Search results for "LIPIDS"

showing 10 items of 2228 documents

The role of plasma triglyceride/high-density lipoprotein cholesterol ratio to predict cardiovascular outcomes in chronic kidney disease.

2015

Background Cardiovascular disease (CVD) risk is substantially increased in subjects with chronic kidney disease (CKD). The Triglycerides (TG) to High-Density Lipoprotein Cholesterol (HDL-C) ratio is an indirect measure of insulin resistance and an independent predictor of cardiovascular risk. No study to date has been performed to evaluate whether the TG/HDL-C ratio predicts CVD risk in patients with CKD. Methods A total of 197 patients (age 53 ± 12 years) with CKD Stages 1 to 5, were enrolled in this longitudinal, observational, retrospective study. TG/HDL-C ratio, HOMA-IR indexes, serum asymmetric dimethyl arginine (ADMA), high sensitivity C-reactive protein (CRP), parathyroid hormone (PT…

AdultMalemedicine.medical_specialtyFlow mediated dilatationTriglyceride to HDL-cholesterol ratioEndocrinology Diabetes and MetabolismAsymmetric dimethyl arginineClinical BiochemistryRenal functionClinical nutritionArginineGastroenterologychemistry.chemical_compoundEndocrinologyInsulin resistanceHigh-density lipoproteinInternal medicineChronic kidney diseasemedicineHumansLongitudinal StudiesRenal Insufficiency ChronicSerum AlbuminTriglyceridesAgedRetrospective StudiesBiochemistry medicalbiologybusiness.industryCholesterolResearchBiochemistry (medical)C-reactive proteinCholesterol HDLMiddle Agedmedicine.diseaseEndocrinologyC-Reactive ProteinchemistryCardiovascular DiseasesParathyroid Hormonebiology.proteinlipids (amino acids peptides and proteins)FemalebusinessBiomarkersKidney diseaseLipidologyGlomerular Filtration RateLipids in health and disease
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Association between glucokinase regulatory protein (GCKR) and apolipoprotein A5 (APOA5) gene polymorphisms and triacylglycerol concentrations in fast…

2008

Background: Hypertriglyceridemia is a risk factor for cardiovascular disease. Variation in the apolipoprotein A5 (APOA5) and glucokinase regulatory protein (GCKR) genes has been associated with fasting plasma triacylglycerol. Objective: We investigated the combined effects of the GCKR rs780094C→T, APOA5 −1131T→C, and APOA5 56C→G single nucleotide polymorphisms (SNPs) on fasting triacylglycerol in several independent populations and the response to a high-fat meal and fenofibrate interventions. Design: We used a cross-sectional design to investigate the association with fasting triacylglycerol in 8 populations from America, Asia, and Europe (n = 7730 men and women) and 2 intervention studies…

AdultMalemedicine.medical_specialtyGenotypeGene-Nutrient InteractionsMedicine (miscellaneous)Blood lipidsSingle-nucleotide polymorphismPolymorphism Single NucleotideYoung AdultFenofibrateGene FrequencyRisk FactorsInternal medicineHyperlipidemiamedicineHumansGenetic Predisposition to DiseaseApolipoproteins ATriglyceridesAdaptor Proteins Signal TransducingAgedHypolipidemic AgentsHypertriglyceridemiaNutrition and DieteticsFenofibrateGlucokinase regulatory proteinbiologyGlucokinaseHypertriglyceridemianutritional and metabolic diseasesGenetic VariationFastingMiddle Agedmedicine.diseasePostprandial PeriodDietary FatsPostprandialEndocrinologyCross-Sectional StudiesTreatment OutcomeApolipoprotein A-Vbiology.proteinlipids (amino acids peptides and proteins)Femalemedicine.drugThe American journal of clinical nutrition
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Effect of the -420C/G variant of the resistin gene promoter on metabolic syndrome, obesity, myocardial infarction and kidney dysfunction.

