Search results for "Lipid Metabolism"

showing 10 items of 359 documents

Paraoxonase-1 deficiency is associated with severe liver steatosis in mice fed a high-fat high-cholesterol diet: a metabolomic approach.

2013

Oxidative stress is a determinant of liver steatosis and the progression to more severe forms of disease. The present study investigated the effect of paraoxonase-1 (PON1) deficiency on histological alterations and hepatic metabolism in mice fed a high-fat high-cholesterol diet. We performed nontargeted metabolomics on liver tissues from 8 male PON1-deficient mice and 8 wild-type animals fed a high-fat, high-cholesterol diet for 22 weeks. We also measured 8-oxo-20-deoxyguanosine, reduced and oxidized glutathione, malondialdehyde, 8-isoprostanes and protein carbonyl concentrations. Results indicated lipid droplets in 14.5% of the hepatocytes of wild-type mice and in 83.3% of the PON1-deficie…

Malemedicine.medical_specialtyBiologymedicine.disease_causeDiet High-FatBiochemistrychemistry.chemical_compoundMiceLipid dropletInternal medicineNonalcoholic fatty liver diseasemedicineAnimalsMetabolomicsAmino AcidsOrotic AcidTriglycerideLipid peroxideAryldialkylphosphataseGeneral Chemistrymedicine.diseaseMalondialdehydeLipid MetabolismGlutathioneFatty LiverMice Inbred C57BLOxidative StressEndocrinologyCholesterolGlucosechemistryLiverUrea cycleSteatosisOxidative stressBiomarkersJournal of proteome research
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Long-chain 3-hydroxyacyl-CoA dehydrogenase deficiency: a severe fatty acid oxidation disorder

1994

3-Hydroxyacyl-CoA dehydrogenase deficiency is a newly recognised fatty acid oxidation disorder with a usually fatal outcome. We present a further patient who presented with hypoketotic hypoglycaemia, hepatopathy, secondary carnitine deficiency and increased plasma long-chain acylcarnitines. 3-Hydroxydicarboxylic aciduria was present and the diagnosis confirmed in cultured skin fibroblasts. Our patient is compared with those reported in the literature with respect to clinical symptoms, differential diagnosis and possible therapeutic regimens.

Malemedicine.medical_specialtyCardiomyopathyLipid Metabolism Inborn ErrorsFatal OutcomeInternal medicineCarnitineMedicineHumansBeta oxidationchemistry.chemical_classificationCultured skinbusiness.industryLiver DiseasesInfant Newborn3-Hydroxyacyl CoA DehydrogenasesMitochondrial MyopathiesClinical Enzyme Testsmedicine.diseaseDehydrogenase deficiencyHypoglycemiaEnzymeEndocrinologychemistrySecondary carnitine deficiencyPediatrics Perinatology and Child HealthDifferential diagnosisbusinessCardiomyopathiesLong-Chain-3-Hydroxyacyl-CoA DehydrogenaseEuropean journal of pediatrics
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Effects of dietary treatment of rats with eicosapentaenoic acid or docosahexaenoic acid on hepatic lipid metabolism

1998

(1) Effects of dietary treatment of male albino rats with eicosapentaenoic acid (EPA) or docosahexaenoic acid on hepatic mitochondrial lipid metabolism have been investigated. (2) Mitochondria isolated from rats given these treatments were shown to have increased ability to respire on acyl-CoA esters in the presence of malonate. This effect was expressed with most of the long-chain acyl-CoA esters used as substrates. When malonate in the incubations was replaced with malate, mitochondria from treated animals were found to exhibit diminished rates of respiration on polyunsaturated acyl-CoA esters, in particular linolenoyl-, eicosapentaenoyl- and docosahexaenoyl-CoA. This phenomenon could not…

Malemedicine.medical_specialtyDocosahexaenoic AcidsMitochondria LiverBiochemistryLipid peroxidationchemistry.chemical_compoundFish OilsInternal medicinemedicineAnimalsRats WistarMolecular Biologychemistry.chemical_classificationFatty acidLipid metabolismCell BiologyMetabolismLipid MetabolismEicosapentaenoic acidDietRatsEndocrinologyEicosapentaenoic AcidLiverchemistryBiochemistryDocosahexaenoic acidCarnitine biosynthesislipids (amino acids peptides and proteins)Lipid PeroxidationCarnitine palmitoyltransferase IResearch ArticleBiochemical Journal
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Obesogen effect of bisphenol S alters mRNA expression and DNA methylation profiling in male mouse liver

2020

International audience; Environmental pollution is increasingly considered an important factor involved in the obesity incidence. Endocrine disruptors (EDs) are important actors in the concept of DOHaD (Developmental Origins of Health and Disease), where epigenetic mechanisms play crucial roles. Bisphenol A (BPA), a monomer used in the manufacture of plastics and resins is one of the most studied obesogenic endocrine disruptor. Bisphenol S (BPS), a BPA substitute, has the same obesogenic properties, acting at low doses with a sex-specific effect following perinatal exposure. Since the liver is a major organ in regulating body lipid homeostasis, we investigated gene expression and DNA methyl…

