Search results for "Luteolin"

showing 10 items of 31 documents

Effects of Indole-3-Carbinol and Flavonoids Administered Separately and in Combination on Nitric Oxide Production and iNOS Expression in Rats

2010

Beneficial effects of natural compounds are often attributed to modulation of NO production; however effects produced by plant extracts do not correlate with effects of purified components. The goal of our work was to study ability of flavonoids and indole-3-carbinol, as well as their combinations to modify NO production, iNOS gene and protein expression in rat tissues. Baicalein and luteolin decreased NO concentration in both intact and LPS-treated animals. Baicalein decreased iNOS gene expression. Luteolin decreased NO production in the liver and heart and number of iNOS-positive cells in the liver of LPS-treated animals. Combination of the two substances did not decrease the NO synthesis…

Enzyme biosynthesisSpleenGeneral MedicineGeneral ChemistryPharmacologyBiologyNitric oxideBaicaleinchemistry.chemical_compoundmedicine.anatomical_structurechemistryBiochemistryGene expressionmedicineIndole-3-carbinolQuercetinLuteolinChinese Medicine
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Structure-activity relationships of polymethoxyflavones and other flavonoids as inhibitors of non-enzymic lipid peroxidation

1990

Polymethoxylated flavones and C-glycosyl derivatives isolated from medicinal plants besides other flavonoid compounds were studied for their influence on lipid peroxidation induced by FeSO4+ cysteine in rat liver microsomes. A number of hydroxyflavones (e.g. luteolin); C-glycosyl-flavones (e.g. orientin); methoxyflavones (e.g. gardenin D) and flavonols (e.g. datiscetin), as well as the flavanol leucocyanidol and the biflavone amentoflavone behaved as inhibitors of non-enzymic lipid peroxidation. Structure-activity relationships were established and it was observed that the structural features for active polyhydroxylated compounds were different from those of polymethoxylated flavones, antip…

FlavonoidsPharmacologychemistry.chemical_classificationOrientinFlavonoidAmentoflavoneBiologyBiochemistryFlavonesRatscarbohydrates (lipids)Lipid peroxidationStructure-Activity Relationshipchemistry.chemical_compoundFlavonolschemistryBiochemistryLipophilicityMicrosomes LiverAnimalsLipid PeroxidationLuteolinBiochemical Pharmacology
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Topoisomerase II inhibition and high yield of endoreduplication induced by the flavonoids luteolin and quercetin

2006

Luteolin and quercetin are widely distributed plant flavonoids that possess a variety of chemical and biological activities, including free-radical scavenging and antioxidant activity. Recently, both flavonoids have been reported to inhibit DNA topoisomerases I and II (topo I and topo II), a property that, together with their ability to induce DNA and chromosome damage, has made them candidate anticancer compounds. In the present study, we confirmed that both compounds are topo II inhibitors by conducting a comparative study of their effect on topo II activity from Chinese hamster ovary AA8 cells. Because interference with the function of topo II to resolve DNA entanglement at the end of re…

Health Toxicology and MutagenesisFlavonoidAntineoplastic AgentsToxicologyTopoisomerase II InhibitorModels BiologicalPolyploidychemistry.chemical_compoundChromosome SegregationCricetinaeGeneticsTopoisomerase II InhibitorsAnimalsEndoreduplicationheterocyclic compoundsEnzyme InhibitorsLuteolinCells CulturedGenetics (clinical)Chromosome AberrationsFlavonoidsEnzyme Inhibitors/pharmacologychemistry.chemical_classificationbiologyTopoisomeraseChinese hamster ovary cellAntineoplastic Agents/adverse effectsDNA Topoisomerases Type IIchemistryBiochemistryDNA Damage/drug effectsFlavonoidbiology.proteinQuercetinDNA Topoisomerases Type II/metabolismTopoisomerase-II InhibitorChromosome Segregation/drug effectsQuercetinAntineoplastic Agents/pharmacologyLuteolinDNADNA DamageMutagenesis
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Cucumis sativus l. Seeds ameliorate muscular spasm-induced gastrointestinal and respiratory disorders by simultaneously inhibiting calcium mediated s…

2021

Cucumis sativus L. is globally cultivated as an edible vegetable. Besides its nutritional benefits, it is used in traditional medicines against various ailments. The current study was designed to elucidate the multi-target mechanisms of a C. sativus seeds extract against asthma and diarrhea using network pharmacology along with a molecular docking approach. Furthermore, in-vitro and in-vivo experiments were conducted to verify the mechanistic insight of in silico studies. LC-ESI-MS/MS was performed to identify the bioactive compounds in the extract; later, some compounds were quantified by HPLC. C. sativus seed. EtOH has kaempferol in higher concentration 783.02 µg/g, followed by quercetin …

