Search results for "Locke"

showing 10 items of 332 documents

Gastric relaxation induced by apigenin and quercetin: Analysis of the mechanism of action

2009

Abstract Aims Recently, flavonoids have been shown to cause murine gastric relaxation. In the present study we examined the mechanism of action underlying gastric relaxation induced by apigenin and quercetin in isolated mouse stomach. Main methods The mechanical activity from the whole stomach was detected as changes in the endoluminal pressure and the response to increasing concentrations of both flavonoids were tested before and after different pharmacological treatments. Key findings Apigenin and quercetin-induced a concentration-dependent gastric relaxation, apigenin being more potent than quercetin. The responses were unaffected by 2′5′dideoxyadenosine, an inhibitor of adenylate cyclas…

Malemedicine.medical_specialtyCarbacholNifedipineMuscle Relaxationchemistry.chemical_elementCalcium antagonistIn Vitro TechniquesMuscarinic AgonistsCalciumPharmacologySettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyPotassium ChlorideMicechemistry.chemical_compoundSmooth muscleInternal medicineCyclic AMPmedicineAnimalsheterocyclic compoundsApigeninGeneral Pharmacology Toxicology and PharmaceuticsCyclic GMPCyclic nucleotide phosphodiesteraseChemistryRyanodine receptorStomachMuscle SmoothGeneral MedicineCalcium Channel BlockersMice Inbred C57BLEndocrinologyMechanism of actionGastric toneApigeninFlavonoidCalciumCarbacholQuercetinmedicine.symptomQuercetinIntracellularMuscle Contractionmedicine.drugLife Sciences
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Effects of papaverine on human isolated bladder muscle

1990

Papaverine is a non-specific smooth muscle relaxant and is thought to act at a site beyond the receptor sites on the cell membrane. In this study the relaxing properties of papaverine were tested in isolated muscle strips from the human bladder dome. In carbachol-induced contractions papaverine, even in high concentrations of 10(-4) mol/l had virtually no effects on peak tension generation, whereas the fading was accelerated and the steady state tension at 30 min. was reduced by about 54%. In contrast, high potassium-induced contractions were relaxed by papaverine in a concentration-dependent way; a concentration of papaverine of 10(-4) mol/l produced full relaxation. These findings might p…

Malemedicine.medical_specialtyCarbacholUrologyPotassiumUrinary Bladderchemistry.chemical_elementIn Vitro TechniquesCalciumCell membranePapaverineInternal medicinemedicineHumansReceptorPapaverineCalcium channelMuscle SmoothMiddle AgedCalcium Channel Blockersmedicine.anatomical_structureEndocrinologychemistryPotassiumCarbacholFemalemedicine.symptomMuscle Contractionmedicine.drugMuscle contractionUrological Research
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Tumour-like presentation of atypical posterior reversible encephalopathy syndrome with prominent brainstem involvement

2020

Typical posterior reversible encephalopathy syndrome (PRES) is a clinical-neuroradiological entity characterised by bilateral white matter oedema, which is usually symmetrical and totally reversible in 2–3 weeks. A 46-year-old man presented with a persistent headache and visual blurring in the right eye. On admission, the clinical examination revealed minimal unsteadiness of gait and elevated blood pressure. A brain MRI showed a hyperintense signal on T2-weighted sequences in the whole brainstem, extended to the spinal cord (C2–C6), the left insula and the right cerebellum. When his blood pressure was controlled, his symptoms gradually improved. The follow-up MRI scan at 3 weeks revealed a …

Malemedicine.medical_specialtyCerebellumNeurologyNifedipinePhysical examination030218 nuclear medicine & medical imagingWhite matterDiagnosis Differential03 medical and health sciences0302 clinical medicineRamiprilmedicineHumansAntihypertensive AgentsUnusual Presentation of More Common Disease/Injuryneuroimagingmedicine.diagnostic_testbusiness.industryneurologyBrain stem/cerebellumDoxazosinPosterior reversible encephalopathy syndromeGeneral MedicineMiddle AgedSpinal cordmedicine.diseaseCalcium Channel BlockersMagnetic Resonance ImagingWhite Matterradiologymedicine.anatomical_structureSettore MED/26 - NeurologiaRadiologyBrainstemPosterior Leukoencephalopathy SyndromeDifferential diagnosisbusinessneuro-oncology030217 neurology & neurosurgeryBrain Stem
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CXCR2 blockade impairs angiotensin II-induced CC chemokine synthesis and mononuclear leukocyte infiltration.

