Search results for "Lung"

showing 10 items of 2389 documents

Spectrum of collateral findings in multislice CT coronary angiography.

2007

Purpose. The aim of the study was to investigate the prevalence of the noncardiac collateral findings during multislice computed tomography coronary angiography (MSCT-CA). Materials and methods. Six hundred and seventy patients undergoing MSCT-CA with 16-slice and 64-slice CT scanners for suspected atherosclerotic disease of the coronary arteries were retrospectively reviewed. All data sets obtained with a large field of view (FOV) were analysed by two radiologists using standard mediastinal and lung window settings. Collateral findings were divided according to clinical importance into nonsignificant, remarkable and compulsory to be investigated. Results. Eighty-five percent of patients re…

MaleRadiography AbdominalCoronary angiographymedicine.medical_specialtyTime FactorsMultislice CT Coronary Angiography Collateral findings Incidental findingsCoronary DiseaseCoronary AngiographySensitivity and SpecificityCoronary artery diseaseCollateral findingsElectrocardiographyRisk FactorsImage Processing Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imagingAgedRetrospective StudiesNeuroradiologyLungmedicine.diagnostic_testMultislice CT Coronary Angiographybusiness.industryUltrasoundInterventional radiologyGeneral MedicineMiddle Agedmedicine.diseaseIncidental findingsCoronary arteriesmedicine.anatomical_structureData Interpretation StatisticalRadiological weaponFemaleRadiography ThoracicRadiologyMultislice CT Coronary Angiography; Collateral findings; Incidental findingsbusinessTomography Spiral ComputedFollow-Up Studies
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β2Integrin deficiency yields unconventional double-negative T cells distinct from mature classical natural killer T cells in mice

2009

Expressed on leucocytes, beta(2) integrins (CD11/CD18) are specifically involved in leucocyte function. Using a CD18-deficient (CD18(-/-)) mouse model, we here report on their physiological role in lymphocyte differentiation and trafficking. CD18(-/-) mice present with a defect in the distribution of lymphocytes with highly reduced numbers of naïve B and T lymphocytes in inguinal and axillary lymph nodes. In contrast, cervical lymph nodes were fourfold enlarged harbouring unconventional T-cell receptor-alphabeta (TCR-alphabeta) and TCR-gammadelta CD3(+) CD4(-) CD8(-) (double-negative; DN) T cells that expanded in situ. Using adoptive transfer experiments, we found that these cells did not h…

MaleReceptors Antigen T-Cell alpha-betaT cellImmunologyCD1chemical and pharmacologic phenomenaBiologyLymphocyte ActivationImmunophenotypingMiceInterleukin 21T-Lymphocyte SubsetsImmune TolerancemedicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellLungLymphatic DiseasesMice KnockoutB-LymphocytesZAP70Receptors Antigen T-Cell gamma-deltahemic and immune systemsOriginal ArticlesNatural killer T cellAdoptive TransferMolecular biologyCoculture TechniquesChemotaxis Leukocytemedicine.anatomical_structureLiverCD18 AntigensImmunologyNatural Killer T-CellsFemaleLymph NodesLymphocyte Culture Test MixedImmunology
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A novel CXCR4 antagonist counteracts paradoxical generation of cisplatin-induced pro-metastatic niches in lung cancer.

2021

Platinum-based chemotherapy remains widely used in advanced non-small cell lung cancer (NSCLC) despite experimental evidence of its potential to induce long-term detrimental effects, including the promotion of pro-metastatic microenvironments. In this study, we investigated the interconnected pathways underlying the promotion of cisplatin-induced metastases. In tumor-free mice, cisplatin treatment resulted in an expansion in the bone marrow of CCR2+CXCR4+Ly6Chigh inflammatory monocytes (IMs) and an increase in lung levels of stromal SDF-1, the CXCR4 ligand. In experimental lung metastasis assays, cisplatin-induced IMs promoted the extravasation of tumor cells and the expansion of CD133+CXCR…

MaleReceptors CXCR4Stromal cellLung NeoplasmsSettore MED/08 - Anatomia PatologicaMonocytesMetastasisMiceCarcinoma Non-Small-Cell LungCell Line TumorDrug DiscoveryGeneticsMedicineSettore MED/05 - Patologia ClinicaAnimalsHumansDrug InteractionsAC133 AntigenNeoplasm MetastasisLung cancerMolecular BiologyPharmacologyCisplatinCXCR4 antagonistchemotherapy combination therapy inflammatory monocytes lung cancer stem cells metastasis peptide anti-CXCR4 SDF-1/CXCR4 axisbusiness.industrymedicine.diseasePrimary tumorXenograft Model Antitumor AssaysExtravasationChemokine CXCL12medicine.anatomical_structureRAW 264.7 CellsA549 CellsCancer researchNeoplastic Stem CellsMolecular MedicineBone marrowCisplatinbusinessPeptidesmedicine.drugMolecular therapy : the journal of the American Society of Gene Therapy
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Co-option of Neutrophil Fates by Tissue Environments

