Search results for "Lymphocyte Subsets"

showing 10 items of 212 documents

Understanding the Role of T Cells in CNS Homeostasis.

2015

T cells within the central nervous system (CNS) have been generally considered pathogenic, especially in the context of neuroinflammatory disease. However, recent findings have revealed varied functions for T cells in the healthy CNS, as well as more complex roles for these cells in infection and injury than previously appreciated. Here we review evidence indicating important roles for different T cell subsets in the maintenance of CNS homeostasis. We examine the contribution of T cells in limiting inflammation and damage upon CNS injury, infection, and in neurodegeneration, and discuss the current understanding of the cellular and molecular mechanisms involved. Insight into these processes…

Central Nervous SystemT cellT-LymphocytesImmunologyCentral nervous systemContext (language use)InflammationDiseaseBiologyLymphocyte Depletion03 medical and health sciences0302 clinical medicineT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsHomeostasisHumansNeurodegenerationmedicine.diseaseCns injurymedicine.anatomical_structureImmunologymedicine.symptomNeurogenic Inflammation030217 neurology & neurosurgeryHomeostasis030215 immunologyTrends in immunology
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Cytokines in Colitis-Associated Cancer: Potential Drug Targets?

2008

In inflammatory bowel disease (IBD), such as UC and CD, the development of colorectal carcinoma can be initiated through chronic inflammation, depending on the duration and severity of the disease. Growing evidence supports a role for various cytokines, released by epithelial and immune cells, in the pathogenesis of colitis associated cancer (CAC). For instance, TNF-alpha has been recently shown to promote tumor development in experimental colitis. Due to its role in the pathogenesis of IBD, TNF-alpha blockade has become one of the cornerstones of IBD therapy. Thus, anti-TNF-alpha strategies could also provide effective anti-tumor therapies. TGF-beta has been shown to attenuate an anti-tumo…

Colorectal cancerImmunologyInflammationInflammatory bowel diseasePathogenesisImmune systemT-Lymphocyte SubsetsAnimalsHumansImmunology and AllergyMedicineColitisPharmacologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryCancerGeneral MedicineColitisInflammatory Bowel Diseasesmedicine.diseaseInterleukin-12Interleukin-10ImmunologyCytokinesTumor necrosis factor alphamedicine.symptomColorectal NeoplasmsbusinessInflammation & Allergy - Drug Targets
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Hierarchical modeling for rare event detection and cell subset alignment across flow cytometry samples.

2013

Flow cytometry is the prototypical assay for multi-parameter single cell analysis, and is essential in vaccine and biomarker research for the enumeration of antigen-specific lymphocytes that are often found in extremely low frequencies (0.1% or less). Standard analysis of flow cytometry data relies on visual identification of cell subsets by experts, a process that is subjective and often difficult to reproduce. An alternative and more objective approach is the use of statistical models to identify cell subsets of interest in an automated fashion. Two specific challenges for automated analysis are to detect extremely low frequency event subsets without biasing the estimate by pre-processing…

Computer scienceAdaptive Immunitycomputer.software_genre0302 clinical medicineSingle-cell analysisEnumerationBiology (General)Immune ResponseEvent (probability theory)0303 health sciencesEcologymedicine.diagnostic_testT CellsStatisticsFlow Cytometry3. Good healthComputational Theory and MathematicsData modelModeling and SimulationMedicineData miningImmunotherapyResearch ArticleTumor ImmunologyQH301-705.5Immune CellsImmunologyContext (language use)BiostatisticsModels BiologicalFlow cytometry03 medical and health sciencesCellular and Molecular NeuroscienceGeneticsmedicineHumansSensitivity (control systems)Statistical MethodsImmunoassaysMolecular BiologyBiologyEcology Evolution Behavior and Systematics030304 developmental biologybusiness.industryImmunityReproducibility of ResultsPattern recognitionStatistical modelImmunologic SubspecialtiesLymphocyte SubsetsImmunologic TechniquesClinical ImmunologyArtificial intelligencebusinesscomputerMathematics030215 immunologyPLoS computational biology
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γδ T cells as a potential tool in colon cancer immunotherapy

2014

γδ T cells are capable of recognizing tumor cells and exert potent cellular cytotoxicity against a large range of tumors, including colon cancer. However, tumors utilize numerous strategies to escape recognition or killing by patrolling γδ T cells, such a downregulation of NKG2D ligands, MICA/B and ULBPs. Therefore, the combined upregulation of T-cell receptorand NKG2D ligands on tumor cells and induction of NKG2D expression on γδ T cells may greatly enhance tumor killing and unlock the functions of γδ T cells. Here, we briefly review current data on the mechanisms of γδ T-cell recognition and killing of colon cancer cells and propose that γδ T cells may represent a promising target for the…