2007

Objective—Angiotensin II (Ang-II) and mononuclear leukocytes are involved in atherosclerosis. This study reports the inhibition of Ang-II–induced mononuclear cell recruitment by CXCR2 antagonism and the mechanisms involved.Methods and Results—Ang-II (1 nmol/L, i.p. in rats) induced CXC and CC chemokines, followed by neutrophil and mononuclear cell recruitment. Administration of the CXCR2 antagonist, SB-517785-M, inhibited the infiltration of both neutrophils (98%) and mononuclear cells (60%). SB-517785-M had no effect on the increase in CXC chemokine levels but reduced MCP-1, RANTES, and MIP-1α release by 66%, 63%, and 80%, respectively. Intravital microscopy showed that pretreatment with S…

Malemedicine.medical_specialtyChemokineCXCR3Peripheral blood mononuclear cellLosartanReceptors Interleukin-8BRats Sprague-DawleyChemokine receptorInternal medicinemedicineCell AdhesionCCL17AnimalsHumansCXC chemokine receptorsSplanchnic CirculationChemokine CCL7Chemokine CCL4Chemokine CCL5Cells CulturedChemokine CCL2Chemokine CCL3InflammationbiologyAngiotensin IIMicrocirculationEndothelial CellsMacrophage Inflammatory ProteinsAtherosclerosisAngiotensin IIMonocyte Chemoattractant ProteinsRatsMononuclear cell infiltrationChemotaxis LeukocyteEndocrinologyNeutrophil Infiltrationbiology.proteinLeukocytes MononuclearCardiology and Cardiovascular MedicineAngiotensin II Type 1 Receptor BlockersArteriosclerosis, thrombosis, and vascular biology
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Differentiation of calcium antagonists with respect to their effects in normal and skinned taenia caeci preparations

1987

Abstract In taenia preparations, depolarized by a K+-rich medium, Ca2+ caused contraction and cinnarizine (0.4–100 μM), trifluoperazine (2–100 μM) and verapamil (0.02–10 μM) caused concentration-dependent antagonism of Ca2+, displacing the Ca2+ log concentration-effect curve to the right and depressing the maximal response. Equieffective (IC75) antispasmogenic concentrations were selected. The antispasmogenic effects of verapamil were readily offset by removing the drug from the bathing fluid but those of the other drugs were not. The calcium antagonists (antispasmogenic IC75) were then tested for spasmolytic activity in tissues generating tension in response to the EC80 of Ca2+. Verapamil …

Malemedicine.medical_specialtyCinnarizineContraction (grammar)Guinea PigsPharmaceutical Sciencechemistry.chemical_elementTrifluoperazineIn Vitro TechniquesCalciumCinnarizineGuinea pigCalmodulinInternal medicinemedicineAnimalsPharmacologyDose-Response Relationship DrugbiologyParasympatholyticsCalcium Channel Blockersbiology.organism_classificationTrifluoperazineIntestinesEndocrinologyVerapamilchemistryTaeniaVerapamilCalciumFemaleIntracellularMuscle Contractionmedicine.drugJournal of Pharmacy and Pharmacology
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Effects of Ca2+ channel antagonists in guinea-pig normal and skinned gall bladder.

1993

CaCl2 (0.01-50 mM, in K(+)-depolarized tissues), KCl (0.1-100 mM) and acetylcholine (1 nM-10 mM) produced concentration-dependent contractions of guinea-pig isolated gall bladder. Nifedipine (1-100 microM), verapamil (1-100 microM), diltiazem (1-100 microM), cinnarizine (1-100 microM), and flunarizine (1-100 microM) each produced a concentration-related inhibition of the log concentration-effect curve for CaCl2. The rank order of potencies of these antagonists, measured as the IC50 against Ca2+ (50 mM)-induced contraction of depolarized gall bladder, was diltiazem (0.25 microM)or = verapamil (0.8 microM) approximately nifedipine (1.2 microM)cinnarizine (25 microM) approximately flunarizine …

Malemedicine.medical_specialtyContraction (grammar)CinnarizineGuinea PigsIn Vitro TechniquesPotassium ChlorideCalcium ChlorideNifedipineInternal medicinemedicineAnimalsDiltiazemFlunarizinePharmacologyChemistryCell MembraneGallbladderMuscle SmoothCalcium Channel BlockersAcetylcholineEndocrinologyVerapamilFemalemedicine.symptomAcetylcholinemedicine.drugMuscle contractionMuscle ContractionEuropean journal of pharmacology
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Calcium dependence of the contraction produced by endothelin (ET-1) in isolated guinea-pig trachea.

1990

Endothelin (ET-1, 1 pM to 0.1 microM) produced a concentration-dependent contraction of isolated guinea-pig trachea. BAY K 8644 (1 microM) did not significantly alter the concentration-response curve for ET-1. Incubation with nicardipine (10 microM) partly inhibited responses to low concentrations (10 pM to 1 nM) of ET-1 while verapamil (10 microM) and diltiazem (10 microM) were ineffective. La3+ (10 microM) and Cd2+ (10 microM) preferentially depressed the responses evoked by high concentrations (30 nM-0.1 microM) of ET-1 without affecting the responses evoked by low concentrations of the peptide. Incubation in Ca2(+)-free (with EDTA, 1 mM) medium resulted in suppression of the responses e…

Malemedicine.medical_specialtyContraction (grammar)NicardipineGuinea PigsIndomethacinchemistry.chemical_elementCalciumBiologyIn Vitro TechniquesPotassium ChlorideGuinea pigInternal medicinemedicineExtracellularAnimalsDiltiazemPharmacologyEndothelinsMuscle Smooth3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterCalcium Channel BlockersTracheaEndocrinologychemistryVerapamilCalciumEndothelin receptormedicine.drugMuscle ContractionEuropean journal of pharmacology
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U-46619-induced potentiation of noradrenergic constriction in the human saphenous vein: antagonism by thromboxane receptor blockade.