2020

Classically considered short-lived and purely defensive leukocytes, neutrophils are unique in their fast and moldable response to stimulation. This plastic behavior may underlie variable and even antagonistic functions during inflammation or cancer, yet the full spectrum of neutrophil properties as they enter healthy tissues remains unexplored. Using a new model to track neutrophil fates, we found short but variable lifetimes across multiple tissues. Through analysis of the receptor, transcriptional, and chromatin accessibility landscapes, we identify varying neutrophil states and assign non-canonical functions, including vascular repair and hematopoietic homeostasis. Accordingly, depletion…

MaleReceptors CXCR4Transcription GeneticAngiogenesisNeutrophilsMedizinNeovascularization PhysiologicInflammationBiologyCXCR4General Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineSingle-cell analysismedicineNuclear Receptor Subfamily 4 Group A Member 1AnimalsCell LineageReceptorLung030304 developmental biology0303 health sciencesInnate immune systemChromatinChromatinCell biologyHematopoiesisIntestinesMice Inbred C57BLOrgan SpecificityFemalemedicine.symptomSingle-Cell AnalysisTranscriptome030217 neurology & neurosurgeryHomeostasisCell
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Induction of RAGE Shedding by Activation of G Protein-Coupled Receptors

2011

The multiligand Receptor for Advanced Glycation End products (RAGE) is involved in various pathophysiological processes, including diabetic inflammatory conditions and Alzheimers disease. Full-length RAGE, a cell surface-located type I membrane protein, can proteolytically be converted by metalloproteinases ADAM10 and MMP9 into a soluble RAGE form. Moreover, administration of recombinant soluble RAGE suppresses activation of cell surface-located RAGE by trapping RAGE ligands. Therefore stimulation of RAGE shedding might have a therapeutic value regarding inflammatory diseases. We aimed to investigate whether RAGE shedding is inducible via ligand-induced activation of G protein-coupled recep…

MaleReceptors Vasopressinendocrine system diseasesReceptor for Advanced Glycation End Productslcsh:MedicineHydroxamic Acids570 Life sciencesRAGE (receptor)Adenylyl cyclaseADAM10 ProteinMicePhosphatidylinositol 3-Kinaseschemistry.chemical_compoundMolecular Cell BiologyNeurobiology of Disease and RegenerationSignaling in Cellular ProcessesMembrane Receptor SignalingReceptors Immunologiclcsh:ScienceReceptorLungCellular Stress ResponsesCalcium signalingMultidisciplinaryKinaseDipeptidesHormone Receptor SignalingCell biologyMatrix Metalloproteinase 9NeurologyReceptors OxytocinGene Knockdown Techniquescardiovascular systemMatrix Metalloproteinase 2Pituitary Adenylate Cyclase-Activating PolypeptideMedicineRNA InterferenceAdenylyl CyclasesResearch ArticleSignal Transduction570 Biowissenschaftenmedicine.medical_specialtyMAP Kinase Signaling SystemADAM17 ProteinBiologyAlzheimer DiseaseCa2+/calmodulin-dependent protein kinaseInternal medicinemedicineAnimalsHumansProtease InhibitorsCalcium Signalingcardiovascular diseasesBiologyG protein-coupled receptorlcsh:RHEK 293 cellsMembrane Proteinsnutritional and metabolic diseasesCyclic AMP-Dependent Protein KinasesADAM ProteinsG-Protein SignalingHEK293 CellsEndocrinologychemistryProteolysisDementialcsh:QAmyloid Precursor Protein SecretasesMolecular Neurosciencehuman activitiesReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type INeurosciencePLoS ONE
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Effect of indoor nitrogen dioxide on lung function in urban environment

2015

BACKGROUND: High levels of indoor NO2 are associated with increased asthma symptoms and decreased expiratory peak flows in children. We investigated the association of exposure to domestic indoor NO2, objectively measured in winter and spring, with respiratory symptoms and lung function in a sample of adolescents from a southern Mediterranean area. METHODS: From a large school population sample (n=2150) participating in an epidemiological survey in the urban area of the City of Palermo (southern Italy), a sub-sample of 303 adolescents was selected which furnished an enriched sample for cases of current asthma. All subjects were evaluated by a health questionnaire, skin prick tests and spiro…

