Search results for "LIPOPROTEIN"

showing 10 items of 982 documents

Hepatic storage and transport of n-3 and n-6 polyunsaturated fatty acids by very-low-density lipoproteins in growing rats fed low- or adequate-protei…

1997

Protein and essential fatty acid (EFA) deficiencies may both occur in chronic malnutrition and have common symptoms. To determine the interactions between dietary protein intake and EFA availability, rats were fed purified diets containing 20% or 2% casein and 5% as one of four fats (sunflower, soybean, coconut, or salmon oil) that differed particularly in their n-6 and n-3 polyunsaturated fatty acids (PUFAs). Protein malnutrition enhanced hepatic triacylglycerol and cholesterol concentrations while decreasing hepatic protein and phospholipid contents and mass and components of very-low-density lipoprotein (VLDL). The ratio of PUFAs to saturated fatty acids (SFAs) was consistently depressed…

Malemedicine.medical_specialtyVery low-density lipoproteinPhospholipidMedicine (miscellaneous)Lipoproteins VLDLBiologydigestive systemchemistry.chemical_compoundDietary Fats UnsaturatedEssential fatty acidFatty Acids Omega-6Protein DeficiencyInternal medicineFatty Acids Omega-3medicineAnimalsFood scienceRats Wistarchemistry.chemical_classificationNutrition and DieteticsCholesterolKwashiorkornutritional and metabolic diseasesfood and beveragesFatty acidmedicine.diseaseDietary FatsRatsApolipoproteinsEndocrinologyLiverchemistryFatty Acids Unsaturatedlipids (amino acids peptides and proteins)Dietary ProteinsLipoproteinPolyunsaturated fatty acidThe American Journal of Clinical Nutrition
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Dietary protein deficiency affects n-3 and n-6 polyunsaturated fatty acids hepatic storage and very low density lipoprotein transport in rats on diff…

1994

Fatty livers and the similarity between the skin lesions in kwashiorkor and those described in experimental essential fatty acid (EFA) deficiency have led to the hypothesis that protein and EFA deficiencies may both occur in chronic malnutrition. The relationship between serum very low density lipoprotein (VLDL) and hepatic lipid composition was studied after 28 d of protein depletion to determine the interactions between dietary protein levels and EFA availability. Rats were fed purified diets containing 20 or 2% casein and 5% fat as either soybean oil rich in EFA, or salmon oil rich in eicosapentaenoic (EPA) and docosahexaenoic (DHA) acids, or hydrogenated coconut, oil poor in EFA. Animal…

Malemedicine.medical_specialtyVery low-density lipoproteinfood.ingredientLow proteinDocosahexaenoic AcidsBiologyLipoproteins VLDLBiochemistrySoybean oilchemistry.chemical_compoundfoodEssential fatty acidInternal medicineCaseinFatty Acids Omega-6Protein DeficiencyFatty Acids Omega-3medicineAnimalsFood scienceRats WistarPhospholipidsTriglycerideschemistry.chemical_classificationFatty Acids EssentialOrganic Chemistryfood and beveragesFatty acidBiological TransportCell BiologyDietary FatsDietRatsEndocrinologychemistryEicosapentaenoic AcidLiverFatty Acids Unsaturatedlipids (amino acids peptides and proteins)Arachidonic acidDietary ProteinsPolyunsaturated fatty acidLipids
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Regulation of lipid flux between liver and adipose tissue during transient hepatic steatosis in carnitine-depleted rats

2007

Rats with carnitine deficiency due to trimethylhydrazinium propionate (mildronate) administered at 80 mg/100 g body weight per day for 10 days developed liver steatosis only upon fasting. This study aimed to determine whether the transient steatosis resulted from triglyceride accumulation due to the amount of fatty acids preserved through impaired fatty acid oxidation and/or from up-regulation of lipid exchange between liver and adipose tissue. In liver, mildronate decreased the carnitine content by approximately 13-fold and, in fasted rats, lowered the palmitate oxidation rate by 50% in the perfused organ, increased 9-fold the triglyceride content, and doubled the hepatic very low density …