Cytotoxicity ImmunologicColorectal cancermedicine.medical_treatmentImmunologyNkg2d ligandsLarge rangeLigandsDownregulation and upregulationT-Lymphocyte SubsetsmedicineHumansImmunology and AllergyIn patientCell-mediated cytotoxicitybusiness.industryReceptors Antigen T-Cell gamma-deltaImmunotherapymedicine.diseaseNKG2Dgamma delta cellcolon cancerOncologyColonic NeoplasmsImmunologyNeoplastic Stem CellsImmunotherapybusinessImmunotherapy
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Combining dasatinib with dexamethasone long-term leads to maintenance of antiviral and antileukemia specific cytotoxic T cell responses in vitro

2012

Maintaining graft versus leukemia (GvL) and antivirus responses of cytotoxic T cells (CTLs) while suppressing graft-versus-host disease (GvHD) remains a challenge after allogeneic bone marrow transplantation. Clinical observations indicate that combining glucocorticoids with multi-tyrosine-kinase inhibitors could be a successful therapeutic approach. We and others have shown that the BCR-ABL/SRC kinase inhibitor dasatinib may enhance or suppress T cells in vitro. In this report, we evaluated combination effects of dasatinib and dexamethasone on CD3 + and virus-specific CD8 + T cells directly ex vivo and on antigen-specific leukemia-reactive and alloreactive CD8 + T cell clones. Functional o…

Cytotoxicity ImmunologicHerpesvirus 4 HumanCancer ResearchNaive T cellT cellDasatinibDrug Evaluation PreclinicalReceptors Antigen T-CellCytomegalovirusApoptosisT-Cell Antigen Receptor SpecificityBiologyLymphocyte ActivationCell DegranulationDexamethasoneAntigenHLA AntigensT-Lymphocyte SubsetsGeneticsmedicineHumansCytotoxic T cellAntigens ViralProtein Kinase InhibitorsMolecular BiologyCells CulturedDegranulationDrug SynergismT-Lymphocytes Helper-InducerCell BiologyHematologyDasatinibThiazolesPyrimidinesmedicine.anatomical_structureImmunologyCancer researchCytokinesK562 CellsMemory T cellCell DivisionCD8Signal TransductionT-Lymphocytes Cytotoxicmedicine.drugExperimental Hematology
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Biochemical markers in Celiac disease.

2009

Celiac Disease is a worldwide spread condition affecting 1:100-1:200 individuals. It is a permanent food intolerance to ingested gluten in genetically predisposed subjects. In this review we analyze the biochemical markers of the disease going from laboratory findings to histology passing through genetics. Gluten intolerance is a unique model of autoimmune disease in which we can recognize the main environmental factor (gluten) and the more complex genetic background. In additional way, serological markers for monitoring the disease and a safe and effective therapy (gluten free diet) are also available. In deed the environmental factor such as gluten intake is necessary to trigger the disea…

DiseaseHuman leukocyte antigenGeneral Biochemistry Genetics and Molecular BiologyPathogenesisSettore MED/38 - Pediatria Generale E SpecialisticaGluten free dietmedicineHumanschemistry.chemical_classificationAutoimmune diseaseGeneral Immunology and Microbiologybusiness.industrynutritional and metabolic diseasesGluten intoleranceEpithelial Cellsmedicine.diseaseGlutendigestive system diseasesLymphocyte SubsetsFood intoleranceCeliac DiseaseSerologychemistryImmunologyGluten freebusinessBiomarkersFrontiers in bioscience (Scholar edition)
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Homing and memory patterns of human gammadelta T cells in physiopathological situations.

2004

Vgamma9Vdelta2 are a heterogeneous population of T cells and comprise distinct naive, memory and effector populations that can be distinguished on the basis of surface marker expression and effector functions. We review here these recently studied features of Vgamma9Vdelta2 T lymphocyte biology and the roles they play in infectious and autoimmune diseases.

EffectorImmunologyT lymphocyteBiologymedicine.disease_causeInfectionsLymphocyte ActivationMicrobiologyAutoimmunityAutoimmune DiseasesHeterogeneous populationInfectious DiseasesT-Lymphocyte SubsetsSurface markerImmunologymedicineHumansTuberculosisEffector functionsMalaria FalciparumProtozoal diseaseImmunologic MemoryHoming (hematopoietic)Microbes and infection
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MAPK3 deficiency drives autoimmunity via DC arming.