MaleRespiratory Tract DiseasesQuestionnairBiochemistrychemistry.chemical_compoundAdolescents' healthIndoor nitrogendioxideEpidemiologyRespiratory functionRespiratory systemChildLungLung functionRespiratory diseaseGeneral Environmental ScienceAir Pollutantsmedicine.diagnostic_testrespiratory systemRespiratory Function TestsAdolescents' health; Indoor nitrogen dioxide; Questionnaire; Respiratory diseases; Spirometry; 2300; BiochemistryItalyAir Pollution IndoorFemaleSeasonsmedicine.symptomIndoor nitrogen dioxideEnvironmental MonitoringSpirometrymedicine.medical_specialtyAdolescentNitrogen DioxideEnvironmentcomplex mixturesWheezeEnvironmental healthmedicineHumansNitrogen dioxideCitiesAsthma2300Questionnairebusiness.industryEnvironmental Exposuremedicine.diseaserespiratory tract diseasesCross-Sectional StudiesSocioeconomic FactorschemistrySpirometrybusiness
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Prevalence and attributable health burden of chronic respiratory diseases, 1990–2017: a systematic analysis for the Global Burden of Disease Study 20…

2020

Artículo con numerosos autores. Sólo se hace referencia al primero que coincide con el de la UAM y al colectivo

MaleRespiratory diseasesRespiratory Tract DiseasesDiseaseChronic respiratory diseasesGlobal Burden of DiseasePulmonary Disease Chronic Obstructive0302 clinical medicineCost of Illness11. SustainabilityMETABOLIC RISKSEPIDEMIOLOGY030212 general & internal medicineChildCause of deathAged 80 and overCOPDDALYChronic obstructive pulmonary diseaseMortality rateRespiratory disease1. No povertyAge FactorsMiddle AgedDeath causes3. Good healthPREVALENCEHealth risksChild PreschoolCOMPARATIVE RISK-ASSESSMENTFemaledeath and disability worldwideQuality-Adjusted Life YearsTERRITORIESBURDENgrowth in absolute numbersPulmonary and Respiratory MedicineAdultADJUSTED LIFE-YEARSHealth burdensAdolescentMedicina195 COUNTRIESchronic respiratory diseasesArticle1117 Public Health and Health Services03 medical and health sciencesYoung AdultLife ExpectancySex FactorsBurden of Disease Respiratory diseaseSarcoidosis PulmonaryEnvironmental healthmedicineDisability-adjusted life yearHumansCOPDEXPOSURERisk factorMortalityAgedper-capita basisbusiness.industryDISABILITYInfant NewbornInfant1103 Clinical Sciencesasthmamedicine.diseaseAsthmaYears of potential life lost030228 respiratory systemRisk factors13. Climate actionSystematic analysesChronic DiseaseINJURIESHuman medicinePneumoconiosisMorbiditybusinessLung Diseases Interstitial1199 Other Medical and Health Sciences
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Ultra-low tidal volume ventilation-A novel and effective ventilation strategy during experimental cardiopulmonary resuscitation.

2018

Abstract Background The effects of different ventilation strategies during CPR on patient outcomes and lung physiology are still poorly understood. This study compares positive pressure ventilation (IPPV) to passive oxygenation (CPAP) and a novel ultra-low tidal volume ventilation (ULTVV) regimen in an experimental ventricular fibrillation animal model. Study design Prospective randomized controlled trial. Animals 30 male German landrace pigs (16–20 weeks). Methods Ventricular fibrillation was induced in anesthetized and instrumented pigs and the animals were randomized into three groups. Mechanical CPR was initiated and ventilation was either provided by means of standard IPPV (RR: 10/min,…

MaleResuscitationSwinemedicine.medical_treatmentRespiratory physiology030204 cardiovascular system & hematologyEmergency NursingLung injuryAdvanced Cardiac Life SupportReal-Time Polymerase Chain ReactionIntermittent Positive-Pressure Ventilation03 medical and health sciencesRandom Allocation0302 clinical medicinemedicineTidal VolumeAnimalsHumansCardiopulmonary resuscitationTidal volumeAnalysis of VarianceContinuous Positive Airway Pressurebusiness.industryPulmonary Gas Exchange030208 emergency & critical care medicineOxygenationLung Injurymedicine.diseaserespiratory tract diseasesDisease Models AnimalTreatment OutcomeAnesthesiaVentricular fibrillationEmergency MedicineBreathingCardiology and Cardiovascular MedicinebusinessResuscitation
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Restrictive pulmonary dysfunction at spirometry and mortality in the elderly

