Search results for "LIPOPOLYSACCHARIDE"

showing 10 items of 382 documents

The gelatinase MMP-9like is involved in regulation of LPS inflammatory response in Ciona robusta

2019

Matrix metalloproteinases (MMPs) are a family of endopeptidases collectively able to degrade the components of the extracellular matrix (ECM), with important roles in many biological processes, such as embryogenesis, normal tissue remodelling, angiogenesis and wound healing. New views on the function of MMPs reveal that they regulate inflammatory response and therefore might represent an early step in the evolution of the immune system. MMPs can affect the activity of cytokines involved in inflammation including TGF-β and TNF-α. MMPs are widely distributed in all kingdoms of life and have likely evolved from a single-domain protein which underwent successive rounds of duplications. In this …

0301 basic medicineLipopolysaccharidesModels MolecularAngiogenesisSettore BIO/05 - ZoologiaInflammationAquatic ScienceMatrix metalloproteinaseExtracellular matrixCiona robustaMatrix metalloproteinases (MMPs)MMP-2MMP-9PharynxInflammationLPS03 medical and health sciencesDownregulation and upregulationTransforming Growth Factor betamedicineEnvironmental ChemistryGelatinaseAnimalsCiona intestinalisPhylogenyInflammationbiologyTumor Necrosis Factor-alphafungi04 agricultural and veterinary sciencesGeneral Medicinebiology.organism_classificationMatrix MetalloproteinasesCell biologyCiona intestinalis030104 developmental biologyGelatinases040102 fisheries0401 agriculture forestry and fisheriesmedicine.symptomWound healing
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The Ciona intestinalis immune-related galectin genes (CiLgals-a and CiLgals-b) are expressed by the gastric epithelium.

2017

The transcription of two Ciona intestinalis galectin genes (CiLgals-a and CiLgalseb) is uparegulated by LPS in the pharynxis (hemocytes, vessel epithelium, endostilar zones) which is retained the main organ of the immunity. In this ascidian, for the first time we show, by immunohistochemistry and in situ hybridization methods, that these two immune-related genes are expressed in the gastric epithelium of naïve ascidians, whereas the galectins appear to be only contained in the intestine columnar epithelium. In addition, according to previous results on the pharynx, the genes are also expressed and galectins produced by hemocytes scattered in the connective tissue surrounding the gut. The ge…

0301 basic medicineLipopolysaccharidesPathologymedicine.medical_specialtyanimal structuresGalectinsSettore BIO/05 - ZoologiaConnective tissueIn situ hybridizationAquatic Science03 medical and health sciencesDownregulation and upregulationGene expressionotorhinolaryngologic diseasesmedicineGalectin genes expression Ascidians Ciona intestinalis Gastric and intestine epithelia Hemocytes in the connective tissue Immunolocalization In situ hybridizationEnvironmental ChemistryAnimalsCiona intestinalisIntestinal MucosaGeneIn Situ HybridizationGalectin030102 biochemistry & molecular biologybiologyGeneral Medicinebiology.organism_classificationImmunohistochemistryEpitheliumCell biologyCiona intestinalis030104 developmental biologymedicine.anatomical_structurePharynxFishshellfish immunology
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Stress and immune response to bacterial LPS in the sea urchin Paracentrotus lividus (Lamarck, 1816).

2018

The immune system of the sea urchin species Paracentrotus lividus is highly complex and, as yet, poorly understood. P. lividus coelomocytes mediate immune response through phagocytosis and encapsulation of non-self particles, in addition to the production of antimicrobial molecules. Despite this understanding, details of exactly how these processes occur and the mechanisms which drive them are still in need of clarification. In this study, we show how the bacterial lipopolysaccharides (LPS) is able to induce a stress response which increases the levels of the heat shock proteins HSP70 and HSP90 only a few hours after treatment. This study also shows that LPS treatment increases the expressi…