Malemedicine.medical_specialtyVery low-density lipoproteintissu adipeuxAdipose tissuerattus rattusBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicinestéatose hépatiqueCarnitineInternal medicinemedicineAnimalsLipolysisCarnitineRats Wistarpathologie animaleMolecular BiologyBeta oxidationlipide030304 developmental biologychemistry.chemical_classification0303 health sciencesTriglycerideFatty AcidsFatty acidCell Biologyfoiemedicine.diseaseLipidsRats[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry Molecular Biology/Biomolecules [q-bio.BM]Fatty LiverLipoproteins LDLLipoprotein LipaseEndocrinologyAdipose TissueGene Expression RegulationLiverchemistryHepatocytesRATSteatosisTriolein030217 neurology & neurosurgerymedicine.drug
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Long-term outcomes after acute myocardial infarction in patients with familial hypercholesterolemia: The French registry of Acute ST-elevation and no…

2020

Patients with familial hypercholesterolemia (FH) are prone to develop acute myocardial infarction (AMI) at a younger age.The aim of the present study was to assess 5-year outcomes after AMI according to the presence of FH in a large multicenter cohort of patients.The French registry of Acute ST-elevation and non-ST-elevation Myocardial Infarction consists of nationwide surveys recruiting patients over a 1- to 2-month period every 5 years. Patients recruited in 2005 and 2010 were followed up to 5 years.Of 5147 patients discharged alive and in whom FH status could be assessed, 2.8% had probable/definite FH, using an adapted Dutch Lipid Clinic score. They were 12 years younger, on average, tha…

Malemedicine.medical_specialty[SDV]Life Sciences [q-bio]Endocrinology Diabetes and MetabolismFamilial hypercholesterolemia030204 cardiovascular system & hematologyCohort StudiesHyperlipoproteinemia Type II03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicineSurveys and QuestionnairesInternal MedicinemedicineHumansIn patient030212 general & internal medicineMyocardial infarctionRegistriesNon-ST Elevated Myocardial InfarctionStrokeComputingMilieux_MISCELLANEOUSAgedNutrition and Dieteticsbusiness.industryST elevationHazard ratioMiddle Agedmedicine.diseasePrognosisConfidence interval3. Good healthCohortST Elevation Myocardial Infarction[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieFemaleFranceHydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessJournal of clinical lipidology
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Lipoprotein lipase-facilitated uptake of LDL is mediated by the LDL receptor

2007

LPL mediates the uptake of lipoproteins into different cell types independent of its catalytic activity. The mechanism of this process and its physiological relevance are not clear. Taking into account the importance of the endothelial barrier for lipoprotein uptake, in vitro studies with primary aortic endothelial cells from wild-type and low density lipoprotein receptor (LDLR)-deficient (LDLR(-/-)) mice were performed. Addition of LPL almost doubled the uptake of LDL into wild-type cells. However, there was virtually no LPL-mediated change of LDL uptake into LDLR(-/-) cells. Upregulation of LDLR by lipoprotein-deficient serum/lovastatin in wild-type cells resulted in a 7-fold increase of …

Malemedicine.medical_specialtyendotheliumQD415-436BreedingBiochemistrylipidschemistry.chemical_compoundMiceEndocrinologyChylomicron remnantInternal medicinemedicineAnimalscardiovascular diseasesMuscle SkeletalCells CulturedLipoprotein lipaseCholesteroldigestive oral and skin physiologyEndothelial Cellsfood and beveragesnutritional and metabolic diseasescholesterolBiological TransportCell BiologyDietary FatsDietLipoproteins LDLMice Inbred C57BLLipoprotein LipaseEndocrinologychemistryBiochemistryReceptors LDLLow-density lipoproteinLDL receptortransportFemaleProteoglycanslipids (amino acids peptides and proteins)Lovastatinatherosclerosislow density lipoproteinmedicine.drugChylomicronLipoproteinJournal of Lipid Research
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No Improvement of High-Density Lipoprotein (HDL) Vasorelaxant Effect Despite Increase in HDL Cholesterol Concentration in Type 2 Diabetic Patients Tr…