2008

SummaryObjectivesTo evaluate the association between pulmonary restriction and mortality in the elderly, taking into account potential confounders not considered in the past (disability, cognitive dysfunction, diabetes, and visceral obesity).DesignLongitudinal study.SettingCommunity-based.ParticipantsTwelve hundred sixty-five patients (51.9% men) aged 65–97 years old from the Salute Respiratoria nell'Anziano (SaRA) Italian multicentric study.MeasurementsParticipants were divided in 4 groups: normal spirometry (NS): FEV1/FVC≥70%, FVC≥80% of predicted; restrictive ventilatory pattern (RVP): FEV1/FVC≥70%, FVC<80%; obstructive ventilatory pattern (OVP): FEV1/FVC<70%, FVC≥80%, and mixed ventilat…

MaleRiskSpirometryPulmonary and Respiratory MedicineLongitudinal studymedicine.medical_specialtyWaistVital CapacitySettore MED/10 - Malattie Dell'Apparato RespiratorioelderlyLung restrictionPulmonary function testingPulmonary Disease Chronic ObstructiveForced Expiratory VolumeInternal medicineEpidemiologymedicineHumansLung Diseases ObstructiveMortalityGeriatric AssessmentLungStrokeDepression (differential diagnoses)Pulmonary function testsAgedProportional Hazards ModelsAged 80 and overmedicine.diagnostic_testProportional hazards modelbusiness.industryLongitudinal studiesPrognosismedicine.diseaseHealth SurveysItalySpirometryPhysical therapyFemalebusinessFollow-Up StudiesRespiratory Medicine
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Convergent sets of data from in vivo and in vitro methods point to an active role of Hsp60 in chronic obstructive pulmonary disease pathogenesis.

2011

BackgroundIt is increasingly clear that some heat shock proteins (Hsps) play a role in inflammation. Here, we report results showing participation of Hsp60 in the pathogenesis of chronic obstructive pulmonary diseases (COPD), as indicated by data from both in vivo and in vitro analyses.Methods and resultsBronchial biopsies from patients with stable COPD, smoker controls with normal lung function, and non-smoker controls were studied. We quantified by immunohistochemistry levels of Hsp10, Hsp27, Hsp40, Hsp60, Hsp70, Hsp90, and HSF-1, along with levels of inflammatory markers. Hsp10, Hsp40, and Hsp60 were increased during progression of disease. We found also a positive correlation between th…

MaleSTRESSPulmonologyChronic Obstructive Pulmonary DiseasesNeutrophilsBiopsyGene ExpressionCD8-Positive T-Lymphocytesmedicine.disease_causeBiochemistryEpitheliumPulmonary function testingPathogenesisACTIVATIONPulmonary Disease Chronic ObstructiveMolecular Cell BiologyLungCOPDMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionCOPD Hsp60QRCOPD heat shock proteins inflammationMiddle AgedImmunohistochemistrymedicine.anatomical_structureEXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITISMedicineFemalemedicine.symptomInflammation MediatorsSPINAL-CORDResearch ArticleEXPRESSIONanimal structuresCOPD; heat shock proteins; inflammationScienceImmunologyMolecular Sequence DataInflammationBronchichemical and pharmacologic phenomenaHEAT-SHOCK-PROTEIN EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS ACUTE LUNG INJURY SPINAL-CORD CELL-DEATH KAPPA-B HEAT-SHOCK-PROTEIN-60 STRESS EXPRESSION ACTIVATIONKAPPA-BBiologyHEAT-SHOCK-PROTEINMicrobiologycomplex mixturesCell LineACUTE LUNG INJURYMolecular GeneticsIn vivoStress PhysiologicalHeat shock proteinmedicineGeneticsHumansCOPDRNA MessengerBiologyAgedLungMucous MembraneBase SequenceSettore BIO/16 - Anatomia UmanaMacrophagesfungiImmunityTranscription Factor RelAProteinsComputational BiologyChaperonin 60medicine.diseaseChaperone Proteinsrespiratory tract diseasesGene Expression RegulationCELL-DEATHHEAT-SHOCK-PROTEIN-60inflammationImmunologyheat shock proteinsClinical ImmunologyOxidative stressBiomarkers
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