0301 basic medicineLipopolysaccharidesPhagocytosisAntimicrobial peptidesAquatic ScienceParacentrotus lividusAntimicrobical peptide03 medical and health sciencesImmune systemStress PhysiologicalHeat shock proteinbiology.animalEnvironmental ChemistryHSP90AnimalsSea urchinHSP70Heat-Shock ProteinsbiologyEchinoderm04 agricultural and veterinary sciencesGeneral Medicinebiology.organism_classificationHsp90Immunity InnateHsp70Cell biologyThymosin030104 developmental biologyImmune System040102 fisheriesbiology.proteinParacentrotus0401 agriculture forestry and fisheriesCoelomocyteParacentrotus lividuFishshellfish immunology
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The New Structure of Core Oligosaccharide Presented by Proteus penneri 40A and 41 Lipopolysaccharides

2018

The new type of core oligosaccharide in Proteus penneri 40A and 41 lipopolysaccharides has been investigated by 1H and 13C NMR spectroscopy, electrospray ionization mass spectrometry and chemical methods. Core oligosaccharides of both strains were chosen for structural analysis based on the reactivity of LPSs with serum against P. penneri 40A core oligosaccharide–diphtheria toxoid conjugate. Structural analyses revealed that P. penneri 40A and 41 LPSs possess an identical core oligosaccharide.

0301 basic medicineLipopolysaccharidesSpectrometry Mass Electrospray IonizationMagnetic Resonance SpectroscopyStereochemistryElectrospray ionizationOligosaccharidesanti-conjugate serum; core oligosaccharide; lipopolysaccharide; NMR spectroscopy; ESI MS; <i>Proteus penneri</i>Immune seraProteus penneriCatalysisArticleInorganic Chemistrycore oligosaccharidelcsh:Chemistry03 medical and health sciencesStructure-Activity Relationship13c nmr spectroscopyNMR spectroscopyMoleculePhysical and Theoretical ChemistryESI MSMolecular Biologylcsh:QH301-705.5SpectroscopyAntigens Bacterial030102 biochemistry & molecular biologybiologyMolecular StructureChemistryCore oligosaccharideImmune Seraanti-conjugate serumOrganic ChemistrylipopolysaccharideGeneral MedicineNuclear magnetic resonance spectroscopybiology.organism_classificationProteus penneriComputer Science Applicationslcsh:Biology (General)lcsh:QD1-999ConjugateInternational Journal of Molecular Sciences
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Ceftaroline modulates the innate immune and host defense responses of immunocompetent cells exposed to cigarette smoke.

2017

Abstract Background Cigarette smoke, the principal risk factor for chronic obstructive pulmonary disease (COPD), negatively influences the effectiveness of the immune system’s response to a pathogen. The antibiotic ceftaroline exerts immune-modulatory effects in bronchial epithelial cells exposed to cigarette smoke. Aims and methods The present study aims to assess the effects of ceftaroline on TLR2 and TLR4 expression, LPS binding and TNF-α and human beta defensin (HBD2) release in an undifferentiated and PMA-differentiated human monocyte cell line (THP-1) exposed or not to cigarette smoke extracts (CSE). TLR2, TLR4, and LPS binding were assessed by flow cytometry, TNF-α and HBD2 release w…

0301 basic medicineLipopolysaccharidesbeta-DefensinsCell SurvivalCephalosporinLipopolysaccharideToxicologyMonocytes03 medical and health sciencesImmunologic Factor0302 clinical medicineImmune systemCell Line TumorSmokeAnti-Bacterial AgentmedicineHumansImmunologic FactorsInnate immune systemImmunocompetent cellDose-Response Relationship Drugbusiness.industryTumor Necrosis Factor-alphaMonocyteMacrophagesSmokingAntibioticCigarette smokeGeneral MedicineImmunity InnateToll-Like Receptor 2Anti-Bacterial AgentsCephalosporinsHost-Pathogen InteractionToll-Like Receptor 4TLR2030104 developmental biologymedicine.anatomical_structureBeta defensinCell cultureImmunologyHost-Pathogen InteractionsTLR4lipids (amino acids peptides and proteins)Tumor necrosis factor alphabusinessImmunocompetence030215 immunologyToxicology letters
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Macrophage-induced reactive oxygen species promote myometrial contraction and labor-associated mechanisms