Malemedicine.medical_specialtyEnvironmental EngineeringHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]0208 environmental biotechnologyEnvironmental pollution02 engineering and technologyEndocrine Disruptors010501 environmental sciencesBiology01 natural sciencesEpigenesis GeneticPhenolsPregnancyInternal medicineToxicity TestsGene expressionmedicineAnimalsHumansEnvironmental ChemistryObesityRNA MessengerSulfonesEpigeneticsGene0105 earth and related environmental sciencesDose-Response Relationship DrugPublic Health Environmental and Occupational HealthGeneral MedicineGeneral ChemistryDNA MethylationLipid MetabolismPollution3. Good health020801 environmental engineeringMice Inbred C57BLEndocrinologyGene Expression RegulationLiverEndocrine disruptorPrenatal Exposure Delayed EffectsDNA methylationFemaleObesogenhormones hormone substitutes and hormone antagonistsDNA hypomethylation
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Changes in lipid metabolism and antioxidant defense status in spontaneously hypertensive rats and Wistar rats fed a diet enriched with fructose and s…

2005

Abstract Objective Larger doses of fructose and saturated fat have been associated with oxidative stress and development of hypertension. The effects of modest amounts of fructose and saturated fatty acids on oxidative stress are unknown. Methods To increase knowledge on this question, 10-wk-old spontaneously hypertensive rats and Wistar rats were fed for 8 wk with a control diet or an experimental diet enriched with fructose (18%) and saturated fatty acids (11%; FS diet). The total antioxidant status of organs and red blood cells was assayed by monitoring the rate of free radical-induced red blood cell hemolysis. Sensitivity of very low-density lipoprotein and low-density lipoprotein (VLDL…

Malemedicine.medical_specialtyErythrocytesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentSaturated fatGlutathione reductaseBlood PressureFructoseBiologyHemolysisThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationRandom Allocationchemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsRats WistarHypertriglyceridemiachemistry.chemical_classificationNutrition and DieteticsGlutathione peroxidaseFatty AcidsLipid MetabolismAscorbic acidRatsOxidative StressEndocrinologychemistrySaturated fatty acidlipids (amino acids peptides and proteins)Lipid PeroxidationPolyunsaturated fatty acidNutrition
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A genetic variant of the atrial natriuretic peptide gene is associated with cardiometabolic protection in the general community

2011

Objectives We sought to define the cardiometabolic phenotype associated with rs5068, a genetic variant of the atrial natriuretic peptide (ANP) gene. Background The ANP and B-type natriuretic peptide play an important role in cardiorenal homeostasis but also exert metabolic actions. Methods We genotyped 1,608 randomly selected residents from Olmsted County, Minnesota. Subjects were well-characterized. Results Genotype frequencies were: AA 89.9%, AG 9.7%, and GG 0.4%; all subsequent analyses were AA versus AG+GG. The G allele was associated with increased plasma levels of N-terminal pro-atrial natriuretic peptide (p = 0.002), after adjustment for age and sex. The minor allele was also associa…

Malemedicine.medical_specialtyLinkage disequilibriumSettore MED/09 - Medicina InternaGenotypemedicine.drug_classPopulationBlood PressureArticlemetabolic syndromeBody Mass IndexRandom AllocationAtrial natriuretic peptideInternal medicineatrial natriuretic peptidelipid metabolismNatriuretic Peptide BrainHomeostasimedicineNatriuretic peptideHomeostasisHumansAlleleeducationAllelesAgedAlleleeducation.field_of_studycardiometabolic diseasebusiness.industryGenetic VariationDNAMiddle Agedmedicine.diseaseEchocardiography DopplerGenotype frequencyProtein Structure TertiaryMinor allele frequencyEndocrinologyPhenotypeFemaleMetabolic syndromebusinessCardiology and Cardiovascular Medicinenatriuretic peptidesAtrial Natriuretic FactorHuman
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Silibinin modulates lipid homeostasis and inhibits nuclear factor kappa B activation in experimental nonalcoholic steatohepatitis.

2012

Nonalcoholic steatohepatitis (NASH) is associated with increased liver-related mortality. Disturbances in hepatic lipid homeostasis trigger oxidative stress and inflammation (ie, lipotoxicity), leading to the progression of NASH. This study aimed at identifying whether silibinin may influence the molecular events of lipotoxicity in a mouse model of NASH. Eight-week-old db/db mice were fed a methionine-choline deficient (MCD) diet for 4 weeks and treated daily with silibinin (20 mg/kg intraperitoneally) or vehicle. Liver expression and enzyme activity of stearoyl-CoA desaturase-1 and acyl-CoA oxidase, and expression of liver fatty acid-binding protein were assessed. Hepatic levels of reactiv…

