Search results for "Locke"

showing 10 items of 332 documents

Chronic pharmacological treatment in takotsubo cardiomyopathy.

2008

Abstract Background Takotsubo cardiomyopathy is a disorder that has been appreciated only recently. In most of reported cases, this syndrome mimes an acute myocardial infarction. Till this moment no data are available from literature about the treatment in the acute phase of this disease. Aim of the study In our multicentric experience we have retrospectively looked at the benefits of a treatment with ACE-inhibitors, beta-blockers, Aspirin and calcium channels blockers, started until the early phases of the disease and continued for 30 days, in 36 patients affected by Takotsubo cardiomyopathy. We chose as endpoint of the study the efficacy of the used drug in improving left ventricular myoc…

Malemedicine.medical_specialtyHeart diseaseAdrenergic beta-AntagonistsCardiomyopathyAngiotensin-Converting Enzyme InhibitorsTakotsubo CardiomyopathyInternal medicineMedicineHumansMyocardial infarctionAgedRetrospective StudiesAspirinEjection fractionAspirinbusiness.industryRetrospective cohort studymedicine.diseaseCalcium Channel BlockersSurgeryTreatment OutcomeACE inhibitorCardiologyFemaleDifferential diagnosisCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Evidence for the neuronal origin of immunoreactive interleukin-1 beta released by rat hypothalamic explants.

1996

In this study, we have investigated the release of immunoreactive interleukin-1 beta (irIL-1 beta) from the rat hypothalamus in vitro. It was found that (1) tissue explants release sizable amounts of irIL-1 beta (ranging from 0.43 to 0.52 pg/mg of wet tissue) in 20 min incubations; (2) basal release in significantly increased by depolarization induced with 56 mM KCl; (3) K(+)-induced irIL-1 beta release is inhibited by the specific blocker of N-type calcium channels, omega-conotoxin, and by verapamil, but not by nifedipine; (4) K(+)-induced release is also inhibited by the Na+ channel blockers tetrodotoxin and lidocaine; (5) irIL-1 beta release is significantly increased by noradrenalin; su…

Malemedicine.medical_specialtyHypothalamusRadioimmunoassaychemistry.chemical_elementPropranololCalciumIn Vitro TechniquesPotassium ChlorideNorepinephrinePhentolamineNifedipineInternal medicinemedicineAnimalsChannel blockerRats WistarNeuronsVoltage-dependent calcium channelGeneral NeuroscienceDepolarizationCalcium Channel BlockersRatsElectrophysiologyEndocrinologychemistryVerapamilmedicine.drugInterleukin-1Sodium Channel BlockersNeuroscience letters
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A homeostatic mechanism counteracting K+-evoked choline release in adult brain

2002

Choline (Ch) is an essential nutrient as the biosynthetic precursor of acetylcholine (ACh) and phospholipids. Under resting conditions, the intracellular accumulation of Ch (above 10-fold), which is positively charged, is governed by the membrane potential and follows the Nernst equation. Accordingly, in synaptosomes from adult rats during depolarization, we observed a linear relationship between release of free cytoplasmic Ch and KCl concentration (2.7-120 mm). The K(+) -evoked Ch release was Ca(2+) -independent and did not originate from ACh or phospholipid hydrolysis. In superfused brain slices of adult rats, however, a K(+) -induced Ch efflux was absent. Also, under in vivo conditions, …

Malemedicine.medical_specialtyMicrodialysisMicrodialysisIn Vitro TechniquesHippocampusBiochemistryCholineCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicinePotassium Channel BlockersmedicineExtracellularAnimalsHomeostasisCholine4-AminopyridineRats WistarNeurotransmitterBrain ChemistrySynaptosomeMembrane potentialDose-Response Relationship DrugBrainBiological TransportDepolarizationHemicholinium 3RatsEndocrinologychemistryPotassiumExtracellular SpaceAcetylcholineSynaptosomesmedicine.drugJournal of Neurochemistry
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Effects of antidepressants in adrenergic neurotransmission of human vas deferens

2000

Objectives. To evaluate the effects of sertraline, fluoxetine, and amitriptyline on the contractile responses of the human vas deferens muscle elicited by norepinephrine, electrical field stimulation, and KCl, because the therapeutic action of antidepressants may be accompanied by sexual dysfunction related to the contractility of the vas deferens smooth muscle. Methods. Ring segments of the epididymal part of the vas deferens were taken from 32 elective vasectomies and mounted in organ baths for isometric recording of tension. We then studied the effects of sertraline, fluoxetine, and amitriptyline on the neurogenic and agonist-induced contractile responses. Results. Amitriptyline caused c…