2010

DC are professional APC that instruct T cells during the inflammatory course of EAE. We have previously shown that MAPK3 (Erk1) is important for the induction of T-cell anergy. Our goal was to determine the influence of MAPK3 on the capacity of DC to arm T-cell responses in autoimmunity. We report that DC from Mapk3(-/-) mice have a significantly higher membrane expression of CD86 and MHC-II and--when loaded with the myelin oligodendrocyte glycoprotein--show a superior capacity to prime naive T cells towards an inflammatory phenotype than Mapk3(+/+) DC. Nonetheless and as previously described, Mapk3(-/-) mice were only slightly but not significantly more susceptible to myelin oligodendrocyt…

Encephalomyelitis Autoimmune ExperimentalMAP Kinase Signaling SystemOvalbuminImmunologyMedizinAutoimmunityMice TransgenicT-Cell Antigen Receptor SpecificityBiologymedicine.disease_causeAutoimmunityMyelinMiceImmune systemT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsNeuroinflammationGlycoproteinsCD86Mitogen-Activated Protein Kinase 3KinaseHistocompatibility Antigens Class IIDendritic Cellsmedicine.diseaseOligodendrocytePeptide FragmentsSpecific Pathogen-Free OrganismsMice Inbred C57BLmedicine.anatomical_structureRadiation ChimeraImmunologyCytokinesMyelin-Oligodendrocyte GlycoproteinB7-2 AntigenInfiltration (medical)European journal of immunology
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T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma.

2007

T cell functional differentiation is mediated by lineage-specific transcription factors. T helper 17 (Th17) has been recently identified as a distinct Th lineage mediating tissue inflammation. Retinoic acid receptor-related orphan receptor gamma (ROR gamma) was shown to regulate Th17 differentiation; ROR gamma deficiency, however, did not completely abolish Th17 cytokine expression. Here, we report Th17 cells highly expressed another related nuclear receptor, ROR alpha, induced by transforming growth factor-beta and interleukin-6 (IL-6), which is dependent on signal transducer and activator of transcription 3. Overexpression of ROR alpha promoted Th17 differentiation, possibly through the c…

Encephalomyelitis Autoimmune ExperimentalReceptors Retinoic AcidT cellImmunologyRetinoic acidReceptors Cytoplasmic and NuclearElectrophoretic Mobility Shift AssayBiology03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineT-Lymphocyte SubsetsmedicineT helper 17 cellImmunology and AllergyAnimalsCell LineageReceptorMOLIMMUNOTranscription factor030304 developmental biologyOrphan receptor0303 health sciencesReceptors Thyroid HormoneReverse Transcriptase Polymerase Chain ReactionInterleukin-17Cell DifferentiationNuclear Receptor Subfamily 1 Group F Member 1T-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 3Molecular biologyMice Mutant StrainsCell biologymedicine.anatomical_structureInfectious DiseaseschemistryNuclear receptorSTAT proteinTrans-ActivatorsFemale030215 immunologyImmunity
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Microglial activation milieu controls regulatory T cell responses.

2013

Abstract Although mechanisms leading to brain-specific inflammation and T cell activation have been widely investigated, regulatory mechanisms of local innate immune cells in the brain are only poorly understood. In this study, to our knowledge we show for the first time that MHC class II+CD40dimCD86dimIL-10+ microglia are potent inducers of Ag-specific CD4+Foxp3+ regulatory T cells (Tregs) in vitro. Microglia differentially regulated MHC class II expression, costimulatory molecules, and IL-10 depending on the amount of IFN-γ challenge and Ag dose, promoting either effector T cell or Treg induction. Microglia-induced Tregs were functionally active in vitro by inhibiting Ag-specific prolifer…

Encephalomyelitis Autoimmune ExperimentalRegulatory T cellT cellImmunologychemical and pharmacologic phenomenaMice TransgenicLymphocyte ActivationT-Lymphocytes RegulatoryImmune toleranceInterferon-gammaMiceImmune systemT-Lymphocyte SubsetsmedicineImmune ToleranceImmunology and AllergyAnimalsCells CulturedCD86MHC class IIbiologyMicrogliaHistocompatibility Antigens Class IIFOXP3Brainhemic and immune systemsForkhead Transcription FactorsCoculture TechniquesCell biologyInterleukin-10Mice Inbred C57BLmedicine.anatomical_structureCellular Microenvironmentbiology.proteinMicrogliaJournal of immunology (Baltimore, Md. : 1950)
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