In silicoPharmaceutical Sciencechemistry.chemical_elementPharmacologyCalciumArticleJejunum<i>Cucumis sativus</i>chemistry.chemical_compoundAntidiarrheal; Asthma; Bronchodilator; Cucumber; Cucumis sativus; HPLC; LC-ESI-MS/MSPharmacy and materia medicaAntidiarrhealDrug Discoverymedicinebiologyasthma; antidiarrheal; bronchodilator; <i>Cucumis sativus</i>; cucumber; HPLC; LC-ESI-MS/MSCucumberRfood and beveragesLC-ESI-MS/MSbiology.organism_classificationAsthmaRS1-441medicine.anatomical_structurechemistryMolecular MedicineMedicineSignal transductionCucumis sativusHPLCKaempferolQuercetinCucumisLuteolinBronchodilator
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The inhibition by flavonoids of 2-amino-3-methylimidazo[4,5-f]quinoline metabolic activation to a mutagen: a structure-activity relationship study.

1997

The mutagenicity of 2-amino-3-methylimidazo[4,5-f]quinoline (IQ) in Salmonella typhimurium TA98 is inhibited by flavonoids with distinct structure-antimutagenicity relationships (Edenharder, R., I. von Petersdorff I. and R. Rauscher (1993). Antimutagenic effects of flavonoids, chalcones and structurally related compounds on the activity of 2-amino-3-methylimidazo[4,5-f]quinoline (IQ) and other heterocyclic amine mutagens from cooked food, Mutation Res., 287, 261-274). With respect to the mechanism(s) of antimutagenicity, the following results were obtained here. (1) 7-Methoxy- and 7-ethoxyresorufin-O-dealkylase activities in rat liver microsomes, linked to cytochrome P-450-dependent 1A1 and…

MaleCytochrome P-450 CYP1A2 InhibitorsHealth Toxicology and MutagenesisHydroxylationFlavonesRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipFlavonolsCytochrome P-450 Enzyme SystemGeneticsCytochrome P-450 CYP1A1AnimalsMolecular BiologyBiotransformationchemistry.chemical_classificationFlavonoidsMutagenicity Testsfood and beveragesAntimutagenic AgentsMonooxygenaseDiosmetinRatschemistryBiochemistryHydroxyquinolinesMicrosomes LiverQuinolinesOxidoreductasesAntimutagenFlavanoneLuteolinFisetinMutagensMutation research
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Metabolism of apigenin by rat liver phase I and phase II enzymes and by isolated perfused rat liver

2004

The metabolism of apigenin, a low estrogenic flavonoid phytochemical, was investigated in rat using liver models both in vitro (subcellular fractions) and ex vivo (isolated perfused liver). In vitro, phase I metabolism led to the formation of three monohydroxylated derivatives: luteolin which was the major metabolite (K(m) = 22.5 +/- 1.5 microM; V(max) = 5.605 +/- 0.090 nmol/min/mg protein, means +/- S.E.M.), scutellarein, and iso-scutellarein. These oxidative pathways were mediated by cytochrome P450 monooxygenases (P450s). The use of P450 inhibitors and inducers showed that CYP1A1, CYP2B, and CYP2E1 are involved. In vitro studies of phase II metabolism indicated that apigenin underwent co…

MaleFMN ReductaseMetabolite[SDV]Life Sciences [q-bio]Pharmaceutical ScienceIn Vitro TechniquesMethylation030226 pharmacology & pharmacyMass Spectrometry03 medical and health scienceschemistry.chemical_compoundGlucuronides0302 clinical medicineCytochrome P-450 Enzyme SystemAnimalsApigeninEnzyme InhibitorsRats WistarLuteolinBiotransformationChromatography High Pressure LiquidComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidsPharmacologySex Characteristics0303 health sciencesbiologySulfatesScutellareinCytochrome P450MonooxygenaseDiosmetinRats3. Good health[SDV] Life Sciences [q-bio]KineticsLiverBiochemistrychemistryApigeninbiology.proteinRATFemaleSpectrophotometry UltravioletLuteolinNADPDrug metabolismSubcellular Fractions
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Inhibition of ethoxyresorufin deethylase activity by natural flavonoids in human and rat liver microsomes

1990

Several flavones and flavonols (chrysin, quercetin, luteolin, flavone and 7, 8-benzoflavone) were found to inhibit ethoxyresorufin deethylase (EROD) activity in human and rat liver microsomes. In man, molecules without hydroxyl groups are more powerful inhibitors than polyhydroxylated flavonoids (7, 8-benzoflavone greater than flavone greater than chrysin greater than luteolin greater than quercetin greater than morin). In rat, chrysin was the strongest inhibitor and the less effective were morin and 7,8-benzoflavone. For all molecules human microsomes were more sensitive than rat microsomes. The most important difference concerned 7,8-benzoflavone which was 10,000-fold more potent in man.

MaleHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]MorinToxicology030226 pharmacology & pharmacyFlavonesStructure-Activity Relationship03 medical and health scienceschemistry.chemical_compound0302 clinical medicineFlavonolsSpecies SpecificityCytochrome P-450 CYP1A1AnimalsCytochrome P-450 Enzyme InhibitorsHumansStructure–activity relationshipheterocyclic compoundsChrysinComputingMilieux_MISCELLANEOUS030304 developmental biologyFlavonoidschemistry.chemical_classification0303 health sciencesPublic Health Environmental and Occupational HealthRats Inbred StrainsGeneral ChemistryRats3. Good healthchemistryBiochemistryChemistry (miscellaneous)Microsomes LiverMicrosomeRATOxidoreductasesQuercetinLuteolinFood Science
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Altilix

2019

The objective was to evaluate the effects of 6 months of supplementation with Altilix®, containing chlorogenic acid and its derivatives, and luteolin and its derivatives, on cardiovascular risk and hepatic markers in subjects with metabolic syndrome (MetS). A randomized, double-blind, placebo-controlled study was performed in 100 subjects with MetS with a follow-up period of 6 months; 50 subjects were randomized to Altilix® (26 men and 24 women, mean age 63 ± 8 years) and the other 50 to placebo (28 men and 22 women, mean age 63 ± 11 years). Anthropometric, cardiometabolic, and hepatic parameters were assessed at baseline and at the end of follow-up. Carotid intima-media thickness and endot…

MaleMetabolic Syndromecynaranon-alcoholic fatty liver diseaseMiddle AgedArticlecardiovascular diseasesdietary supplementsDouble-Blind MethodLivertype 2Risk Factorsdiabetes mellitusHumansFemaleChlorogenic AcidLuteolinBiomarkersAgedNutrients
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Flavonoids from Artichoke (Cynara scolymus L.) Up-Regulate Endothelial-Type Nitric-Oxide Synthase Gene Expression in Human Endothelial Cells

2004

Nitric oxide (NO) produced by endothelial nitric-oxide synthase (eNOS) represents an antithrombotic and anti-atherosclerotic principle in the vasculature. Hence, an enhanced expression of eNOS in response to pharmacological interventions could provide protection against cardiovascular diseases. In EA.hy 926 cells, a cell line derived from human umbilical vein endothelial cells (HUVECs), an artichoke leaf extract (ALE) increased the activity of the human eNOS promoter (determined by luciferase reporter gene assay). An organic subfraction from ALE was more potent in this respect than the crude extract, whereas an aqueous subfraction of ALE was without effect. ALE and the organic subfraction t…

MaleNitric Oxide Synthase Type IIIRNA StabilityQuinic AcidGene ExpressionCynarosideBiologyUmbilical veinNitric oxideRats Sprague-Dawleychemistry.chemical_compoundEnosCynara scolymusGene expressionAnimalsHumansRNA MessengerPromoter Regions GeneticAortaCells CulturedFlavonoidsPharmacologybiology.organism_classificationMolecular biologyRatsUp-RegulationVasomotor SystemNitric oxide synthasechemistryBiochemistryCell culturebiology.proteinMolecular MedicineEndothelium VascularNitric Oxide SynthaseLuteolinJournal of Pharmacology and Experimental Therapeutics
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Antimutagenic effects of flavonoids, chalcones and structurally related compounds on the activity of 2-amino-3-methylimidazo[4,5-f]quinoline (IQ) and…

1993

Abstract Sixty-four flavonoids were tested for their antimutagenic potencies with respect to IQ in Salmonella typhimurium TA98 and in part also towards MeIQ, MeIQx, Trp-P-2, and Glu-P-1 and in S. typhimurium TA100. Antimutagenic potencies were quantified by the inhibitory dose for 50% reduction of mutagenic activity (ID 50 ). A carbonyl function at C-4 of the flavane nucleus seems to be essential for antimutagenicity: two flavanols and four anthocyanidines were inactive. Again, five isoflavons, except biochanin A, were inactive. Within the other groups of 21 flavones, 16 flavonols and 16 flavanones the parent compounds flavone, flavonol, and flavonone possessed the highest antimutagenic pot…

MaleStereochemistryHealth Toxicology and MutagenesisFlavonoidFlavonesRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipFlavonolsChalconeGeneticsAnimalsCookingMolecular Biologychemistry.chemical_classificationFlavonoidsDose-Response Relationship DrugMutagenicity TestsAntimutagenic AgentsRatschemistryBiochemistryApigeninFlavanonesQuinolinesKaempferolLuteolinFlavanoneAntimutagenMutagensMutation research
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