2020

AbstractAt labor, the myometrium is infiltrated by a massive influx of macrophages that secrete high levels of pro-inflammatory cytokines inducing the expression of specific labor-associated markers. However, the interactions between myocytes and macrophages and the role of macrophages in the myometrium at labor remain to be elucidated. In this work, we studied the role of myometrium-infiltrated macrophages and their interaction with myocytes in lipopolysaccharide-induced preterm labor. A co-culture model of human primary myometrial cells and macrophages was developed and validated. Collagen lattices were used to evaluate myocyte contraction. Differentiation steps were assessed by (i) phall…

0301 basic medicineLipopolysaccharideslabormacrophage03 medical and health scienceschemistry.chemical_compoundTransactivationUterine Contraction0302 clinical medicineMyocyteHumansoxidative stress[SDV.BDD]Life Sciences [q-bio]/Development BiologyCells Culturedmyocytechemistry.chemical_classificationReactive oxygen speciescell culture030219 obstetrics & reproductive medicinebiologySuperoxideMacrophagesMyometriumGap junctionParturitionCell DifferentiationCell BiologyGeneral MedicineHydrogen PeroxidedifferentiationVinculinCoculture TechniquesCell biology030104 developmental biologyReproductive Medicinechemistrybiology.proteinMyometriumFemaleSignal transductionReactive Oxygen Species
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Beta3 adrenergic receptor stimulation in human macrophages inhibits NADPHoxidase activity and induces catalase expression via PPARγ activation

2017

IF 4.521; International audience; The beta3 adrenergic receptor (β3-AR) stimulation plays a protective role against preterm labor by blocking myometrial contraction, cytokine production, remodeling and apoptosis. We previously demonstrated that macrophage-induced ROS production in the myometrium was a key element leading to the induction of all these labor-associated features. We thus aimed to investigate if the β3-AR could be expressed in human macrophages and could trigger its protective role in the myometrium by directly inhibiting ROS production. Using lipopolysaccharide (LPS)-stimulated myometrial samples and cell co-culture experiments, we demonstrated that β3-AR stimulation inhibits …

0301 basic medicineLipopolysaccharidesmedicine.medical_specialtyLipopolysaccharidePPARγPreterm laborMacrophagemedicine.medical_treatmentPeroxisome proliferator-activated receptorStimulationApoptosisAntioxidants03 medical and health scienceschemistry.chemical_compoundTransactivation0302 clinical medicineInternal medicinemedicineHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMolecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biologychemistry.chemical_classificationNADPH oxidasebiologybeta3 adrenergic receptorMacrophagesMyometriumNADPH OxidasesROSCell BiologyCatalaseCoculture Techniques3. Good healthCell biologyPPAR gamma030104 developmental biologyEndocrinologyCytokinechemistryGene Expression RegulationReceptors Adrenergic beta-3biology.proteinMyometriumFemaleSignal transductionReactive Oxygen Species030217 neurology & neurosurgerySignal Transduction
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Gut Microbiota Restricts NETosis in Acute Mesenteric Ischemia-Reperfusion Injury.

