRACELLULAR INFECTIONNatural killer T cellVirologyBOVIS BCGMICEChild PreschoolTuberculosis MeningealImmunologyFemaleImmunologic MemoryCD8RESPONSES
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Leprosy: report of a case with severe peripheral neuropathy

2009

Leprosy (Hansen's disease) is a chronic granulomatous infectious disease, caused by Mycobacterium leprae, with cutaneous and neurological manifestations. Leprosy is very rare in Europe but some cases are reported, especially among people coming from endemic areas. Here, we report a case of Hansen's disease and emphasize the importance of a prompt diagnosis and treatment also in non-endemic areas.

Malemedicine.medical_specialtyNeurologyAdolescentSensory Receptor CellsChronic granulomatousLeprosy neuropathyNeural ConductionDermatologyDiseaseLeprosymedicineHumansMuscle SkeletalSicilyMycobacterium lepraeMotor NeuronsbiologyElectromyographybusiness.industryPeripheral Nervous System DiseasesGeneral Medicinebiology.organism_classificationmedicine.diseaseDermatologySenegalPsychiatry and Mental healthPeripheral neuropathyInfectious disease (medical specialty)ImmunologySettore MED/26 - NeurologiaNeurology (clinical)LeprosyNeurosurgerybusinessNeurological Sciences
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Die Lepra in Riga, ihre Ausbreitung und Bekämpfung: Vortrag, gehalten am 22. April 1911 in der Gesellschaft für kommunale Sozialpolitik in Riga

1911

"Als Manuskript gedruckt für die Mitglieder der Gesellschaft für kommunale Sozialpolitik in Riga."

Mycobacterial diseasesLepraLepra - izplatība un novēršana (Rīga)LeprosyMikobakteriālās slimības:MEDICINE::Microbiology immunology infectious diseases::Infectious diseases [Research Subject Categories]SpitālībaMikobaktēriju infekcijas
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Lepromatous leprosy : a review and case report

2006

Podeu consultar la versió en castellà a http://hdl.handle.net/2445/117327

Mycobacterium lepraelepromatous leprosyLepraMouth diseasesLeprosyUNESCO::CIENCIAS MÉDICAStuberculoid leprosyDiagnòstic diferencialDifferential diagnosisMalalties infecciosesMalalties de la bocaCommunicable diseases:CIENCIAS MÉDICAS [UNESCO]
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Comparative Sequence Analysis ofMycobacterium lepraeand the New Leprosy-CausingMycobacterium lepromatosis

2009

ABSTRACTMycobacterium lepromatosisis a newly discovered leprosy-causing organism. Preliminary phylogenetic analysis of its 16S rRNA gene and a few other gene segments revealed significant divergence fromMycobacterium leprae, a well-known cause of leprosy, that justifies the status ofM. lepromatosisas a new species. In this study we analyzed the sequences of 20 genes and pseudogenes (22,814 nucleotides). Overall, the level of matching of these sequences withM. lepraesequences was 90.9%, which substantiated the species-level difference; the levels of matching for the 16S rRNA genes and 14 protein-encoding genes were 98.0% and 93.1%, respectively, but the level of matching for five pseudogenes…

Nonsynonymous substitutionSequence analysisPseudogeneMolecular Sequence Datamedicine.disease_causePolymerase Chain ReactionMicrobiologyMycobacteriumBacterial ProteinsPhylogeneticsLeprosyRNA Ribosomal 16SmedicineMolecular BiologyMycobacterium lepraePhylogenyGeneticsMycobacterium lepromatosisBase CompositionLikelihood FunctionsbiologyPhylogenetic treeComputational BiologySequence Analysis DNAbiology.organism_classificationMycobacterium lepraePseudogenesMycobacteriumJournal of Bacteriology
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