2007

. Objective.  Resistin is an adipokine that has been suggested to be correlated with markers of inflammation and to be predictive of coronary atherosclerosis and type II diabetes in humans. A common single nucleotide polymorphism (SNP) (−420C/G) in the promoter of resistin is associated with increased resistin plasma levels and susceptibility to type II diabetes. The aim of this study was to investigate the association of the -420C/G polymorphism with metabolic syndrome, obesity, myocardial infarction and kidney disease. Design and results.  First we studied 1542 subjects from the PLIC study (a population based cohort). GG carriers showed an higher prevalence of obesity and metabolic syndro…

AdultMalemedicine.medical_specialtyHeart diseaseGenotypeMyocardial InfarctionAdipokineGene ExpressionSingle-nucleotide polymorphismPolymorphism Single NucleotideCohort StudiesInternal medicineInternal MedicinemedicineHumansGenetic Predisposition to DiseaseResistinMyocardial infarctionObesityRNA MessengerPromoter Regions GeneticAgedMetabolic SyndromeFramingham Risk Scorebusiness.industryMiddle Agedmedicine.diseaseLipidsEndocrinologyKidney dysfunction metabolic syndrome myocardial infarction PBMC resistins SNPChronic DiseaseResistinFemaleKidney DiseasesMetabolic syndromebusinessKidney diseaseJournal of internal medicine
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Familial HDL deficiency due to ABCA1 gene mutations with or without other genetic lipoprotein disorders

2004

Mutations in ABCA1 have been shown to be the cause of Tangier disease (TD) and some forms of familial hypoalphalipoproteinemia (HA), two genetic disorders characterized by low plasma HDL levels. Here we report six subjects with low HDL, carrying seven ABCA1 mutations, six of which are previously unreported. Two mutations (R557X and H160FsX173) were predicted to generate short truncated proteins; two mutations (E284K and Y482C) were located in the first extracellular loop and two (R1901S and Q2196H) in the C-terminal cytoplasmic domain of ABCA1. Two subjects found to be compound heterozygotes for ABCA1 mutations did not have overt clinical manifestations of TD. Three subjects, all with prema…

AdultMalemedicine.medical_specialtyHeterozygoteSettore MED/09 - Medicina InternaApolipoprotein BAdolescentPremature coronary artery diseaseTangier diseaseCoronary DiseaseBiologyGene mutationmedicine.disease_causeCompound heterozygosityTangier diseaseInternal medicineGenotypeABCA1 genemedicineHumansChildHypoalphalipoproteinemiaSelection BiasAgedApolipoproteins BGeneticsMutationFamilial defective Apo B (FDB)Apolipoprotein A-ICholesterol HDLnutritional and metabolic diseasesMiddle Agedmedicine.diseaseLipoprotein lipaseTangier disease; Familial HDL deficiency; ABCA1 gene; Familial defective Apo B (FDB); Lipoprotein lipase; Premature coronary artery diseaseEndocrinologyChild PreschoolMutationbiology.proteinlipids (amino acids peptides and proteins)Allelic heterogeneityATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineFamilial HDL deficiencyATP Binding Cassette Transporter 1
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In the Identification of Cardiovascular Risk With the SCORE Model, Could We Recommend Its Calculation Interchangeably With Total Cholesterol or Ather…

2011

The SCORE table indiscriminately recommends the use of total cholesterol (SCORE-TC) or atherogenic index (SCORE-AI) for calculating cardiovascular (CV) risk. We evaluated reliability and agreement between both methods and the clinical implications for the identification of high CV risk. Observational study (n = 8942) in a 40- to 65-year-old population. Spearman’s Rho correlation was 0.987 (P < .001), the agreement intraclass correlation coefficient was 0.671 (IC 95% 0.413–0.796; with Bland–Altman’s method, the average of the differences between models was 0.74. Kappa index was poor, 0.297 (P < .001) and positive specific agreement was 0.31. Discrepancies fitted individuals with high CV risk…