2014

Abstract Context: High-density lipoproteins (HDLs) from type 2 diabetic patients are unable to counteract the inhibitory effect of oxidized low-density lipoproteins (ox-LDLs) on vasorelaxation. We hypothesized that glitazones, which improve glycemic control and dyslipidemia, could correct this abnormality. Objectives and Design: We compared the ability of HDL from controls (n = 12) and from type 2 diabetic patients before and after 6 months of treatment with either rosiglitazone (n = 11) or pioglitazone (n = 8) to counteract the inhibitory effect of ox-LDL on vasodilatation of rabbit aorta rings. Results: Rosiglitazone induced a decrease in hemoglobin A1c (7.7% ± 1.1% vs 9.8% ± 1.0%, P = .0…

Malemedicine.medical_specialtymedicine.drug_classEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)BiochemistryRosiglitazonechemistry.chemical_compoundEndocrinologyHigh-density lipoproteinInternal medicineDiabetes mellitusmedicineAnimalsHumansHypoglycemic AgentsThiazolidinedioneAortaAgedDyslipidemiasPioglitazoneCholesterolbusiness.industryCholesterol HDLBiochemistry (medical)Middle Agedmedicine.diseaseLipoproteins LDLVasodilationEndocrinologyDiabetes Mellitus Type 2chemistryFemaleThiazolidinedionesEndothelium VascularRabbitsLipoproteins HDLRosiglitazonebusinessPioglitazoneDyslipidemiamedicine.drugThe Journal of Clinical Endocrinology & Metabolism
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Real-world study: Escalating targeted lipid-lowering treatment with PCSK9-inhibitors and lipoprotein apheresis.

2018

INTRODUCTION Proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibition with monoclonal antibodies has complemented the armamentarium of lipid-lowering therapy (LLT) before the final step of commencing chronic lipoprotein apheresis (LA). Data are scarce on patients who, after escalation of LLT with PCSK9 antibodies, have commenced chronic LA or PCSK9 antibody treatment during ongoing long-term LA. PATIENTS AND METHODS In this study, a cohort of 110 patients with established atherosclerotic cardiovascular disease (ASCVD) due to hypercholesterolemia or concomitant lipoprotein(a)-hyperlipoproteinemia, who received PCSK9 antibodies for the first time during routine care, were consecutivel…

Malemedicine.medical_specialtymedicine.drug_classLipoproteinsHypercholesterolemia030204 cardiovascular system & hematologyMonoclonal antibodyGastroenterology03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansEnzyme InhibitorsAdverse effectbiologybusiness.industryPCSK9PCSK9 InhibitorsAntibodies MonoclonalHematologyGeneral MedicineLipoprotein(a)Cholesterol LDLMiddle AgedAtherosclerosisCombined Modality TherapyLipidsDiscontinuationConcomitantCohortbiology.proteinBlood Component RemovalFemaleAntibodyProprotein Convertase 9business030215 immunologyLipoprotein(a)Journal of clinical apheresis
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Lipoprotein(a) and long-term recurrent infarction after an episode of ST-segment elevation acute myocardial infarction

2020

Background In established ischemic heart disease, the relationship between lipoprotein(a) and new cardiovascular events showed contradictory results. Our aim was to assess the relationship between lipoprotein(a) and very long-term recurrent myocardial infarction (MI) after an index episode of ST-segment elevation acute myocardial infarction (STEMI). Methods We included 435 consecutive STEMI patients discharged from October 2000 to June 2003 in a single teaching center. The relationship between lipoprotein(a) at discharge and recurrent MI was evaluated through negative binomial regression and Cox regression analysis. Results The mean age was 65 years (55-74 years), 25.5% were women, 34.7% we…