2001

Objective: We investigated the potentiating effect of U-46619, a synthetic analogue of thromboxane A2 (TXA2), on the adrenergic responses in human saphenous vein. Methods: Saphenous vein rings were obtained from 35 patients undergoing coronary artery bypass surgery. The rings were suspended in organ bath chambers for isometric recording of tension. Results: U-46619 (10−10–3×10−7 mol/l) produced concentration-dependent and endothelium-independent contractile responses. U-46619 (10−10 mol/l) potentiated the contractions elicited by electrical stimulation and potassium chloride, and produced leftward shifts of the concentration–response curve for noradrenaline. The TXA2 receptor antagonist SQ-…

Malemedicine.medical_specialtyDihydropyridinesNifedipinePhysiologymedicine.drug_classThromboxaneReceptors ThromboxaneAdrenergicIn Vitro TechniquesPotassium ChlorideThromboxane receptorThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2Physiology (medical)Internal medicinemedicineHumansVasoconstrictor AgentsSaphenous VeinAgedVoltage-dependent calcium channelDose-Response Relationship DrugChemistryCalcium channelDihydropyridineDrug SynergismMiddle AgedReceptor antagonistCalcium Channel BlockersElectric StimulationStimulation ChemicalEndocrinology15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidFemaleEndothelium VascularCardiology and Cardiovascular Medicinemedicine.drugCardiovascular research
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Elevation of plasma viscosity induces sustained NO-mediated dilation in the hamster cremaster microcirculation in vivo

1997

We studied whether a flow-independent increase of luminal wall shear stress (WSS) could dilate hamster arterioles in vivo and which endothelial mediators are potentially involved. To this end the plasma viscosity was elevated by exchanging blood for dextran-erythrocyte solution thereby augmenting WSS. Diameters of small and large arterioles as well as red blood cell velocities were measured before and after exchange of blood for solutions of identical haematocrit containing either high- (HMWD) or low-molecular weight dextran (LMWD). The potential role of endothelial autacoids was investigated by local application of the NO-synthase inhibitor NG-nitro-L-arginine (L-NNA), the inhibitor of cyc…

Malemedicine.medical_specialtyEndotheliumPhysiologyClinical BiochemistryPlasma SubstitutesHamsterGenitalia MaleNitric OxideMicrocirculationPlasmachemistry.chemical_compoundIn vivoCricetinaePhysiology (medical)Internal medicinePotassium Channel BlockersmedicineAnimalsBlood TransfusionCyclooxygenase InhibitorsMesocricetusMusclesDextransAnatomyBlood ViscosityMolecular WeightVasodilationArteriolesRed blood cellDextranmedicine.anatomical_structureEndocrinologychemistrycardiovascular systemDilation (morphology)Stress MechanicalNitric Oxide SynthaseErythrocyte TransfusionAutacoidcirculatory and respiratory physiologyPfl�gers Archiv European Journal of Physiology
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Effects of calcium channel blockers on gastric emptying and acid secretion of the rat in vivo.

1986

Abstract Experiments were designed to evaluate the effects of three calcium channel blockers (verapamil, diltiazem and cinnarizine) on gastric emptying and secretion in the rat. Pretreatment with the calcium blockers delayed gastric emptying of phenol red in a dose-dependent manner. Verapamil was the most effective of the agents tested. Verapamil and diltiazem inhibited gastric acid secretion in the pylorus-ligated rat without affecting pepsin output. Cinnarizine was ineffective in this model. When the perfused lumen of the anaesthetized rat was used, verapamil was found to inhibit responses to carbachol or histamine more than those to pentagastrin. Further, we found a greater sensitivity t…

Malemedicine.medical_specialtyGastric motilitychemistry.chemical_elementBlood PressureCalciumBiologyGastric AcidInternal medicinemedicineAnimalsAnesthesiaDiltiazemPylorusPharmacologyGastric emptyingDose-Response Relationship DrugCalcium channeldigestive oral and skin physiologyRats Inbred StrainsCalcium Channel BlockersRatsPentagastrinPerfusionEndocrinologychemistryGastric Emptyingcardiovascular systemGastric acidVerapamilFemalemedicine.drugResearch ArticleBritish journal of pharmacology
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