AdultMalemedicine.medical_specialtyIndex (economics)Intraclass correlationConcordancePopulationRisk AssessmentKappa indexCorrelationInternal medicineTotal cholesterolmedicineHumanseducationAgededucation.field_of_studyModels Statisticalbusiness.industryGeneral MedicineMiddle AgedAtherosclerosisLipidsCholesterolCardiovascular DiseasesSpainFemaleObservational studybusinessRevista Española de Cardiología (English Edition)
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Association of selected ABC gene family single nucleotide polymorphisms with postprandial lipoproteins: results from the population-based Hortega stu…

2009

The aim of the study was to determine the influence of twenty single nucleotide polymorphisms (SNPs) of the ABCA1, ABCG1, ABCG5 and ABCG8 genes on the plasmatic concentrations of total cholesterol (TC), HDL and LDL cholesterol (HDLc, LDLc) in the postprandial state with a representative Spanish Caucasian population (1473 individuals, 50.0% women, ages ranging 21-85 years). In men, subjects with the AA genotype of the ABCA1 rs2230806 (R219K) polymorphism were associated with increased plasma LDLc levels, while the ABCA1 haplotype, which included the rs2230806 A allele, was associated with higher TC and LDLc plasma concentrations. In women, significant relationships were found between rs18935…

AdultMalemedicine.medical_specialtyLipoproteinsBlood lipidsSingle-nucleotide polymorphismHyperlipidemiasBiologyPolymorphism Single Nucleotidechemistry.chemical_compoundHigh-density lipoproteinPolymorphism (computer science)Internal medicineGenotypemedicineHumansATP Binding Cassette Transporter Subfamily G Member 5AllelesATP Binding Cassette Transporter Subfamily G Member 1AgedGeneticsAged 80 and overCholesterolHaplotypeATP Binding Cassette Transporter Subfamily G Member 8Cholesterol HDLMiddle AgedAtherosclerosisPostprandial PeriodPostprandialEndocrinologyCholesterolchemistryHaplotypesSpainlipids (amino acids peptides and proteins)ATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineATP Binding Cassette Transporter 1Atherosclerosis
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Resistance Training Induces Antiatherogenic Effects on Metabolomic Pathways

2019

INTRODUCTION Arising evidence suggests that resistance training has the potential to induce beneficial modulation of biomarker profile. To date, however, only immediate responses to resistance training have been investigated using high-throughput metabolomics whereas the effects of chronic resistance training on biomarker profile have not been studied in detail. METHODS A total of 86 recreationally active healthy men without previous systematic resistance training background were allocated into (i) a resistance training (RT) group (n = 68; age, 33 ± 7 yr; body mass index, 28 ± 3 kg·m) and (ii) a non-RT group (n = 18; age, 31 ± 4 yr; body mass index, 27 ± 3 kg·m). Blood samples were collecte…

AdultMalemedicine.medical_specialtyMagnetic Resonance SpectroscopyWeight LiftingApolipoprotein BLipoproteinsPhysical Therapy Sports Therapy and Rehabilitation030204 cardiovascular system & hematologyBody Mass Index03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsInternal medicinemedicineMetabolomeHumansMetabolomicsOrthopedics and Sports MedicineLongitudinal StudiesMuscle StrengthAmino Acids2. Zero hungerbiologymedicine.diagnostic_testCholesterolbusiness.industryFatty AcidsResistance TrainingCholesterol LDL030229 sport sciencesAtherosclerosisLipidsDiet3. Good healthCholesterolEndocrinologychemistryBody CompositionLean body massbiology.proteinBiomarker (medicine)businessLipid profileBody mass indexBiomarkersLipoproteinMedicine &amp; Science in Sports &amp; Exercise
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Blood lipids, homocysteine, stress factors, and vitamins in clinically stable multiple sclerosis patients