Malemedicine.medical_specialtyNifedipineAdrenergic receptorAmitriptylineUrologyAdrenergicSynaptic TransmissionNorepinephrine (medication)Vas DeferensNifedipineCulture TechniquesFluoxetineSertralineInternal medicinemedicineHumansAmitriptylineSertralineFluoxetineDose-Response Relationship Drugbusiness.industryVas deferensCalcium Channel BlockersAntidepressive AgentsReceptors AdrenergicEndocrinologymedicine.anatomical_structureCalciumbusinessMuscle Contractionmedicine.drugUrology
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Effect of Divalent Cations on the Contractile Response of Rat Aorta to Depolarization before and after Nifedipine Treatment

1996

The influence of the divalent cations, Ca2+, Mg2+ and Ba2+, on the contractile response of the rat aorta to KCl and on the recovery of this response after nifedipine treatment was analyzed. KCl (80 mmol/l) promoted a two-phase (phasic and tonic) contractile response in Krebs solution but, as expected, no contractile response in Ca(2+)-free medium. In Mg(2+)-free medium, the phasic response to KCl was unaffected but the tonic one decreased slowly, suggesting that a long incubation time in the absence of Mg2+ (65 min) promotes a loss of or a change in the intracellular distribution of this ion that modifies Ca2+ entry through L channels or Ca2+ handling. Ba2+ (1.8 mmol/l) contracted the rat a…

Malemedicine.medical_specialtyNifedipineCations DivalentAorta ThoracicMuscle Smooth VascularPotassium ChlorideDivalentTonic (physiology)NifedipineInternal medicinemedicine.arterymedicineAnimalsRats WistarPharmacologychemistry.chemical_classificationAnalysis of VarianceAortaChemistryContractile responseDepolarizationGeneral MedicineCalcium Channel BlockersRatsEndocrinologyKrebs solutionIntracellularMuscle Contractionmedicine.drugPharmacology
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Endothelin-1-induced potentiation of adrenergic responses in the rabbit pulmonary artery: role of thromboxane A(2).

2001

Abstract To examine whether low concentrations of endothelin-1 potentiate the vasocontrictor response to adrenergic stimulation, we recorded the isometric response of rings of rabbit pulmonary artery to electrical stimulation and noradrenaline. Endothelin-1 (10 −10 M) potentiated the contractions induced by electrical stimulation and noradrenaline. The endothelin ET B receptor antagonist (2,6-dimethylpiperidinecarbonyl-γ-methyl-Leu- N in -[Methoxycarbonyl]- d -Trp- d -Nle) (BQ-788, 10 −6 M), but not the endothelin ET A receptor antagonist cyclo( d -Asp-Pro- d -Val-Leu- d -TRP) (BQ-123, 10 −6 M), inhibited the potentiating effects of endothelin-1. Pretreatment with the cyclooxygenase inhibit…

Malemedicine.medical_specialtyNifedipineThromboxanemedicine.drug_classAdrenergicPulmonary ArteryThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2PiperidinesInternal medicinemedicineAnimalsVasoconstrictor AgentsAntihypertensive AgentsPharmacologybiologyDose-Response Relationship DrugEndothelin-1Receptors EndothelinReceptor antagonistCalcium Channel BlockersEndothelin 1Receptor Endothelin BElectric StimulationEndocrinologychemistryVasoconstrictionbiology.proteinCyclooxygenaseThromboxane-A synthaseRabbitsEndothelin receptorOligopeptidesEuropean journal of pharmacology
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Differential effects of calcium channel antagonists (omega-conotoxin GVIA, nifedipine, verapamil) on the electrically-evoked release of [3H]acetylcho…

1990

Electrically-evoked release of [3H]acetylcholine from autonomic neurons (myenteric plexus), motoneurons (phrenic nerve) and the central nervous system (neocortex) was investigated in the presence and absence of the calcium channel antagonists omega-conotoxin GVIA, nifedipine and verapamil, whereby the same species (rat) was used in all experiments. Release of [3H]acetylcholine was measured after incubation of the tissue with [3H]choline. omega-Conotoxin GVIA markedly reduced (70%) the evoked release of [3H]acetylcholine from the myenteric plexus of the small intestine (IC50: 0.7 nmol/l) with a similar potency at 3 and 10 Hz stimulation. An increase in the extracellular calcium concentration…

Malemedicine.medical_specialtyNifedipinechemistry.chemical_elementMollusk VenomsMyenteric PlexusCalciumAutonomic Nervous Systemcomplex mixturesNifedipineomega-Conotoxin GVIAInternal medicinemedicineAnimalsMyenteric plexusPhrenic nervePharmacologyCerebral CortexMotor NeuronsVoltage-dependent calcium channelCalcium channelRats Inbred StrainsGeneral MedicineCalcium Channel BlockersAcetylcholineElectric StimulationRatsPhrenic NerveEndocrinologynervous systemchemistryVerapamilAnesthesiaVerapamilFemaleAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Voltage-Dependent Effects of Barnidipine in Rat Vascular Smooth Muscle