Malemedicine.medical_specialtymedicine.medical_treatmentInfarction030204 cardiovascular system & hematologyRate ratio03 medical and health sciences0302 clinical medicineRecurrenceInternal medicineFibrinolysismedicineRisk of mortalityHumansST segment030212 general & internal medicineMyocardial infarctionAgedRetrospective Studiesbiologybusiness.industryIncidenceGeneral MedicineLipoprotein(a)Middle Agedmedicine.diseaseConfidence intervalSpainbiology.proteinCardiologyST Elevation Myocardial InfarctionFemaleCardiology and Cardiovascular MedicinebusinessBiomarkersFollow-Up StudiesForecastingLipoprotein(a)Coronary Artery Disease
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Improvement of fibrinolysis and plasma lipoprotein levels induced by gemfibrozil in hypertriglyceridemia.

1995

A randomized double-blind study was carried out with gemfibrozil (600 mg b.i.d.) vs placebo in 20 patients (twelve males and eight females, age 52 +/- 3 years, BMI 24.2 +/- 0.4) suffering from primary hypertriglyceridemia (Fredrickson's type IV). Each group was treated for a 12 week period with gemfibrozil (n = 10) or placebo (n = 10) patients) in a double-blind fashion. Total cholesterol, HDL-cholesterol (HDL-C) and its subfractions (HDL2-C and HDL3-C), blood glucose, Apolipoproteins A1 and B, fibrinogen, plasminogen, factor VII, t-PA:Ag and PAI activity pre- and post-venous occlusion (VO) were determined. In the gemfibrozil-treated group a significant decrease of total cholesterol and tri…

Malemedicine.medical_specialtymedicine.medical_treatmentLipoproteinsFibrinogenchemistry.chemical_compoundDouble-Blind MethodInternal medicineFibrinolysismedicineGemfibrozilHumansTriglyceridesApolipoproteins BHypertriglyceridemiaTriglycerideApolipoprotein A-Ibusiness.industryCholesterolFibrinolysisHypertriglyceridemiaReverse cholesterol transportCholesterol HDLFibrinogenPlasminogenHematologyGeneral MedicineFactor VIIMiddle Agedmedicine.diseaseEndocrinologyCholesterolchemistryTissue Plasminogen Activatorlipids (amino acids peptides and proteins)FemaleGemfibrozilbusinessLipoproteinmedicine.drugBlood coagulationfibrinolysis : an international journal in haemostasis and thrombosis
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Dextran-sulfate-adsorption of atherosclerotic lipoproteins from whole blood or separated plasma for lipid-apheresis--comparison of performance charac…

2007

For many years dextran sulfate adsorption (DSA) treatment of separated plasma has been an established technology for low-density lipoprotein (LDL)-elimination. Recently a system for the treatment of whole blood based on DSA was introduced into clinical practice. To further characterize DSA treatment of whole blood, the performance characteristics of both DSA modalities were compared at two investigational sites with two alternative LDL apheresis systems being already in routine clinical use. In prospective cross-over design, DSA whole blood treatment was compared with a whole blood polyacrylate LDL adsorption system (DALI) in one study group. DSA for plasma treatment was compared with Lipid…

Malemedicine.medical_specialtymedicine.medical_treatmentUrologyAcrylic ResinsFibrinogenchemistry.chemical_compoundmedicineHumansWhole bloodbusiness.industryCholesterolDextran SulfateBlood Component RemovalFibrinogenHematologyGeneral MedicineCholesterol LDLHemoperfusionAtherosclerosisSurgerybody regionsApheresischemistryLDL apheresisBlood Component RemovalFemalebusinessFiltrationmedicine.drugLipoproteinJournal of clinical apheresis
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