2009

Abstract Multiple Sclerosis (MS) patients present a decrease of antioxidants and neuroprotective and immunoregulatory vitamins and an increase of total homocysteine (tHcy), cholesterol (CHL), HDL-cholesterol, and of cellular stress markers, variably associated with the different phases of the disease. We compared the blood levels of uric acid, folic acid, vitamins B12, A, and E, tHcy, CHL, HDL-cholesterol, and triglycerides in forty MS patients during a phase of clinical inactivity with those of eighty healthy controls, matched for age and sex. We found higher levels of tHcy (p = 0.032) and of HDL-cholesterol (p = 0.001) and lower levels of vitamin E (p = 0.001) and the ratio vitamin E/CHL …

AdultMalemedicine.medical_specialtyMultiple SclerosisAdolescentHomocysteineEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryBlood lipidsClinical nutritionBiologyAntioxidantschemistry.chemical_compoundFolic AcidEndocrinologyHigh-density lipoproteinSettore BIO/10 - BiochimicaInternal medicinemedicineHumansShort PaperVitamin B12Homocysteinelcsh:RC620-627TriglyceridesBiochemistry medicalCholesterolVitamin EBiochemistry (medical)VitaminsMiddle AgedLipidsUric Acidlcsh:Nutritional diseases. Deficiency diseasesEndocrinologychemistryCase-Control StudiesUric acidSettore MED/26 - NeurologiaFemalelipids (amino acids peptides and proteins)Lipids in Health and Disease
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Changes in plasma lipids and low-density lipoprotein peak particle size during and after acute myocardial infarction

2002

In summary, LDL peak particle size variations occur very early after AMI, and therefore this parameter, which may be considered a genetically determined risk factor, must be measured not before 2 to 3 months after the acute event.

AdultMalemedicine.medical_specialtyMyocardial Infarctionchemistry.chemical_compoundInternal medicineBlood plasmaPlasma lipidsmedicineHumansMyocardial infarctionLow-Density Lipoprotein Acute Myocardial InfarctionRisk factorAgedAged 80 and overbusiness.industryCholesterolCholesterol HDLCholesterol LDLMiddle Agedmedicine.diseaseLipidsCoronary heart diseaseEndocrinologychemistryLow-density lipoproteinCardiologyParticle sizeCardiology and Cardiovascular MedicinebusinessThe American Journal of Cardiology
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Inverse correlation between plasma oxysterol and LDL-cholesterol levels in hepatitis C virus-infected patients

2012

Background: Hepatitis C virus infection is characterised by enhanced oxidative stress, which can be measured quantitatively by plasma oxysterol concentration. These molecules may affect lipid metabolism through the activation of Liver X Receptors. Hepatitis C virus exploits host lipid metabolism to facilitate its replication and diffusion. In our study we aimed to evaluate and highlight the potential pathogenetic role of oxysterols, 7-ketocholesterol and 7-β-hydroxycholesterol, in hepatitis C virus-related lipid dysmetabolism. Methods: The study was performed in 42 patients with chronic hepatitis C (93% genotype 1b) and 38 non-alcoholic fatty liver disease patients. Plasma oxysterols 7-keto…

AdultMalemedicine.medical_specialtyOxysterolHepatitis C virusHepacivirusIsotope dilutionmedicine.disease_causechronic hepatitis c infection; lipid metabolism; non-alcoholic fatty liver disease; oxidative stressNon-alcoholic Fatty Liver DiseaseInternal medicinemedicinepolycyclic compoundsoxysterol hcv nafldHumansoxysterols sterols cholesterol mass spectrometry metabolomicsLiver X receptorKetocholesterolsHepatologybusiness.industryFatty liverGastroenterologyLipid metabolismHepatitis CCholesterol LDLMiddle Agedmedicine.diseaseHepatitis CChronic hepatitis C infectionHydroxycholesterolsFatty LiverOxidative StressEndocrinologyLipid metabolismBiochemistryMultivariate AnalysisLinear Modelslipids (amino acids peptides and proteins)FemalebusinessOxidative stress
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