2020

Objective: Recruitment of neutrophils and formation of neutrophil extracellular traps (NETs) contribute to lethality in acute mesenteric infarction. To study the impact of the gut microbiota in acute mesenteric infarction, we used gnotobiotic mouse models to investigate whether gut commensals prime the reactivity of neutrophils towards formation of neutrophil extracellular traps (NETosis). Approach and Results: We applied a mesenteric ischemia-reperfusion (I/R) injury model to germ-free (GF) and colonized C57BL/6J mice. By intravital imaging, we quantified leukocyte adherence and NET formation in I/R-injured mesenteric venules. Colonization with gut microbiota or monocolonization with Esch…

0301 basic medicineMaleExtracellular TrapsMesenteric infarctionLipopolysaccharideNeutrophilsGut floraExtracellular Traps03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAcute mesenteric ischemiaVenulesmedicineCell AdhesionEscherichia coliLeukocytesAnimalsGerm-Free LifeLeukocyte RollingMesenteryCells CulturedMice Knockoutbiologybusiness.industryNeutrophil extracellular trapsbiology.organism_classificationmedicine.diseaseGastrointestinal MicrobiomeMice Inbred C57BLToll-Like Receptor 4Disease Models Animal030104 developmental biologychemistryNeutrophil Infiltration030220 oncology & carcinogenesisMesenteric IschemiaReperfusion InjuryImmunologyHost-Pathogen InteractionsFemaleCardiology and Cardiovascular MedicinebusinessReperfusion injuryBacillus subtilisSignal TransductionArteriosclerosis, thrombosis, and vascular biology
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The pituitary gland prevents shock-associated death by controlling multiple inflammatory mediators

2018

Abstract Bacterial infections cause a major burden of disease worldwide. Sepsis and septic shock are life-threatening complications of infections. The hypothalamic-pituitary-adrenal (HPA) axis initiates the release of endogenous glucocorticoids that modulate the host stress response and acute inflammation during septic shock. It is an ongoing controversial debate, if therapeutic manipulations of the HPA axis could benefit the clinical situation in the context of shock. Here, we have studied Long Evans rats with hypophysectomy followed by endotoxic shock. The shock-associated lethality was substantially higher in hypophysectomized rats as compared to control mice after cranial sham surgery (…

0301 basic medicineMalePituitary glandmedicine.medical_specialtyLipopolysaccharidemedicine.medical_treatmentBiophysicsInflammationBiochemistryArticleSepsis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsRats Long-EvansMolecular BiologyInflammationSeptic shockbusiness.industryCell Biologymedicine.diseaseShock SepticMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureEndocrinologyCytokinechemistry030220 oncology & carcinogenesisShock (circulatory)Pituitary GlandCytokinesTumor necrosis factor alphamedicine.symptomInflammation Mediatorsbusiness
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Klebsiella pneumoniae Lipopolysaccharides Serotype O2afg Induce Poor Inflammatory Immune Responses Ex Vivo

2021

Currently, Klebsiella pneumoniae is a pathogen of clinical relevance due to its plastic ability of acquiring resistance genes to multiple antibiotics. During K. pneumoniae infections, lipopolysaccharides (LPS) play an ambiguous role as they both activate immune responses but can also play a role in immune evasion. The LPS O2a and LPS O2afg serotypes are prevalent in most multidrug resistant K. pneumoniae strains. Thus, we sought to understand if those two particular LPS serotypes were involved in a mechanism of immune evasion. We have extracted LPS (serotypes O1, O2a and O2afg) from K. pneumoniae strains and, using human monocytes ex vivo, we assessed the ability of those LPS antigens to in…

0301 basic medicineMicrobiology (medical)SerotypeChemokineQH301-705.5Klebsiella pneumoniae<i>Klebsiella pneumoniae</i>030106 microbiologyMicrobiologyArticleNF-κBMicrobiology03 medical and health scienceschemistry.chemical_compoundImmune systemAntigenVirologyantimicrobial resistanceBiology (General)Pathogenimmune evasionbiologylipopolysaccharideNF-κBSettore CHIM/06 - Chimica Organicalipopolysaccharidesbiology.organism_classificationKlebsiella pneumoniae030104 developmental biologychemistrynosocomial infectionbiology.proteinlipids (amino acids peptides and proteins)Ex vivoMicroorganisms
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