Malemedicine.medical_specialtyMice ObeseSilibininmedicine.disease_causeAntioxidantsTranslational Research BiomedicalMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineNonalcoholic fatty liver diseasemedicineTBARSAnimalsHomeostasisNASH MCD Silibinin lipotoxicity.Reactive nitrogen speciesLiver injurychemistry.chemical_classificationReactive oxygen speciesAnti-Inflammatory Agents Non-SteroidalBiochemistry (medical)NF-kappa BPublic Health Environmental and Occupational HealthGeneral MedicineLipid Metabolismmedicine.diseaseCholine DeficiencyFatty LiverDisease Models AnimalOxidative StressEndocrinologyLiverchemistryLipotoxicitySilybinOxidative stressSilymarin
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Fatty Acid Profiles in Demented Patients: Identification of Hexacosanoic Acid (C26:0) as a Blood Lipid Biomarker of Dementia

2014

Background: Several lipid metabolism alterations have been described in the brain and plasma of Alzheimer’s disease (AD) patients, suggesting a relation between lipid metabolism alteration and dementia. 15 16 Objective: We attempted to identify blood fatty acids as biomarkers of dementia. 17 Methods: Fatty acid profiles were established using gas chromatography with or without mass spectrometry on matched plasma and red blood cells (RBCs) of demented patients diagnosed with AD, vascular dementia, or other dementia, and compared with a control group of elderly individuals. The severity of dementia was evaluated with the Mini-Mental State Examination test. 18 19 20 Results: Fatty acid analysi…

Malemedicine.medical_specialtyPathologyChromatography GasErythrocytesBlood lipidsNeuropsychological TestsBiologyMass Spectrometrychemistry.chemical_compoundInternal medicinemedicineHumansDementiaVascular dementiaAgedAged 80 and overchemistry.chemical_classificationFatty acid metabolismGeneral NeuroscienceFatty AcidsFatty acidLipid metabolismGeneral MedicineMiddle AgedPeroxisomemedicine.diseasePsychiatry and Mental healthClinical PsychologyEndocrinologyROC CurvechemistryBiomarker (medicine)DementiaFemaleGeriatrics and GerontologyMental Status ScheduleJournal of Alzheimer's Disease
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Mass Concentration of Plasma Phospholipid Transfer Protein in Normolipidemic, Type IIa Hyperlipidemic, Type IIb Hyperlipidemic, and Non–Insulin-Depen…

1999

Abstract —Mean plasma phospholipid transfer protein (PLTP) concentrations were measured for the first time by using a competitive enzyme-linked immunosorbent assay. PLTP mass levels and phospholipid transfer activity values, which were significantly correlated among normolipidemic plasma samples ( r =0.787, P <0.0001), did not differ between normolipidemic subjects (3.95±1.04 mg/L and 575±81 nmol · mL −1 · h −1 , respectively; n=30), type IIa hyperlipidemic patients (4.06±0.84 mg/L and 571±43 nmol · mL −1 · h −1 , respectively; n=36), and type IIb hyperlipidemic patients (3.90±0.79 mg/L and 575±48 nmol · mL −1 · h −1 , respectively; n=33). No significant correlations with plasma lipid p…

Malemedicine.medical_specialtyPhospholipidEnzyme-Linked Immunosorbent AssayHyperlipidemiasCarbohydrate metabolismchemistry.chemical_compoundReference ValuesPhospholipid transfer proteinInternal medicineDiabetes mellitusCholesterylester transfer proteinBlood plasmamedicineHumansPhospholipid Transfer ProteinsGlycoproteinsbiologyChemistryImmune SeraOsmolar ConcentrationMembrane ProteinsLipid metabolismmedicine.diseaseLipidsCholesterol Ester Transfer ProteinsType iibEndocrinologyDiabetes Mellitus Type 2biology.proteinFemaleCarrier ProteinsCardiology and Cardiovascular MedicineArteriosclerosis, Thrombosis, and Vascular Biology
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Short-term exposure to sublethal tebuconazole induces physiological impairment in male zebrafish (Danio rerio).

2010

Abstract The aim of the present study was to assess the physiological response of male zebrafish Danio rerio to the fungicide tebuconazole and recovery in fungicide-free water. Acute toxicity tests were carried out and the median lethal concentration (LC 50 ) from 24 to 96 h was calculated. The fish were exposed to a sublethal fungicide concentration of 230 μg/L for 7 or 14 days and allowed to recover for 7 or 14 more days, respectively. Whole-body levels of vitellogenins, triglycerides, cholesterol, glucose, lactate and proteins as well as the activities γ-glutamil transpeptidase (γ-GT), alanin aminotransferase (AlAT), alkaline phosphatase (AP) and lactate dehydrogenase (LDH) were assayed;…

Malemedicine.medical_specialtyTime FactorsHealth Toxicology and MutagenesisBiologyLethal Dose 50chemistry.chemical_compoundVitellogeninVitellogeninsFish physiologyInternal medicineLactate dehydrogenasemedicineToxicity Tests AcuteAnimalsZebrafishTebuconazoleBody WeightPublic Health Environmental and Occupational HealthGeneral MedicineEnvironmental ExposureRecovery of FunctionTriazolesLipid MetabolismPollutionAcute toxicityEnzymesFungicides IndustrialEndocrinologyGlucosechemistryToxicitybiology.proteinLactatesAlkaline phosphataseVitellogeninsBiomarkersEcotoxicology and environmental safety
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