2003

The effects of the dihydropyridine nifedipine and its more lipophilic congener, barnidipine, were investigated in smooth muscle preparations from the rat in resting and depolarizing conditions. Both drugs relaxed precontracted aortic rings more potently in depolarizing conditions, barnidipine being more potent than nifedipine. Currents through Ca 2+ channels in rat vascular smooth muscle cells (A7r5) and in isolated rat cardiomyocytes were reduced more potently by both drugs at a holding potential of-40 mV than at -80 mV. However, barnidipine and nifedipine were more effective in reducing the current in A7r5 cells than in cardiomyocytes. The IC 50 obtained in aortic rings and in A7r5 cells …

Malemedicine.medical_specialtyPatch-Clamp TechniquesVascular smooth muscleBarnidipineNifedipinechemistry.chemical_elementPharmacologyCalciumMuscle Smooth VascularRats Sprague-DawleyNifedipineInternal medicinemedicineAnimalsMyocyteCells CulturedPharmacologyChemistryDihydropyridineDepolarizationCalcium Channel BlockersRatsEndocrinologyMechanism of actioncardiovascular systemFemalemedicine.symptomCardiology and Cardiovascular Medicinemedicine.drugJournal of Cardiovascular Pharmacology
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Glucagon-like peptide-2 relaxes mouse stomach through vasoactive intestinal peptide release.

2009

Glucagon-like peptide-2 (GLP-2) influences different aspects of the gastrointestinal function, including epithelial growth, digestion, absorption, motility, and blood flow. Intraluminal pressure from isolated mouse stomach was recorded to investigate whether GLP-2 affects gastric tone and to analyze its mechanism of action. Regional differences between diverse parts of the stomach were also examined using circular muscular strips from fundus and antrum. In the whole stomach, GLP-2 (0.3–100 nM) produced concentration-dependent relaxation with a maximum that was about 75% of relaxation to 1 μM isoproterenol (IC50 = 2.5 nM). This effect was virtually abolished by desensitization of GLP-2 rece…

Malemedicine.medical_specialtyPhysiologyVasoactive intestinal peptideGastric motilityMotilityTetrodotoxinIn Vitro TechniquesPeptide hormoneBiologySettore BIO/09 - FisiologiaMiceenteric nervous systemPhysiology (medical)Internal medicineGlucagon-Like Peptide 2Pyloric AntrummedicineAnimalsChymotrypsingastric motilityGastric FundusEnzyme InhibitorsSympathomimeticsHepatologyStomachdigestive oral and skin physiologyIsoproterenolGastroenterologygastrointestinal hormoneGlucagon-like peptide-2Mice Inbred C57BLVIPNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structureGastric EmptyingGastrointestinal hormoneGastrointestinal functionhormones hormone substitutes and hormone antagonistsSodium Channel BlockersVasoactive Intestinal Peptide
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Efficacy of the long-acting nitro vasodilator pentaerithrityl tetranitrate in patients with chronic stable angina pectoris receiving anti-anginal bac…

2013

Background The organic nitrate pentaerithrityl tetranitrate (PETN) has been shown to have ancillary properties that prevent the development of tolerance and endothelial dysfunction. This randomized, double-blind, placebo-controlled, multicentre study (‘CLEOPATRA’ study) was designed to investigate the anti-ischaemic efficacy of PETN 80 mg b.i.d. (morning and mid-day) over placebo in patients with chronic stable angina pectoris. Methods and results A total of 655 patients were evaluated in the intention-to-treat population, randomized to PETN (80 mg b.i.d., n = 328) or placebo ( n = 327) and completed the study. Patients underwent treadmill exercise tests at randomization, after 6 and 12 wee…

Malemedicine.medical_specialtyRandomizationmedicine.drug_classVasodilator AgentsPopulationAdrenergic beta-AntagonistsPlacebo-controlled studyPlaceboAdrenergic beta-AntagonistsMedication AdherenceDouble-Blind MethodmedicineClinical endpointHumansPentaerythritol TetranitrateAngina StableEndothelial dysfunctioneducationBeta blockereducation.field_of_studyExercise Tolerancebusiness.industryMiddle Agedmedicine.diseaseSurgeryTreatment OutcomeAnesthesiaDelayed-Action PreparationsChronic DiseaseExercise TestFemaleCardiology and Cardiovascular MedicinebusinessEuropean heart journal
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